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EMBO Molecular Medicine, ISSN 1757-4676, 03/2018, Volume 10, Issue 3, p. n/a
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 27, pp. 8173 - 8180
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 8, p. e40690
.... Furthermore, treating metastatic melanoma cells with the drug, Elesclomol, which induces cancer cell apoptosis through oxidative stress, we document by way of stable isotope labeling with amino acids in cell culture... 
SURVIVAL | OXYGEN | METABOLISM | LIPOGENESIS | MULTIDISCIPLINARY SCIENCES | GROWTH | CELL LINES | FLUX | EXPRESSION | Melanoma - metabolism | Hydrazines - pharmacology | Hydrazines - therapeutic use | Reactive Oxygen Species - metabolism | Humans | Antineoplastic Agents - therapeutic use | Mitochondria - metabolism | Melanoma - pathology | Mitochondria - drug effects | Glycolysis - drug effects | Oxidative Phosphorylation - drug effects | Mitochondrial Proteins - metabolism | Melanoma - drug therapy | Cell Respiration - drug effects | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Oxidative Stress - drug effects | Molecular Targeted Therapy - methods | Proteins | Care and treatment | Melanoma | Development and progression | Research | Metastasis | Comparative analysis | Health aspects | Cancer | Cell culture | Oxidative stress | Reactive oxygen species | Phosphorylation | Dehydrogenases | Amino acids | Melanocytes | Biology | Cancer therapies | Metastases | Skin cancer | Mitochondria | Cell growth | Bioenergetics | Stable isotopes | Oxygen probes | Drug dosages | Oxygen | Pharmacology | Metabolism | Medicine | Oxidative phosphorylation | Womens health | Medical prognosis | Glycolysis | Hypoxia | Electron transport | Apoptosis
Journal Article
Pharmaceutical Research, ISSN 0724-8741, 6/2015, Volume 32, Issue 6, pp. 1975 - 1992
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 2012, Volume 52, Issue 1, pp. 198 - 207
Statins, the widely prescribed cholesterol-lowering drugs for the treatment of cardiovascular disease, cause adverse skeletal muscle side effects ranging from fatigue to fatal rhabdomyolysis... 
Oxidative stress | Mitochondria | Hydrogen peroxide | Simvastatin | Mitochondrial respiration | Superoxide | Human skeletal muscle | Apoptosis | COA REDUCTASE INHIBITORS | COENZYME-A REDUCTASE | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | INDUCE APOPTOSIS | MUSCLE-CELLS | STATIN-ASSOCIATED MYOPATHY | PERMEABILITY TRANSITION | LIPID-LOWERING DRUGS | MYOTOXICITY | ENDOCRINOLOGY & METABOLISM | Simvastatin - adverse effects | Gene Expression - drug effects | Oxidative Stress | Apoptosis - drug effects | Humans | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Electron Transport Complex I - metabolism | Oxygen Consumption - physiology | Dose-Response Relationship, Drug | Myoblasts - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Myoblasts - metabolism | Electron Transport Complex I - genetics | Cell Respiration - drug effects | Myoblasts - cytology | Superoxides - metabolism | Muscular Diseases - chemically induced | Respiratory System Agents - pharmacology | Muscular Diseases - metabolism | Mitochondria - metabolism | Muscular Diseases - pathology | Mitochondria - drug effects | Anticholesteremic Agents - adverse effects | Adenosine Diphosphate - pharmacology | Hydrogen Peroxide - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Oxygen Consumption - drug effects | Muscle Fibers, Skeletal - cytology | Primary Cell Culture | Proto-Oncogene Proteins c-bcl-2 - genetics | Enzymes | Low density lipoproteins | Dimethyl sulfoxide | Dosage and administration | Mitochondrial DNA | Permeability | Glutamate | Cells | Muscles | Drugs | Cell culture | Myotubes | Bcl-2 protein | Fatigue | Oxygen consumption | Nuclei | Skeletal muscle | Myoblasts | Atrophy | Electron transport chain | statins | Cardiovascular diseases | Electron transport | mitochondria | hydrogen peroxide | simvastatin | apoptosis | superoxide | oxidative stress | mitochondrial respiration | human skeletal muscle
Journal Article
Toxicology, ISSN 0300-483X, 2015, Volume 331, pp. 47 - 56
Highlights • HepG2 cells were grown in glucose medium or mitochondrial respiration-inducing galactose medium... 
Emergency | HepG2 | Mitochondrial respiration | Reactive oxygen species | Sodium valproate | Mitochondrial toxicity | Hepatotoxicity | OXIDATIVE-PHOSPHORYLATION | DNA-POLYMERASE-GAMMA | METABOLITES | TOXICITY | ACID-INDUCED HEPATOTOXICITY | CULTURED RAT HEPATOCYTES | SUPEROXIDE-DISMUTASE 2 | PHARMACOLOGY & PHARMACY | EXPERIENCE | TOXICOLOGY | MUTATIONS | FATAL LIVER-FAILURE | Mitochondrial Diseases - pathology | Galactose - metabolism | Reactive Oxygen Species - metabolism | Mitochondria, Liver - metabolism | Humans | Mitochondrial Diseases - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | Membrane Potential, Mitochondrial - drug effects | Valproic Acid - toxicity | Dose-Response Relationship, Drug | Oxidative Phosphorylation - drug effects | Time Factors | Adenosine Triphosphate - metabolism | Mitochondrial Diseases - chemically induced | Cell Respiration - drug effects | Pyruvic Acid - metabolism | Cell Death - drug effects | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Hepatocytes - drug effects | Superoxide Dismutase - metabolism | Mitochondria, Liver - pathology | Anticonvulsants - toxicity | Hep G2 Cells | Mitochondria, Liver - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Electron Transport Chain Complex Proteins - metabolism | Glucose - metabolism | Cell Proliferation - drug effects | Oxidative Stress - drug effects | Superoxide | Glucose | Galactose | Dextrose | Cells | Index Medicus | Proteins | Pyruvates | Sodium | Cell death | In vitro testing | Respiration | Patients
Journal Article
Journal Article