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PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0132977
.... Recent evidences have highlighted the correlation of BORIS/CTCFL expression with poor overall survival of different cancer patients... 
TESTIS ANTIGEN BORIS | HEPATOCELLULAR-CARCINOMA | SIDE POPULATION | IN-VITRO | INDUCED APOPTOSIS | TELOMERASE | MULTIDISCIPLINARY SCIENCES | IMPRINTED SITES | CTCF | IDENTIFICATION | TUMOR-INITIATING CELLS | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | RNA, Small Interfering - genetics | Cell Proliferation | Epithelial Cells - metabolism | Polycomb Repressive Complex 1 - metabolism | Homeodomain Proteins - metabolism | Humans | Retinal Dehydrogenase - metabolism | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | SOXB1 Transcription Factors - metabolism | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-3 - genetics | Polycomb Repressive Complex 1 - genetics | Telomerase - genetics | ATP-Binding Cassette Transporters - genetics | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Isoenzymes - metabolism | Hyaluronan Receptors - metabolism | ATP-Binding Cassette Transporters - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Telomerase - metabolism | Neoplasm Proteins - genetics | DNA-Binding Proteins - antagonists & inhibitors | Nanog Homeobox Protein | Signal Transduction | Isoenzymes - genetics | Spheroids, Cellular - metabolism | Epithelial Cells - pathology | Retinal Dehydrogenase - genetics | DNA-Binding Proteins - genetics | Organ Specificity | Hyaluronan Receptors - genetics | Homeodomain Proteins - genetics | Phenotype | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Biomarkers, Tumor - genetics | Cell Movement | RNA, Small Interfering - metabolism | RNA | Genes | Colorectal cancer | Stem cells | Genetic aspects | Gene expression | Cancer | Protein binding | Neurosciences | Germ cells | Telomerase reverse transcriptase | Populations | Dyes | Mesenchyme | Oct-4 protein | Gene regulation | Oncology | Spheres | Drug resistance | Tissues | Metastases | DNA-binding protein | CD44 antigen | Cell cycle | Life sciences | Colon | Telomerase | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Antigens | Cell survival | Tumor cells | Invasiveness | Cervix | Survival | Brain research | Molecular modelling | Medical prognosis | Epigenetics | Breast | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Journal of clinical oncology, ISSN 1527-7755, 2013, Volume 31, Issue 34, pp. 4333 - 4342
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2013, Volume 123, Issue 6, pp. 2488 - 2501
...). Strikingly, patients with HPV-positive OPSCC are highly curable with ionizing radiation and have better survival compared with HPV-negative patients, but the underlying molecular mechanisms remain poorly understood... 
CHROMOSOMAL ALTERATIONS | MEDICINE, RESEARCH & EXPERIMENTAL | RETINOIC ACID | DNA METHYLATION | ONCOGENIC HUMAN-PAPILLOMAVIRUS | PROMOTER METHYLATION | EPIGENETIC INACTIVATION | TONSILLAR CANCER | POSITIVE HEAD | TUMOR-SUPPRESSOR | HIGH VIRAL LOAD | Human papillomavirus 16 - physiology | Oligonucleotide Array Sequence Analysis | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - virology | Humans | Gene Expression Regulation, Neoplastic | Oropharyngeal Neoplasms - mortality | Transcriptome | Case-Control Studies | DNA Methylation | Carcinoma, Squamous Cell - mortality | Papillomavirus Infections - virology | Receptors, AMPA - genetics | Promoter Regions, Genetic | Oropharyngeal Neoplasms - genetics | Kaplan-Meier Estimate | Proportional Hazards Models | GATA4 Transcription Factor - genetics | Oropharyngeal Neoplasms - virology | Papillomavirus Infections - mortality | Retinal Dehydrogenase - genetics | Transcription Factors - genetics | Sequence Analysis, DNA | Homeodomain Proteins - genetics | Papillomavirus Infections - genetics | Host-Pathogen Interactions | Disease-Free Survival | CpG Islands | HeLa Cells | Human papillomavirus 16 - genetics | Complications and side effects | Squamous cell carcinoma | Care and treatment | Causes of | Physiological aspects | Genetic aspects | Diagnosis | Research | Methylation | Papillomavirus infections | Cluster analysis | Disease | Decision making | Genes | Values | Principal components analysis | Patients | Ethics | Human papillomavirus | DNA methylation | Software | Behavior | Genetic testing | Cervical cancer
Journal Article
International journal of cancer, ISSN 0020-7136, 2018, Volume 143, Issue 11, pp. 2871 - 2883
... in cell survival, including those for cell proliferation and migration. Of the total breast cancer patients, 70% were reported to be positive for ER. Therefore... 
breast cancer | tamoxifen resistance | estrogen receptor | miR‐1915‐3p | SETD1A | miR-1915-3p | ACTIVATION | PATTERNS | MECHANISMS | HSETD1A | THERAPY | ONCOLOGY | METHYLTRANSFERASE COMPLEX | CXXC FINGER PROTEIN-1 | HISTONE MODIFICATIONS | PROGRESSION | Cell Cycle - genetics | Apoptosis - drug effects | Humans | Cell Survival - genetics | Apoptosis - genetics | Cell Movement - genetics | MCF-7 Cells | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - genetics | Cell Survival - drug effects | Cell Proliferation - genetics | Histone-Lysine N-Methyltransferase - genetics | Up-Regulation - genetics | Down-Regulation - drug effects | Breast Neoplasms - drug therapy | Down-Regulation - genetics | Up-Regulation - drug effects | Cell Movement - drug effects | Drug Resistance, Neoplasm - genetics | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Tamoxifen - pharmacology | Cell Line, Tumor | Cell Proliferation - drug effects | MicroRNAs - genetics | Cell Cycle - drug effects | Drug Resistance, Neoplasm - drug effects | Cell proliferation | Chromatin | Cell survival | Leukocyte migration | Genes | Estrogens | Methyltransferase | Estrogen receptors | Breast cancer | Tamoxifen | Survival | Metastases | Lysine | Transcription activation | Cell cycle | Methylation | Cell migration | Histone H3 | Cancer | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2012, Volume 109, Issue 11, pp. 4251 - 4256
...) model of human cancer whereby amplification of certain lineage transcription factors predisposes a survival mechanism in tumor cells... 
Cell growth | Hepatocytes | Genes | Genomics | Cell lines | Cells | Oncogenes | Tumors | Apoptosis | Cancer | Lineage-survival oncogene | Transcriptional reprogramming | P38α and tnf-related apoptosis-inducing ligand activation | Extrinsic apoptosis pathway | LUNG | transcriptional reprogramming | MULTIDISCIPLINARY SCIENCES | ENDODERM | BARRETTS-ESOPHAGUS | lineage-survival oncogene | CANCER | ADDICTION | AMPLIFICATION | p38 alpha and TNF-related apoptosis-inducing ligand activation | extrinsic apoptosis pathway | EMBRYONIC STEM-CELLS | TRANSCRIPTION FACTOR GENES | DIFFERENTIATION | EXPRESSION | Adenocarcinoma - pathology | Cell Proliferation | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Cell Survival - genetics | Apoptosis - genetics | Esophageal Neoplasms - pathology | Barrett Esophagus - pathology | Cell Transformation, Neoplastic - genetics | DNA Fragmentation | Adenocarcinoma - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Genes, Neoplasm - genetics | Barrett Esophagus - genetics | Cell Lineage - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Cell Adhesion | GATA6 Transcription Factor - metabolism | Genome, Human - genetics | Comparative Genomic Hybridization | Esophageal Neoplasms - genetics | Cell Line, Tumor | Gene Amplification - genetics | GATA6 Transcription Factor - genetics | Chromosomes, Human, Pair 18 - genetics | TNF-Related Apoptosis-Inducing Ligand - genetics | RNA, Small Interfering - metabolism | Adenocarcinoma | Genetic transformation | Physiological aspects | Development and progression | Genetic aspects | Research | Genetic transcription | Binding proteins | Biological Sciences | p38α and TNF-related apoptosis-inducing ligand activation
Journal Article
Nature genetics, ISSN 1546-1718, 2009, Volume 41, Issue 11, pp. 1238 - 1242
Lineage-survival oncogenes are activated by somatic DNA alterations in cancers arising from the cell lineages in which these genes play a role in normal development(1,2... 
CONSISTENT | AMPLIFICATION | DEPENDENCY | GENE | ADENOCARCINOMAS | GENETICS & HEREDITY | DIFFERENTIATION | IDENTIFICATION | EXPRESSION | CANCER | REVEALS | Gene mutations | Lung cancer | Genetic aspects | Research | Health aspects | Risk factors | Esophageal cancer | Proteins | Medical research | Genetic testing | Molecular biology | Fluorescence in situ hybridization
Journal Article