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Experimental Cell Research, ISSN 0014-4827, 07/2016, Volume 345, Issue 2, pp. 206 - 217
Journal Article
Cancer Discovery, ISSN 2159-8274, 07/2017, Volume 7, Issue 7, pp. 736 - 749
Journal Article
Nature Communications, ISSN 2041-1723, 08/2014, Volume 5, Issue 1, pp. 4629 - 4629
Whether the Hippo pathway contributes to cell lineage transition under pathological conditions, especially tumorigenesis, remains largely unknown. Here we show... 
YES-ASSOCIATED PROTEIN | ORGAN SIZE | HEPATOCELLULAR-CARCINOMA | STEM-LIKE CELLS | MULTIDISCIPLINARY SCIENCES | HIPPO PATHWAY | SIZE-CONTROL | DIFFERENTIATION | CANCER | ADENOSQUAMOUS CARCINOMA | TUMOR-SUPPRESSOR PATHWAY | Protein-Serine-Threonine Kinases - deficiency | Adenocarcinoma - pathology | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Cell Transdifferentiation - genetics | Lung Neoplasms - physiopathology | Zinc Finger E-box Binding Homeobox 2 | Trans-Activators - physiology | Repressor Proteins - physiology | Cell Transdifferentiation - physiology | Adenocarcinoma - physiopathology | Tumor Suppressor Proteins - genetics | Up-Regulation - physiology | Trans-Activators - genetics | Female | Adenocarcinoma - genetics | Phosphoproteins - physiology | Disease Models, Animal | Lung Neoplasms - genetics | Transcription Factors - physiology | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Mice, Transgenic | Phosphoproteins - genetics | Transcription Factors - genetics | Carcinoma, Squamous Cell - physiopathology | Homeodomain Proteins - genetics | Down-Regulation - physiology | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing - physiology | Animals | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Mice | Homeodomain Proteins - physiology | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2011, Volume 108, Issue 41, pp. 17016 - 17021
Earlier work has shown that pre-B cells can be converted into macrophages by the transcription factor CCAAT/enhancer binding protein α at very high... 
T lymphocytes | B lymphocytes | Epithelial cells | Genes | Stromal cells | Stem cells | Cell lines | Macrophages | Progenitor cells | Hematopoietic stem cells | Cell reprogramming | Hematopoietic differentiation lineage commitment | Cell fate decision | PROGENITOR CELLS | STEM-CELLS | DEFINED FACTORS | MULTIDISCIPLINARY SCIENCES | NEUTROPHILS | PLURIPOTENCY | FIBROBLASTS | cell reprogramming | C/EBP-ALPHA | cell fate decision | IN-VIVO | DIFFERENTIATION | hematopoietic differentiation lineage commitment | HEMATOPOIETIC STEM | Precursor Cells, B-Lymphoid - metabolism | RNA, Messenger - genetics | Cells, Cultured | Transcriptome | Cell Transdifferentiation - genetics | Macrophages - cytology | RNA, Messenger - metabolism | fms-Like Tyrosine Kinase 3 - genetics | Macrophages - metabolism | Animals | Cell Transdifferentiation - physiology | T-Lymphocytes - cytology | T-Lymphocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - physiology | Precursor Cells, B-Lymphoid - cytology | Erythrocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Erythrocytes - cytology | Proto-Oncogene Proteins c-kit - genetics | Mice | Megakaryocytes - cytology | Megakaryocytes - metabolism | Cell Lineage - genetics | Genes, cdc | Index Medicus | Biological Sciences | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | hematopoietic differentiation | Medicinska och farmaceutiska grundvetenskaper | Cell and Molecular Biology | lineage commitment | Cell- och molekylärbiologi
Journal Article
Plant Physiology, ISSN 0032-0889, 7/2010, Volume 153, Issue 3, pp. 906 - 914
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, pp. e33766 - e33766
MicroRNAs (miRNAs) are non-coding RNAs that regulate gene expression in post-transcriptional fashion, and emerging studies support their importance in... 
TRANSCRIPTION FACTORS | FIBROSIS | TGF-BETA | SMAD3 | RNA-SEQ | MYOGENESIS | BIOLOGY | DUCHENNE MUSCULAR-DYSTROPHY | DIFFERENTIATION | MICRORNAS | INJURED SKELETAL-MUSCLE | RNA, Small Interfering - genetics | MicroRNAs - antagonists & inhibitors | Transcriptome | MicroRNAs - metabolism | Cell Transdifferentiation - genetics | Smad3 Protein - metabolism | YY1 Transcription Factor - metabolism | Myofibroblasts - metabolism | Myoblasts, Skeletal - cytology | Smad3 Protein - genetics | Cell Transdifferentiation - physiology | Base Sequence | Repressor Proteins - metabolism | Myoblasts, Skeletal - metabolism | Cell Line | Promoter Regions, Genetic | Down-Regulation | MyoD Protein - metabolism | Polycomb-Group Proteins | Smad3 Protein - antagonists & inhibitors | Myofibroblasts - cytology | Animals | Models, Biological | Signal Transduction - physiology | Mice | MicroRNAs - genetics | Transforming Growth Factor beta - metabolism | Bone morphogenetic proteins | Genetic aspects | MicroRNA | Genetic transcription | Transforming growth factors | Genes | Post-transcription | Health sciences | Pathogenesis | Transforming growth factor-b | Clinical trials | Smad3 protein | Kinases | Muscular dystrophy | Gene sequencing | Proteins | Cell growth | Rodents | Cell adhesion | Cell cycle | Extracellular matrix | Trends | Inhibition | Hypertension | MyoD protein | Gynecology | MiRNA | Muscles | Gene expression | Ribonucleic acid--RNA | Obstetrics | Biological activity | Myogenesis | Myoblasts | Polycomb group proteins | Pathology | Musculoskeletal system | Signaling | Hypotheses | Differentiation | Index Medicus | RNA | Ribonucleic acid
Journal Article
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 731 - 741.e4
Background & Aims Animal studies have indicated that pancreatic exocrine acinar cells have phenotypic plasticity. In rodents, acinar cells can differentiate... 
Gastroenterology and Hepatology | Metaplasia | Reprogramming | Exocrine Pancreas | Tissue Engineering | BETA-CELLS | INTRAEPITHELIAL NEOPLASIA | HUMAN EXOCRINE PANCREAS | ADULT HUMAN PANCREAS | IN-VITRO | NOTCH SIGNALING PATHWAY | GENERATION | MICE | GROWTH-FACTOR | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | Cell Proliferation | Chymotrypsin - metabolism | Humans | Glycoproteins - metabolism | Carbonic Anhydrase II - metabolism | Ki-67 Antigen - metabolism | Cell Transdifferentiation - genetics | RNA, Messenger - metabolism | Pancreatic Ducts - metabolism | Antigens, CD - metabolism | Cell Transdifferentiation - physiology | Peptides - metabolism | Pancreas, Exocrine - cytology | Cell Lineage - genetics | Genes, Reporter | Hepatocyte Nuclear Factor 1-beta - metabolism | Plant Lectins - pharmacokinetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Green Fluorescent Proteins - metabolism | Promoter Regions, Genetic | Pancreatic Ducts - cytology | Transduction, Genetic | Cell Survival | Cells, Cultured | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Signal Transduction - genetics | AC133 Antigen | Cell Lineage - physiology | Phenotype | Signal Transduction - physiology | Pancreas, Exocrine - metabolism | Keratin-19 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Keratin | Epidermal growth factor | Analysis | Fluorescein | Fluorescence | Cystic fibrosis | Diabetes | Universities and colleges | Research | Protein kinases | Index Medicus | Abridged Index Medicus
Journal Article