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2009, 1. Aufl., ISBN 0387896104, xiv, 296
.... In particular, we now know that many cancers contain a fraction of self-renewing, drug- and radiation-resistant cells... 
Stem Cell Transplantation | adverse effects | Teratoma | therapy | Neoplasms | etiology | Treatment | Etiology | Stem cells | Cancer cells | Cancer
Book
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 12/2012, Volume 110, Issue 3, pp. 972 - 977
Metformin, the first-line drug for treating diabetes, inhibits cellular transformation and selectively kills cancer stem cells in breast cancer cell lines... 
T lymphocytes | Cell growth | Phosphorylation | Stem cells | Cell lines | Breast cancer | Diabetes | Heterologous transplantation | Tumors | Cancer | Cancer treatment | Cancer prevention | Inflammation | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - pathology | Neoplastic Stem Cells - drug effects | Humans | NF-kappa B - metabolism | Breast Neoplasms - metabolism | Inflammation - metabolism | Hypoglycemic Agents - administration & dosage | Neoplastic Stem Cells - metabolism | Anticarcinogenic Agents - administration & dosage | Neoplastic Stem Cells - pathology | Female | Stress, Physiological - drug effects | Metformin - administration & dosage | Anticarcinogenic Agents - pharmacology | STAT3 Transcription Factor - metabolism | Doxorubicin - administration & dosage | Cell Line | Metformin - pharmacology | Breast Neoplasms - drug therapy | Cell Transformation, Neoplastic - metabolism | Hypoglycemic Agents - pharmacology | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Feedback, Physiological - drug effects | Inflammation - prevention & control | Mice | Cell Transformation, Neoplastic - drug effects | Cell Transformation, Neoplastic - pathology | Prevention | Cancer cells | Physiological aspects | Dosage and administration | Hypoglycemic agents | Metformin | Research | Cell transformation | Health aspects | Index Medicus | Biological Sciences | cancer treatment | cancer prevention | inflammation
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 5, pp. 1119 - 1133.e4
.... Methods We studied the effects of nicotine administration on pancreatic cancer development in Kras +/LSLG12Vgeo ;Elas-tTA/tetO-Cre (Ela-KRAS) mice, Kras +/LSLG12D ;Trp53+/LSLR172H;Pdx-1-Cre (KPC) mice... 
Gastroenterology and Hepatology | Metastasis | Pancreas | Progenitor Cells | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Carcinoma, Pancreatic Ductal - metabolism | Epithelial-Mesenchymal Transition - drug effects | Mice, 129 Strain | Carcinoma, Pancreatic Ductal - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Pancreatic Neoplasms - chemically induced | Liver Neoplasms - secondary | Nicotinic Agonists - toxicity | Proto-Oncogene Proteins c-akt - metabolism | Neoplastic Cells, Circulating - drug effects | GATA6 Transcription Factor - deficiency | Liver Neoplasms - genetics | Pancreatic Neoplasms - pathology | Pancreas - pathology | alpha7 Nicotinic Acetylcholine Receptor - genetics | Pancreas - metabolism | Mice, Knockout | Acinar Cells - pathology | Signal Transduction - drug effects | Mice, Nude | Liver Neoplasms - metabolism | Cell Line, Tumor | alpha7 Nicotinic Acetylcholine Receptor - metabolism | Mutation | Carcinoma, Pancreatic Ductal - secondary | Carcinoma, Pancreatic Ductal - chemically induced | Neoplastic Cells, Circulating - pathology | Neoplastic Cells, Circulating - metabolism | Cell Transformation, Neoplastic - chemically induced | Extracellular Signal-Regulated MAP Kinases - metabolism | Carcinoma, Pancreatic Ductal - prevention & control | Tumor Suppressor Protein p53 - genetics | Transfection | Proto-Oncogene Proteins p21(ras) - deficiency | Gene Expression Regulation, Neoplastic - drug effects | Pancreatic Neoplasms - prevention & control | Tumor Cells, Cultured | Proto-Oncogene Proteins p21(ras) - metabolism | Acinar Cells - metabolism | Cell Dedifferentiation - drug effects | Metformin - pharmacology | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Pancreas - drug effects | Pancreatic Neoplasms - genetics | Nicotine - toxicity | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | GATA6 Transcription Factor - metabolism | Animals | Acinar Cells - drug effects | GATA6 Transcription Factor - genetics | Cell Transformation, Neoplastic - pathology | Pancreatic cancer | Stem cells | Nicotine | Index Medicus | Abridged Index Medicus
Journal Article
Toxicology and applied pharmacology, ISSN 0041-008X, 01/2015, Volume 282, Issue 1, pp. 9 - 19
The incidence of lung diseases, including cancer, caused by cigarette smoke is increasing, but the molecular mechanisms of gene regulation induced by cigarette... 
lncRNAs | Epithelial-mesenchymal transition (EMT) | Inflammation | Cancer stem cells (CSCs) | Carcinogenesis | Cigarette smoke extract (CSE) | LncRNAs | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Smoking - adverse effects | Up-Regulation | Epithelial Cells - metabolism | Neoplastic Stem Cells - drug effects | Epithelial Cells - drug effects | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - drug effects | Lung Neoplasms - pathology | Autocrine Communication - drug effects | Dose-Response Relationship, Drug | Transfection | Lung Neoplasms - etiology | Neoplastic Stem Cells - metabolism | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Female | Lung - metabolism | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Lung Neoplasms - genetics | Lung - pathology | Smoke - adverse effects | Epithelial Cells - pathology | RNA, Long Noncoding - genetics | Cell Transformation, Neoplastic - metabolism | Animals | Signal Transduction - drug effects | Mice, Nude | Lung - drug effects | Mice | Cell Transformation, Neoplastic - pathology | RNA, Long Noncoding - metabolism | Cell Line, Transformed | Antisense RNA | Stem cells | Cancer | Smoking | Index Medicus | ANTIBODIES | TOBACCO SMOKES | CARCINOGENESIS | NEOPLASMS | RNA | CELL CULTURES | INFLAMMATION | LUNGS | STEM CELLS | GENE REGULATION | 60 APPLIED LIFE SCIENCES
Journal Article
Biochemical pharmacology, ISSN 0006-2952, 10/2016, Volume 117, pp. 35 - 45
[Display omitted] The neoplastic transformation of cells and inflammation are processes that contribute to tumor initiation... 
NF-κB | JQ-1 | Bromodomain | Neoplastic transformation | Brd4 | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Humans | E1A-Associated p300 Protein - genetics | Tetradecanoylphorbol Acetate - analogs & derivatives | E1A-Associated p300 Protein - metabolism | Promoter Regions, Genetic - drug effects | Tetradecanoylphorbol Acetate - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Carcinogens - toxicity | Carcinogens - antagonists & inhibitors | Protein Processing, Post-Translational - drug effects | RNA Interference | Cyclooxygenase 2 - genetics | E1A-Associated p300 Protein - antagonists & inhibitors | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Anticarcinogenic Agents - pharmacology | Cell Line | Epidermis - metabolism | Epidermis - pathology | Cyclooxygenase 2 - chemistry | Epidermis - drug effects | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Cell Transformation, Neoplastic - metabolism | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Acetylation - drug effects | Animals | Tetradecanoylphorbol Acetate - toxicity | Epidermis - immunology | Nuclear Proteins - antagonists & inhibitors | Cyclooxygenase 2 - metabolism | Genes, Reporter - drug effects | Mice | Cell Transformation, Neoplastic - drug effects | Histones - metabolism | Cell Transformation, Neoplastic - pathology | Antimitotic agents | Antineoplastic agents | Epigenetic inheritance | Index Medicus
Journal Article
Nature (London), ISSN 1476-4687, 02/2012, Volume 483, Issue 7390, pp. 484 - 488
Journal Article
Oncogene, ISSN 1476-5594, 12/2014, Volume 34, Issue 37, pp. 4821 - 4833
Journal Article
Biomaterials, ISSN 0142-9612, 02/2012, Volume 33, Issue 5, pp. 1462 - 1476
Abstract Glioblastomas (GBMs) are the most common primary brain tumors with poor prognosis. CD133 has been considered a putative marker of cancer stem cells... 
Advanced Basic Science | Dentistry | miR145 | Drug-resistance | Polyurethane-short branch polyethylenimine | Glioblastoma | MiR145 | Engineering | Materials Science | Technology | Engineering, Biomedical | Materials Science, Biomaterials | Science & Technology | Dacarbazine - therapeutic use | Neoplastic Stem Cells - drug effects | Humans | Middle Aged | 3' Untranslated Regions - genetics | Molecular Sequence Data | Polyurethanes - chemical synthesis | Male | MicroRNAs - metabolism | Gene Expression Profiling | Glioblastoma - radiotherapy | SOXB1 Transcription Factors - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Dacarbazine - pharmacology | Base Sequence | Polyethyleneimine - chemistry | Dacarbazine - analogs & derivatives | Neoplastic Stem Cells - pathology | Female | Gene Expression Regulation, Neoplastic - drug effects | Polyethyleneimine - chemical synthesis | Gene Transfer Techniques | Polyurethanes - chemistry | Down-Regulation - drug effects | Down-Regulation - genetics | Radiation Tolerance - drug effects | Polyethyleneimine - analogs & derivatives | Glioblastoma - pathology | Octamer Transcription Factor-3 - metabolism | Aged | MicroRNAs - genetics | Cell Transformation, Neoplastic - drug effects | Cell Transformation, Neoplastic - pathology | Glioblastoma - drug therapy | Drug Resistance, Neoplasm - drug effects | Medical colleges | Care and treatment | Biological products | Brain tumors | Genes | Drug resistance | Prevention | MicroRNA | Gliomas | Nuclear radiation | Analysis | Stem cells | Cancer | Index Medicus
Journal Article