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Nature, ISSN 0028-0836, 01/2010, Volume 463, Issue 7279, pp. 318 - 325
The inference of transcriptional networks that regulate transitions into physiological or pathological cellular states remains a central challenge in systems... 
SURVIVAL | GROWTH-FACTOR RECEPTOR | NEURAL STEM-CELLS | GLIOBLASTOMA | MULTIDISCIPLINARY SCIENCES | DISEASE | C-MYC | MECHANISMS | DIFFERENTIATION | EXPRESSION | BINDING | Neurons - pathology | Prognosis | Glioma - diagnosis | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Gene Regulatory Networks | Glioma - genetics | Cell Differentiation - genetics | Cell Transformation, Neoplastic - genetics | Neoplasm Invasiveness - pathology | Glioma - pathology | Transcription, Genetic | Neurons - metabolism | Cellular Reprogramming - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Reproducibility of Results | Brain Neoplasms - diagnosis | CCAAT-Enhancer-Binding Protein-beta - genetics | Computational Biology | Brain Neoplasms - genetics | Mesenchymal Stromal Cells - metabolism | Mice, SCID | Cell Transformation, Neoplastic - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Animals | Cell Line, Tumor | Mice, Inbred NOD | Mesoderm - metabolism | Mice | Mesenchymal Stromal Cells - pathology | Cell Transformation, Neoplastic - pathology | Neoplasm Invasiveness - genetics | Mesoderm - pathology | Transcription factors | Gliomas | Physiological aspects | Genetic aspects | Research | Genetic transcription | Risk factors | Proteins | Genotype & phenotype | Brain | Reverse engineering | Brain cancer | Regression analysis | Gene expression | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 10/2015, Volume 526, Issue 7571, pp. 131 - 135
Despite major advances in understanding the molecular and genetic basis of cancer, metastasis remains the cause of >90% of cancer-related mortality(1).... 
INHIBITION | COMMITMENT | MULTIDISCIPLINARY SCIENCES | GROWTH | GENES | MARKERS | IDENTIFICATION | FATE | TUMORS | TRANSCRIPTOME ANALYSIS | MAMMARY-GLAND | Neoplastic Stem Cells - drug effects | Epithelial Cells - drug effects | Humans | Gene Expression Profiling | Epithelial-Mesenchymal Transition - genetics | Cell Differentiation - genetics | Flow Cytometry | Neoplasm Metastasis - drug therapy | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Cyclin-Dependent Kinases - antagonists & inhibitors | Single-Cell Analysis | Disease Models, Animal | Cell Separation | Epithelial Cells - pathology | Mice, SCID | Breast Neoplasms - drug therapy | Disease Progression | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Differentiation - drug effects | Neoplasm Metastasis - pathology | Genes, myc - genetics | Cell Line, Tumor | Mice, Inbred NOD | Cell Proliferation - drug effects | Mesoderm - metabolism | Mice | Cell Transformation, Neoplastic - drug effects | Cell Cycle - drug effects | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Stem cells | Cancer cells | Development and progression | Breast cancer | Metastasis | Observations | Health aspects | Bone marrow | Principal components analysis | Gene expression | Tumors | Apoptosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 3, pp. 972 - 977
Metformin, the first-line drug for treating diabetes, inhibits cellular transformation and selectively kills cancer stem cells in breast cancer cell lines. In... 
T lymphocytes | Cell growth | Phosphorylation | Stem cells | Cell lines | Breast cancer | Diabetes | Heterologous transplantation | Tumors | Cancer | Cancer treatment | Cancer prevention | Inflammation | cancer treatment | RESPIRATORY-CHAIN | METABOLIC SYNDROME | ACTIVATION | cancer prevention | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | CYCLE ARREST | DECREASES | CHEMOTHERAPY | BREAST-CANCER | DIABETIC-PATIENTS | inflammation | AMPK | Inflammation - pathology | Neoplastic Stem Cells - drug effects | Humans | NF-kappa B - metabolism | Breast Neoplasms - metabolism | Inflammation - metabolism | Hypoglycemic Agents - administration & dosage | Neoplastic Stem Cells - metabolism | Anticarcinogenic Agents - administration & dosage | Neoplastic Stem Cells - pathology | Female | Stress, Physiological - drug effects | Metformin - administration & dosage | Anticarcinogenic Agents - pharmacology | STAT3 Transcription Factor - metabolism | Doxorubicin - administration & dosage | Cell Line | Metformin - pharmacology | Breast Neoplasms - drug therapy | Cell Transformation, Neoplastic - metabolism | Hypoglycemic Agents - pharmacology | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Feedback, Physiological - drug effects | Inflammation - prevention & control | Mice | Cell Transformation, Neoplastic - drug effects | Cell Transformation, Neoplastic - pathology | Prevention | Cancer cells | Physiological aspects | Dosage and administration | Hypoglycemic agents | Metformin | Research | Cell transformation | Health aspects | Signal transduction | Metabolism | Index Medicus | Biological Sciences
Journal Article
STEM CELLS, ISSN 1066-5099, 04/2014, Volume 32, Issue 4, pp. 983 - 997
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2011, Volume 108, Issue 19, pp. 7950 - 7955
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 20, Issue 1, pp. 11 - 24
Journal Article