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Nature, ISSN 0028-0836, 12/2007, Volume 450, Issue 7172, pp. 1096 - 1099
Journal Article
Oncogene, ISSN 0950-9232, 07/2016, Volume 35, Issue 29, pp. 3807 - 3816
Infection of human B lymphocytes by Epstein-Barr virus (EBV) leads to the establishment of immortalized lymphoblastoid cell lines (LCLs) that are widely used... 
ACTIVATION | SIGNAL-BINDING-PROTEIN | NUCLEAR ANTIGEN | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | TYROSINE KINASES | INDUCTION | CELL BIOLOGY | GENOMIC INSTABILITY | ONCOLOGY | MEMBRANE-PROTEIN 1 | GENETICS & HEREDITY | NF-KAPPA-B | EXPRESSION | Phosphorylation | Viral Matrix Proteins - genetics | Herpesvirus 4, Human - genetics | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Immunoblotting | Viral Proteins - metabolism | B-Lymphocytes - virology | Cell Transformation, Viral - genetics | Viral Matrix Proteins - metabolism | B-Lymphocytes - pathology | Epstein-Barr Virus Nuclear Antigens - metabolism | B-Lymphocytes - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cell Proliferation - genetics | Epstein-Barr Virus Nuclear Antigens - genetics | Herpesvirus 4, Human - physiology | Cells, Cultured | Gene Expression Regulation | Cell Transformation, Viral - physiology | Viral Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Host-Pathogen Interactions | DNA Damage | Microscopy, Fluorescence | Oxidative stress | Usage | DNA damage | Genetic aspects | Epstein-Barr virus | Genetic transcription | Gene therapy | Health aspects | Methods | Cellular biology | Lymphocytes | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Blood, ISSN 0006-4971, 04/2015, Volume 125, Issue 16, pp. 2530 - 2543
Epigenetic events that are essential drivers of lymphocyte transformation remain incompletely characterized. We used models of Epstein-Barr virus (EBV)-induced... 
PRMT5 | LINE ESTABLISHMENT | METHYLATION | ACTIVATION | GENE | TRANSCRIPTION | LATENT MEMBRANE-PROTEIN-1 | HEMATOLOGY | NF-KAPPA-B | EXPRESSION | EPSTEIN-BARR-VIRUS | Small Molecule Libraries - pharmacology | Cell Transformation, Viral - drug effects | Humans | Protein-Arginine N-Methyltransferases - antagonists & inhibitors | MicroRNAs - metabolism | Host-Pathogen Interactions - drug effects | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - genetics | Transcription Factor RelA - genetics | Lymphoma - metabolism | RNA Interference | Tumor Suppressor Proteins - genetics | B-Lymphocytes - virology | Cell Transformation, Viral - genetics | Protein-Arginine N-Methyltransferases - genetics | B-Lymphocytes - metabolism | Tumor Suppressor Proteins - metabolism | Histone Deacetylases - genetics | Herpesvirus 4, Human - physiology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Lymphoma - genetics | Histone Deacetylases - metabolism | Transcriptome - drug effects | Transcriptome - genetics | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Protein-Arginine N-Methyltransferases - metabolism | B-Lymphocytes - drug effects | Microscopy, Confocal | Animals | Transcription Factor RelA - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - metabolism | MicroRNAs - genetics | Cell Line, Transformed | Lymphoma - virology | Lymphoid Neoplasia
Journal Article
Genome Research, ISSN 1088-9051, 2009, Volume 19, Issue 3, pp. 438 - 451
Journal Article
Oncogene, ISSN 0950-9232, 04/2008, Volume 27, Issue 18, pp. 2575 - 2582
Human papillomaviruses (HPVs) are involved in the pathogenesis of cancer of the cervix (CaCx). MicroRNA (miRNA) expression analysis using Ambion (Austin, TX,... 
microRNA | HPVs | Oncogene | Cervical cancer | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | oncogene | cervical cancer | CANCER | P53 | HPV | CELL BIOLOGY | GENOMIC INSTABILITY | ONCOLOGY | LAMININ-5 | GENETICS & HEREDITY | GENE-EXPRESSION | TUMOR-SUPPRESSOR | Cell Adhesion Molecules - genetics | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Regulation, Neoplastic | Human papillomavirus 6 - genetics | Papillomavirus E7 Proteins | Gene Expression Profiling | Repressor Proteins - antagonists & inhibitors | Oncogene Proteins, Viral - antagonists & inhibitors | Uterine Cervical Neoplasms - metabolism | Cell Transformation, Viral - genetics | Female | Oncogene Proteins, Viral - genetics | Cell Adhesion Molecules - biosynthesis | Repressor Proteins - genetics | MicroRNAs - biosynthesis | Uterine Cervical Neoplasms - genetics | Oncogene Proteins, Viral - biosynthesis | Human papillomavirus 16 - metabolism | Human papillomavirus 6 - metabolism | Papillomavirus Infections - genetics | RNA, Neoplasm - biosynthesis | Repressor Proteins - biosynthesis | Papillomavirus Infections - metabolism | RNA, Neoplasm - genetics | Uterine Cervical Neoplasms - virology | Biomarkers, Tumor - genetics | MicroRNAs - genetics | HeLa Cells | Human papillomavirus 16 - genetics | Biomarkers, Tumor - biosynthesis | Papillomaviruses | Complications and side effects | RNA | Physiological aspects | Genetic aspects | Research | Health aspects | Papillomavirus infections | Risk factors | Cell culture | Oncology | Genetics | Human papillomavirus | Ribonucleic acid--RNA
Journal Article
PLoS Pathogens, ISSN 1553-7366, 08/2013, Volume 9, Issue 8, p. e1003536
Journal Article