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Growth factors, ISSN 0897-7194, 1988
Journal
Hypertension, ISSN 0194-911X, 10/2009, Volume 54, Issue 4, pp. 877 - 884
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 08/2007, Volume 6, Issue 8, pp. 2158 - 2167
Insulin-like growth factor-I receptor (IGF-IR) and its ligands, IGF-I and IGF-II, are up-regulated in a variety of human cancers. In tumors, such as... 
Protein tyrosine kinases | Small Molecules and Other Therapeutics Agents | Autocrine-paracrine signaling | COLORECTAL-CANCER RISK | BREAST-CANCER | SARCOMA-CELLS | ONCOLOGY | PLASMA-LEVELS | IGF-BINDING PROTEIN-3 | FACTOR SYSTEM | ANTIBODY | ANTITUMOR-ACTIVITY | EXPRESSION | TUMORIGENESIS | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Humans | Pyrazines - administration & dosage | Imidazoles - chemistry | Imidazoles - administration & dosage | Autocrine Communication - drug effects | Antineoplastic Agents - administration & dosage | Imidazoles - pharmacokinetics | Protein Kinase Inhibitors - chemistry | Female | Antineoplastic Agents - pharmacokinetics | Antineoplastic Agents - pharmacology | Phosphorylation - drug effects | Protein Kinase Inhibitors - pharmacokinetics | Tumor Stem Cell Assay | Imidazoles - pharmacology | Antineoplastic Agents - chemistry | Enzyme Activation - drug effects | Insulin-Like Growth Factor II - metabolism | Blood Glucose - drug effects | Xenograft Model Antitumor Assays | Protein Kinase Inhibitors - administration & dosage | Pyrazines - chemistry | Animals | Signal Transduction - drug effects | Mice, Nude | Pyrazines - pharmacokinetics | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Colorectal Neoplasms - pathology | Pyrazines - pharmacology | Index Medicus
Journal Article
Cytokine & growth factor reviews, ISSN 1359-6101, 1996
Journal
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e76133 - e76133
Human insulin-like growth factor 1 Ec (IGF-1Ec), also called mechano growth factor (MGF), is a splice variant of insulin-like growth factor 1 (IGF-1), which... 
CELLS | MECHANICAL OVERLOAD | HORMONE | BONE-GROWTH | GENE | MULTIDISCIPLINARY SCIENCES | MUSCLE FUNCTION | MGF EXPRESSION | E-PEPTIDE ACTIONS | ARTICULAR CHONDROCYTES | MONOCLONAL-ANTIBODIES | Chondrocytes - cytology | Alternative Splicing | Humans | Peptides - genetics | Molecular Sequence Data | Muscle, Skeletal - metabolism | Insulin-Like Growth Factor I - genetics | Muscle, Skeletal - cytology | RNA, Messenger - metabolism | Chondrocytes - drug effects | Cell Nucleus - metabolism | Peptides - metabolism | Growth Plate - cytology | Swine | Growth Plate - drug effects | Chondrocytes - metabolism | Amino Acid Sequence | Gene Expression | RNA, Messenger - genetics | Growth Plate - metabolism | Peptides - pharmacology | Sequence Homology, Amino Acid | Mechanotransduction, Cellular | Sequence Alignment | Animals | Cytosol - metabolism | Cell Proliferation - drug effects | Primary Cell Culture | Insulin-Like Growth Factor I - metabolism | Peptides | Analysis | Cell culture | Alternative splicing | Transplants & implants | Insulin-like growth factor I | Insulin-like growth factors | Tissues | Nuclei | Proteins | Cartilage | Growth plate | Embryology | Cell growth | Aging | Physiology | Muscles | Histology | Anatomy & physiology | Insulin | Medicine | Chondrocytes | Nuclei (cytology) | Mechanical stimuli | Cytoplasm | Cancer | Hypertrophy | Index Medicus
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 11/2003, Volume 163, Issue 4, pp. 801 - 812
Spinal muscular atrophy (SMA), a common autosomal recessive form of motoneuron disease in infants and young adults, is caused by mutations in the survival... 
Growth cones | Axons | Neurites | Messenger RNA | Neurons | Heterogeneous nuclear ribonucleoproteins | Cell lines | Actins | Antibodies | PC12 cells | β-actin | RNA transport | SMN | HnRNP R | SMA | SURVIVAL | GRY-RBP | hnRNP R | PROGRESSIVE-MOTOR-NEURONOPATHY | CYTOPLASMIC LOCALIZATION | beta-actin | BINDING PROTEIN | SNRNP BIOGENESIS | SINGLE NUCLEOTIDE | CELL-DEATH | CELL BIOLOGY | MOUSE MODEL | MISSENSE MUTATION | Spinal Cord - metabolism | Spinal Cord - growth & development | Nerve Tissue Proteins - deficiency | 3' Untranslated Regions - genetics | Cell Survival - genetics | Axons - physiology | RNA-Binding Proteins | RNA, Messenger - metabolism | PC12 Cells | Actins - genetics | Cell Differentiation - genetics | Motor Neurons - cytology | Spinal Cord - cytology | Cyclic AMP Response Element-Binding Protein | Disease Models, Animal | Nerve Tissue Proteins - physiology | Survival of Motor Neuron 1 Protein | Heterogeneous-Nuclear Ribonucleoproteins - metabolism | Rats | Mice, Transgenic | SMN Complex Proteins | Nerve Tissue Proteins - genetics | Heterogeneous-Nuclear Ribonucleoproteins - genetics | Motor Neurons - metabolism | Animals | Growth Cones - metabolism | Growth Cones - ultrastructure | Mice | Muscular system | Cellular biology | Neurological disorders | SMN1 gene | spinal muscular atrophy | Smn protein | Heterogeneous-nuclear ribonucleoprotein R | Index Medicus | SMA; SMN; RNA transport; β-actin; hnRNP R
Journal Article
Progress in growth factor research, ISSN 0955-2235, 1989
Journal
Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2003, Volume 278, Issue 45, pp. 44966 - 44974
Journal Article
Oncogene, ISSN 0950-9232, 12/2010, Volume 29, Issue 49, pp. 6485 - 6498
Journal Article