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Cold Spring Harbor Protocols, ISSN 1940-3402, 04/2016, Volume 2016, Issue 4, pp. 343 - 346
Adherent cells detach from cell culture plates during cell death. This characteristic can be used for the indirect quantification of cell death and to... 
Gentian Violet - metabolism | Staining and Labeling - methods | Cells, Cultured - physiology | Cytological Techniques - methods | Cell Survival
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2004, Volume 24, Issue 30, pp. 6799 - 6809
The accumulation of beta-amyloid (Abeta) is one of the etiological factors in Alzheimer's disease ( AD). It has been assumed that the underlying mechanism... 
PI3-K | CREB | MAPK | BDNF | Cortical neurons | β-amyloid | ACTIVATED PROTEIN-KINASE | ALZHEIMERS-DISEASE | MAP-KINASE | ELEMENT-BINDING PROTEIN | SYNAPTIC PLASTICITY | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | beta-amyloid | cortical neurons | SIGNALING PATHWAY | IN-VIVO | LONG-TERM POTENTIATION | TRANSGENIC MICE | Alzheimer Disease - etiology | Brain-Derived Neurotrophic Factor - antagonists & inhibitors | Phosphorylation | Oncogene Protein p21(ras) - physiology | Amyloid beta-Peptides - pharmacology | Receptor, trkB - physiology | Glycogen Synthase Kinase 3 beta | Peptide Fragments - pharmacology | Cerebral Cortex - cytology | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Brain-Derived Neurotrophic Factor - pharmacology | Transfection | Neurons - physiology | Phospholipase C gamma | Amyloid beta-Peptides - administration & dosage | Extracellular Signal-Regulated MAP Kinases - physiology | Neurons - drug effects | Proto-Oncogene Proteins - metabolism | Cells, Cultured - drug effects | Nerve Tissue Proteins - physiology | ets-Domain Protein Elk-1 | Protein-Serine-Threonine Kinases - physiology | Peptide Fragments - administration & dosage | Cells, Cultured - physiology | Rats | Enzyme Activation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Peptide Fragments - chemical synthesis | Proto-Oncogene Proteins c-akt | Transcription Factors - metabolism | Animals | MAP Kinase Signaling System - drug effects | Amyloid beta-Peptides - chemical synthesis | Signal Transduction - drug effects | Alzheimer Disease - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Proto-Oncogene Proteins - physiology | Phosphatidylinositol 3-Kinases - physiology | Protein Processing, Post-Translational | Type C Phospholipases - physiology | Cellular | Molecular
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2004, Volume 24, Issue 48, pp. 10963 - 10973
The profound neuroprotection observed in poly(ADP-ribose) polymerase-1 (PARP-1) null mice to ischemic and excitotoxic injury positions PARP-1 as a major... 
NMDA | Neuronal death | PARP-1 | Apoptosis-inducing factor | Excitotoxicity | nNOS | ACTIVATION | NITRIC-OXIDE SYNTHASE | POLY(ADP-RIBOSE) POLYMERASE GENE | NEUROSCIENCES | CELL-DEATH | DISRUPTION | MICE RESISTANT | NAD(+) DEPLETION | neuronal death | apoptosis-inducing factor | excitotoxicity | SYNTHESIZED IN-VITRO | MITOCHONDRIAL RELEASE | CEREBRAL-ISCHEMIA | Caspases - physiology | Proto-Oncogene Proteins c-bcl-2 - physiology | Intracellular Membranes - physiology | Apoptosis - drug effects | Apoptosis Inducing Factor | Cells, Cultured - cytology | Poly(ADP-ribose) Polymerases - physiology | Cerebral Cortex - cytology | Corpus Striatum - metabolism | Transfection | Membrane Proteins - physiology | Neurons - physiology | Active Transport, Cell Nucleus | Neurons - drug effects | Membrane Potentials - drug effects | Cells, Cultured - drug effects | Cells, Cultured - physiology | Glutamic Acid - physiology | Nitric Oxide - physiology | Mitochondria - metabolism | Cytochromes c - analysis | Genes, bcl-2 | Enzyme Activation - drug effects | Proto-Oncogene Proteins c-bcl-2 - biosynthesis | Mice, Knockout | Flavoproteins - physiology | Injections | Animals | Excitatory Amino Acid Agonists - toxicity | Poly(ADP-ribose) Polymerases - genetics | alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid - toxicity | Cerebral Cortex - embryology | Signal Transduction - drug effects | Cysteine Proteinase Inhibitors - pharmacology | Corpus Striatum - drug effects | Poly(ADP-ribose) Polymerases - deficiency | Signal Transduction - physiology | Mice | Apoptosis - physiology | DNA Damage | Poly (ADP-Ribose) Polymerase-1 | N-Methylaspartate - toxicity | Neurobiology of Disease
Journal Article
Cancer Research, ISSN 0008-5472, 11/2011, Volume 71, Issue 21, pp. 6857 - 6866
Most ductal breast carcinoma cells are weakly invasive in vitro and in vivo, suggesting that components of their microenvironment may facilitate a transition... 
MIGRATION | TRANSITION | COLLAGEN | ONCOLOGY | BRANCHING MORPHOGENESIS | GENE-EXPRESSION | TUMOR | CELL-LINES | CARCINOMA | IN-SITU | REVEALS | Neoplasm Transplantation | Fibroblasts - physiology | Spheroids, Cellular - transplantation | Cells, Cultured - cytology | Coculture Techniques | Humans | Neoplasm Invasiveness - prevention & control | Culture Media, Conditioned - pharmacology | Tumor Microenvironment - physiology | Spheroids, Cellular - physiology | Cell Movement - physiology | Neovascularization, Pathologic - pathology | Carcinoma, Ductal, Breast - classification | Extracellular Matrix - physiology | cdc42 GTP-Binding Protein - antagonists & inhibitors | Carcinoma, Ductal, Breast - pathology | Female | Breast Neoplasms - classification | Breast - cytology | RNA, Small Interfering - pharmacology | Tumor Cells, Cultured - drug effects | Collagen - physiology | Cells, Cultured - physiology | cdc42 GTP-Binding Protein - physiology | Mice, SCID | Neoplasm Invasiveness - physiopathology | Cell Movement - drug effects | Extracellular Matrix - ultrastructure | Animals | Carcinoma, Intraductal, Noninfiltrating - pathology | Breast Neoplasms - pathology | cdc42 GTP-Binding Protein - genetics | Fibroblasts - cytology | Mice | Protein Kinase Inhibitors - pharmacology | Tumor Cells, Cultured - physiology | Tumor Cells, Cultured - cytology | microenvironment | Breast cancer | invasion | metastasis | heterogeneity
Journal Article
Circulation, ISSN 0009-7322, 01/2005, Volume 111, Issue 1, pp. 11 - 20
Background - Human embryonic stem cells (hESCs) derived from blastocysts can propagate indefinitely in culture while maintaining pluripotency, including the... 
Cardiac development | Viruses | Gene therapy | Pacemakers | Stem cells | pacemakers | stem cells | viruses | CARDIAC & CARDIOVASCULAR SYSTEMS | GENE DELIVERY | MYOCYTES | gene therapy | LENTIVIRAL VECTORS | BIOLOGICAL PACEMAKER | LIDOCAINE | IN-VIVO | CANINE HEART | CONDUCTION | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | cardiac development | EXPRESSION | Lidocaine - pharmacology | Heart Ventricles - cytology | Cells, Cultured - cytology | Giant Cells - drug effects | Heart - physiology | Humans | Electrophysiology | Myocardial Contraction - drug effects | Green Fluorescent Proteins - genetics | Action Potentials | Cell Fusion | HIV-1 - physiology | Pericardium - physiology | Heart Conduction System - physiology | Female | Cell Differentiation | Genes, Reporter | Organ Culture Techniques | Heart Conduction System - cytology | Green Fluorescent Proteins - analysis | Guinea Pigs | Pluripotent Stem Cells - cytology | Transduction, Genetic | Myocytes, Cardiac - cytology | Pluripotent Stem Cells - physiology | Defective Viruses - genetics | Adrenergic beta-Agonists - pharmacology | Cells, Cultured - physiology | Rats | Giant Cells - cytology | Pyrimidines - pharmacology | HIV-1 - genetics | Defective Viruses - physiology | Myocytes, Cardiac - transplantation | Animals | Myocytes, Cardiac - drug effects | Genetic Vectors - pharmacology | Myocytes, Cardiac - physiology | Isoproterenol - pharmacology | Mice
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 02/2005, Volume 25, Issue 8, pp. 1904 - 1913
Alzheimer's disease ( AD) is characterized by enhanced beta amyloid peptide ( betaA) deposition along with glial activation in senile plaques, selective... 
Neuroprotection | Cannabinoids | Neurotoxicity | Alzheimer's disease | β-amyloid | Microglia | SELECTIVE ANTAGONIST | HUMAN BRAIN | MESSENGER-RNA EXPRESSION | NITRIC-OXIDE SYNTHASE | NEUROSCIENCES | beta-amyloid | CB2 RECEPTORS | neurotoxicity | neuroprotection | AMYLOID-BETA-PEPTIDE | IN-VIVO | cannabinoids | PRECURSOR PROTEIN | microglia | RECEPTOR-BINDING | TRANSGENIC MICE | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Receptor, Cannabinoid, CB2 - physiology | Rats, Wistar | Receptor, Cannabinoid, CB2 - agonists | Peptide Fragments - toxicity | Cells, Cultured - cytology | Coculture Techniques | Humans | Middle Aged | Injections, Intraventricular | Motor Activity - drug effects | Male | Benzoxazines | Plaque, Amyloid - drug effects | Receptor, Cannabinoid, CB1 - physiology | Microglia - physiology | Neuroprotective Agents - pharmacology | Receptor, Cannabinoid, CB1 - agonists | Protein Processing, Post-Translational - drug effects | Alzheimer Disease - prevention & control | Nerve Degeneration - prevention & control | Dronabinol - analogs & derivatives | Amyloid beta-Peptides - metabolism | Neurons - physiology | Female | Dronabinol - pharmacology | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Alzheimer Disease - physiopathology | Cells, Cultured - drug effects | Microglia - drug effects | Amyloid beta-Peptides - toxicity | Cannabinoids - administration & dosage | Morpholines - administration & dosage | Cells, Cultured - physiology | Morpholines - pharmacology | Rats | Maze Learning - drug effects | Cannabinoids - pharmacology | Naphthalenes - pharmacology | Animals | Signal Transduction - drug effects | Naphthalenes - administration & dosage | Plaque, Amyloid - metabolism | Aged | Nitric Oxide - metabolism | Neurobiology of Disease
Journal Article
PLoS Medicine, ISSN 1549-1277, 04/2007, Volume 4, Issue 4, pp. 675 - 689
In multiple sclerosis, inflammation can successfully be prevented, while promoting repair is still a major challenge. Microglial cells, the resident phagocytes... 
MEDICINE, GENERAL & INTERNAL | Genetic Therapy | Neurons - pathology | Nitric Oxide Synthase Type II - biosynthesis | Receptors, Immunologic - physiology | Humans | Myeloid Cells - physiology | Cells, Cultured - transplantation | Genetic Vectors - therapeutic use | Multiple Sclerosis | Membrane Glycoproteins - physiology | Flow Cytometry | Microglia - physiology | Female | Lentivirus - genetics | Myeloid Cells - drug effects | Monocytes - physiology | Cytokines - genetics | Genes, Reporter | Disease Models, Animal | Encephalomyelitis, Autoimmune, Experimental - surgery | Promoter Regions, Genetic | Encephalomyelitis, Autoimmune, Experimental - pathology | Transduction, Genetic | Mice, Inbred C57BL | Gene Expression Regulation | Genes, Synthetic | Cells, Cultured - physiology | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Hematopoietic Stem Cell Transplantation | Genetic Vectors - genetics | Membrane Glycoproteins - genetics | Cell Lineage | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Nitric Oxide Synthase Type II - genetics | Cell Differentiation - drug effects | Interleukin-10 - genetics | Interleukin-10 - biosynthesis | Myeloid Cells - transplantation | Mice | Receptors, Immunologic - genetics | Cytokines - biosynthesis | Phagocytosis | Apoptosis | Cell Movement | Multiple sclerosis | Biochemical genetics | Inflammation | Research | Neuroglia | Central nervous system | Neurological Disorders | Neurology | Neuroscience | Immunology | Immunology and Allergy
Journal Article