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Nature (London), ISSN 1476-4687, 2017, Volume 546, Issue 7658, pp. 431 - 435
...). Resistance can result from secondary mutations(3,4), but in other cases there is no clear genetic cause, raising the possibility of non-genetic rare cell variability(5-11... 
METASTATIC MELANOMA | BRAF INHIBITOR | SUBPOPULATIONS | CHROMATIN | MULTIDISCIPLINARY SCIENCES | SINGLE MAMMALIAN-CELLS | STATE | MECHANISMS | MUTATIONS | VEMURAFENIB | PLASTICITY | Transcription, Genetic - drug effects | Humans | Male | Transcription Factor AP-1 - metabolism | DNA-Binding Proteins - metabolism | Melanoma - genetics | Female | Indoles - pharmacology | Epigenesis, Genetic - drug effects | Gene Expression Regulation, Neoplastic - drug effects | SOXE Transcription Factors - deficiency | Single-Cell Analysis | Cellular Reprogramming - genetics | ErbB Receptors - metabolism | In Situ Hybridization, Fluorescence | Nuclear Proteins - metabolism | Signal Transduction - genetics | Melanoma - pathology | Sulfonamides - pharmacology | Cellular Reprogramming - drug effects | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Genetic Markers - drug effects | Vemurafenib | Drug Resistance, Neoplasm - genetics | Animals | Signal Transduction - drug effects | Genetic Markers - genetics | Cell Line, Tumor | Oncogene Protein p65(gag-jun) - metabolism | Drug Resistance, Neoplasm - drug effects | SOXE Transcription Factors - genetics | Antimitotic agents | Cell interaction | Dosage and administration | Antineoplastic agents | Drug resistance | Observations | Health aspects | Subpopulations | Transcription factors | Substance abuse treatment | Variability | Activator protein 1 | Melanoma | Genomes | Kinases | Gene expression | Sox10 protein | Signal transduction | Signaling | Converting | Population | Mutation | Differentiation | Deoxyribonucleic acid--DNA | Cancer
Journal Article
Cell Cycle, ISSN 1538-4101, 2014, Volume 11, Issue 16, pp. 3019 - 3035
.... Mechanistically, a loss of Cav-1 induces the metabolic reprogramming of stromal cells, with increased autophagy/mitophagy, mitochondrial dysfunction and aerobic glycolysis... 
autophagy | paracrine signaling | myofibroblast | cancer associated fibroblast | the field effect | cancer metabolism | "Pied-Piper of Hamelin" | mitophagy | autocrine signaling | tumor stroma | TGF beta | aerobic glycolysis | oxidative stress | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Oxidative stress | Autocrine signaling | Myofibroblast | Tumor stroma | Paracrine signaling | Cancer associated fibroblast | Mitophagy | Autophagy | Cancer metabolism | The field effect | Aerobic glycolysis | Pied-Piper of Hamelin | SMOOTH MUSCLE ACTIN | TGF- beta | METASTASIS | CELL BIOLOGY | BREAST-CANCER | CAVEOLIN-1 EXPRESSION | EPITHELIAL-CELLS | MICROENVIRONMENT | TRANSFORMING GROWTH-FACTOR-BETA-1 | Immunohistochemistry | Receptors, Transforming Growth Factor beta - genetics | Oxidative Stress | Stromal Cells - pathology | Coculture Techniques | Humans | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Breast Neoplasms - metabolism | Cellular Reprogramming | Mitochondria - genetics | Cell Transformation, Neoplastic - genetics | Female | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Autocrine Communication | Paracrine Communication | Lactic Acid - metabolism | Stromal Cells - metabolism | Oxidative Phosphorylation | Protein-Serine-Threonine Kinases - genetics | Caveolin 1 - genetics | Mitochondria - metabolism | Mitochondria - pathology | Fibroblasts - pathology | Cell Transformation, Neoplastic - metabolism | Caveolin 1 - metabolism | Gene Expression Regulation, Enzymologic | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Breast Neoplasms - pathology | Mice, Nude | Cell Line, Tumor | Glycolysis | Ligands | Mice | Neovascularization, Pathologic - metabolism | Transforming Growth Factor beta - metabolism | Report
Journal Article
Stem cells (Dayton, Ohio), ISSN 1066-5099, 2014, Volume 32, Issue 2, pp. 364 - 376
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 483, Issue 7391, pp. 598 - 602
Generation of induced pluripotent stem cells (iPSCs) by somatic cell reprogramming involves global epigenetic remodelling(1... 
CELLS | LEUKEMIA | PRC2 | HISTONE METHYLATION | MULTIDISCIPLINARY SCIENCES | PLURIPOTENT | Chromatin - metabolism | Homeodomain Proteins - metabolism | Humans | Methyltransferases - metabolism | Methyltransferases - genetics | Methyltransferases - biosynthesis | YY1 Transcription Factor - metabolism | Repressor Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Kruppel-Like Transcription Factors - metabolism | YY1 Transcription Factor - antagonists & inhibitors | Induced Pluripotent Stem Cells - cytology | Cellular Reprogramming - genetics | Repressor Proteins - metabolism | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Nanog Homeobox Protein | Transcription Factors - antagonists & inhibitors | DNA Methylation - genetics | Polycomb-Group Proteins | Proto-Oncogene Proteins c-myc - metabolism | Enhancer of Zeste Homolog 2 Protein | Transcription Factors - metabolism | Polycomb Repressive Complex 2 | Methyltransferases - antagonists & inhibitors | Fibroblasts - cytology | RNA, Small Interfering | Histones - metabolism | Methylation | Chromatin - genetics | RNA-Binding Proteins - metabolism | Physiological aspects | Chromatin | Genetic aspects | Research | Methyltransferases | Embryonic stem cells | Enzymes | Efficiency | Stem cells | Epigenetics | Kinases | Gene expression | Apoptosis
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2016, Volume 167, Issue 7, pp. 1719 - 1733.e12
Journal Article
Cell (Cambridge), ISSN 0092-8674, 2018, Volume 172, Issue 1-2, pp. 162 - 175.e14
Journal Article