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Frontiers in Oncology, ISSN 2234-943X, 12/2017, Volume 7, p. 306
The concept that cancer is a metabolic disease is now well acknowledged: many cancer cell types rely mostly on glucose and some amino acids, especially... 
Mitochondria | Proteoliposome | Drug design | Metabolism | Plasma membrane | Tumors | 1 LAT1 | plasma membrane | drug design | mitochondria | AMINO-ACID TRANSPORTERS | proteoliposome | MAMMALIAN-CELLS | MOUSE MODELS | tumors | ONCOLOGY | ASCT2 | ATB(0,+) SLC6A14 | THERAPEUTIC TARGET | metabolism | UP-REGULATION | TUMOR-GROWTH | EXPRESSION | Genetic aspects | Carrier proteins | Health aspects | Carcinogenesis | Cellular control mechanisms
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0181687
Oxidative stress resulting from endogenous and exogenous sources causes damage to cellular components, including genomic and mitochondrial DNA. Oxidative DNA... 
SKELETAL-MUSCLE | ALZHEIMERS-DISEASE BRAIN | FATTY-ACID TRANSPORT | BASE EXCISION-REPAIR | OXIDATIVE DAMAGE | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | SER326CYS POLYMORPHISM | SACCHAROMYCES-CEREVISIAE | PARKINSONS-DISEASE | DNA, Mitochondrial - metabolism | Mice, Inbred C57BL | DNA Glycosylases - genetics | Male | Muscle, Skeletal - metabolism | Gene Expression Profiling | Mitochondria - metabolism | Physical Endurance - physiology | Mitochondrial Dynamics - physiology | DNA Glycosylases - deficiency | Running - physiology | Animals | Adiposity | DNA Damage - physiology | Mitochondria - genetics | Pyruvic Acid - metabolism | Models, Animal | Muscle, Skeletal - pathology | Fatty Acids - metabolism | Lipid Metabolism - physiology | Muscle Strength - physiology | Oxidative stress | DNA damage | Mitochondrial DNA | DNA glycosylases | Research | Metabolic syndrome X | Gene expression | Tricarboxylic acid cycle | Health sciences | Oxidative metabolism | Genes | Lipids | DNA glycosylase | Kinases | Base excision repair | Fission | High fat diet | Fragmentation | Purines | Alterations | Fatty liver | Rodents | Occupational health | Physiology | Endurance | Lipid metabolism | Grip strength | Damage | Deposition | Deoxyribonucleic acid--DNA | Carbohydrates | Markers | Carbohydrate metabolism | Metabolism | Insulin | Fatty acids | Skeletal muscle | Steatosis | Musculoskeletal system | OGG1 protein | Insulin resistance | Mice | Diabetes | Metabolic disorders | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2011, Volume 300, Issue 2, pp. 308 - 317
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 03/2011, Volume 300, Issue 3, pp. C447 - C455
Brooks C, Cho S, Wang C, Yang T, Dong Z. Fragmented mitochondria are sensitized to Bax insertion and activation during apoptosis. Am J Physiol Cell Physiol... 
Cytochrome c | Mitochondria outer membrane permeabilization | Mitochondrial dynamics | TRANSLOCATION | mitochondria outer membrane permeabilization | PHYSIOLOGY | FUSION | TUBULAR CELL APOPTOSIS | INJURY | DEATH | mitochondrial dynamics | FAMILY | CELL BIOLOGY | BCL-2 | DYNAMICS | FISSION | cytochrome c | MORPHOLOGY | Mitochondrial Diseases - pathology | Microtubule-Associated Proteins - genetics | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Humans | Mitochondrial Diseases - metabolism | Membrane Fusion - genetics | Mitochondrial Proteins - genetics | Gene Knockdown Techniques - methods | Mitochondrial Membrane Transport Proteins | Mitochondrial Proteins - metabolism | Apoptosis Regulatory Proteins - genetics | Membrane Transport Proteins - metabolism | RNA Interference - physiology | bcl-2-Associated X Protein - genetics | Mitochondrial Diseases - genetics | Stress, Physiological - physiology | Cell Line | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Mice | Apoptosis - physiology | HeLa Cells | Enzyme Activation - genetics | Physiological aspects | Mitochondria | Research | Apoptosis | Cellular and Mitochondrial Metabolism
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2010, Volume 107, Issue 19, pp. 8788 - 8793
Journal Article
Journal Article