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Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
Journal Article
Brain and Development, ISSN 0387-7604, 04/2018, Volume 40, Issue 4, pp. 259 - 267
Journal Article
2017, Advances in Experimental Medicine and Biology, Volume 1002, 23
The cell cycle culminates in mitosis with the purpose of dividing the cell's DNA content equally over two daughter cells. Error-free segregation relies on... 
MEDICINE, RESEARCH & EXPERIMENTAL | CHROMOSOME INSTABILITY | DNA-DAMAGE | Cell division | Aneuploidy | SPINDLE ASSEMBLY CHECKPOINT | INNER CENTROMERE | Chromosomal instability | GENOME STABILITY | Microcephaly | MOSAIC VARIEGATED ANEUPLOIDY | CELL BIOLOGY | INTELLECTUAL DISABILITY | Kinetochore | CENTROSOME AMPLIFICATION | Development | CENP-F | MITOTIC CHECKPOINT | Cancer
Book Chapter
Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1214 - 1223
The midbody is a singular organelle formed between daughter cells during cytokinesis and required for their final separation. Midbodies persist in cells long... 
NEURAL PROGENITORS | STEM-CELLS | ASYMMETRIC INHERITANCE | CYTOKINESIS | MEMBRANE | CENTROSOME | DISEASE | DEGRADATION | LITHIUM | DIVISION | CELL BIOLOGY | Embryonic Stem Cells - metabolism | Cell Proliferation | Coculture Techniques | Humans | Recombinant Fusion Proteins - metabolism | Cellular Reprogramming | Lysosomes - metabolism | Transfection | Neoplastic Stem Cells - metabolism | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Cell Division | Cell Cycle Proteins - genetics | Neoplastic Stem Cells - pathology | Cell Differentiation | Autophagy - genetics | Nuclear Proteins - genetics | Induced Pluripotent Stem Cells - metabolism | Calcium-Binding Proteins - metabolism | Induced Pluripotent Stem Cells - pathology | Cell Line | Chromosomal Proteins, Non-Histone - metabolism | Organelles - pathology | Cell Cycle Proteins - metabolism | Nuclear Proteins - metabolism | Cell Transformation, Neoplastic - metabolism | Centrosome - metabolism | Chromosomal Proteins, Non-Histone - genetics | Proteins - genetics | Animals | Proteins - metabolism | Embryonic Stem Cells - pathology | Mice | HeLa Cells | Cell Transformation, Neoplastic - pathology | Organelles - metabolism | Calcium-Binding Proteins - genetics | Physiological aspects | Cell division | Autophagy (Cytology) | Research | Stem cells
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 86 - 100
It has been proposed that an aggressive secondary cancer stem cell population arises from a primary cancer stem cell population through acquisition of... 
STEM-CELLS | INSTABILITY | EPITHELIAL-CELLS | ONCOLOGY | GROUP PROTEIN EZH2 | GENE-EXPRESSION | SELF-RENEWAL | HYPOXIC CANCER-CELLS | DNA-REPAIR | IDENTIFICATION | IN-VITRO PROPAGATION | CELL BIOLOGY | Cell Hypoxia - physiology | Gene Expression - genetics | Neoplastic Stem Cells - drug effects | Humans | Spheroids, Cellular - pathology | DNA Breaks, Double-Stranded | Benzenesulfonates - pharmacology | Neoplastic Stem Cells - metabolism | DNA Damage - genetics | Neoplastic Stem Cells - pathology | Phosphorylation - drug effects | Benzimidazoles - therapeutic use | Rad51 Recombinase - metabolism | Cell Survival - drug effects | Breast Neoplasms - drug therapy | beta Catenin - metabolism | Enhancer of Zeste Homolog 2 Protein | Polycomb Repressive Complex 2 | Centrosome - pathology | Signal Transduction - drug effects | Models, Biological | Cell Line, Tumor | Benzimidazoles - pharmacology | Mice, Inbred NOD | Breast Neoplasms - diagnosis | Mice | Niacinamide - analogs & derivatives | Gene Expression - drug effects | Phenylurea Compounds | Extracellular Signal-Regulated MAP Kinases - metabolism | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Female | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - physiology | Cell Survival - physiology | Proto-Oncogene Proteins c-raf - genetics | Rad51 Recombinase - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mice, SCID | Proto-Oncogene Proteins c-raf - metabolism | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Chromosome Aberrations | Signal Transduction - physiology | Transplantation, Heterologous - pathology | Cell Proliferation - drug effects | Pyridines - pharmacology
Journal Article