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Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects | Neuroepithelial Cells/virology | Life Sciences | Brain/pathology | Zika Virus/drug effects | Brain/embryology | Mitochondria/drug effects | Fetus/virology | Neocortex/pathology | Neuroglia/pathology | Cell Death/drug effects | Mitosis/drug effects | Transcription, Genetic/drug effects | Nucleosides/pharmacology | Neural Stem Cells/enzymology | Neural Stem Cells/ultrastructure | Neuroepithelial Cells/ultrastructure | Receptor Protein-Tyrosine Kinases/metabolism | Microcephaly/virology | Proto-Oncogene Proteins/metabolism | Neuroprotective Agents/pharmacology | Zika Virus/ultrastructure | Neuroepithelial Cells/immunology | Brain/virology | Immunity, Innate/drug effects | Spinal Cord/pathology | Zika Virus/physiology | Neuroglia/virology | Microbiology and Parasitology | Zika Virus Infection/pathology | Neurons/virology | Zika Virus Infection/virology | Neural Stem Cells/virology | Centrosome/drug effects | Protein Kinase Inhibitors/pharmacology | Centrosome/metabolism | Phosphorylation/drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e100819 - e100819
Beclin 1 interacts with UV-irradiation-resistance-associated gene (UVRAG) to form core complexes that induce autophagy. While cells with defective autophagy... 
RECOMBINATION | REPAIR | INHIBITION | GENE | INDUCED APOPTOSIS | MULTIDISCIPLINARY SCIENCES | ATM | AUTOPHAGY | LC3 | CYCLE | Gamma Rays | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Centrosome - drug effects | Intracellular Signaling Peptides and Proteins - metabolism | Centrosome - radiation effects | Autophagy - drug effects | DNA Breaks, Double-Stranded - radiation effects | Gene Knockdown Techniques | Cell Nucleus - metabolism | DNA Breaks, Double-Stranded - drug effects | Protein Binding - drug effects | Cytoprotection - drug effects | Protein Binding - radiation effects | Membrane Proteins - metabolism | Colorectal Neoplasms - metabolism | Beclin-1 | Rad51 Recombinase - metabolism | Tumor Suppressor Proteins - metabolism | Apoptosis Regulatory Proteins - metabolism | Centrosome - metabolism | Autophagy-Related Protein 5 | Cell Line, Tumor | Fluorouracil - pharmacology | DNA Damage | Tumor Suppressor p53-Binding Protein 1 | Cell Nucleus - drug effects | Colorectal Neoplasms - pathology | Cytoprotection - radiation effects | Prevention | Ionizing radiation | DNA damage | DNA | Colorectal cancer | Genetic aspects | Tubulins | Cancer | Homologous recombination | Colorectal carcinoma | Radiation | Irradiated | Homology | Cytotoxicity | Biology | Kinases | Autophagy | DNA repair | Cancer therapies | Genomic instability | Deletion mutant | Proteins | Tubulin | Clonal deletion | Cell cycle | Deletion | Tumorigenesis | Chromosomes | Deoxyribonucleic acid--DNA | Radiation effects | Stability | U.V. radiation | Tumor cells | Cell division | siRNA | Metabolism | Ultraviolet radiation | Chemotherapy | Cell death | Irradiation | Hypoxia | Radiation damage | Phagocytosis | Tumors | Apoptosis | Index Medicus | Deoxyribonucleic acid
Journal Article
Cell Stem Cell, ISSN 1934-5909, 06/2015, Volume 16, Issue 6, pp. 684 - 698
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 07/2015, Volume 14, Issue 7, pp. 1693 - 1704
The aurora kinases constitute one family of serine/threonine kinases whose activity is essential for mitotic progression. The aurora kinases are frequently... 
RADIATION RESPONSE | APOPTOSIS | ONCOLOGY | PHASE-III | A KINASE | FRACTIONATED-IRRADIATION | CONCURRENT CHEMORADIATION | COMBINATION | DOCETAXEL | SMALL-MOLECULE INHIBITOR | CELL-DEATH | Gene Expression Regulation, Enzymologic - drug effects | Lung Neoplasms - drug therapy | Aurora Kinase B - metabolism | Apoptosis - drug effects | Apoptosis - radiation effects | Humans | Centrosome - drug effects | Lung Neoplasms - radiotherapy | Lung Neoplasms - pathology | Green Fluorescent Proteins - genetics | Centrosome - radiation effects | Chemoradiotherapy | Aurora Kinase A - metabolism | RNA Interference | Genomic Instability - drug effects | Female | Gene Expression Regulation, Neoplastic - drug effects | Aurora Kinase A - genetics | Genomic Instability - genetics | Aurora Kinase B - genetics | Cell Survival - drug effects | Genomic Instability - radiation effects | Green Fluorescent Proteins - metabolism | Gene Expression Regulation, Enzymologic - radiation effects | HCT116 Cells | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Xenograft Model Antitumor Assays - methods | Cell Survival - radiation effects | Blotting, Western | Naphthalenes - pharmacology | Animals | Cell Line, Tumor | Mice, Inbred NOD | Cell Proliferation - drug effects | Gene Expression Regulation, Neoplastic - radiation effects | Benzodioxoles - pharmacology | Microscopy, Fluorescence | Cell Proliferation - radiation effects
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 2013, Volume 202, Issue 5, pp. 779 - 792
Journal Article
Journal Article
Anticancer Research, ISSN 0250-7005, 06/2018, Volume 38, Issue 6, pp. 3393 - 3400
Background/Aim: Owing to the frequent observation of centrosome amplification in human cancers, cancer cells have a unique mechanism to suppress detrimental... 
Centrosome amplification | Radiosensitizer | Breast cancer | Radiotherapy | Centrosome clustering | centrosome amplification | IN-VITRO | AMPLIFICATION | INSTABILITY | ONCOLOGY | radiotherapy | breast cancer | radiosensitizer | INDUCTION | CIP2A | INHIBITOR | Humans | Spindle Apparatus - radiation effects | Cell Survival - genetics | Centrosome - drug effects | Centrosome - radiation effects | Breast Neoplasms - metabolism | Spindle Apparatus - metabolism | MCF-7 Cells | RNA Interference | Kinesin - genetics | Female | Griseofulvin - pharmacology | Fibroblasts - metabolism | Cell Division - genetics | Cell Line | Cell Survival - drug effects | Radiation-Sensitizing Agents - pharmacology | Cell Division - radiation effects | Cell Survival - radiation effects | Cell Division - drug effects | Centrosome - metabolism | Kinesin - metabolism | Phenanthrenes - pharmacology | Breast Neoplasms - genetics | Fibroblasts - radiation effects | Breast Neoplasms - pathology | Fibroblasts - drug effects | Cell Line, Tumor | Spindle Apparatus - drug effects | Biotechnology | Radiation effects | Radiosensitivity | Centrosomes | siRNA | Radiation therapy | Tumor cell lines | Clustering | Amplification | Ionizing radiation | Cell lines | Fibroblasts | Breast | Irradiation | Sensitivity enhancement | Division | Inhibition | Immunofluorescence | Spindles | Viability | Radiation damage | Cancer | Tumors
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 06/2003, Volume 63, Issue 12, pp. 3211 - 3220
Journal Article
ACS Nano, ISSN 1936-0851, 03/2014, Volume 8, Issue 3, pp. 2196 - 2205
Journal Article
Cancer Cell, ISSN 1535-6108, 12/2007, Volume 12, Issue 6, pp. 514 - 527
Journal Article
Journal Article
British Journal of Cancer, ISSN 0007-0920, 01/2008, Volume 98, Issue 2, pp. 345 - 355
Journal Article
Journal Article