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BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 06/2016, Volume 1861, Issue 6, pp. 491 - 500
Journal Article
Cancer Cell, ISSN 1535-6108, 05/2012, Volume 21, Issue 5, pp. 601 - 613
The proto-oncogene is mis-expressed in various types of human brain tumors. To clarify how developmental and regional differences influence transformation, we... 
PROGENITORS | SOX9 EXPRESSION | ONCOLOGY | SONIC HEDGEHOG | MOUSE MODEL | EMBRYONIC LETHALITY | PROLIFERATION | GENERATION | MUTATIONS | PEDIATRIC MEDULLOBLASTOMA | ASTROCYTES | CELL BIOLOGY | Oncogene Proteins - genetics | Cell Proliferation | Humans | Zinc Finger Protein Gli2 | Brain Stem - metabolism | Hedgehog Proteins - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Glioma - pathology | Medulloblastoma - pathology | Cell Differentiation | N-Myc Proto-Oncogene Protein | Cerebellar Neoplasms - pathology | Prosencephalon - metabolism | Cerebellar Neoplasms - metabolism | Biomarkers - metabolism | Neuroectodermal Tumors, Primitive - metabolism | SOX9 Transcription Factor - metabolism | Transduction, Genetic | Signal Transduction | Oncogene Proteins - metabolism | Brain Neoplasms - genetics | Mice, Transgenic | Medulloblastoma - metabolism | Gestational Age | Cell Lineage | Mice, Nude | Brain Stem - embryology | Mice | Mutation | Brain Neoplasms - pathology | Brain Neoplasms - metabolism | Glioma - metabolism | Cerebellum - embryology | Hedgehog Proteins - genetics | Kruppel-Like Transcription Factors - metabolism | Female | Nuclear Proteins - genetics | Spheroids, Cellular | Proto-Oncogene Proteins - metabolism | Cerebellum - metabolism | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Neural Stem Cells - pathology | Animals | Cell Transformation, Neoplastic - pathology | Prosencephalon - embryology | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Neuroectodermal Tumors, Primitive - pathology | Gliomas | Children's hospitals | Oncology, Experimental | Brain tumors | Stem cells | Transplantation | Universities and colleges | Research | Statistics | Tumors | Cancer | N-MYC | glioma | SOX9 | medulloblastoma | neural stem cells | GFAP | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal Article
Endocrinology, ISSN 0013-7227, 02/2016, Volume 157, Issue 2, pp. 737 - 751
Journal Article
Cerebral cortex (New York, N.Y. : 1991), ISSN 1047-3211, 2014, Volume 24, Issue 10, pp. 2784 - 2795
Journal Article
Nature Medicine, ISSN 1078-8956, 2014, Volume 20, Issue 8, pp. 886 - 896
In Alzheimer's disease (AD), memory impairment is the most prominent feature that afflicts patients and their families. Although reactive astrocytes have been... 
MEDICINE, RESEARCH & EXPERIMENTAL | GLUTAMATE RELEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAO-B | CELL BIOLOGY | PUTRESCINE | CHANNEL | MONOAMINE-OXIDASE-B | SELEGILINE | CULTURED ASTROCYTES | L-DEPRENYL | MICE | DENTATE GYRUS | Memory - drug effects | RNA, Small Interfering - genetics | Alzheimer Disease - complications | Amyloid beta-Peptides - pharmacology | Humans | Ion Channels - genetics | Memory Disorders - metabolism | Male | Peptide Fragments - pharmacology | Monoamine Oxidase - genetics | GABA Antagonists - therapeutic use | Bestrophins | Eye Proteins - genetics | Putrescine - pharmacology | Disease Models, Animal | Astrocytes - drug effects | Monoamine Oxidase Inhibitors - pharmacology | Dentate Gyrus - metabolism | Cerebellum - metabolism | Mice, Inbred C57BL | Alzheimer Disease - drug therapy | Mice, Transgenic | Hippocampus - cytology | Mice, Inbred ICR | Hippocampus - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Maze Learning | Memory Disorders - drug therapy | Memory Disorders - etiology | Eye Proteins - metabolism | Ion Channels - metabolism | gamma-Aminobutyric Acid - biosynthesis | Alzheimer Disease - metabolism | Receptors, GABA - metabolism | Plaque, Amyloid - metabolism | Mice | Astrocytes - metabolism | Monoamine Oxidase - metabolism | Care and treatment | Monoamine oxidase | Analysis | Physiological aspects | GABA | Diagnosis | Alzheimer's disease | Risk factors | Models | Alzheimers disease | Memory | Rodents
Journal Article
Nature Neuroscience, ISSN 1097-6256, 06/2005, Volume 8, Issue 6, pp. 723 - 729
The cerebellum is critical for motor coordination and cognitive function and is the target of transformation in medulloblastoma, the most common malignant... 
IN-VITRO | WHITE-MATTER | SONIC HEDGEHOG | NEURONS | CNS | CENTRAL-NERVOUS-SYSTEM | GROWTH-FACTOR | PRECURSOR CELLS | NEUROSCIENCES | MOUSE CEREBELLUM | BRAIN-TUMORS | Oligodendroglia - metabolism | Multipotent Stem Cells - metabolism | Glycoproteins - metabolism | Peptides - genetics | Nerve Growth Factors - metabolism | Spheroids, Cellular - cytology | Cerebellum - physiology | Cell Lineage - drug effects | Hedgehog Proteins | Nerve Growth Factors - pharmacology | Stem Cell Transplantation | Neuroglia - cytology | Multipotent Stem Cells - drug effects | Peptides - metabolism | Interneurons - cytology | Spheroids, Cellular - drug effects | Trans-Activators - pharmacology | Oligodendroglia - cytology | Medulloblastoma - genetics | Cell Differentiation - physiology | Basic Helix-Loop-Helix Transcription Factors | Animals, Newborn | Astrocytes - cytology | Biomarkers - metabolism | Glycoproteins - genetics | Cell Separation | Cerebellum - metabolism | Spheroids, Cellular - metabolism | Medulloblastoma - physiopathology | Mice, Transgenic | Medulloblastoma - metabolism | Transcription Factors - genetics | AC133 Antigen | Cell Lineage - physiology | Nerve Tissue Proteins - metabolism | Animals | Interneurons - metabolism | Cell Differentiation - drug effects | Multipotent Stem Cells - cytology | Cerebellum - cytology | Neuroglia - metabolism | Trans-Activators - metabolism | Mice | Antigens, CD | Astrocytes - metabolism | Cerebellum | Cell proliferation | Stem cell research | Neural transmission | Regulation | Research | Structure
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2014, Volume 45, pp. 80 - 97
Highlights • Transgenic IL-10 production in the CNS induces a specific activated microglial phenotype. • IL-10-induced microglia shows increased Iba1, CD11b,... 
Psychiatry | Allergy and Immunology | CD150 | Transgenic mice | Cytokine | Neuroinflammation | LTP | Iba1 | F4/80 | CD11b | GFAP | Microglial plasticity | Gliosis | IL-10R | CD16/32 | TGF-BETA | DEVELOPING RAT-BRAIN | IMMUNOLOGY | EXPRESSION PATTERNS | TUMOR-NECROSIS-FACTOR | MULTIPLE-SCLEROSIS | CENTRAL-NERVOUS-SYSTEM | PRO-INFLAMMATORY CYTOKINES | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | NEUROSCIENCES | TRAUMATIC BRAIN-INJURY | TRANSCRIPTION FACTOR | Glial Fibrillary Acidic Protein - genetics | Microglia - metabolism | CA1 Region, Hippocampal - cytology | Neurons - cytology | RNA, Messenger - metabolism | Cerebral Cortex - cytology | Cerebral Cortex - metabolism | Receptors, IgG - metabolism | Synapses - metabolism | CA3 Region, Hippocampal - metabolism | Antigens, Differentiation - metabolism | CA1 Region, Hippocampal - metabolism | Microfilament Proteins - metabolism | Neurons - metabolism | Astrocytes - cytology | Calcium-Binding Proteins - metabolism | Microglia - cytology | Dendrites - metabolism | CA3 Region, Hippocampal - cytology | Cerebellum - metabolism | Mice, Transgenic | Long-Term Potentiation - genetics | Hippocampus - cytology | Hippocampus - metabolism | Phenotype | Animals | Interleukin-10 - genetics | Cerebellum - cytology | Neural Pathways - metabolism | Mice | CD11b Antigen - metabolism | Astrocytes - metabolism | Animal genetic engineering | Genetic aspects | Interleukins | Neurons
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2011, Volume 13, Issue 1, pp. 40 - 48
Journal Article
Journal of Cell Science, ISSN 0021-9533, 03/2005, Volume 118, Issue 6, pp. 1267 - 1277
Nectins are Ca2+-independent immunoglobulin-like cell-cell adhesion molecules and comprise a family of four members. At the mossy fiber terminals of... 
Axon | Necl-1 | Glia | Myelin | Nectin | Schwann cell | myelin | TUMOR-SUPPRESSOR TSLC1 | ADHERENS JUNCTION | LOCALIZATION | PROTEIN | DEVELOPING MOUSE | AFADIN | glia | CELL BIOLOGY | LUNG-CANCER | EPITHELIAL-CELLS | GENE | axon | nectin | HUMAN HOMOLOG | Immunohistochemistry | Immunoglobulins - physiology | Cadherins - metabolism | Calcium - metabolism | Humans | Calcium - chemistry | Brain - metabolism | Immunoglobulins - metabolism | Tissue Distribution | Cell Adhesion Molecule-1 | Time Factors | Membrane Proteins - physiology | Antigens, Neoplasm - metabolism | Neurons - metabolism | Dimerization | Silver Staining | Schwann Cells - metabolism | Cell Adhesion | Cell Adhesion Molecules - metabolism | Plasmids - metabolism | Blotting, Western | Guanylate Kinases | Mice | Nucleoside-Phosphate Kinase - metabolism | Immunoprecipitation | Exons | Actins - metabolism | Cytoplasm - metabolism | Neoplasm Proteins - metabolism | Microscopy, Immunoelectron | Myelin Sheath - metabolism | Membrane Proteins - metabolism | Protein Structure, Tertiary | Cell Line | Gene Library | Cerebellum - metabolism | Electrophoresis, Polyacrylamide Gel | Axons - metabolism | Cell Communication | Nuclear Proteins - metabolism | Transcription Factors - metabolism | Hippocampus - metabolism | Two-Hybrid System Techniques | Animals | Presynaptic Terminals - metabolism | Protein Binding | Neuroglia - metabolism | Calcium-Calmodulin-Dependent Protein Kinases - metabolism | Microscopy, Fluorescence | Tumor Suppressor Proteins
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 08/2011, Volume 18, Issue 8, pp. 1356 - 1365
We have recently reported that the bioactive lipid sphingosine-1-phosphate (S1P), usually signaling proliferation and anti-apoptosis induces neuronal death... 
neurodegeneration | calpain | apoptosis | cis-4-methylsphingosine | release | Sphingosine-1-phosphate | Ca2+-release | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FUNCTIONAL-CHARACTERIZATION | SPHINGOSINE 1-PHOSPHATE | CELL BIOLOGY | SPHINGOLIPID METABOLISM | CYTOCHROME-C | POSTMITOTIC NEURONS | AMYLOID-BETA | CELL-CYCLE | MOLECULAR-CLONING | Lysophospholipids - metabolism | Neurons - pathology | Calpain - metabolism | Caspase 9 - metabolism | Calcium - metabolism | tau Proteins - metabolism | Calpain - genetics | Nerve Degeneration - chemically induced | Nerve Degeneration - metabolism | Neurons - physiology | Sphingosine - metabolism | Aldehyde-Lyases - genetics | Neurons - drug effects | Mice, Inbred C57BL | Phosphotransferases (Alcohol Group Acceptor) - genetics | Mitochondria - metabolism | Caspase 12 - metabolism | Nerve Degeneration - pathology | Cyclin-Dependent Kinase 5 - metabolism | Mice, Knockout | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Sphingosine - pharmacology | Sphingosine - analogs & derivatives | Animals | Cerebellum - cytology | Aldehyde-Lyases - metabolism | Cell Cycle - physiology | Glycosphingolipids - metabolism | Signal Transduction - physiology | Mice | Apoptosis - physiology | Sphingosine - chemistry | Cerebellum | Phosphorylation | Calcium | Central nervous system | Cysteine proteinase | Caspase | Calpain | Lipids | Anatomy | Metabolism | Cyclin-dependent kinase 5 | Sphingosine 1-phosphate | Neurotoxicity | Glycosphingolipids | Tau protein | Neurodegeneration | Phosphate | Cell cycle | Endoplasmic reticulum | Index Medicus | Original Paper
Journal Article