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Journal Article
PLoS genetics, ISSN 1553-7404, 10/2015, Volume 11, Issue 10, pp. e1005575 - e1005575
Ciliopathies are a group of human disorders caused by dysfunction of primary cilia, ubiquitous microtubule-based organelles involved in transduction of... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Abnormalities, Multiple - metabolism | Abnormalities, Multiple - pathology | Polycystic Kidney Diseases - genetics | Retina - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Mixed Function Oxygenases - metabolism | rab GTP-Binding Proteins - genetics | Cerebellum - abnormalities | Encephalocele - metabolism | Gene Knockdown Techniques | Polycystic Kidney Diseases - pathology | Retinitis Pigmentosa | Kidney Diseases, Cystic - genetics | Nuclear Proteins - genetics | Eye Abnormalities - metabolism | Abnormalities, Multiple - genetics | Encephalocele - genetics | rab GTP-Binding Proteins - metabolism | Polycystic Kidney Diseases - metabolism | Cilia - pathology | Signal Transduction | Cerebellum - metabolism | Nuclear Proteins - metabolism | Zebrafish | Eye Abnormalities - genetics | Cilia - metabolism | Protein Transport - genetics | Cerebellum - pathology | Cilia - genetics | Kidney Diseases, Cystic - pathology | Proteins - genetics | Kidney Diseases, Cystic - metabolism | Animals | Ciliary Motility Disorders - pathology | Proteins - metabolism | Encephalocele - pathology | Eye Abnormalities - pathology | Retina - abnormalities | Ciliary Motility Disorders - metabolism | Mutation | Mixed Function Oxygenases - genetics | Retina - pathology | Ciliary Motility Disorders - genetics | Physiological aspects | Phenotype | Cell organelles | Cilia and ciliary motion | Analysis | Index Medicus | Research & development | Proteins | R&D | Signal transduction | Genotype & phenotype | Photoreceptors | Grants | Localization | Experiments | Defects
Journal Article
Nature neuroscience, ISSN 1097-6256, 08/2011, Volume 14, Issue 8, pp. 1009 - 1016
Permanent damage to white matter tracts, comprising axons and myelinating oligodendrocytes, is an important component of brain injuries of the newborn that... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Humans | Ki-67 Antigen - metabolism | Male | Brain Injuries - metabolism | Wnt Proteins - metabolism | Oligodendroglia - drug effects | Hypoxia-Ischemia, Brain - therapy | Infant, Newborn | Organ Culture Techniques | Disease Models, Animal | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Brain Injuries - therapy | Multiple Sclerosis - therapy | Myelin Proteins - genetics | beta Catenin - metabolism | Heterocyclic Compounds, 3-Ring - therapeutic use | Spinal Cord - physiology | Axin Protein | Mice | Myelin Sheath - ultrastructure | beta-Galactosidase - genetics | Cerebellum - ultrastructure | Myelin Proteins - metabolism | Corpus Callosum - metabolism | Spinal Cord - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Myelin Proteins - therapeutic use | Cerebellum - drug effects | Myelin Sheath - drug effects | Hypoxia-Ischemia, Brain - pathology | Cerebral Cortex - cytology | Cytoskeletal Proteins - deficiency | Dose-Response Relationship, Drug | Oligodendroglia - physiology | Wnt Proteins - genetics | beta-Galactosidase - metabolism | Adult | Cytoskeletal Proteins - metabolism | Female | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Neurons - drug effects | Cell Differentiation - physiology | Hypoxia-Ischemia, Brain - metabolism | Microscopy, Electron, Transmission | Myelin Sheath - pathology | Gene Expression Regulation - genetics | Cerebellum - metabolism | Cells, Cultured | Gene Expression Regulation - physiology | Corpus Callosum - drug effects | Nerve Tissue Proteins - genetics | beta Catenin - genetics | Multiple Sclerosis - complications | Nerve Tissue Proteins - metabolism | Animals | Cell Differentiation - drug effects | Multiple Sclerosis - pathology | Stem Cells - drug effects | Brain Injuries - etiology | Lysophosphatidylcholines - toxicity | Postmortem Changes | Infants (Newborn) | Brain | Care and treatment | Physiological aspects | Research | Binding proteins | Health aspects | Injuries | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e30151 - e30151
Foxp2(R552H) knock-in (KI) mouse pups with a mutation related to human speech-language disorders exhibit poor development of cerebellar Purkinje cells and... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Immunohistochemistry | Cell Adhesion Molecules - genetics | Humans | Immunoblotting | Male | Immunoglobulins - genetics | Cerebellum - abnormalities | Immunoglobulins - metabolism | Synapses - metabolism | Cell Adhesion Molecule-1 | Forkhead Transcription Factors - metabolism | Female | Child | Repressor Proteins - metabolism | Dendrites - metabolism | Purkinje Cells - metabolism | Vesicular Glutamate Transport Protein 1 - metabolism | Cerebellum - metabolism | Mice, Inbred C57BL | Speech Disorders - genetics | Repressor Proteins - genetics | Vocalization, Animal | Genotype | Mice, Transgenic | Forkhead Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion Molecules - metabolism | Mice, Knockout | Ultrasonics | Animals | Child Development Disorders, Pervasive - genetics | Mice | Mutation | Dendrites - genetics | Autism | Brain | Genetic aspects | RNA | Cerebellum | Pediatrics | Vocalization behavior | Nervous system | Fibers | Proteins | Foxp2 protein | Language | Purkinje cells | Rodents | Mother-offspring interactions | Dendrites | Communication | Immunoglobulins | Parallel fibers | Gene expression | Offspring | Brain research | Speech | Bilingualism | Immunoreactivity | Synapses | Index Medicus
Journal Article
Cellular signalling, ISSN 0898-6568, 01/2018, Volume 42, pp. 281 - 291
Deficits in brain function that are associated with aging and age-related diseases benefit very little from currently available therapies, suggesting a better... 
Cerebellum | PDE8 | PDE7 | Mild cognitive impairment | PDE1 | Memory | Cortex | PDE3 | cGMP | Parkinson's Disease | cAMP | Huntington's disease | Striatum | Age-related cognitive decline | Cyclic nucleotides | PDE11 | Aging | Alzheimer's disease | Hippocampus | Phosphodiesterase | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Humans | Natriuretic Peptide, Brain - metabolism | Heterotrimeric GTP-Binding Proteins - metabolism | Cyclic GMP-Dependent Protein Kinases - metabolism | Heterotrimeric GTP-Binding Proteins - genetics | Cyclic GMP-Dependent Protein Kinases - genetics | Brain - metabolism | Cyclic AMP-Dependent Protein Kinases - genetics | Aging - genetics | Guanine Nucleotide Exchange Factors - metabolism | Atrial Natriuretic Factor - genetics | Cyclic AMP - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Guanine Nucleotide Exchange Factors - genetics | Natriuretic Peptide, Brain - genetics | Neurodegenerative Diseases - pathology | Atrial Natriuretic Factor - metabolism | Signal Transduction | Gene Expression Regulation | Neurodegenerative Diseases - genetics | Adenylyl Cyclases - metabolism | Neurodegenerative Diseases - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Cyclic GMP - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Brain - pathology | Adenylyl Cyclases - genetics | Aging - metabolism | Brain | Medical colleges | Neurosciences | Analysis | Cyclic adenylic acid | G proteins | Adenylic acid | Protein kinases | Protein binding | Natriuretic peptides | Index Medicus | Huntington’s Disease | memory | cerebellum | striatum | cyclic nucleotides | Parkinson’s Disease | age-related cognitive decline | cyclase | cortex | PDE5 | Alzheimer’s disease | hippocampus | PDE2 | PDE4 | natriuretic peptide | PDE9 | PDE10 | mild cognitive impairment | aging | phosphodiesterase
Journal Article