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1. Full Text Possible Origins and Distribution of Immunoreactive Nitric Oxide Synthase-Containing Nerve Fibers in Cerebral Arteries
by Nozaki, Kazuhiko and Moskowitz, Michael A and Maynard, Kenneth I and Koketsu, Naoki and Dawson, Ted M and Bredt, David S and Snyder, Solomon H
Journal of cerebral blood flow and metabolism, ISSN 0271-678X, 01/1993, Volume 13, Issue 1, pp. 70 - 79
Parasympathetic | Nitric oxide synthase | Sphenopalatine ganglion | Cerebral artery | Nitric oxide | Vasoactive intestinal polypeptide | Endocrinology & Metabolism | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Hematology | Science & Technology | Fundamental and applied biological sciences. Psychology | Vertebrates: nervous system and sense organs | Biological and medical sciences | Cerebral circulation. Blood-brain barrier. Choroid plexus. Cerebrospinal fluid. Circumventricular organ. Meninges | Circle of Willis - innervation | Ganglia, Parasympathetic - enzymology | Immunohistochemistry | Calcitonin Gene-Related Peptide - analysis | Vasoactive Intestinal Peptide - analysis | Rats | Male | Nerve Fibers - enzymology | Rats, Sprague-Dawley | Ganglia, Parasympathetic - cytology | Animals | Cerebral Arteries - chemistry | Amino Acid Oxidoreductases - analysis | Cerebral Arteries - enzymology | Cerebral Arteries - innervation | Neurons - enzymology | Nitric Oxide Synthase | Denervation | Index Medicus
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2. Full Text Pharmacological Inhibition of Phospholipase D Protects Mice From Occlusive Thrombus Formation and Ischemic Stroke—Brief Report
by Stegner, David and Thielmann, Ina and Kraft, Peter and Frohman, Michael A and Stoll, Guido and Nieswandt, Bernhard
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 09/2013, Volume 33, Issue 9, pp. 2212 - 2217
phospholipase D | thrombosis | 5-fluoro-2-indolyl des-chlorohalopemide | cerebral stroke | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Carotid Artery Diseases - prevention & control | Infarction, Middle Cerebral Artery - physiopathology | Infarction, Middle Cerebral Artery - prevention & control | Carotid Artery Diseases - physiopathology | Blood Platelets - enzymology | Recovery of Function | Dose-Response Relationship, Drug | Time Factors | Infarction, Middle Cerebral Artery - genetics | Indoles - pharmacology | Phospholipase D - genetics | Thrombosis - blood | Thrombosis - prevention & control | Blood Platelets - drug effects | Domperidone - analogs & derivatives | Integrins - blood | Disease Models, Animal | Hemostasis - drug effects | Thrombosis - physiopathology | Phospholipase D - deficiency | Mice, Inbred C57BL | Thrombosis - enzymology | Enzyme Inhibitors - pharmacology | Phospholipase D - antagonists & inhibitors | Infarction, Middle Cerebral Artery - blood | Mice, Knockout | Fibrinolytic Agents - pharmacology | Animals | Carotid Artery Diseases - enzymology | Carotid Artery Diseases - blood | Carotid Artery Diseases - genetics | Domperidone - pharmacology | Thrombosis - genetics | Mice | Infarction, Middle Cerebral Artery - enzymology | Index Medicus
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3. Full Text NADPH-oxidase activity is elevated in penumbral and non-ischemic cerebral arteries following stroke
by Miller, Alyson A and Dusting, Gregory J and Roulston, Carli L and Sobey, Christopher G
Brain research, ISSN 0006-8993, 2006, Volume 1111, Issue 1, pp. 111 - 116
Oxidative stress | Cerebral vasculature | Stroke | Cerebral ischemia | NADPH-oxidase | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurology | Biological and medical sciences | Vascular diseases and vascular malformations of the nervous system | Medical sciences | Infarction, Middle Cerebral Artery - physiopathology | Reactive Oxygen Species - metabolism | Cerebral Cortex - blood supply | Cerebrovascular Circulation - physiology | Rats, Wistar | NADPH Oxidases - metabolism | Male | Stroke - physiopathology | Cerebral Cortex - physiopathology | Brain Ischemia - enzymology | Basilar Artery - physiopathology | Time Factors | Up-Regulation - physiology | Middle Cerebral Artery - physiopathology | Superoxides - metabolism | Functional Laterality - physiology | Disease Models, Animal | Basilar Artery - enzymology | Cerebral Cortex - enzymology | Reperfusion Injury - enzymology | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Cerebral Arteries - physiopathology | Brain Ischemia - physiopathology | Onium Compounds - pharmacology | Stroke - enzymology | Middle Cerebral Artery - enzymology | Animals | Cerebral Arteries - enzymology | Reperfusion Injury - physiopathology | Infarction, Middle Cerebral Artery - enzymology | Stroke (Disease) | Enzymes | Oxides | Hydrogen peroxide | Analysis | Arteries | Index Medicus
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4. Full Text Catalpol stimulates VEGF production via the JAK2/STAT3 pathway to improve angiogenesis in rats’ stroke model
by Dong, Wan and Xian, Yang and Yuan, Wang and Huifeng, Zhu and Tao, Wang and Zhiqiang, Liu and Shan, Feng and Ya, Fu and Hongli, Wang and Jinghuan, Wang and Lei, Qin and Li, Zou and Hongyi, Qi
Journal of ethnopharmacology, ISSN 0378-8741, 09/2016, Volume 191, pp. 169 - 179
Angiogenesis | JAK2/STAT3 | Catalpol | VEGF | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Chemistry, Medicinal | Integrative & Complementary Medicine | Plant Sciences | Science & Technology | Infarction, Middle Cerebral Artery - physiopathology | Iridoid Glucosides - pharmacology | Neovascularization, Physiologic - drug effects | Up-Regulation | Phosphorylation | Transcriptional Activation | Brain - enzymology | Male | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Cerebral Arteries - drug effects | Neuroprotective Agents - pharmacology | Time Factors | Janus Kinase 2 - metabolism | Infarction, Middle Cerebral Artery - drug therapy | Angiogenesis Inducing Agents - pharmacology | Erythropoietin - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Promoter Regions, Genetic | Cerebrovascular Circulation - drug effects | Brain - physiopathology | RNA, Messenger - genetics | Cerebral Arteries - physiopathology | Infarction, Middle Cerebral Artery - pathology | Rats, Sprague-Dawley | Brain - drug effects | Animals | Signal Transduction - drug effects | Cerebral Arteries - enzymology | Cerebral Arteries - pathology | Brain - pathology | Protein Binding | Infarction, Middle Cerebral Artery - enzymology | Receptors, Erythropoietin - metabolism | Stroke (Disease) | Drugstores | Nervous system diseases | Medicine, Chinese | Endothelial growth factors | Pharmacy | Brain | Yuan (China) | Injuries | Index Medicus
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5. Full Text Which NADPH Oxidase Isoform Is Relevant for Ischemic Stroke? The Case for Nox 2
by Kahles, Timo and Brandes, Ralf P
Antioxidants & redox signaling, ISSN 1523-0864, 04/2013, Volume 18, Issue 12, pp. 14 - 1417
Forum Review Articles | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Reactive Oxygen Species | Oxidative Stress | Reperfusion Injury - enzymology | Humans | Brain - enzymology | NADPH Oxidase 2 | Stroke - enzymology | Brain Ischemia - enzymology | Brain - blood supply | Membrane Glycoproteins - physiology | Animals | Cerebral Arteries - enzymology | Neurons - enzymology | Cerebral Arteries - pathology | Brain - pathology | NADPH Oxidases - physiology | Isoenzymes - physiology | Stroke (Disease) | Physiological aspects | Analysis | NADP (Coenzyme) | Index Medicus | Forum Review
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6. Full Text Carcinoembryonic Antigen–Related Cell Adhesion Molecule 1 Inhibits MMP-9–Mediated Blood–Brain–Barrier Breakdown in a Mouse Model for Ischemic Stroke
by Ludewig, Peter and Sedlacik, Jan and Gelderblom, Mathias and Bernreuther, Christian and Korkusuz, Yücel and Wagener, Christoph and Gerloff, Christian and Fiehler, Jens and Magnus, Tim and Horst, Andrea Kristina
Circulation research, ISSN 0009-7330, 09/2013, Volume 113, Issue 8, pp. 1013 - 1022
Vascular system injuries | Animal model of human disease remodeling | Inflammation | Ischemic stroke | Neutrophils | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Infarction, Middle Cerebral Artery - physiopathology | Infarction, Middle Cerebral Artery - prevention & control | Capillary Permeability | Humans | Carcinoembryonic Antigen - genetics | Male | Infarction, Middle Cerebral Artery - immunology | Motor Activity | Neutrophil Infiltration | Sulfones - pharmacology | Antigens, CD - metabolism | Brain Edema - enzymology | Carcinoembryonic Antigen - metabolism | Atrophy | Behavior, Animal | Matrix Metalloproteinase 9 - metabolism | Flow Cytometry | Time Factors | Heterocyclic Compounds, 1-Ring - pharmacology | Infarction, Middle Cerebral Artery - genetics | Inflammation Mediators - metabolism | Brain Edema - immunology | Disease Models, Animal | Neutrophils - pathology | Blood-Brain Barrier - immunology | Neutrophil Activation | Neutrophils - enzymology | Brain Edema - pathology | Mice, Inbred C57BL | Neutrophils - drug effects | Neutrophils - immunology | Infarction, Middle Cerebral Artery - pathology | Blood-Brain Barrier - drug effects | Cell Adhesion Molecules - metabolism | Mice, Knockout | Blood-Brain Barrier - pathology | Magnetic Resonance Imaging | Matrix Metalloproteinase Inhibitors - pharmacology | Animals | Blood-Brain Barrier - enzymology | Neurologic Examination | Mice | Infarction, Middle Cerebral Artery - enzymology | Microscopy, Fluorescence | Index Medicus
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7. Full Text Marked Induction of Calcium-Independent Nitric Oxide Synthase Activity After Focal Cerebral Ischemia
by Iadecola, Costantino and Xu, Xiaohong and Zhang, Fangyi and El-Fakahany, Esam E and Ross, M. Elizabeth
Journal of cerebral blood flow and metabolism, ISSN 0271-678X, 01/1995, Volume 15, Issue 1, pp. 52 - 59
Constitutive nitric oxide synthase (cNOS) | Spontaneously hypertensive rats | Reverse-transcription polymerase chain reaction | Inducible NOS | Lipopolysaccharide antigen | Middle cerebral artery | Endocrinology & Metabolism | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Hematology | Science & Technology | Neurology | Biological and medical sciences | Vascular diseases and vascular malformations of the nervous system | Medical sciences | Rats, Inbred SHR | Neurons - pathology | Cerebral Cortex - enzymology | Calcium - pharmacology | Biopterin - pharmacology | Glial Fibrillary Acidic Protein - analysis | Astrocytes - pathology | Rats | Male | Ischemic Attack, Transient - enzymology | RNA, Messenger - metabolism | Biopterin - analogs & derivatives | Lung - enzymology | Amino Acid Oxidoreductases - genetics | Animals | Spleen - enzymology | NADPH Dehydrogenase - analysis | Neurons - enzymology | Lipopolysaccharides - pharmacology | Astrocytes - chemistry | Nitric Oxide Synthase | Amino Acid Oxidoreductases - biosynthesis | Kinetics | Ischemic Attack, Transient - pathology | Index Medicus
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8. Full Text NEU1 and NEU3 sialidase activity expressed in human lung microvascular endothelia: NEU1 restrains endothelial cell migration, whereas NEU3 does not
by Cross, Alan S and Hyun, Sang Won and Miranda-Ribera, Alba and Feng, Chiguang and Liu, Anguo and Nguyen, Chinh and Zhang, Lei and Luzina, Irina G and Atamas, Sergei P and Twaddell, William S and Guang, Wei and Lillehoj, Erik P and Puché, Adam C and Huang, Wei and Wang, Lai-Xi and Passaniti, Antonino and Goldblum, Simeon E
The Journal of biological chemistry, ISSN 0021-9258, 05/2012, Volume 287, Issue 19, pp. 15966 - 15980
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Humans | Kidney - enzymology | Neuraminidase - genetics | Substrate Specificity | Immunoblotting | Cell Nucleus - enzymology | Lung - enzymology | Cytosol - enzymology | Hymecromone - analogs & derivatives | Flow Cytometry | RNA Interference | Carotid Arteries - enzymology | Hymecromone - pharmacology | Aorta - enzymology | Cell Line | Endothelial Cells - metabolism | Neuraminidase - metabolism | Reverse Transcriptase Polymerase Chain Reaction | N-Acetylneuraminic Acid - metabolism | Gene Expression Regulation, Enzymologic | Cell Membrane - enzymology | Microscopy, Confocal | Cerebral Arteries - enzymology | N-Acetylneuraminic Acid - analogs & derivatives | Neuraminidase - antagonists & inhibitors | Endothelial Cells - enzymology | Cell Movement | Index Medicus | Sialic Acid | Glycobiology | Signal Transduction | Endothelial Dysfunction | Lung | Vascular Biology | Migration | Neuraminidase | Glycobiology and Extracellular Matrices | Wound Healing | Sialidase
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