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Free Radical Biology and Medicine, ISSN 0891-5849, 12/2013, Volume 65, pp. 1012 - 1022
Disruption of the blood–brain barrier (BBB) and cerebral edema are the major pathogenic mechanisms leading to neurological dysfunction and death after ischemic... 
Stroke | Nrf2 | Sulforaphane | Heme oxygenase | Gliovascular complex | Preconditioning | Blood–brain barrier | Blood-brain barrier | OXIDATIVE STRESS | ISCHEMIC BRAIN | ARTERY OCCLUSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | Hems oxygenase | HEME OXYGENASE-1 GENE | BEHAVIORAL-ASSESSMENT | PERMEABILITY TRANSITION | TRANSCRIPTION FACTOR NRF2 | ENDOCRINOLOGY & METABOLISM | RAT-BRAIN | NEUROVASCULAR PROTECTION | MOLECULAR-MECHANISMS | Neuroprotective Agents - therapeutic use | Up-Regulation | Heme Oxygenase-1 - metabolism | Oxidative Stress | Male | Infarction, Middle Cerebral Artery - metabolism | Cerebral Arteries - drug effects | Heme Oxygenase-1 - genetics | Psychomotor Disorders - metabolism | Isothiocyanates - therapeutic use | Neuroprotective Agents - pharmacology | NF-E2-Related Factor 2 - genetics | Infarction, Middle Cerebral Artery - drug therapy | Membrane Proteins - metabolism | Microvessels - enzymology | Psychomotor Disorders - prevention & control | Reperfusion Injury - metabolism | Isothiocyanates - pharmacology | Gene Expression | Signal Transduction | Membrane Proteins - genetics | Blood-Brain Barrier - drug effects | Rats, Sprague-Dawley | Blood-Brain Barrier - metabolism | Blood-Brain Barrier - pathology | Animals | Reperfusion Injury - prevention & control | Cerebral Arteries - enzymology | NF-E2-Related Factor 2 - metabolism | Stroke (Disease) | Antioxidants | Analysis | Antigens | Brain | Heme | Injuries | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 04/2007, Volume 101, Issue 2, pp. 566 - 576
The blood–brain barrier (BBB) formed by brain microvascular endothelial cells (BMVEC) regulates the passage of molecules and leukocytes in and out of the... 
blood–brain barrier | protein tyrosine kinase | matrix metalloproteinases | protein tyrosine phosphatase | brain endothelial cell | oxidative stress | Protein tyrosine kinase | Oxidative stress | Matrix metalloproteinases | Blood-brain barrier | Brain endothelial cell | Protein tyrosine phosphatase | blood-brain barrier | HUMAN STROKE | FACTOR RECEPTOR TRANSACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | VASCULAR-PERMEABILITY | TIGHT JUNCTION | NEUROSCIENCES | SIGNAL-TRANSDUCTION | MULTIPLE-SCLEROSIS | ENDOTHELIAL-CELLS | RHO GTPASES | EXTRACELLULAR-MATRIX | Reactive Oxygen Species - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Oxidative Stress - physiology | Reactive Oxygen Species - pharmacology | Blood-Brain Barrier - physiopathology | Basement Membrane - pathology | Chemotaxis, Leukocyte - physiology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Encephalitis - physiopathology | Chemotaxis, Leukocyte - drug effects | Up-Regulation - physiology | Basement Membrane - enzymology | Tight Junctions - metabolism | Encephalitis - enzymology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Cerebral Arteries - physiopathology | Basement Membrane - metabolism | Blood-Brain Barrier - enzymology | Neurodegenerative Diseases - physiopathology | Cerebral Arteries - enzymology | Endothelial Cells - cytology | Matrix Metalloproteinases - metabolism | Endothelial Cells - enzymology | Enzyme Activation - physiology | Neurodegenerative Diseases - enzymology | Neurosciences | Biochemistry | Oxidation | Brain research | Kinases | Neurological disorders | Index Medicus
Journal Article
Journal of Ethnopharmacology, ISSN 0378-8741, 09/2016, Volume 191, pp. 169 - 179
Catalpol is the main active component of the radix from Libosch, which has pleiotropic protective effects in neurodegenerative diseases, ischemic stroke,... 
Angiogenesis | JAK2/STAT3 | Catalpol | VEGF | TYROSINE-PHOSPHATASE SHP-1 | CHEMISTRY, MEDICINAL | ARTERY OCCLUSION | FOCAL CEREBRAL-ISCHEMIA | REPERFUSION INJURY | PLANT SCIENCES | STAT3 ACTIVATION | PLASMINOGEN-ACTIVATOR | SIGNALING PATHWAY | INTEGRATIVE & COMPLEMENTARY MEDICINE | ERYTHROPOIETIN PROMOTES | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | ENDOTHELIAL GROWTH-FACTOR | Infarction, Middle Cerebral Artery - physiopathology | Iridoid Glucosides - pharmacology | Neovascularization, Physiologic - drug effects | Up-Regulation | Phosphorylation | Transcriptional Activation | Brain - enzymology | Male | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Cerebral Arteries - drug effects | Neuroprotective Agents - pharmacology | Time Factors | Janus Kinase 2 - metabolism | Infarction, Middle Cerebral Artery - drug therapy | Angiogenesis Inducing Agents - pharmacology | Erythropoietin - metabolism | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Promoter Regions, Genetic | Cerebrovascular Circulation - drug effects | Brain - physiopathology | RNA, Messenger - genetics | Cerebral Arteries - physiopathology | Infarction, Middle Cerebral Artery - pathology | Rats, Sprague-Dawley | Brain - drug effects | Animals | Signal Transduction - drug effects | Cerebral Arteries - enzymology | Cerebral Arteries - pathology | Brain - pathology | Protein Binding | Infarction, Middle Cerebral Artery - enzymology | Receptors, Erythropoietin - metabolism | Stroke (Disease) | Drugstores | Nervous system diseases | Medicine, Chinese | Endothelial growth factors | Pharmacy | Brain | Yuan (China) | Injuries | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2013, Volume 33, Issue 9, pp. 2212 - 2217
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2018, Volume 38, Issue 3, pp. 610 - 621
OBJECTIVE—Cigarette smoke exposure (CSE) is a risk factor for cerebral aneurysm (CA) formation, but the molecular mechanisms are unclear. Although CSE is known... 
Oxygen | Smoke | Subarachnoid hemorrhage | Aneurysm | Atherosclerosis | Rupture | atherosclerosis | aneurysm | RISK-FACTORS | smoke | NATURAL-HISTORY | rupture | subarachnoid hemorrhage | IN-VIVO | UNRUPTURED INTRACRANIAL ANEURYSMS | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CORONARY-HEART-DISEASE | HEMATOLOGY | oxygen | ISCHEMIC-STROKE | MOLECULAR-MECHANISMS | Vascular Remodeling | Oxidative Stress | Myocytes, Smooth Muscle - pathology | NADPH Oxidases - metabolism | Aneurysm, Ruptured - prevention & control | Male | Intracranial Aneurysm - prevention & control | Acetophenones - pharmacology | NADPH Oxidases - genetics | Intracranial Aneurysm - enzymology | Dilatation, Pathologic | Disease Models, Animal | Cigarette Smoking - adverse effects | Signal Transduction | Intracranial Aneurysm - genetics | Myocytes, Smooth Muscle - enzymology | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | NADPH Oxidase 1 - genetics | Antioxidants - pharmacology | Rats, Sprague-Dawley | Mice, Knockout | Muscle, Smooth, Vascular - pathology | Phenotype | Aneurysm, Ruptured - enzymology | Animals | Cerebral Arteries - enzymology | Cerebral Arteries - pathology | NADPH Oxidase 1 - metabolism | Aneurysm, Ruptured - pathology | Intracranial Aneurysm - pathology | Aneurysm, Ruptured - genetics | Muscle, Smooth, Vascular - enzymology | Index Medicus | Hypertension | stress | cigarette | Vascular Biology | cerebrovascular | Smooth muscle | Inflammation | phenotypic modulation | Cerebral Aneurysms | stroke | reactive | species | Ischemic Stroke | oxidative | Cerebrovascular Disease | Intracranial Hemorrhage
Journal Article
Hypertension, ISSN 0194-911X, 03/2013, Volume 61, Issue 3, pp. 647 - 654
Sex steroid hormones estradiol and progesterone play an important role in vascular adaptations during pregnancy. However, little is known about the role of... 
blood pressure | eNOS phosphorylation | prostacyclin | vasodilation | mesenteric arteries | hypertension | endothelium-derived hyperpolarizing factor | NITRIC-OXIDE SYNTHESIS | ANGIOTENSIN-II | MESENTERIC-ARTERY | UTERINE ARTERY | BLOOD-PRESSURE | PERIPHERAL VASCULAR DISEASE | ANDROGEN LEVELS | CEREBRAL-ARTERIES | GROWTH RESTRICTION | RENAL INJURY | VASODILATOR PRODUCTION | Mesenteric Arteries - physiology | Phosphorylation | Adaptation, Physiological - drug effects | Testosterone - physiology | Nitric Oxide Synthase Type III - analysis | Endothelium, Vascular - drug effects | Mesenteric Arteries - drug effects | Nitroprusside - pharmacology | Endothelium, Vascular - enzymology | Testosterone - pharmacology | Threonine - metabolism | Hypertension, Pregnancy-Induced - drug therapy | Endothelium, Vascular - physiology | Arterial Pressure | Epoprostenol - pharmacology | Female | Nitric Oxide Synthase Type III - metabolism | Testosterone - blood | Hypertension, Pregnancy-Induced - enzymology | Vasodilator Agents - pharmacology | Acetylcholine - pharmacology | Adaptation, Physiological - physiology | Rats | Hypertension, Pregnancy-Induced - physiopathology | Nitric Oxide - physiology | Rats, Sprague-Dawley | Serine - metabolism | Nitric Oxide Synthase Type III - antagonists & inhibitors | Pregnancy | Animals | Mesenteric Arteries - enzymology | Biological Factors - pharmacology | Index Medicus
Journal Article
Journal Article
Brain Research, ISSN 0006-8993, 2006, Volume 1111, Issue 1, pp. 111 - 116
Reactive oxygen species play a role in neuronal damage following cerebral ischemia–reperfusion. We tested whether activity of the superoxide-generating enzyme,... 
Oxidative stress | Cerebral vasculature | Stroke | Cerebral ischemia | NADPH-oxidase | SUPEROXIDE | MICROGLIAL CELLS | BLOOD-FLOW | stroke | NEUROSCIENCES | cerebral vasculature | VASCULAR SMOOTH-MUSCLE | SENSORIMOTOR DEFICITS | NEURONAL DAMAGE | CONSCIOUS RATS | NEUROPROTECTIVE AGENT | cerebral ischemia | IN-VIVO | ISCHEMIC-STROKE | oxidative stress | Infarction, Middle Cerebral Artery - physiopathology | Reactive Oxygen Species - metabolism | Cerebral Cortex - blood supply | Cerebrovascular Circulation - physiology | Rats, Wistar | NADPH Oxidases - metabolism | Male | Stroke - physiopathology | Cerebral Cortex - physiopathology | Brain Ischemia - enzymology | Basilar Artery - physiopathology | Time Factors | Up-Regulation - physiology | Middle Cerebral Artery - physiopathology | Superoxides - metabolism | Functional Laterality - physiology | Disease Models, Animal | Basilar Artery - enzymology | Cerebral Cortex - enzymology | Reperfusion Injury - enzymology | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Cerebral Arteries - physiopathology | Brain Ischemia - physiopathology | Onium Compounds - pharmacology | Stroke - enzymology | Middle Cerebral Artery - enzymology | Animals | Cerebral Arteries - enzymology | Reperfusion Injury - physiopathology | Infarction, Middle Cerebral Artery - enzymology | Stroke (Disease) | Enzymes | Oxides | Hydrogen peroxide | Analysis | Arteries | Index Medicus
Journal Article
Brain Research, ISSN 0006-8993, 2009, Volume 1292, pp. 123 - 135
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2008, Volume 28, Issue 48, pp. 12788 - 12797
Activated protein C (APC) is a serine protease with anticoagulant and direct cytoprotective activities. Early postischemic APC application activates the... 
Angiogenesis | Neuroprotection | Serine protease | Activated protein C | Neurogenesis | Transient cerebral ischemia | FUNCTIONAL RECOVERY | ARTERY OCCLUSION | angiogenesis | FOCAL CEREBRAL-ISCHEMIA | CELL-PROLIFERATION | SUBVENTRICULAR ZONE | serine protease | NEUROSCIENCES | NEURONAL DAMAGE | transient cerebral ischemia | TISSUE-PLASMINOGEN-ACTIVATOR | neuroprotection | IN-VIVO | neurogenesis | EMBOLIC STROKE | activated protein C | SEVERE SEPSIS | Neovascularization, Physiologic - drug effects | Recovery of Function - drug effects | Nerve Regeneration - physiology | Blood-Brain Barrier - physiopathology | Brain - enzymology | Male | Cell Movement - physiology | Protein C - pharmacology | Brain - metabolism | Brain Ischemia - enzymology | Recovery of Function - physiology | Neurogenesis - drug effects | Tissue Plasminogen Activator - toxicity | Brain Infarction - drug therapy | Disease Models, Animal | Brain Infarction - enzymology | Reperfusion Injury - enzymology | Brain - physiopathology | Mice, Inbred C57BL | Cerebral Arteries - physiopathology | Treatment Outcome | Brain Ischemia - physiopathology | Brain Infarction - physiopathology | Blood-Brain Barrier - drug effects | Receptor, PAR-1 - genetics | Blood-Brain Barrier - metabolism | Mice, Knockout | Cell Movement - drug effects | Neovascularization, Physiologic - physiology | Animals | Cerebral Arteries - enzymology | Cerebral Arteries - metabolism | Neurogenesis - physiology | Brain Ischemia - drug therapy | Reperfusion Injury - physiopathology | Cell Proliferation - drug effects | Mice | Protein C - metabolism | Enzyme Activation - physiology | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2012, Volume 287, Issue 19, pp. 15966 - 15980
Journal Article