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Book
Journal of the American College of Cardiology, ISSN 0735-1097, 2012, Volume 60, Issue 5, pp. 397 - 403
Objectives The purpose of this study was describe the cardiovascular phenotype of the aneurysms-osteoarthritis syndrome (AOS) and to provide clinical... 
Cardiovascular | Internal Medicine | aneurysm | aorta | genetics | SMAD3 | cerebrovascular disorders | FIBROSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | CARDIOMYOPATHY | HEART-DISEASE | STIFFNESS | BICUSPID AORTIC-VALVE | MUTATIONS | MARFANS-SYNDROME | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Aortic Aneurysm, Thoracic - diagnostic imaging | Humans | Middle Aged | Image Interpretation, Computer-Assisted | Male | Cardiovascular Diseases - genetics | Cause of Death | Osteoarthritis - mortality | Young Adult | Smad3 Protein - genetics | Cardiovascular Diseases - diagnostic imaging | Cardiovascular Diseases - mortality | Adult | Aortic Aneurysm, Thoracic - mortality | Female | Genomic Structural Variation - genetics | Aneurysm - diagnostic imaging | Imaging, Three-Dimensional | Peptide Fragments - genetics | Natriuretic Peptide, Brain - genetics | Vascular Stiffness - genetics | Osteoarthritis - diagnostic imaging | Osteoarthritis - genetics | Syndrome | Aneurysm - genetics | Cerebrovascular Disorders - mortality | Pregnancy | Phenotype | Adolescent | Cerebrovascular Disorders - diagnostic imaging | Chromosome Aberrations | Survival Analysis | Aneurysm, Dissecting - diagnostic imaging | Aged | Cerebrovascular Disorders - genetics | Aneurysm - mortality | Aortography | Genes, Dominant - genetics | Aneurysm, Dissecting - mortality | Cohort Studies | Medical colleges | Peptides | Atrial fibrillation | Aneurysms | Genetic aspects | Universities and colleges | Cardiology | Transforming growth factors | Marfan syndrome | Natriuretic peptides | Osteoarthritis | Medical genetics | Studies | Genotype & phenotype | Sinuses | Medical imaging | Mortality | Tomography | Arthritis | Dissection | Patients | Age | Abdomen | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2010, Volume 107, Issue 51, pp. 22290 - 22295
Modern functional imaging techniques of the brain measure local hemodynamic responses evoked by neuronal activity. Capillary pericytes recently were suggested... 
Brain | Arterioles | Dilatation | Imaging | Blood vessels | Flow velocity | Hyperemia | Capillaries | Endothelial cells | Blood flow | Electrophysiology | Cortical spreading depolarization | Cerebral blood flow | Neurovascular coupling | RAT | MULTIDISCIPLINARY SCIENCES | neurovascular coupling | ISCHEMIA | MICROVESSELS | RESPONSES | electrophysiology | CEREBELLAR CORTEX | FLUCTUATIONS | NITRIC-OXIDE | GABA | cerebral blood flow | REFLOW | CEREBRAL-BLOOD-FLOW | cortical spreading depolarization | Hyperemia - metabolism | Capillaries - pathology | Male | Cerebrovascular Disorders - metabolism | Brain - blood supply | Hyperemia - pathology | Vasodilation - genetics | Pericytes - pathology | Thromboxane A2 - antagonists & inhibitors | Female | Microscopy, Fluorescence, Multiphoton | Capillaries - metabolism | GABA-A Receptor Antagonists - pharmacology | Vasoconstrictor Agents - pharmacology | Cerebrovascular Disorders - physiopathology | Thromboxane A2 - genetics | Brain - physiopathology | Pericytes - metabolism | Mice, Transgenic | Bicuculline - pharmacology | Blood Flow Velocity - genetics | Thromboxane A2 - metabolism | Arterioles - pathology | Animals | Arterioles - metabolism | Hyperemia - physiopathology | Receptors, GABA-A - genetics | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Brain - pathology | Cerebrovascular Disorders - genetics | Mice | Receptors, GABA-A - metabolism | Vasodilation - drug effects | Cerebrovascular Disorders - pathology | Hyperemia - genetics | Blood Flow Velocity - drug effects | Measurement | Connective tissue cells | Research | Properties | Arteries | Methods | Convulsions & seizures | Neurons | Rodents | Erythrocytes | Index Medicus | Neuroimaging | Brain slice preparation | Thromboxane A2 | pericytes | Contractility | Cortex | hyperemia | Data processing | Prostaglandin F2 alpha | gamma -Aminobutyric acid A receptors | Microscopy | Hemodynamics | Bicuculline | Seizures | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 1, pp. 74 - 79
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2011, Volume 108, Issue 12, pp. 5063 - 5068
Increasing evidence indicates that cerebrovascular dysfunction plays a pathogenic role in Alzheimer's dementia (AD). Amyloid-β (Aβ), a peptide central to the... 
Oxidases | Oxidative stress | Brain | Reactive oxygen species | Blocking antibodies | Scavenger receptors | Mice | Amyloids | Alzheimers disease | Endothelial cells | Functional hyperemia | Receptor for advanced glycation end-products | Alzheimer's disease | Nox2 | NADPH OXIDASE | MICROGLIAL ACTIVATION | ALZHEIMERS-DISEASE | MEMBRANE | MULTIDISCIPLINARY SCIENCES | MONOCYTES | functional hyperemia | MECHANISMS | BLOOD-BRAIN-BARRIER | MICE | RADICALS | PRECURSOR PROTEIN | receptor for advanced glycation end-products | CD36 Antigens - genetics | Oxidative Stress | Humans | NADPH Oxidases - metabolism | Male | Cerebrovascular Disorders - metabolism | Cerebrovascular Circulation - genetics | CD36 Antigens - metabolism | Amyloid beta-Peptides - genetics | Amyloid beta-Peptides - metabolism | NADPH Oxidases - genetics | Female | Peptide Fragments - genetics | Cerebrovascular Disorders - physiopathology | Alzheimer Disease - physiopathology | Peptide Fragments - metabolism | Cerebrovascular Circulation - drug effects | Somatosensory Cortex - physiopathology | Antibodies, Neutralizing - pharmacology | Mice, Transgenic | Animals | Alzheimer Disease - metabolism | Protein Binding | Cerebrovascular Disorders - genetics | Mutation | Alzheimer Disease - genetics | Somatosensory Cortex - metabolism | Enzymes | Neurodegenerative diseases | Vasodilators | Transgenic mice | beta -Amyloid | Cortex (somatosensory) | CD36 antigen | Amyloid precursor protein | Arteries | NAD(P)H oxidase | scavenger receptors | Cerebral blood flow | Dementia disorders | Membrane glycoproteins | Topical application | Index Medicus | Biological Sciences
Journal Article
Journal Article
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2017, Volume 21, Issue 12, pp. 3705 - 3717
Oestrogens have been reported to attenuate acute inflammation in sepsis. In this study, the effects of long‐term oestrogen replacement with 17β‐oestradiol (E 2... 
oestrogen | septic shock | autophagy | HSP70 | inflammation | endotoxaemia | HO‐1 | ovariectomy | multiple organ dysfunction syndrome | oxidative stress | HO-1 | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG INJURY | SEPSIS | ANTIOXIDANT SUPPLEMENTATION | CELL BIOLOGY | PLASMINOGEN-ACTIVATOR | ESTROGEN PROTECTS | GENE-EXPRESSION | ETHYL PYRUVATE | INFLAMMATORY RESPONSE | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Heme Oxygenase-1 - metabolism | Cerebrovascular Disorders - drug therapy | Endotoxemia - genetics | Tumor Necrosis Factor-alpha - genetics | Cerebrovascular Disorders - metabolism | Endotoxemia - pathology | Transcription Factor RelA - genetics | Autophagy - drug effects | Lipopolysaccharides | Heme Oxygenase-1 - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Rhabdomyolysis - drug therapy | Rhabdomyolysis - pathology | Female | Renal Insufficiency - drug therapy | Autophagy - genetics | Plasminogen Activator Inhibitor 1 - genetics | Interleukin-6 - metabolism | Estradiol - pharmacology | Renal Insufficiency - genetics | Autophagy-Related Protein 12 - genetics | Drug Administration Schedule | Interleukin-6 - genetics | Ovariectomy | Anti-Inflammatory Agents - pharmacology | Endotoxemia - drug therapy | Gene Expression Regulation | Rats | HSP70 Heat-Shock Proteins - genetics | Renal Insufficiency - metabolism | Autophagy-Related Protein 12 - metabolism | Rats, Sprague-Dawley | HSP70 Heat-Shock Proteins - metabolism | Rhabdomyolysis - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Transcription Factor RelA - metabolism | Renal Insufficiency - pathology | Rhabdomyolysis - genetics | Cerebrovascular Disorders - genetics | Endotoxemia - metabolism | Cerebrovascular Disorders - pathology | Nitric Oxide Synthase Type II - metabolism | Myoglobin | Intravenous administration | Renal function | Estrogens | Menopause | Activation | Damage prevention | Renal tubules | Autophagy | Interleukin 6 | Infusion | Rodents | Intravenous infusion | Heart diseases | Endotoxemia | Glutathione | NF-κB protein | Haem | Multiple organ dysfunction syndrome | Contractility | Heat shock proteins | Muscles | Heme oxygenase (decyclizing) | Hsp70 protein | Muscle contraction | Nitric-oxide synthase | Skeletal muscle | Oxygenase | Myocardium | Plasma levels | Phagocytosis | Index Medicus | Original
Journal Article