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Molecular Neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2451 - 2467
...) disruption and synaptic dysfunction, leading to AD pathology is not clear. Therefore, we hypothesized that the inhibition of neuronal NMDA-R by H2S and MK801 mitigate... 
Cerebrovascular pathology | Neurology | Neurosciences | Biomedicine | Blood–brain barrier dysfunction | Alzheimer’s disease | Neurobiology | Homocysteine | Cell Biology | Dementia | COGNITIVE PERFORMANCE | NEUROSCIENCES | MATRIX METALLOPROTEINASES | INDUCED MEMORY IMPAIRMENT | RISK-FACTOR | Blood-brain barrier dysfunction | RECEPTOR TRAFFICKING | MATRIX-METALLOPROTEINASE-9 | RAT-BRAIN | Alzheimer's disease | PLASMA HOMOCYSTEINE | EXPRESSION | Memory - drug effects | Glial Fibrillary Acidic Protein - genetics | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Blood-Brain Barrier - physiopathology | Claudin-5 - metabolism | Microvessels - pathology | Male | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Cadherins - genetics | Synapses - drug effects | Alzheimer Disease - physiopathology | Hydrogen Sulfide - pharmacology | Cerebrovascular Circulation - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Alzheimer Disease - drug therapy | Microvessels - drug effects | Claudin-5 - genetics | Cystathionine beta-Synthase - metabolism | Permeability | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Hydrogen Sulfide - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Intercellular Adhesion Molecule-1 - genetics | Avoidance Learning - drug effects | Alzheimer Disease - genetics | Dizocilpine Maleate - pharmacology | Hydrogen sulfide | Methyl aspartate | Brain | RNA | Neurons | Intermediate filament proteins | cerebrovascular pathology | dementia | blood brain barrier dysfunction
Journal Article
Lancet neurology, ISSN 1474-4422, 2008, Volume 7, Issue 11, pp. 1044 - 1055
... of various movement disorders. The most widely recognised finding for movement disorders has been an increase in echogenicity of the substantia nigra, an area of the midbrain that is affected in idiopathic Parkinson's disease (PD... 
Neurology | REAL-TIME SONOGRAPHY | RESTLESS LEGS SYNDROME | SUBSTANTIA-NIGRA HYPERECHOGENICITY | ADULT-ONSET DYSTONIA | BASAL GANGLIA | IN-VIVO DETECTION | IDIOPATHIC PARKINSONS-DISEASE | ESSENTIAL TREMOR | CLINICAL NEUROLOGY | BRAIN PARENCHYMA SONOGRAPHY | PROGRESSIVE SUPRANUCLEAR PALSY | Predictive Value of Tests | Basal Ganglia - diagnostic imaging | Lateral Ventricles - pathology | Humans | Basal Ganglia Cerebrovascular Disease - diagnostic imaging | Ultrasonography, Doppler, Transcranial - methods | Lateral Ventricles - diagnostic imaging | Lateral Ventricles - physiopathology | Mesencephalon - physiopathology | Movement Disorders - diagnostic imaging | Mesencephalon - pathology | Diagnosis, Differential | Parkinson Disease - pathology | Parkinson Disease - diagnostic imaging | Basal Ganglia - physiopathology | Movement Disorders - pathology | Brain - physiopathology | Basal Ganglia - pathology | Parkinson Disease - physiopathology | Mesencephalon - diagnostic imaging | Ultrasonography, Doppler, Transcranial - trends | Basal Ganglia Cerebrovascular Disease - pathology | Movement Disorders - physiopathology | Basal Ganglia Cerebrovascular Disease - physiopathology | Brain - pathology | Neuroimaging | Parkinson's disease | Scanning | Mesencephalon | Neurodegenerative diseases | Brain stem | Lentiform nucleus | Substantia nigra | Differential diagnosis | Ultrasound | Movement disorders
Journal Article
2005, Supportive care series, ISBN 0198529732, xiii, 476
This book provides a practical, evidence-based overview of the supportive care of patients with urological failure, focusing on chronic symptoms such as... 
methods | therapy | Quality of Life | Urologic Diseases | Palliative Care | Palliative treatment | Chronic Disease | Urinary organs | Diseases | Patient Care and End-of-Life Decision Making | Pain Management and Palliative Pharmacology | Internal medicine | Neurological | Urinary | Hematuria | Infertility | Urological failure | Supportive care | Symptomatic | Chronic prostatitis
Book
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-9071, 2018, Volume 76, Issue 2, pp. 283 - 300
Cerebrovascular disorders are underlain by perturbations in cerebral blood flow and abnormalities in blood vessel structure... 
Life Sciences | Biomedicine, general | Biochemistry, general | Life Sciences, general | Hemorrhagic cerebrovascular disease | Small vessel disease | Genetics | Model organisms | Cerebrovascular disease | Cell Biology | FAMILIAL INTRACRANIAL ANEURYSMS | ANEURYSMAL SUBARACHNOID HEMORRHAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HOMOZYGOUS CADASIL PATIENT | MATCHED HETEROZYGOUS PATIENTS | SOLUBLE GUANYLATE-CYCLASE | CELL BIOLOGY | CEREBRAL CAVERNOUS MALFORMATIONS | SMALL-VESSEL DISEASE | GRANULAR OSMIOPHILIC MATERIAL | AUTOSOMAL-DOMINANT ARTERIOPATHY | GENOME-WIDE ASSOCIATION | Cerebral Small Vessel Diseases - genetics | CADASIL - pathology | Humans | Moyamoya Disease - pathology | Cerebral Small Vessel Diseases - pathology | CADASIL - genetics | Cerebral Amyloid Angiopathy - pathology | Amyloid beta-Protein Precursor - genetics | Cerebrovascular Disorders - diagnostic imaging | Receptor, Notch3 - genetics | Adenosine Triphosphatases - genetics | Cerebrovascular Disorders - genetics | Cerebral Amyloid Angiopathy - genetics | SOXF Transcription Factors - genetics | Ubiquitin-Protein Ligases - genetics | Cerebrovascular Disorders - pathology | Moyamoya Disease - diagnostic imaging | Moyamoya Disease - genetics | Molecular modelling | Cerebral blood flow | Pathogenesis | Abnormalities | Disorders | Blood vessels | Lesions | Cerebrovascular diseases | Blood | Blood flow
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