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2013, First edition., ISBN 9780316234801, x, 323 pages
The devastating truth about the effects of wheat, sugar, and carbs on the brain, with a 30-day plan to achieve optimum health. 
Nutritional aspects | Brain | Carbohydrates | Energy metabolism | Metabolism | Diseases
Book
2012, Contemporary neurology series, ISBN 0195394275, Volume 82., xvi, 211 p., [12] p. of plates
Book
Nature Communications, ISSN 2041-1723, 04/2016, Volume 7, Issue 1, p. 11185
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e78439
Neuroinflammation mediated by the activated microglia is suggested to play a pivotal role in the pathogenesis of hypoxic brain injury; however, the underlying... 
MURINE BV-2 CELLS | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | RECEPTOR | NEURAL PROGENITOR CELLS | RAT-BRAIN | DIFFERENTIATION | INFLAMMATORY RESPONSE | EXPRESSION | AMEBOID MICROGLIA | INNATE IMMUNITY | Amyloid Precursor Protein Secretases - genetics | Microglia - metabolism | Transcription Factor HES-1 | Homeodomain Proteins - metabolism | NF-kappa B - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Hypoxia - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Gene Expression Regulation, Developmental | Microglia - pathology | Membrane Proteins - metabolism | TNF Receptor-Associated Factor 6 - genetics | Immunoglobulin J Recombination Signal Sequence-Binding Protein - genetics | Intracellular Signaling Peptides and Proteins - genetics | Animals, Newborn | Immunoglobulin J Recombination Signal Sequence-Binding Protein - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Membrane Proteins - genetics | Dipeptides - pharmacology | Enzyme Inhibitors - pharmacology | Myeloid Differentiation Factor 88 - genetics | Rats | Toll-Like Receptor 4 - genetics | Receptor, Notch1 - metabolism | Toll-Like Receptor 4 - metabolism | Homeodomain Proteins - genetics | Amyloid Precursor Protein Secretases - metabolism | Hypoxia - genetics | Animals | NF-kappa B - genetics | Hypoxia - pathology | TNF Receptor-Associated Factor 6 - metabolism | Cell Line, Tumor | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Receptor, Notch1 - antagonists & inhibitors | Cell Hypoxia - genetics | Receptor, Notch1 - genetics | Myeloid Differentiation Factor 88 - metabolism | Brain | Mental disorders | Laboratories | Pathogenesis | Crosstalk | Activation | Neurogenesis | Signal transduction | Embryology | Head injuries | Pathways | Rodents | Cerebrum | Toll-like receptors | Microglial cells | Inhibition | Pretreatment | Casualties | Translocation | NF-κB protein | Adenosine | TRAF6 protein | Cytokines | Histology | Exposure | Inflammation | TLR4 protein | Gene expression | Microglia | Medicine | Studies | Signaling | Brain research | DELTA protein | Stem cells | MyD88 protein | Hypoxia | Ligands | Brain damage | Notch protein | Brain injury | Secretase
Journal Article
Diabetes, ISSN 0012-1797, 07/2012, Volume 61, Issue 7, pp. 1831 - 1837
Journal Article
2002, International review of neurobiology, ISBN 0123668522, Volume 51., xiv, 555 p., [10] p. of plates
Regulation of glucose at the biochemical level affects every area of the brain, and has impact from cellular to behavioral brain function. It plays an... 
Brain | Pathophysiology | Depression, Mental | Physiology | Glucose | Diabetes | Metabolism
Book
Molecular Pharmaceutics, ISSN 1543-8384, 01/2013, Volume 10, Issue 1, pp. 289 - 296
Journal Article
1976, 2d ed. --, ISBN 9780470013762, 384
Book
Biological Trace Element Research, ISSN 0163-4984, 9/2013, Volume 154, Issue 3, pp. 387 - 395
Chronic overexposure to cobalt (Co) may result in neurotoxic effects, but the mechanism of Co-induced neurotoxicity is not yet well established. Our study was... 
Life Sciences | Biochemistry, general | Cerebellum | Biotechnology | Nutrition | Cobalt chloride | Cerebrum | Oncology | Suckling rats | Histological studies | Antioxidant enzyme profiles | OXIDATIVE STRESS | LIPID-PEROXIDATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | SELENIUM | DAMAGE | FREE-RADICALS | CELL-DEATH | PROTECTIVE ROLE | ENDOCRINOLOGY & METABOLISM | TISSUES | BRAIN | Rats, Wistar | Antioxidants - metabolism | Cerebellum - drug effects | Oxidants - metabolism | Protein Carbonylation - drug effects | Advanced Oxidation Protein Products - metabolism | Butyrylcholinesterase - metabolism | Animals, Suckling | Cerebrum - metabolism | Female | DNA Fragmentation - drug effects | Cerebrum - pathology | Superoxide Dismutase - metabolism | Cerebrum - drug effects | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Administration, Oral | Cerebellum - metabolism | Adenosine Triphosphatases - metabolism | Rats | Cobalt - administration & dosage | Cobalt - blood | Random Allocation | GPI-Linked Proteins - metabolism | Prenatal Exposure Delayed Effects - metabolism | Cerebellum - pathology | Hydrogen Peroxide - metabolism | Catalase - metabolism | Pregnancy | Cobalt - toxicity | Animals | Acetylcholinesterase - metabolism | Prenatal Exposure Delayed Effects - chemically induced | Antioxidants | Neurotoxicity | Histology | Cobalt | Rodents | Adenosine triphosphatase | Index Medicus
Journal Article
Biological Trace Element Research, ISSN 0163-4984, 12/2017, Volume 180, Issue 2, pp. 275 - 284
This study investigated the toxicity of rats exposed to lead acetate (AcPb) during the second phase of brain development (8–12 days postnatal) in hematological... 
Biochemistry, general | Biotechnology | AChE | Nutrition | Oncology | Hematological | Zinc | Pups | PBG-synthase | Life Sciences | NAC | Lead | Cerebral | ACETYLCHOLINESTERASE ACTIVITY | OXIDATIVE DAMAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE LEVELS | IN-VITRO | POSTNATAL-GROWTH | ENDOCRINOLOGY & METABOLISM | CHELATING-AGENTS | SUCKLING RATS | BRAIN GROWTH | AMINOLEVULINIC-ACID DEHYDRATASE | BINDING | Environmental Pollutants - metabolism | Neuroprotective Agents - metabolism | Neuroprotective Agents - pharmacokinetics | Chlorides - metabolism | Injections, Subcutaneous | Neurons - metabolism | Environmental Pollutants - toxicity | Acetylcysteine - administration & dosage | GPI-Linked Proteins - antagonists & inhibitors | Zinc Compounds - therapeutic use | Lead - toxicity | Animals, Newborn | Biomarkers - metabolism | Lead Poisoning, Nervous System - metabolism | Lead - metabolism | Biomarkers - blood | Random Allocation | Blood-Brain Barrier - metabolism | Toxicokinetics | Porphobilinogen Synthase - blood | Chlorides - administration & dosage | Neurons - enzymology | Porphobilinogen Synthase - antagonists & inhibitors | Zinc Compounds - administration & dosage | Acetylcholinesterase - metabolism | Lead Poisoning, Nervous System - blood | Organometallic Compounds - administration & dosage | Neuroprotective Agents - therapeutic use | Rats, Wistar | Environmental Pollutants - blood | Lead Poisoning, Nervous System - prevention & control | Zinc Compounds - metabolism | Cerebrum - metabolism | Acetylcysteine - therapeutic use | Drug Therapy, Combination | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Cerebrum - drug effects | Nerve Tissue Proteins - antagonists & inhibitors | Tissue Distribution - drug effects | Chlorides - therapeutic use | Blood-Brain Barrier - drug effects | GPI-Linked Proteins - metabolism | Nerve Tissue Proteins - metabolism | Cerebrum - enzymology | Animals | Zinc Compounds - pharmacokinetics | Lead - blood | Chlorides - pharmacokinetics | Brain | Zinc chloride | Hematology | Toxicity | Rats | Acetylcysteine | Exposure | Saline solutions | Sodium chloride | Acetylcholinesterase | Blood | Neurotoxicity | Cerebrum | Hemoglobin | Biomarkers | Inhibition | Pretreatment | Index Medicus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 04/2017, Volume 32, Issue 4, pp. 757 - 769
ABSTRACT In 1985, we briefly reported infant sisters with a unique, lethal, autosomal recessive disorder designated congenital sclerosing osteomalacia with... 
CRANIOFACIAL DYSOSTOSIS | OSTEOMALACIA | RICKETS | DENTIN MATRIX PROTEIN | KINASE | PHOSPHOPROTEOME | CROUZON SYNDROME | CRANIOSYNOSTOSIS | HYPOPHOSPHATEMIA | SIBLING PROTEINS | OSTEOPONTIN | METOPIC SUTURE | MINERALIZATION | OSTEOSCLEROSIS | OSTEOPETROSIS | TRIGONOCEPHALY | AUTOSOMAL RECESSIVE SYNDROME | INTRACRANIAL CALCIFICATION | HYPOPLASTIC NOSE | SECRETED PROTEINS | ENDOCRINOLOGY & METABOLISM | OSTEOSCLEROTIC BONE DYSPLASIA | ANHYDRASE-II DEFICIENCY | CASEIN KINASE | RENAL TUBULAR-ACIDOSIS | Abnormalities, Multiple - metabolism | Calcinosis - genetics | Microcephaly - genetics | Cerebrum - diagnostic imaging | Casein Kinase I - genetics | Osteomalacia - diagnostic imaging | Humans | Calcinosis - diagnostic imaging | Male | Cleft Palate - genetics | Exophthalmos - genetics | Osteosclerosis - metabolism | Adult | Cerebrum - metabolism | Female | Osteomalacia - metabolism | Osteosclerosis - genetics | Calcinosis - metabolism | Cleft Palate - diagnostic imaging | Cerebrum - pathology | Osteosclerosis - diagnostic imaging | Abnormalities, Multiple - genetics | Extracellular Matrix Proteins - metabolism | Infant, Newborn | Extracellular Matrix Proteins - genetics | Microcephaly - metabolism | Abnormalities, Multiple - diagnostic imaging | Exophthalmos - diagnostic imaging | Microcephaly - diagnostic imaging | Osteomalacia - genetics | Exophthalmos - metabolism | Casein Kinase I - metabolism | Cleft Palate - metabolism | Medical research | Dysplasia | Genetic disorders | Calcification | Medicine, Experimental | Genetics | Genetic aspects | Histochemistry
Journal Article
Archives of Physiology and Biochemistry, ISSN 1381-3455, 05/2017, Volume 123, Issue 3, pp. 165 - 174
Context: Vanillin is known to possess important antioxidant activity. Objective: The current study was conducted to establish the therapeutic efficiency of... 
potassium bromate | genes expression | fatty acids | vanillin | oxidative stress | Cerebrum | SPRAGUE-DAWLEY RATS | OXYGEN SPECIES PRODUCTION | WISTAR RATS | PHYSIOLOGY | LIPID-PEROXIDATION | INDUCED APOPTOSIS | GLUTATHIONE-PEROXIDASE | ANGIOTENSIN-II | LIPOIC ACID | ENDOCRINOLOGY & METABOLISM | LIPOPHYLIC VITAMINS | SUPEROXIDE-DISMUTASE | Tumor Necrosis Factor-alpha - metabolism | Sodium-Potassium-Exchanging ATPase - genetics | Superoxide Dismutase - genetics | Butyrylcholinesterase - genetics | Neurotoxicity Syndromes - genetics | Ca(2+) Mg(2+)-ATPase - metabolism | Tumor Necrosis Factor-alpha - genetics | Ca(2+) Mg(2+)-ATPase - genetics | Interleukin-1beta - genetics | Depression - genetics | Protein Carbonylation - drug effects | Neurotoxicity Syndromes - drug therapy | Depression - metabolism | Neuroprotective Agents - pharmacology | Butyrylcholinesterase - metabolism | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Bromates - toxicity | Depression - chemically induced | Lipid Peroxidation - drug effects | Behavior, Animal - drug effects | Cerebrum - metabolism | Cerebrum - physiopathology | Fatty Acids - metabolism | Interleukin-6 - metabolism | Superoxide Dismutase - metabolism | Cerebrum - drug effects | Glutathione Peroxidase - metabolism | Interleukin-6 - genetics | Benzaldehydes - pharmacology | Antioxidants - pharmacology | Glutathione Peroxidase - genetics | Gene Expression Regulation - drug effects | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Depression - prevention & control | Cyclooxygenase 2 - metabolism | Neurotoxicity Syndromes - physiopathology | Neurotoxicity Syndromes - metabolism | Mice | Oxidative Stress - drug effects | Index Medicus
Journal Article