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Journal Article
Journal Article
by Sudha, K and Rao, A. V and Rao, S and Rao, A
Free radical toxicity and antioxidants in Parkinson's disease, 12/2003
Erythrocyte lipid peroxidation, oxidative hemolysis, erythrocyte antioxidant enzymes, viz. superoxide dismutase, glutathione reductase, glutathione peroxidase,... 
Parkinson's disease, Antioxidant enzymes, Ceruloplasmin, Antioxidant vitamins
Journal
Trace elements status in children with bronchial asthma, 03/2004
Zinc, copper and magnesium are important elements in the preservation of immune resistance. Zinc and copper are both required for numerous biochemical... 
Medicine | Trace elements, bronchial asthma, ceruloplasmin. gm04002
Journal
EFFECT OF ESTROGEN ON PLASMA CERULOPLASMIN LEVEL IN RATS EXPOSED TO ACUTE STRESS, 04/2004
Background: Plasma ceruloplasmin, a copper containing protein, belongs to a class called acute phase proteins. Reduced level of ceruloplasmin was associated... 
Medicine | ms04025 Ceruloplasmin, Estradiol valerate, Acute stress
Journal
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 53, Issue 6, pp. 1101 - 1107
Background & Aims Aceruloplasminemia is a rare autosomal recessive neurodegenerative disease associated with brain and liver iron accumulation which typically... 
Gastroenterology and Hepatology | Iron overload | Iron chelation | Neurodegeneration | Metabolic liver disease | Fibrosis | OVERLOAD | OXIDATIVE STRESS | HOMEOSTASIS | FERROPORTIN | HEPCIDIN | COPPER | MECHANISMS | CERULOPLASMIN GENE | GASTROENTEROLOGY & HEPATOLOGY | HEMOCHROMATOSIS | Ceruloplasmin - deficiency | Benzoates - therapeutic use | Humans | Middle Aged | Male | Neurodegenerative Diseases - drug therapy | RNA, Messenger - metabolism | Brain - metabolism | Hepcidins | Female | Antimicrobial Cationic Peptides - genetics | Triazoles - therapeutic use | Iron Chelating Agents - therapeutic use | Iron Metabolism Disorders - drug therapy | Liver - metabolism | RNA, Messenger - genetics | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Iron - metabolism | Ceruloplasmin - genetics | Homozygote | Iron Metabolism Disorders - genetics | Pedigree | Ceruloplasmin - metabolism | Iron Metabolism Disorders - metabolism | Consanguinity | Mutation | Medical colleges | Nervous system diseases | Transferrin | Liver diseases | Gene mutations | Genetic aspects | Diabetes | Index Medicus | NAFLD, non alcoholic fatty liver disease | GPI, glycosylphosphatidylinositol | CP, ceruloplasmin | ACP, aceruloplasmin | MRI, magnetic resonance imaging | RT-PCR, reverse transcription PCR | PCR, polymerase chain reaction | Bp, basepairs | DMS, dysmetabolic siderosis
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e89003 - e89003
We have used an in vitro model system to probe the iron transport pathway across the brain microvascular endothelial cells (BMVEC) of the blood-brain barrier... 
MOLECULAR-MECHANISM | GLYCOSYLPHOSPHATIDYLINOSITOL-ANCHORED FORM | OXIDATIVE STRESS | IN-VITRO | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | FERROXIDASE ACTIVITY | BARRIER | CENTRAL-NERVOUS-SYSTEM | AMYLOID PRECURSOR PROTEIN | RAT-BRAIN | Hepcidins - metabolism | Up-Regulation | Ceruloplasmin - chemistry | Humans | Hepcidins - secretion | RNA, Messenger - metabolism | Brain - blood supply | Neuroglia - cytology | Ubiquitination | Cation Transport Proteins - metabolism | Astrocytes - cytology | Microvessels - cytology | Cell Line | Paracrine Communication | Endothelial Cells - metabolism | RNA, Messenger - genetics | Solubility | Neuroglia - secretion | Iron - metabolism | Blood-Brain Barrier - metabolism | Protein Transport | Endothelial Cells - cytology | Ceruloplasmin - secretion | Ceruloplasmin - metabolism | Hepcidins - genetics | Neuroglia - metabolism | Astrocytes - metabolism | Astrocytes - secretion | Gliomas | Analysis | Cells | Endothelium | Ceruloplasmin | Cell culture | Brain | Oxidative stress | Transferrin | Brain tumors | Trafficking | Homeostasis | Iron | Biochemistry | mRNA | Mammalian cells | Proteins | Blood-brain barrier | Permease | Rodents | Glioma cells | Efflux | Ferroxidase | Secretion | Astrocytes | Gene expression | Endothelial cells | Exports | Internalization | Microvasculature | Mutation | Alzheimers disease | Hepcidin | Index Medicus
Journal Article
Journal of Nutrition, ISSN 0022-3166, 10/2010, Volume 140, Issue 10, pp. 1728 - 1735
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2015, Volume 135, Issue 5, pp. 958 - 974
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 05/2018, Volume 28, Issue 14, pp. 1323 - 1337
Aims: Ceruloplasmin (CP), a ferrous oxidase enzyme, plays an important role in regulating iron metabolism and redox reactions. Previous studies showed that CP... 
Original Research Communications | ceruloplasmin | apoptosis | iron | Alzheimer's disease | oxidative stress | β-amyloid | HOMEOSTASIS | INHERITED NEURODEGENERATIVE DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | COGNITIVE IMPAIRMENT | AMYLOID-BETA PRODUCTION | beta-amyloid | ANCHORED FORM | IRON-METABOLISM | ENDOCRINOLOGY & METABOLISM | TRANSGENIC MOUSE MODEL | SUPEROXIDE-DISMUTASE | BRAIN | PARKINSONS-DISEASE | Ceruloplasmin - deficiency | Humans | Alzheimer Disease - therapy | Male | Mice, Transgenic | Neuroprotective Agents - metabolism | Mice, Knockout | Ceruloplasmin - genetics | Animals | Alzheimer Disease - metabolism | Aged, 80 and over | Ceruloplasmin - metabolism | Aged | Mice | Mice, Inbred BALB C | Alzheimer Disease - genetics | Disease Models, Animal | Ceruloplasmin | Brain | Oxidative stress | Neuroprotection | Animal models | Parkinson's disease | Bax protein | Bcl-2 protein | Pathogenesis | Substantia nigra | Memory | Innovations | Transgenic | Ventricles (cerebral) | Iron | Gene deletion | Accumulation | CP gene | Signal transduction | Pathways | Redox reactions | Clonal deletion | Restoration | Rodents | Deletion | Movement disorders | Neurodegenerative diseases | Extracellular signal-regulated kinase | Pharmacology | Divalent metal transporter-1 | Injection | Gene expression | Metabolism | Chemical compounds | Signaling | Brain damage | Ventricle | Alzheimers disease | Hippocampus | Apoptosis | Index Medicus
Journal Article