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Biochimica et biophysica acta. Molecular and cell biology of lipids, ISSN 1388-1981, 2015, Volume 1851, Issue 6, pp. 844 - 856
Journal Article
Current Medicinal Chemistry, ISSN 0929-8673, 04/2013, Volume 20, Issue 10, pp. 1241 - 1285
Ion channel targeted drugs have always been related with either the central nervous system (CNS), the peripheral nervous system, or the cardiovascular system.... 
ASIC | TRP | Central nervous system | P2X | KCNQ | NMDA | CNS | Ion channels | CHEMISTRY, MEDICINAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | GATED SODIUM-CHANNELS | SUBTYPE-SELECTIVE AGONIST | D-ASPARTATE RECEPTOR | ion channels | ACID-SENSING ION-CHANNEL-1 | central nervous system | GLYCINE ANTAGONIST GV150526 | TARANTULA TOXIN PSALMOTOXIN-1 | TRAUMATIC BRAIN-INJURY | ISCHEMIC CELL-DEATH | PHARMACOLOGY & PHARMACY | PERIPHERAL NEUROPATHIC PAIN | TRANSGENIC MOUSE MODEL | Ligand-Gated Ion Channels - antagonists & inhibitors | Transient Receptor Potential Channels - antagonists & inhibitors | Calcium Channels - metabolism | Humans | Calcium Channel Blockers - therapeutic use | Potassium Channel Blockers - therapeutic use | Sodium Channel Blockers - pharmacology | Calcium Channel Blockers - pharmacology | Ion Channels - antagonists & inhibitors | Potassium Channels - metabolism | Animals | Ion Channels - metabolism | Sodium Channels - chemistry | Calcium Channels - chemistry | Central Nervous System Diseases - drug therapy | Sodium Channels - metabolism | Calcium Channel Blockers - chemistry | Sodium Channel Blockers - therapeutic use | Potassium Channels - chemistry | Sodium Channel Blockers - chemistry | Transient Receptor Potential Channels - metabolism | Ligand-Gated Ion Channels - metabolism | Potassium Channel Blockers - chemistry | Potassium Channel Blockers - pharmacology
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 7, p. e0180154
Ion channels regulate a variety of physiological processes and represent an important class of drug target. Among the many methods of studying ion channel... 
TARGET | DYE | KV1.3 | NA(V)1.7 | MODULATORS | MULTIDISCIPLINARY SCIENCES | EXTREME PAIN DISORDER | PATCH-CLAMP | MUTATIONS | Cricetulus | High-Throughput Screening Assays - instrumentation | NAV1.3 Voltage-Gated Sodium Channel - genetics | NAV1.1 Voltage-Gated Sodium Channel - genetics | NAV1.5 Voltage-Gated Sodium Channel - metabolism | NAV1.1 Voltage-Gated Sodium Channel - metabolism | NAV1.4 Voltage-Gated Sodium Channel - metabolism | Kv1.3 Potassium Channel - genetics | NAV1.2 Voltage-Gated Sodium Channel - metabolism | NAV1.3 Voltage-Gated Sodium Channel - metabolism | Patch-Clamp Techniques - methods | Kv1.3 Potassium Channel - deficiency | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Sodium Channels - metabolism | NAV1.7 Voltage-Gated Sodium Channel - genetics | NAV1.5 Voltage-Gated Sodium Channel - genetics | Drug Evaluation, Preclinical | Transgenes | Potassium Channel Blockers - pharmacology | CHO Cells | High-Throughput Screening Assays - methods | Gene Expression | NAV1.4 Voltage-Gated Sodium Channel - genetics | NAV1.2 Voltage-Gated Sodium Channel - genetics | Rats | Sodium Channel Blockers - pharmacology | Membrane Potentials - physiology | Animals | T-Lymphocytes - cytology | NAV1.6 Voltage-Gated Sodium Channel - metabolism | NAV1.7 Voltage-Gated Sodium Channel - metabolism | NAV1.6 Voltage-Gated Sodium Channel - genetics | Sodium Channels - genetics | Primary Cell Culture | Patch-Clamp Techniques - instrumentation | Ion channels | Information management | Drug discovery | T cells | Modules | Electrophysiology | Patching | Lymphocytes T | Boundaries | Assaying | Channels | Step voltage | Immunology | Pain | Lymphocytes | Sodium channels (voltage-gated) | Physiology | Potassium channels (voltage-gated) | Gold | Platforms | Data points | Automation | Cloning | Pharmacology | Sodium | Antigen-presenting cells | Ligands | Mutation | Recording | Potassium
Journal Article
Circulation Research, ISSN 0009-7330, 02/2007, Volume 100, Issue 3, pp. 342 - 353
A large body of evidence has accrued indicating that voltage-gated Ca channel subtypes, including L-, T-, N-, and P/Q-type, are present within renal vascular... 
Renal microcirculation | Mibefradil | channels | Afferent arteriole | channel blockers | Efferent arteriole | Renal disease | Voltage-dependent Ca | Efonidipine | Ca2+ channel blockers | N-TYPE | CARDIAC & CARDIOVASCULAR SYSTEMS | EFFERENT ARTERIOLES | efonidipine | SPONTANEOUSLY HYPERTENSIVE-RATS | RHO-KINASE INHIBITOR | renal microcirculation | ANGIOTENSIN-II | T-TYPE | renal disease | mibefradil | DEPENDENT CALCIUM-CHANNELS | CARDIAC L-TYPE | RENAL MICROVASCULAR CONSTRICTION | efferent arteriole | voltage-dependent Ca2+ channels | PERIPHERAL VASCULAR DISEASE | afferent arteriole | HEMATOLOGY | IN-VIVO VISUALIZATION | Arterioles - physiology | Kidney - physiology | Protein Subunits | Antihypertensive Agents - pharmacology | Kidney - blood supply | Humans | Calcium Channel Blockers - therapeutic use | Hypertension - drug therapy | Antihypertensive Agents - classification | Calcium Channels - physiology | Calcium Channels, T-Type - chemistry | Neurotransmitter Agents - secretion | Calcium Channels - drug effects | Calcium Channels, N-Type - drug effects | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Calcium Channels - classification | Rats | Calcium Channels, L-Type - physiology | Antihypertensive Agents - therapeutic use | Arterioles - drug effects | Disease Progression | Antihypertensive Agents - adverse effects | Mice, Knockout | Calcium Signaling - drug effects | Diabetes Mellitus - physiopathology | Models, Biological | Calcium Channels - chemistry | Calcium Channels, T-Type - drug effects | Mice | Vasodilation - drug effects | Hydronephrosis - physiopathology | Calcium Channel Blockers - adverse effects | Calcium Signaling - physiology | Cardiovascular Diseases - drug therapy | Renal Circulation - physiology | Microcirculation - drug effects | Microcirculation - physiology | Renal Circulation - drug effects | Blood Pressure - drug effects | Calcium Channels, L-Type - chemistry | Kidney Diseases - metabolism | Calcium Channels, T-Type - physiology | Renin - secretion | Aldosterone - physiology | Kidney Diseases - drug therapy | Renin-Angiotensin System - physiology | Calcium Channel Blockers - pharmacology | Hypertension - physiopathology | Animals | Calcium Channels, L-Type - drug effects | Calcium Channels, N-Type - physiology | Calcium Channels, N-Type - chemistry
Journal Article
Stem cells (Dayton, Ohio), ISSN 1549-4918, 2005, Volume 23, Issue 3, pp. 371 - 382
This study characterized functional ion channels in cultured undifferentiated human mesenchymal stem cells (hMSCs) from bone marrow with whole‐cell patch clamp... 
Transient outward K+ current | Heag K+ current | TTX‐sensitive Na+ current | Human mesenchymal stem cells | Ion channels | Ca2+‐activated K+ current | current | Transient outward K | Heag K | activated K | TTX-sensitive Na | ACTIVATION | K+ CURRENT-DENSITY | RAT | ionchannels | DROSOPHILA | CELL BIOLOGY | IN-VITRO | ONCOLOGY | human mesenchymal stem cells | ETHER | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GO-GO CHANNELS | HEART FUNCTION | TTX sensitive Na+ current | transient outward K+ current | heag K(+)current | HEMATOLOGY | EXPRESSION | Ca2+-activated K+ current | POTASSIUM CHANNEL | Large-Conductance Calcium-Activated Potassium Channels | Shal Potassium Channels | Potassium Channels, Voltage-Gated - physiology | Gene Expression - genetics | Humans | NAV1.7 Voltage-Gated Sodium Channel | Bone Marrow Cells - physiology | Ion Channels - genetics | Ion Channels - physiology | Sodium Channels - physiology | RNA, Messenger - metabolism | Potassium Channels, Calcium-Activated - genetics | Mesenchymal Stromal Cells - cytology | Potassium Channels, Voltage-Gated - drug effects | Bone Marrow Cells - drug effects | Cell Differentiation | Tetrodotoxin - pharmacology | 4-Aminopyridine - pharmacology | Mesenchymal Stromal Cells - physiology | Potassium Channels, Voltage-Gated - genetics | Membrane Potentials - drug effects | Mesenchymal Stromal Cells - drug effects | Nifedipine - pharmacology | Potassium Channels, Calcium-Activated - physiology | Bone Marrow Cells - cytology | RNA, Messenger - genetics | Cells, Cultured | Ether-A-Go-Go Potassium Channels | Calcium Channels, L-Type - physiology | Sodium Channels - drug effects | Nerve Tissue Proteins - genetics | Peptides - pharmacology | Potassium Channels - genetics | Patch-Clamp Techniques | Calcium Channels, L-Type - genetics | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits | Calcium Channels, L-Type - drug effects | Potassium Channels, Calcium-Activated - drug effects | Kv1.4 Potassium Channel | Sodium Channels - genetics
Journal Article
Journal of neurochemistry, ISSN 0022-3042, 2016, Volume 139, Issue S1, pp. 156 - 178
Dopamine‐releasing neurons within the Substantia nigra (SN DA) are particularly vulnerable to degeneration compared to other dopaminergic neurons. The... 
D2‐autoreceptor‐coupled GIRK2 channels | Cav1.3 L‐type/Cav3.1 T‐type calcium channels | neuronal calcium sensor NCS‐1 | ambroxol | A‐type Kv4.3/KChip3 potassium channels | Kir6.2/SUR1 potassium channels | Cav1.3 L-type/Cav3.1 T-type calcium channels | neuronal calcium sensor NCS-1 | A-type Kv4.3/KChip3 potassium channels | D2-autoreceptor-coupled GIRK2 channels | SENSITIVE POTASSIUM CHANNELS | OXIDANT STRESS | CA2+ CHANNELS | GLUCOCEREBROSIDASE MUTATIONS | KChip3 potassium channels | D2-AUTORECEPTOR RESPONSES | SUR1 potassium channels | 1 T-type calcium channels | Kir6 | Cav3 | Cav1 | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | A-type Kv4 | NEUROSCIENCES | 3L-type | 2 | 3 | ALPHA-SYNUCLEIN FORM | LOCUS-COERULEUS | MPTP MOUSE MODEL | MITOCHONDRIAL-DNA DELETIONS | Stress, Physiological - physiology | Parkinson Disease - pathology | Animals | Calcium - physiology | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Dopaminergic Neurons - metabolism | Humans | Ion Channels - physiology | Parkinson Disease - metabolism | Substantia Nigra - metabolism | Health Status | Calcium channels | Stress (Physiology) | Parkinson's disease | Potassium channels | Neurons | Dopamine | Calcium | Animal behavior | Rodents | Bandwidths | Parkinsons disease | Homeostasis | Physiology | Metabolism | Kir6.2 | Cav3.1 T‐type calcium channels | Cav1.3 L‐type | Jörg B. Schulz and John Hardy | Review | Parkinson Disease. Guest Editors | A‐type Kv4.3
Journal Article
Circulation. Arrhythmia and electrophysiology, ISSN 1941-3084, 2017, Volume 10, Issue 4, pp. e003560 - e003560
BACKGROUND—The widely used macrolide antibiotic azithromycin increases risk of cardiovascular and sudden cardiac death, although the underlying mechanisms are... 
calcium channel | sodium channels | mice | potassium channels | pharmacology | TORSADES-DE-POINTES | SODIUM | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | MACROLIDE | TRAFFICKING | ARRHYTHMIA | QT-INTERVAL PROLONGATION | IN-VIVO | CHANNELS | MUTATIONS | Arrhythmias, Cardiac - chemically induced | KCNQ1 Potassium Channel - genetics | Cricetulus | Humans | KCNQ1 Potassium Channel - metabolism | Azithromycin - toxicity | NAV1.5 Voltage-Gated Sodium Channel - metabolism | Arrhythmias, Cardiac - physiopathology | Action Potentials | Dose-Response Relationship, Drug | Young Adult | Heart Rate - drug effects | Transfection | Potassium Channels, Voltage-Gated - metabolism | Time Factors | NAV1.5 Voltage-Gated Sodium Channel - drug effects | HEK293 Cells | KCNQ1 Potassium Channel - antagonists & inhibitors | Female | Potassium Channels, Inwardly Rectifying - antagonists & inhibitors | Potassium Channel Blockers - toxicity | Anti-Bacterial Agents - toxicity | NAV1.5 Voltage-Gated Sodium Channel - genetics | CHO Cells | Potassium Channels, Voltage-Gated - genetics | Sodium Channel Blockers - toxicity | Arrhythmias, Cardiac - metabolism | Rabbits | Mice, Inbred C57BL | Potassium Channels, Inwardly Rectifying - genetics | Potassium Channels, Voltage-Gated - antagonists & inhibitors | Telemetry | Calcium Channel Blockers - toxicity | Animals | Calcium Channels, L-Type - genetics | Myocytes, Cardiac - drug effects | Calcium Channels, L-Type - drug effects | Myocytes, Cardiac - metabolism | Calcium Channels, L-Type - metabolism | Potassium Channels, Inwardly Rectifying - metabolism | Electrocardiography, Ambulatory | Ion Channels |