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Hepatology, ISSN 0270-9139, 09/2011, Volume 54, Issue 3, pp. 931 - 939
Distinguishing drug‐induced liver injury (DILI) from idiopathic autoimmune hepatitis (AIH) can be challenging. We performed a standardized histologic... 
CRITERIA | GASTROENTEROLOGY & HEPATOLOGY | PROGNOSIS | Liver - pathology | Biopsy | Humans | Middle Aged | Adult | Female | Male | Aged | Chemical and Drug Induced Liver Injury - pathology | Hepatitis, Autoimmune - pathology | Gallbladder diseases | Inflammation | Discrimination | Liver | Rodents | Neutrophils | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 03/2012, Volume 61, Issue 3, pp. 416 - 426
ObjectiveMonocyte chemoattractant protein-1 (MCP-1, CCL2), the primary ligand for chemokine receptor C–C chemokine receptor 2 (CCR2), is increased in livers of... 
FIBROSIS | STEATOSIS | INSULIN-RESISTANCE | INFLAMMATION | BONE-MARROW | DISEASE | MONOCYTE SUBSETS | MICE | SPIEGELMER | GASTROENTEROLOGY & HEPATOLOGY | CCR2 | Liver - pathology | Carbon Tetrachloride | Liver Cirrhosis, Experimental - etiology | Bone Marrow Cells - physiology | Male | Chemical and Drug Induced Liver Injury, Chronic - drug therapy | Liver Cirrhosis, Experimental - prevention & control | Non-alcoholic Fatty Liver Disease | Chemokine CCL2 - physiology | Bone Marrow Cells - drug effects | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury, Chronic - complications | Macrophages - physiology | Aptamers, Nucleotide - pharmacology | Drug Evaluation, Preclinical - methods | Acute Disease | Cytokines - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Fatty Liver - prevention & control | Aptamers, Nucleotide - therapeutic use | Chemotaxis - drug effects | Chemical and Drug Induced Liver Injury, Chronic - pathology | Disease Progression | Fatty Liver - drug therapy | Animals | Chemical and Drug Induced Liver Injury - complications | Chemical and Drug Induced Liver Injury - drug therapy | Macrophages - drug effects | Mice | Chemokine CCL2 - antagonists & inhibitors | Fatty Liver - etiology | Care and treatment | Liver diseases | Fibrosis | Models | Research | Macrophages | Chemokine receptors | Studies | Cytokines | Polyethylene glycol | Rodents | Liver | Clinical trials | Tumor necrosis factor-TNF | Inflammation | Diabetes | Metabolic disorders | Chemokines | Immunohistochemistry | CC chemokine receptors | Flow cytometry | Animal models | Leukocyte migration | RNA | Carbon tetrachloride | Oligonucleotides | Blood | Metastases | Interleukin 6 | Fatty liver | CCR2 protein | Degeneration | Lipogenesis | Digestive tract | steatosis | Injuries | Triglycerides | CCL4 protein | Chemotaxis | Diets | Monocytes | gamma -Interferon | Monocyte chemoattractant protein 1 | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 539 - 550
In contrast to microbially triggered inflammation, mechanisms promoting sterile inflammation remain poorly understood. Damage-associated molecular patterns... 
MEDICINE, RESEARCH & EXPERIMENTAL | DANGER SIGNALS | STERILE INFLAMMATION | CHROMATIN PROTEIN HMGB1 | IN-VIVO | SEPTIC SHOCK | MOBILITY GROUP BOX-1 | RECEPTOR | DEFICIENT MICE | CELL-DEATH | TRANSGENIC MICE | Acetaminophen - adverse effects | Inflammation - chemically induced | Leukocyte Elastase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tumor Necrosis Factor-alpha - genetics | Analgesics, Non-Narcotic - pharmacology | Hepatocytes - pathology | Hepatocytes - metabolism | Neutrophil Infiltration | fas Receptor - metabolism | Necrosis - pathology | HMGB1 Protein - genetics | Shock, Septic - metabolism | Inflammation - metabolism | Acetaminophen - pharmacology | Analgesics, Non-Narcotic - adverse effects | HMGB1 Protein - metabolism | fas Receptor - genetics | Chemical and Drug Induced Liver Injury - pathology | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Neutrophils - pathology | Shock, Septic - genetics | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Macrophages - pathology | Shock, Septic - chemically induced | Leukocyte Elastase - genetics | Necrosis - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mice, Knockout | Necrosis - chemically induced | Macrophages - metabolism | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammation - genetics | Mice | Necrosis - genetics | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Physiological aspects | Causes of | Inflammation | Genetic aspects | Cell death | Necrosis | Chromosomal proteins | Mass spectrometry | Analysis | Proteins | Studies | Rodents | Mortality | Gangrene | Homeostasis | Mitochondrial DNA | Laboratory animals | Deoxyribonucleic acid--DNA | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
American Journal of Gastroenterology, ISSN 0002-9270, 01/2017, Volume 112, Issue 1, pp. 18 - 35
Clinicians are required to assess abnormal liver chemistries on a daily basis. The most common liver chemistries ordered are serum alanine aminotransferase... 
UNITED-STATES | TRANSPEPTIDASE ACTIVITY | AMERICAN ASSOCIATION | GAMMA-GLUTAMYL-TRANSFERASE | CHRONIC HEPATITIS-C | SERUM ALANINE AMINOTRANSFERASE | TRANSAMINASE-ACTIVITY | CORONARY-HEART-DISEASE | ALPHA-1-ANTITRYPSIN DEFICIENCY | GASTROENTEROLOGY & HEPATOLOGY | UPPER LIMITS | Non-alcoholic Fatty Liver Disease - pathology | Liver - pathology | Humans | Hemochromatosis - blood | Liver Diseases - pathology | Hepatolenticular Degeneration - diagnosis | Alanine Transaminase - blood | Liver Diseases, Alcoholic - diagnosis | Hemochromatosis - pathology | Hepatitis, Viral, Human - blood | Hepatitis, Autoimmune - pathology | Non-alcoholic Fatty Liver Disease - blood | Chemical and Drug Induced Liver Injury - blood | Chemical and Drug Induced Liver Injury - pathology | alpha 1-Antitrypsin Deficiency - pathology | Alkaline Phosphatase - blood | Liver Diseases - blood | Liver Diseases, Alcoholic - pathology | Hepatitis, Autoimmune - diagnosis | Liver - metabolism | Cholestasis - diagnosis | Hepatolenticular Degeneration - blood | Chemical and Drug Induced Liver Injury - diagnosis | Hepatitis, Viral, Human - pathology | alpha 1-Antitrypsin Deficiency - diagnosis | Cholestasis - blood | alpha 1-Antitrypsin Deficiency - blood | Cholestasis - pathology | Bilirubin - blood | Hepatolenticular Degeneration - pathology | Aspartate Aminotransferases - blood | Biopsy | Hepatitis, Autoimmune - blood | Non-alcoholic Fatty Liver Disease - diagnosis | Liver Diseases - diagnosis | Hemochromatosis - diagnosis | Hepatitis, Viral, Human - diagnosis | Liver Diseases, Alcoholic - blood | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 7, pp. 1763 - 1774
Background & Aims The NACHT, LRR, and pyrin domain–containing protein 3 (NLRP3) inflammasome induces inflammation in response to organ injury, but little is... 
Gastroenterology and Hepatology | Innate Immune Response | Immune Regulation | Pancreas | Mouse Model | ACTIVATION | NLRP3 INFLAMMASOME | GPR81 | AGONISTS | GENE | TLR9 | MICE | HEPATOTOXICITY | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | SEVERITY | Liver - pathology | Inflammasomes - metabolism | Receptors, G-Protein-Coupled - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Male | NF-kappa B - metabolism | Monocytes - immunology | Lipopolysaccharides | Liver - immunology | Liver - drug effects | RNA Interference | Interleukin-1beta - metabolism | Toll-Like Receptors - drug effects | Anti-Inflammatory Agents - administration & dosage | Toll-Like Receptors - metabolism | Cytoprotection | Disease Models, Animal | Galactosamine | Chemical and Drug Induced Liver Injury - prevention & control | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Liver - metabolism | Injections, Intraperitoneal | Pancreas - pathology | Pancreas - metabolism | Pancreas - immunology | Toll-Like Receptor 4 - metabolism | Chemical and Drug Induced Liver Injury - immunology | Monocytes - drug effects | Macrophages - metabolism | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Sodium Lactate - pharmacology | beta-Arrestins | Mice | Receptors, G-Protein-Coupled - genetics | RNA, Small Interfering - metabolism | Monocytes - metabolism | Pancreatitis - prevention & control | Arrestins - metabolism | Dose-Response Relationship, Drug | Pancreatitis - genetics | Transfection | Inflammasomes - drug effects | Sodium Lactate - administration & dosage | Pancreatitis - immunology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Macrophages - immunology | Cell Line | Immunity, Innate - drug effects | Toll-Like Receptor 4 - drug effects | Mice, Inbred C57BL | Pancreas - drug effects | Chemical and Drug Induced Liver Injury - genetics | Pancreatitis - chemically induced | Animals | Carrier Proteins - metabolism | beta-Arrestin 2 | Inflammasomes - immunology | Macrophages - drug effects | Pancreatitis - pathology | Pancreatitis - metabolism | Ceruletide | Lactates | Gastrointestinal diseases | Inflammation | Index Medicus | Abridged Index Medicus
Journal Article