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Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 8598 - 12
Inflammation plays an important role in the pathogenesis of renal and cardiovascular disease in renovascular hypertension (RVH). Ccl2 is an important mediator... 
Hypertension | CC chemokine receptors | Nuclear magnetic resonance--NMR | Kidneys | Renal artery | Stenosis | Leukocytes (mononuclear) | Inflammation | Blood flow | Atrophy | Magnetic resonance imaging | CCR2 protein | Perfusion | Surgery | Rodents | Fibrosis | Functional magnetic resonance imaging | Hypoxia | Cardiovascular diseases | Monocyte chemoattractant protein 1
Journal Article
Journal Article
CIRCULATION, ISSN 0009-7322, 04/2008, Volume 117, Issue 13, pp. 1649 - 1657
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2012, Volume 123, Issue 6, pp. 954 - 962
The induction of ischemic tolerance by preconditioning provides a platform to elucidate endogenous mechanisms of stroke protection. In these studies, we... 
sphingosine‐1‐phosphate | chemokines | neuroprotection | bioactive lipids | sphingosine-1-phosphate | CANCER-CELLS | ISCHEMIC TOLERANCE | ACTIVATION | INDUCIBLE FACTOR-I | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | RECEPTOR | NEUROSCIENCES | HUMAN ENDOTHELIAL-CELLS | HIF-1-ALPHA | UP-REGULATION | TRANSCRIPTION FACTOR | Male | Ischemic Preconditioning - methods | Chemokine CCL2 - physiology | Female | Hypoxia-Ischemia, Brain - therapy | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Receptors, Chemokine - genetics | Hypoxia-Ischemia, Brain - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Chemokine CCL2 - genetics | Phosphotransferases (Alcohol Group Acceptor) - genetics | Stroke - drug therapy | Chemokine CCL2 - deficiency | Mice, Knockout | Receptors, Chemokine - deficiency | Stroke - enzymology | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Phenotype | Stroke - metabolism | Animals | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | CX3C Chemokine Receptor 1 | Stroke (Disease) | Gene expression | Sphingosine | Proteins | Neurochemistry | Signal transduction | Brain | Stroke | Rodents | Kinases | Index Medicus | Hypoxia-inducible factors | Translation | sphingosine kinase | Data processing | cobalt chloride | Cobalt | epigenetics | Sphingosine 1-phosphate | Ischemia | Feedback | FTY720 | Hypoxia | genomics | Chemokines | Monocyte chemoattractant protein 1
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, pp. e22818 - e22818
We have shown previously that a para-inflammatory response exists at the retinal/choroidal interface in the aging eye; and this response plays an important... 
MIGRATION | CELLS | MAINTENANCE | MULTIDISCIPLINARY SCIENCES | FACTOR-H POLYMORPHISM | MACULAR DEGENERATION | EXPERIMENTAL AUTOIMMUNE UVEORETINITIS | COMPLEMENT FACTOR-B | ACCUMULATION | MODEL | SUBRETINAL MICROGLIA | Receptors, CCR2 - genetics | Tumor Necrosis Factor-alpha - metabolism | Leukocytes, Mononuclear - metabolism | Microscopy, Electron, Transmission | Retinal Diseases - genetics | Retinal Pigment Epithelium - metabolism | Retina - metabolism | Chemokine CCL2 - genetics | Retinal Diseases - metabolism | Chemokine CCL2 - deficiency | Mice, Knockout | Autoimmune Diseases - genetics | Retinal Pigment Epithelium - pathology | Animals | Retina - ultrastructure | Interleukin-10 - metabolism | Retinal Diseases - pathology | Female | Mice | Receptors, CCR2 - deficiency | Autoimmune Diseases - metabolism | Retina - pathology | Dendritic Cells - metabolism | Nitric Oxide Synthase Type II - metabolism | Retinal degeneration | Physiological aspects | Inflammation | Stress (Psychology) | Cell death | CC chemokine receptors | Complement component C3b | Genes | Homeostasis | Retina | Inflammatory response | Activation | Complement | Macular degeneration | Cell activation | CCR2 protein | Lasers | Breakdowns | Rodents | Light | Aging | Physiology | Degeneration | Age | Immune system | Lymphatic system | Free radicals | Cytokines | Mortality | Interleukin 12 | Tumor necrosis factor-α | Chemotaxis | Nitric-oxide synthase | Hypotheses | Monocytes | Complement activation | Tumor necrosis factor | Interleukin 10 | Photoreceptors | Phagocytosis | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 05/2010, Volume 184, Issue 10, pp. 5571 - 5581
Journal Article
EMBO reports, ISSN 1469-221X, 12/2016, Volume 17, Issue 12, pp. 1738 - 1752
Sickness behavior defines the endocrine, autonomic, behavioral, and metabolic responses associated with infection. While inflammatory responses were suggested... 
weight loss | CCR2 signaling pathway | melanin‐concentrating hormone | CCL2 chemokine | neuroinflammation | melanin-concentrating hormone | MESSENGER-RNA EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ARCUATE NUCLEUS | CELL BIOLOGY | ENERGY-BALANCE | BODY-WEIGHT | INDUCED SICKNESS BEHAVIOR | INDUCED ANOREXIA | REGULATE FEEDING-BEHAVIOR | RAT-BRAIN | CHEMOKINE RECEPTORS | Chemokine CCL2 - immunology | Lipopolysaccharides - immunology | Melanins - metabolism | Inflammation - metabolism | Pituitary Hormones - metabolism | Hypothalamic Hormones - immunology | Chemokine CCL2 - metabolism | Neurons - metabolism | Cytokines - genetics | Cytokines - immunology | Signal Transduction | Melanins - immunology | Neurons - immunology | Chemokine CCL2 - genetics | Melanins - genetics | Chemokine CCL2 - deficiency | Receptors, CCR2 - metabolism | Animals | Hypothalamus - metabolism | Hypothalamic Hormones - metabolism | Weight Loss | Pituitary Hormones - genetics | Hypothalamic Hormones - genetics | Mice | Pituitary Hormones - immunology | Illness Behavior | Cytokines - biosynthesis | Inflammation | Behavior | Metabolism | Neurons | Index Medicus | Life Sciences | Neurons and Cognition | Biochemistry, Molecular Biology | Neurobiology | Biomolecules | Cellular Biology | Cell Behavior | Neuroscience | Immunology | Scientific Reports | Scientific Report
Journal Article