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Toxicology Letters, ISSN 0378-4274, 10/2012, Volume 214, Issue 2, pp. 91 - 98
► Microglial activation by NPs. ► Microglial activation on neurons. ► Related signaling pathway. Once nanoparticles enter the central nervous system (CNS),... 
PC12 cell | Coculture | Neurodegeneration | Inorganic nanoparticle | Microglia | APOPTOSIS | TRANSLOCATION | ACTIVATION | PARTICLES | NITRIC-OXIDE SYNTHASE | DOPAMINERGIC-NEURONS | NEUROTOXICITY | TOXICOLOGY | PC12 CELLS | NF-KAPPA-B | EXPRESSION | Inflammation - chemically induced | Microglia - metabolism | Neurons - pathology | Nitric Oxide Synthase Type II - biosynthesis | Neurodegenerative Diseases - immunology | Brain - metabolism | Inflammation - metabolism | Microglia - immunology | Chemokine CCL3 - genetics | Chemokine CCL3 - biosynthesis | Microglia - pathology | Neurons - metabolism | Neurons - drug effects | Real-Time Polymerase Chain Reaction | Cell Survival - drug effects | Durapatite - toxicity | Ferric Compounds - toxicity | Microscopy, Electron, Transmission | Neurodegenerative Diseases - pathology | Microglia - drug effects | Nanoparticles - ultrastructure | Neurons - immunology | Rats | Chemokine CCL2 - genetics | Inflammation - immunology | Neurodegenerative Diseases - metabolism | Nanoparticles - toxicity | Rats, Sprague-Dawley | Blotting, Western | Brain - drug effects | DNA - genetics | Titanium - toxicity | DNA - chemistry | Animals | Neurodegenerative Diseases - chemically induced | Silicon Dioxide - toxicity | Nitric Oxide Synthase Type II - genetics | Brain - pathology | Chemokine CCL2 - biosynthesis | Brain - immunology | Nanoparticles | Tyrosine | Neurons | Nitric oxide | Brain damage | Macrophages | Enzyme-linked immunosorbent assay
Journal Article
Glia, ISSN 0894-1491, 12/2017, Volume 65, Issue 12, pp. 1900 - 1913
Brain‐intrinsic degenerative cascades are a proposed factor driving inflammatory lesion formation in multiple sclerosis (MS) patients. We recently described a... 
EAE | cuprizone | inflammation | multiple sclerosis | MULTIPLE-SCLEROSIS LESIONS | ADULT BRAIN | BARRIER PERMEABILITY | SPINAL-CORD | NEUROSCIENCES | ACUTE AXONAL DAMAGE | INDUCED CNS DEMYELINATION | TERM-FOLLOW-UP | CENTRAL-NERVOUS-SYSTEM | ANIMAL-MODEL | T-CELLS | Myelin-Oligodendrocyte Glycoprotein - immunology | Receptors, CCR2 - genetics | Freund's Adjuvant - toxicity | Gene Expression - drug effects | Gene Expression - genetics | Peptide Fragments - toxicity | Receptors, Interleukin-8A - genetics | Encephalomyelitis, Autoimmune, Experimental - immunology | Glial Fibrillary Acidic Protein - metabolism | Myelin-Oligodendrocyte Glycoprotein - toxicity | Peptide Fragments - immunology | Amyloid beta-Protein Precursor - metabolism | Monocytes - pathology | Microglia - pathology | Female | Disease Models, Animal | Monocytes - ultrastructure | Encephalitis - genetics | Mice, Inbred C57BL | Encephalitis - pathology | Mice, Transgenic | Disease Progression | Intercellular Adhesion Molecule-1 - metabolism | Receptors, CCR2 - metabolism | Animals | Microglia - ultrastructure | Receptors, Interleukin-8A - metabolism | Encephalomyelitis, Autoimmune, Experimental - complications | Intercellular Adhesion Molecule-1 - genetics | Sesquiterpenes - toxicity | Luminescent Proteins - genetics | Mice | Encephalitis - etiology | Luminescent Proteins - metabolism | Animal experimentation | Brain | Multiple sclerosis | Encephalomyelitis | Fluorescence | Brain damage | Genetic engineering | Gene expression | Cerebellum | Spinal cord | Forebrain | Substantia grisea | Recruitment | Cuprizone | Demyelination | Extravasation | Cascades | Rodents | Lesions | Cortex | Red fluorescent protein | Transgenic mice | CCL3 protein | Inflammation | Substantia alba | Experimental allergic encephalomyelitis | Microglia | Monocytes | CXCL10 protein | Green fluorescent protein | Leukocytes (granulocytic) | Monocyte chemoattractant protein 1 | Chemokines | Index Medicus
Journal Article
Journal of Biomedical Materials Research Part B: Applied Biomaterials, ISSN 1552-4973, 01/2014, Volume 102, Issue 1, pp. 149 - 159
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 7, p. e11869
Background: Direct cellular contact with stimulated T cells is a potent mechanism that induces cytokine production in human monocytes in the absence of an... 
RHEUMATOID-ARTHRITIS | PLATELET MICROPARTICLES | LYMPH NODE | MULTIPLE-SCLEROSIS | CONTACT-ACTIVATION | RECEPTOR ANTAGONIST PRODUCTION | BIOLOGY | NECROSIS-FACTOR-ALPHA | ENDOTHELIAL MICROPARTICLES | DIFFERENTIAL REGULATION | HIGH-DENSITY-LIPOPROTEINS | Cell Line | Microscopy, Electron, Scanning | Cytokines - metabolism | Humans | Cells, Cultured | Monocytes - metabolism | T-Lymphocytes - ultrastructure | Lipoproteins, HDL - pharmacology | Monocytes - drug effects | T-Lymphocytes - metabolism | Interleukin-1beta - metabolism | Interleukin 1 Receptor Antagonist Protein - metabolism | Cell-Derived Microparticles - metabolism | Cell-Derived Microparticles - ultrastructure | Monocytes - ultrastructure | Rheumatoid factor | Multiple sclerosis | Cytokines | Development and progression | Arthritis | Blood lipids | T cells | Political aspects | Brain | Shear stresses | Microparticles | Proteins | Inhibition | Binding | Cellular manufacture | Blood & organ donations | Internal medicine | CCL3 protein | Cultures | Endothelium | Pathology | Hospitals | Tumor necrosis factor | Rheumatoid arthritis | Biomarkers | Ligands | Monocyte chemoattractant protein 1 | Shedding | Flow cytometry | Membranes | Laboratories | Carbon tetrachloride | Lymphocytes T | Cell surface | Inflammatory diseases | Cell adhesion & migration | Interleukin 6 | Immunology | Lymphocytes | Interleukin 1 | Lipoproteins (high density) | Interleukin 8 | Interleukin 1 receptor antagonist | Cloning | Neutrophils | Inflammation | Cell membranes | CCL4 protein | Endothelial cells | Medicine | Cytometry | Monocytes | Lipoproteins | Chemokines | Cell membranes (biology)
Journal Article
Cellular Signalling, ISSN 0898-6568, 01/2018, Volume 42, pp. 88 - 96
Cell migration towards a chemotactic stimulus relies on the re-arrangement of the cytoskeleton, which is triggered by activation of small G proteins RhoA, Rac1... 
Chemokine receptor | Antagonist | Rac1 | Chemotaxis | Ligand bias | ACTIVATION | METASTASIS | CANCER | CELL BIOLOGY | INDUCED CELL-MIGRATION | G-BETA-GAMMA | INTERNALIZATION | CCR5 | AXIS | ACTS | Humans | Receptors, CCR5 - genetics | cdc42 GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - metabolism | rhoA GTP-Binding Protein - genetics | Pyrones - pharmacology | Quinolines - pharmacology | DNA-Binding Proteins - metabolism | Receptors, CCR5 - metabolism | Peptides - chemical synthesis | MCF-7 Cells | Receptors, CXCR4 - genetics | Aminoquinolines - pharmacology | Amino Acid Sequence | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Jurkat Cells | Gene Expression Regulation | THP-1 Cells | Chemokine CCL3 - pharmacology | Transcription Factors - antagonists & inhibitors | Chemotaxis - drug effects | Cytoskeleton - ultrastructure | Pyrimidines - pharmacology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Peptides - pharmacology | Receptors, CXCR4 - metabolism | Transcription Factors - metabolism | Receptors, CCR1 - genetics | Chemokine CXCL12 - pharmacology | cdc42 GTP-Binding Protein - genetics | Receptors, CCR1 - metabolism | rac1 GTP-Binding Protein - antagonists & inhibitors | Cytoskeleton - metabolism | Cytoskeleton - drug effects | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Phosphates | G proteins | T cells | Muscle proteins | Membrane proteins
Journal Article
Biomaterials, ISSN 0142-9612, 2009, Volume 31, Issue 4, pp. 648 - 657
Abstract This study compares the effects of two polymers currently being marketed on commercially available drug-eluting stents, PVDF-HFP fluorinated copolymer... 
Advanced Basic Science | Dentistry | Endothelialization | Biocompatibility | Phosphorylcholine | Stent | Thrombogenicity | Fluorinated copolymer | MATERIALS SCIENCE, BIOMATERIALS | MEDIATED PLATELET-AGGREGATION | CANINE MODEL | STRUT THICKNESS | ENGINEERING, BIOMEDICAL | DIAMOND-LIKE CARBON | IN-VITRO EVALUATION | IMPLANTATION | SURFACE | FIBRONECTIN ADSORPTION | DRUG-ELUTING STENTS | BLOOD | Thrombosis - chemically induced | Monocytes - cytology | Humans | Male | Monocytes - metabolism | Drug-Eluting Stents - adverse effects | Endothelial Cells - ultrastructure | Fluorocarbon Polymers - pharmacology | Thrombomodulin - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Phosphorylcholine - pharmacology | Chemokine CCL3 - metabolism | Chemokine CCL4 - metabolism | Chemokine CCL2 - metabolism | Chemokine CCL5 - metabolism | Interleukin-6 - metabolism | Granulocyte Colony-Stimulating Factor - metabolism | Cell Line | Rabbits | Microscopy, Electron, Scanning | Endothelial Cells - metabolism | Cell Adhesion - drug effects | Fluorocarbon Polymers - chemistry | Monocytes - drug effects | Animals | Endothelial Cells - cytology | Phosphorylcholine - chemistry | Polymers - chemistry | Endothelial Cells - drug effects | Coatings industry | Biological products | Analysis | Physicians (General practice) | Polymer industry | Coatings | Stent (Surgery) | Endothelium | Index Medicus
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 04/2009, Volume 26, Issue 4, pp. 549 - 561
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