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Journal Article
Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 850 - 858
The success of antitumor immune responses depends on the infiltration of solid tumors by effector T cells, a process guided by chemokines. Here we show that in... 
CD26 EXPRESSION | CELLS | CXCL10 | CHEMOKINE ACTIVITY | IN-VIVO | RECEPTOR | IMMUNOLOGY | CD26/DIPEPTIDYL PEPTIDASE-IV | PROTEINS | CANCER | POSTTRANSLATIONAL MODIFICATION | Dipeptidyl Peptidase 4 - metabolism | Immunotherapy - methods | Male | Adoptive Transfer | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Cell Movement - immunology | Flow Cytometry | Lymphocytes - immunology | Neoplasms, Experimental - immunology | Chemokine CXCL10 - immunology | Neoplasms, Experimental - genetics | Female | Sitagliptin Phosphate | Dipeptidyl Peptidase 4 - immunology | Chemokines - immunology | Lymphocytes - metabolism | Receptors, CXCR3 - metabolism | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Dipeptidyl Peptidase 4 - genetics | Mice, Transgenic | Mice, Knockout | Triazoles - pharmacology | Cell Movement - drug effects | Animals | Receptors, CXCR3 - immunology | Cell Line, Tumor | Chemokines - metabolism | Mice, Inbred BALB C | Pyrazines - pharmacology | Chemokine CXCL10 - metabolism | Care and treatment | Usage | Immunotherapy | Development and progression | Inflammation | Health aspects | Chemokines | Tumors | Neoplasms, Experimental/genetics | Immunotherapy/methods | Cell Movement/drug effects | Neoplasms, Experimental/therapy | Lymphocytes/metabolism | Lymphocytes/immunology | Dipeptidyl Peptidase 4/genetics | Life Sciences | Chemokine CXCL10/immunology | Immunology | Pyrazines/pharmacology | Dipeptidyl Peptidase 4/immunology | Dipeptidyl-Peptidase IV Inhibitors/pharmacology | Chemokines/metabolism | Receptors, CXCR3/immunology | Receptors, CXCR3/metabolism | Neoplasms, Experimental/immunology | Triazoles/pharmacology | Cell Movement/immunology | Chemokines/immunology | Dipeptidyl Peptidase 4/metabolism | Chemokine CXCL10/metabolism
Journal Article
Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7577, pp. 249 - 253
Epigenetic silencing including histone modifications and DNA methylation is an important tumorigenic mechanism. However, its role in cancer immunopathology and... 
Polycomb Repressive Complex 2 - antagonists & inhibitors | CD8-Positive T-Lymphocytes - cytology | Immunotherapy - methods | Prognosis | Histones - chemistry | Chemokine CXCL9 - biosynthesis | Humans | Ovarian Neoplasms - pathology | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Th1 Cells - immunology | DNA (Cytosine-5-)-Methyltransferases - metabolism | Th1 Cells - metabolism | Chemokine CXCL10 - biosynthesis | Chemokine CXCL10 - immunology | Female | Epigenesis, Genetic - drug effects | Lysine - metabolism | Tumor Cells, Cultured | Chemokines - immunology | Tumor Escape - immunology | Chemokines - biosynthesis | DNA (Cytosine-5-)-Methyltransferase 1 | Gene Silencing | Chemokine CXCL9 - immunology | Chemokine CXCL9 - genetics | Chemokines - genetics | Enhancer of Zeste Homolog 2 Protein | Ovarian Neoplasms - enzymology | Xenograft Model Antitumor Assays | B7-H1 Antigen - metabolism | Animals | Chemokine CXCL10 - genetics | Ovarian Neoplasms - therapy | Mice | Histones - metabolism | CD8-Positive T-Lymphocytes - immunology | Polycomb Repressive Complex 2 - metabolism | DNA Methylation - drug effects | Lymphocytes, Tumor-Infiltrating - immunology | Ovarian Neoplasms - immunology | checkpoint | CXCL9 | histone modification | chemotherapy | epigenetics | Chemokine | CXCL10 | DNMT | T cell therapy | DNA methylation | PD-L1 | B7-H1 | cancer | trafficking | PD-1 | EZH2
Journal Article
Nature Immunology, ISSN 1529-2908, 03/2014, Volume 15, Issue 3, pp. 231 - 238
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 03/2010, Volume 159, Issue 3, pp. 338 - 343
Summary Type 1 diabetes results from a T cell-mediated destruction of insulin-producing pancreatic β cells. Little is known on local factors contributing to... 
CXCL10 | type 1 diabetes | IP-10 | CXCR3 | autoreactive T cells | IP‐10 | Autoreactive T cells | Type 1 diabetes | IMMUNE-RESPONSE | NOD MICE | AUTOANTIGENS | ALLOGRAFT | BETA-CELLS | RISK | MELLITUS | IMMUNOLOGY | INFILTRATION | T-CELL RESPONSES | INSULIN EPITOPE | Lymph Nodes - pathology | Humans | Child, Preschool | Male | Diabetes Mellitus, Type 1 - therapy | Enterovirus Infections - immunology | Chemokine CXCL10 - immunology | Adult | Enterovirus Infections - therapy | Female | Retrospective Studies | Diabetes Mellitus, Type 1 - immunology | T-Lymphocytes - pathology | Diabetes Mellitus, Type 1 - virology | Gene Expression Regulation - immunology | Diabetes Mellitus, Type 1 - pathology | Inflammation - virology | Inflammation - immunology | Lymph Nodes - immunology | Insulin-Secreting Cells - immunology | Inflammation - therapy | Insulin-Secreting Cells - virology | Interferon-gamma - immunology | Adolescent | Receptors, CXCR3 - immunology | Enterovirus - immunology | T-Lymphocytes - immunology | Enterovirus Infections - pathology | Insulin-Secreting Cells - pathology | Immunity, Cellular | Interleukin-10 - immunology | Autoimmunity | Diabetics | Pancreatic beta cells | Autoantigens | Inflammation | Biological response modifiers | T cells | Infections | Diabetes | Patients | Viral infections | Chemokines | Immune system | Insulitis | Leukocyte migration | Diabetes mellitus | Data processing | Lymphocytes T | CXCR3 protein | a-Interferon | Chemokine receptors | Lymph nodes | Beta cells | Infection | Interleukins | CXCL10 protein | Remission | Islets of Langerhans | Pancreas | Translational Studies
Journal Article
Journal Article
Hepatology, ISSN 0270-9139, 08/2014, Volume 60, Issue 2, pp. 487 - 496
The pathogenesis of hepatitis C virus (HCV) infection is strongly influenced by the nature of the host's antiviral immunity. Counterintuitively, elevated serum... 
NATURAL-KILLER-CELLS | INJECTION-DRUG USERS | INTERFERON | VIRUS-INFECTION | NK CELLS | APPARENT RESISTANCE | PROTEIN-10 | REACTIVITY | RIBAVIRIN | GASTROENTEROLOGY & HEPATOLOGY | HCV INFECTION | Dipeptidyl Peptidase 4 - metabolism | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Hepatitis C, Chronic - virology | Male | Interferon-gamma - metabolism | T-Lymphocytes - virology | Young Adult | T-Lymphocytes - metabolism | Chemokine CXCL10 - immunology | Interleukin-10 - metabolism | Killer Cells, Natural - immunology | Adult | Female | Adaptive Immunity - immunology | Dipeptidyl Peptidase 4 - immunology | Chemokine CXCL10 - blood | Killer Cells, Natural - virology | Immunity, Innate - immunology | Interferon-gamma - immunology | Hepatitis C, Chronic - immunology | T-Lymphocytes - immunology | Killer Cells, Natural - metabolism | Interleukin-10 - immunology | Longitudinal Studies | Hepatitis | Infections | Drug therapy | Hepatology | Chemokines | Immune system | Killer Cells, Natural/immunology | Interferon-gamma/immunology | Chemokine CXCL10/blood | Life Sciences | Chemokine CXCL10/immunology | Immunology | Hepatitis C, Chronic/immunology | Interleukin-10/immunology | T-Lymphocytes/metabolism | Dipeptidyl Peptidase 4/immunology | Hepatitis C, Chronic/virology | Immunity, Innate/immunology | Adaptive Immunity/immunology | T-Lymphocytes/immunology | Interferon-gamma/metabolism | T-Lymphocytes/virology | Killer Cells, Natural/metabolism | Killer Cells, Natural/virology | Dipeptidyl Peptidase 4/metabolism | Interleukin-10/metabolism
Journal Article
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