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1. Full Text CXCL13–CXCR5 co-expression regulates epithelial to mesenchymal transition of breast cancer cells during lymph node metastasis
by Biswas, Subir and Biswas, Subir and Sengupta, Suman and Sengupta, Suman and Roy Chowdhury, Sougata and Roy Chowdhury, Sougata and Jana, Samir and Jana, Samir and Mandal, Gunjan and Mandal, Gunjan and Mandal, Palash Kumar and Mandal, Palash Kumar and Saha, Nipun and Saha, Nipun and Malhotra, Vivek and Malhotra, Vivek and Gupta, Arnab and Gupta, Arnab and Kuprash, Dmitry V and Kuprash, Dmitry V and Bhattacharyya, Arindam and Bhattacharyya, Arindam
Breast Cancer Research and Treatment, ISSN 0167-6806, 1/2014, Volume 143, Issue 2, pp. 265 - 276
We investigated the expression of –CXC chemokine ligand 13 (CXCL13) and its receptor –CXC chemokine receptor 5 (CXCR5) in 98 breast cancer (BC) patients with... 
Lymph node metastasis | Epithelial to mesenchymal transition | RANKL | Medicine & Public Health | CXCR5 | Oncology | Breast cancer | CXCL13 | ANGIOGENESIS | TUMOR | CXCR5 CHEMOKINE RECEPTOR | NEUROBLASTOMA-CELLS | INVASION | MELANOMA | ONCOLOGY | MAMMARY-GLAND DEVELOPMENT | PROSTATE-CANCER | TRANSCRIPTIONAL REGULATION | EXPRESSION | Antigens, CD - biosynthesis | Furans - pharmacology | Humans | Middle Aged | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Cadherins - biosynthesis | RNA, Messenger - biosynthesis | Lymphatic Metastasis - pathology | Epithelial-Mesenchymal Transition | Biomarkers, Tumor - metabolism | Adult | Female | Indoles - pharmacology | Snail Family Transcription Factors | Chemokine CXCL13 - antagonists & inhibitors | Matrix Metalloproteinase 9 - biosynthesis | Receptors, CXCR5 - metabolism | Signal Transduction | Vimentin - biosynthesis | src-Family Kinases - antagonists & inhibitors | Transcription Factors - biosynthesis | RANK Ligand - biosynthesis | Pyrimidines - pharmacology | Sulfonamides - pharmacology | RANK Ligand - genetics | Receptors, CXCR5 - antagonists & inhibitors | Breast Neoplasms - pathology | Chemokine CXCL13 - biosynthesis | Chemokine CXCL13 - metabolism | Cell Line, Tumor | Receptors, CXCR5 - biosynthesis | Aged | Pyridines - pharmacology | Cell Movement | Metastasis | Gene expression | Health aspects | Intermediate filament proteins | Stem cells
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2. Full Text Early BAFF receptor blockade mitigates murine Sjögren's syndrome: Concomitant targeting of CXCL13 and the BAFF receptor prevents salivary hypofunction
by Sharma, Arjun and Kiripolsky, Jeremy and Klimatcheva, Ekaterina and Howell, Alan and Fereidouni, Farzad and Levenson, Richard and Rothstein, Thomas L and Kramer, Jill M
Clinical Immunology, ISSN 1521-6616, 2016, Volume 164, pp. 85 - 94
Abstract Sjögren's syndrome (SS) is a debilitating autoimmune disease. Patients with SS may develop xerostomia. This process is progressive, and there are no... 
Allergy and Immunology | Sialadenitis | BAFF receptor | Autoantibody | CXCL13 | Sjögren's syndrome | Salivary hypofunction | Sjogren's syndrome | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PHASOR ANALYSIS | MARGINAL ZONE | FACTOR FAMILY BAFF | PERIPHERAL-BLOOD | IMMUNOLOGY | MALT LYMPHOMA | B-CELLS | CELL-ACTIVATING FACTOR | CLASSIFICATION CRITERIA | AUTOANTIBODY PRODUCTION | Salivary Glands - pathology | Sjogren's Syndrome - pathology | Sialadenitis - prevention & control | Sjogren's Syndrome - immunology | Salivary Glands - physiology | B-Cell Activation Factor Receptor - antagonists & inhibitors | Sialadenitis - pathology | Animals | Saliva - metabolism | Antibodies - immunology | Female | Mice, Inbred NOD | Mice | Chemokine CXCL13 - immunology | Sjogren's Syndrome - prevention & control | B-Cell Activation Factor Receptor - immunology | Chemokine CXCL13 - antagonists & inhibitors | Disease Models, Animal | Sialadenitis - immunology | Autoimmunity | Immunoglobulin G | Autoantibodies | Rheumatoid factor | Therapeutics | Cells | Homeopathy | Materia medica and therapeutics | autoantibody | sialadenitis | salivary hypofunction
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3. Full Text Effects of CXCL13 inhibition on lymphoid follicles in models of autoimmune disease
by Finch, Donna K and Ettinger, Rachel and Karnell, Jodi L and Herbst, Ronald and Sleeman, Matthew A
European Journal of Clinical Investigation, ISSN 0014-2972, 05/2013, Volume 43, Issue 5, pp. 501 - 509
The chemokine CXCL13 has a key role in secondary lymphoid tissue orchestration and lymphoid neogenesis. Transgenic mice deficient in CXCL13 or its receptor... 
Antibodies | ectopic follicle | CXCL13 | lymphoid follicle | rheumatoid arthritis | Lymphoid follicle | Ectopic follicle | Rheumatoid arthritis | RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | COLLAGEN-INDUCED ARTHRITIS | CD4 T-CELLS | CELL-ATTRACTING CHEMOKINE-1 | SJOGRENS-SYNDROME | GERMINAL CENTER | SALIVARY-GLANDS | B-CELLS | MEDICINE, GENERAL & INTERNAL | FOLLICULAR-HELPER-CELLS | MULTIPLE-SCLEROSIS | Animals | Autoimmune Diseases - immunology | Mice, Transgenic | Mice | Chemokine CXCL13 - immunology | Receptors, CXCR5 - immunology | Chemokine CXCL13 - antagonists & inhibitors | Disease Models, Animal | Lymphoid Tissue - immunology | Rheumatoid factor | Genetic engineering | Models | Analysis
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4. Full Text Upregulation of (C-X-C motif) ligand 13 (CXCL13) attenuates morphine analgesia in rats with cancer-induced bone pain
by Wang, Shi-Feng and Dong, Cheng-Gong and Yang, Xue and Yin, Jian-Jun
Medical Science Monitor, ISSN 1234-1010, 11/2016, Volume 22, pp. 4612 - 4622
Background: The aim of this study was to investigate the role of chemokine (C-X-C motif) ligand 13 (CXCL13) in morphine tolerance in rats with cancer-induced... 
Chemokine CXCL13 | Rats | Models | Animal | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | TOLERANCE | INVOLVEMENT | SPINAL-CORD | MECHANISMS | NEUROPATHIC PAIN | OPIOID RECEPTOR | MICROGLIA | Models, Animal | CONTRIBUTES | Spinal Cord - metabolism | Analgesia - methods | Mammary Neoplasms, Experimental - physiopathology | Bone Neoplasms - pathology | RNA, Messenger - biosynthesis | Antibodies, Neutralizing - immunology | Bone Neoplasms - physiopathology | Mammary Neoplasms, Experimental - pathology | Female | Bone Neoplasms - drug therapy | Chemokine CXCL13 - immunology | Chemokine CXCL13 - antagonists & inhibitors | Disease Models, Animal | Mammary Neoplasms, Experimental - drug therapy | Morphine - pharmacology | Cancer Pain - physiopathology | RNA, Messenger - genetics | Drug Tolerance | Pain Management - methods | Antibodies, Neutralizing - pharmacology | Random Allocation | Rats, Sprague-Dawley | Cancer Pain - drug therapy | Up-Regulation - drug effects | Animals | Chemokine CXCL13 - biosynthesis
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5. Full Text CXCL13 blockade disrupts B lymphocyte organization in tertiary lymphoid structures without altering B cell receptor bias or preventing diabetes in nonobese diabetic mice
by Henry, Rachel A and Kendall, Peggy L
Journal of Immunology, ISSN 0022-1767, 08/2010, Volume 185, Issue 3, pp. 1460 - 1465
Lymphocytes that invade nonlymphoid tissues often organize into follicle-like structures known as tertiary lymphoid organs (TLOs). These structures resemble... 
RECRUITMENT | ACTIVATION | RHEUMATOID SYNOVITIS | PANCREATIC-ISLETS | NOD MICE | NEOGENESIS | LYMPHOTOXIN-BETA-RECEPTOR | SOMATIC HYPERMUTATION | SJOGRENS-SYNDROME | IMMUNOLOGY | EXPRESSION | Chemokine CXCL13 - physiology | Diabetes Mellitus, Type 1 - prevention & control | Lymphoid Tissue - immunology | Islets of Langerhans - metabolism | Inflammation Mediators - metabolism | B-Lymphocyte Subsets - immunology | Female | Receptors, Antigen, B-Cell - physiology | Inflammation Mediators - antagonists & inhibitors | Diabetes Mellitus, Type 1 - immunology | Organ Culture Techniques | Chemokine CXCL13 - antagonists & inhibitors | Chemokine CXCL13 - genetics | Receptors, CXCR5 - metabolism | B-Lymphocyte Subsets - pathology | Islets of Langerhans - pathology | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - pathology | Islets of Langerhans - immunology | Lymphoid Tissue - metabolism | Pancreas - pathology | Pancreas - metabolism | Pancreas - immunology | Disease Progression | Lymphoid Tissue - pathology | Animals | B-Lymphocyte Subsets - metabolism | Mice, Inbred NOD | Mice
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6. Full Text CXCL13 blockade attenuates lupus nephritis of MRL/lpr mice
by Wu, Xiaobei and Guo, Jialong and Ding, Rui and Lv, Bin and Bi, Liqi
Acta Histochemica, ISSN 0065-1281, 10/2015, Volume 117, Issue 8, pp. 732 - 737
The chemokine CXC ligand 13 protein (CXCL13) is reported to closely related to the disease activity and severity of systemic lupus erythematosus (SLE),... 
MRL/lpr mouse | Lupus nephritis | Cxcl13 | Neutralization | MORTALITY | CELL BIOLOGY | ELEVATED PRODUCTION | IN-VITRO | ERYTHEMATOSUS SLE | RENAL INVOLVEMENT | COHORT | CHEMOKINE CXCL13 | AUTOIMMUNE-DISEASE | EXPRESSION | SEVERITY | Chemokine CXCL13 - physiology | Kidney - drug effects | Autoantibodies - blood | Lupus Nephritis - drug therapy | CD4 Lymphocyte Count | Lupus Nephritis - blood | T-Lymphocytes, Regulatory - immunology | Kidney - metabolism | Mice, Inbred MRL lpr | Animals | Th17 Cells - immunology | Female | Drug Evaluation, Preclinical | Chemokine CXCL13 - antagonists & inhibitors | Lupus | Nephritis | Systemic lupus erythematosus | Immunoglobulin G
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7. Full Text A Combination of Structural and Empirical Analyses Delineates the Key Contacts Mediating Stability and Affinity Increases in an Optimized Biotherapeutic Single-chain Fv (scFv)
by Tu, C and Terraube, V and Tam, ASP and Stochaj, W and Fennell, BJ and Lin, L and Stahl, M and LaVallie, ER and Somers, W and Finlay, WJJ and Mosyak, L and Bard, J and Cunningham, O and Argonne National Lab. (ANL), Argonne, IL (United States). Advanced Photon Source (APS)
JOURNAL OF BIOLOGICAL CHEMISTRY, ISSN 0021-9258, 01/2016, Volume 291, Issue 3, pp. 1267 - 1276
Fully-human single-chain Fv (scFv) proteins are key potential building blocks of bispecific therapeutic antibodies, but they often suffer from... 
BISPECIFIC ANTIBODIES | BIOPHYSICAL PROPERTIES | FRAGMENTS | COMPUTATIONAL DESIGN | FAB | BIOCHEMISTRY & MOLECULAR BIOLOGY | IN-VIVO | LIBRARY | LIGAND | MATURATION | VARIABLE DOMAINS | Protein Aggregates | Antibody Specificity | Complementarity Determining Regions - genetics | Chemokine CXCL13 - chemistry | Humans | Recombinant Fusion Proteins - metabolism | Single-Chain Antibodies - metabolism | Single-Chain Antibodies - genetics | Complementarity Determining Regions - chemistry | X-Ray Diffraction | Antigen-Antibody Complex - metabolism | Protein Stability | Complementarity Determining Regions - metabolism | Chemokine CXCL13 - antagonists & inhibitors | Recombinant Proteins - metabolism | Solubility | Models, Molecular | Recombinant Proteins - chemistry | Antibody Affinity | Recombinant Fusion Proteins - chemistry | Biological Products - chemistry | Antigen-Antibody Complex - chemistry | Chemokine CXCL13 - metabolism | Biological Products - metabolism | Protein Conformation | Kinetics | Mutation | Binding Sites, Antibody | Single-Chain Antibodies - chemistry | Amino Acid Substitution | Index Medicus | antibody engineering | crystal structure | mutagenesis in vitro | Protein Structure and Folding | chemokine | protein stability
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8. Full Text CXCL13 regulates the trafficking of GluN2B-containing NMDA receptor via IL-17 in the development of remifentanil-induced hyperalgesia in rats
by Zhu, M and Yuan, S.T and Yu, W.L and Jia, L.L and Sun, Y
Neuroscience Letters, ISSN 0304-3940, 05/2017, Volume 648, pp. 26 - 33
This study aimed to investigate whether CXCL13 modulated the trafficking of NMDA receptor via interleukin (IL)-17 in a rat model of remifentanil-induced... 
CXCR5 | CXCL13 | Remifentanil-induced hyperalgesia | GluN2B | IL-17 | PHOSPHORYLATION | MECHANISMS | PATHOLOGICAL PAIN | NEUROSCIENCES | OPIOID-INDUCED HYPERALGESIA | NEUROPATHIC PAIN | INDUCED HYPERNOCICEPTION | INFLAMMATORY PAIN | INDUCED POSTOPERATIVE HYPERALGESIA | UP-REGULATION | CONTRIBUTES | Hyperalgesia - chemically induced | Interleukin-17 - administration & dosage | Spinal Cord Dorsal Horn - metabolism | Hyperalgesia - metabolism | Nociception - physiology | Receptors, N-Methyl-D-Aspartate - metabolism | Rats | Male | Protein Transport - drug effects | RNA, Messenger - metabolism | Rats, Sprague-Dawley | Chemokine CXCL13 - administration & dosage | Recombinant Proteins - administration & dosage | Interleukin-17 - metabolism | Spinal Cord Dorsal Horn - drug effects | Animals | Chemokine CXCL13 - metabolism | Piperidines | Nociception - drug effects | Pain Threshold - drug effects | Interleukins | Methyl aspartate | Yuan (China)
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9. Full Text Lymphotoxin-beta receptor blockade reduces CXCL13 in lacrimal glands and improves corneal integrity in the NOD model of Sjögren's syndrome
by Fava, Roy A and Kennedy, Susan M and Wood, Sheryl G and Bolstad, Anne I and Bienkowska, Jadwiga and Papandile, Adrian and Kelly, John A and Mavragani, Clio P and Gatumu, Margaret and Skarstein, Kathrine and Browning, Jeffrey L
Arthritis Research and Therapy, ISSN 1478-6354, 11/2011, Volume 13, Issue 6, pp. R182 - R182
Introduction: In Sjogren's syndrome, keratoconjunctivitis sicca (dry eye) is associated with infiltration of lacrimal glands by leukocytes and consequent... 
Chemokine | Keratoconjuntivitis sicca | Sjogren's syndrome | CXCL13 | NOD mouse | Lymphotoxin-beta | chemokine | keratoconjuntivitis sicca | RHEUMATOLOGY | SALIVARY-GLANDS | TARGET ORGAN | TUMOR-NECROSIS-FACTOR | LYMPHOCYTIC INFILTRATION | LYMPHOID-TISSUE | DENDRITIC CELL NETWORKS | MURINE LUPUS | GERMINAL-CENTERS | CHEMOKINE CXCL13 | AUTOANTIBODY PRODUCTION | Immunohistochemistry | Gene Expression - drug effects | Humans | Middle Aged | Venules - physiology | Tears - metabolism | Endothelium, Vascular - drug effects | Male | Gene Expression Profiling | Sjogren's Syndrome - metabolism | Endothelium, Vascular - physiology | Lymphotoxin beta Receptor - metabolism | Cornea - drug effects | Keratoconjunctivitis Sicca - drug therapy | Mucins - metabolism | Adult | Female | Antibodies, Monoclonal - immunology | Sjogren's Syndrome - genetics | Chemokine CXCL13 - genetics | Lacrimal Apparatus - drug effects | Sjogren's Syndrome - drug therapy | Antibodies, Monoclonal - pharmacology | Keratoconjunctivitis Sicca - metabolism | Keratoconjunctivitis Sicca - genetics | Lacrimal Apparatus - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Cornea - metabolism | Venules - metabolism | Animals | Endothelium, Vascular - metabolism | Chemokine CXCL13 - metabolism | Mice, Inbred NOD | Aged | Lymphotoxin beta Receptor - antagonists & inhibitors | Mice | Microscopy, Fluorescence | Mucins - genetics | Lymphotoxin beta Receptor - immunology | Care and treatment | Lacrimal organs | Physiological aspects | Genetic aspects | Research | Diagnosis | Gene expression | Risk factors | Sjögren’s syndrome
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10. Full Text Baseline CXCL10 and CXCL13 levels are predictive biomarkers for tumor necrosis factor inhibitor therapy in patients with moderate to severe rheumatoid arthritis: A pilot, prospective study
by Han, Bobby Kwanghoon and Kuzin, Igor and Gaughan, John P and Olsen, Nancy J and Bottaro, Andrea
Arthritis Research and Therapy, ISSN 1478-6354, 04/2016, Volume 18, Issue 1, p. 93
Background: TNF inhibitors have been used as a treatment for moderate to severe RA patients. However, reliable biomarkers that predict therapeutic response to... 
CXCL10 | CXCL13 | TNF inhibitor | Rheumatoid arthritis | CHEMOKINE EXPRESSION | CELL CHEMOATTRACTANT CXCL13 | INFLAMMATORY ARTHRITIS | RHEUMATOLOGY | I INTERFERON | DISEASE-ACTIVITY | FACTOR-ALPHA | TNF-ALPHA | RECEPTORS | ASSOCIATION | BLOOD | Chemokine CXCL10 - blood | Prospective Studies | Enzyme-Linked Immunosorbent Assay | Arthritis, Rheumatoid - blood | Humans | Middle Aged | Male | Biomarkers - blood | Chemokine CXCL13 - blood | Adalimumab - therapeutic use | Pilot Projects | Arthritis, Rheumatoid - drug therapy | Adult | Female | Aged | Arthritis, Rheumatoid - immunology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Antirheumatic Agents - therapeutic use | Etanercept - therapeutic use | Usage | Tumor necrosis factor inhibitors | Patient outcomes | Research | Biological markers | Drug therapy
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11. Full Text CXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5
by Jiang, Bao-Chun and Cao, De-Li and Zhang, Xin and Zhang, Zhi-Jun and He, Li-Na and Li, Chun-Hua and Zhang, Wen-Wen and Wu, Xiao-Bo and Berta, Temugin and Ji, Ru-Rong and Gao, Yong-Jing
Journal of Clinical Investigation, ISSN 0021-9738, 02/2016, Volume 126, Issue 2, pp. 745 - 761
Recent studies have implicated chemokines in microglial activation and pathogenesis of neuropathic pain. C-X-C motif chemokine 13 (CXCL13)is a B lymphocyte... 
MEDICINE, RESEARCH & EXPERIMENTAL | MECHANICAL ALLODYNIA | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | CENTRAL SENSITIZATION | LYME NEUROBORRELIOSIS | P2X RECEPTOR | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CHEMOKINE RECEPTOR | CENTRAL-NERVOUS-SYSTEM | TNF-ALPHA | UP-REGULATION | Chemokine CXCL13 - genetics | Neuralgia - genetics | Receptors, CXCR5 - metabolism | Spinal Cord - metabolism | Receptors, CXCR5 - genetics | Humans | Astrocytes - pathology | Neuralgia - metabolism | MicroRNAs - metabolism | Neuralgia - pathology | Mice, Inbred ICR | Mice, Knockout | Animals | MAP Kinase Signaling System - genetics | Spinal Cord - pathology | Chemokine CXCL13 - metabolism | Mice | MicroRNAs - genetics | Astrocytes - metabolism | Studies | Biotechnology | Spinal cord | Lymphatic system | Genes | Rodents | Pain management | Behavior | Chemokines
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12. Full Text The CXCR5 receptor, a new target in inflammation?
by Norman, Peter
Expert Opinion on Therapeutic Patents, ISSN 1354-3776, 10/2008, Volume 18, Issue 10, pp. 1209 - 1210
A method of treating CXCL13-mediated inflammatory diseases by the co-administration of a CXCL13 antagonist and a TNF-α antagonist is claimed. Their use is... 
chemokine | inflammation | pulmonary fibrosis | CXCR5 | asthma | CXCL13 | lupus | COPD | CHEMISTRY, MEDICINAL | PHARMACOLOGY & PHARMACY
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13. Full Text CXCL13 and CCL11 Serum Levels and Lymphoma and Disease Activity in Primary Sjögren's Syndrome
by Nocturne, G and Seror, R and Fogel, O and Belkhir, R and Boudaoud, S and Saraux, A and Larroche, C and Le Guern, V and Gottenberg, J. E and Mariette, X
Arthritis & Rheumatology, ISSN 2326-5191, 12/2015, Volume 67, Issue 12, pp. 3226 - 3233
Objective Non‐Hodgkin's lymphoma (NHL) is a severe complication of primary Sjögren's syndrome (SS). Ectopic germinal centers (GCs) in the salivary glands are... 
GERMINAL CENTER FORMATION | B-CELLS | PATHOGENESIS | ORGANIZATION | EARLY RHEUMATOID-ARTHRITIS | IMMUNE-RESPONSES | CHEMOKINE RECEPTOR | RHEUMATOLOGY | SALIVARY-GLANDS | EXPRESSION | ASSOCIATION | Severity of Illness Index | Lymphoma, Non-Hodgkin - immunology | Multivariate Analysis | Prospective Studies | Lymphocyte Activation | Humans | Middle Aged | Risk Factors | Sjogren's Syndrome - immunology | Male | Complement C4 - immunology | B-Lymphocytes - immunology | Biomarkers | B-Cell Activating Factor - immunology | Female | Aged | Chemokine CCL11 - immunology | Chemokine CXCL13 - immunology | Cohort Studies | Multivariate analysis | Lymphomas | Chemokines | Life Sciences | Human health and pathology | Rhumatology and musculoskeletal system
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14. Full Text RGS13 Regulates Germinal Center B Lymphocytes Responsiveness to CXC Chemokine Ligand (CXCL)12 and CXCL13
by Shi, Geng-Xian and Harrison, Kathleen and Wilson, Gaye Lynn and Moratz, Chantal and Kehrl, John H
The Journal of Immunology, ISSN 0022-1767, 09/2002, Volume 169, Issue 5, pp. 2507 - 2515
Normal lymphoid tissue development and function depend upon directed cell migration. Providing guideposts for cell movement and positioning within lymphoid... 
ALPHA-SUBUNITS | GAIP | DOMAIN | HETEROTRIMERIC G-PROTEINS | GENE | CHEMOTAXIS | GTPASE-ACTIVATING PROTEINS | LYMPHOID ORGANS | FAMILY MEMBERS | IMMUNOLOGY | RECEPTORS | Green Fluorescent Proteins | RGS Proteins - genetics | Germinal Center - immunology | Humans | B-Lymphocyte Subsets - cytology | Molecular Sequence Data | Heterotrimeric GTP-Binding Proteins - metabolism | RGS Proteins - physiology | Recombinant Fusion Proteins - metabolism | RNA, Messenger - biosynthesis | Signal Transduction - immunology | RGS Proteins - metabolism | B-Lymphocyte Subsets - immunology | Germinal Center - cytology | Intracellular Fluid - metabolism | CHO Cells | Chemokine CXCL13 | DNA, Complementary - analysis | Amino Acid Sequence | Chemokine CXCL12 | Chemokines, CXC - antagonists & inhibitors | Cricetinae | Gene Expression Regulation - immunology | Stromal Cells - metabolism | RGS Proteins - biosynthesis | Receptors, Cell Surface - metabolism | Stromal Cells - immunology | Chromosome Mapping | Signal Transduction - genetics | Genetic Markers - immunology | Animals | Germinal Center - metabolism | B-Lymphocyte Subsets - metabolism | K562 Cells | HL-60 Cells | Chemokines, CXC - physiology | Luminescent Proteins - genetics | Mice | HeLa Cells | COS Cells | Luminescent Proteins - metabolism
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