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Journal of Investigative Dermatology, ISSN 0022-202X, 02/2018, Volume 138, Issue 2, pp. 344 - 354
Journal Article
Gut, ISSN 0017-5749, 02/2019, Volume 68, Issue 2, pp. 335 - 346
ObjectivesYttrium-90 (Y90)-radioembolisation (RE) significantly regresses locally advanced hepatocellular carcinoma and delays disease progression. The current... 
SURVIVAL | NATURAL-KILLER | SOLID TUMORS | RADIOEMBOLIZATION | T-CELL DYSFUNCTION | TIM-3 | GASTROENTEROLOGY & HEPATOLOGY | RADIATION | EXPRESSION | TUMOR MICROENVIRONMENT | INFILTRATION | Flow Cytometry - methods | Chemokine CXCL16 - immunology | Singapore | Humans | Carcinoma, Hepatocellular - radiotherapy | Liver Neoplasms - immunology | Male | Treatment Outcome | Antigen-Presenting Cells - immunology | Disease Progression | Leukocytes, Mononuclear - immunology | Liver Neoplasms - radiotherapy | Tumor Necrosis Factor-alpha - immunology | Female | Carcinoma, Hepatocellular - immunology | Granzymes - immunology | Yttrium Radioisotopes | Biomarkers, Tumor - immunology | Chemokine CCL5 - immunology | Lymphocytes, Tumor-Infiltrating - immunology | Disease | PD-1 protein | CD8 antigen | Hepatocellular carcinoma | Leukocytes (mononuclear) | Lymphocytes T | Leukocytes | Cancer therapies | Metastases | Liver cancer | Cell activation | Granzyme B | Next-generation sequencing | Prediction models | Lymphocytes | Immunotherapy | Cell cycle | CXCL16 protein | Peripheral blood mononuclear cells | Natural killer cells | CD56 antigen | Bioinformatics | CCR5 protein | Homing receptors | Cytokines | Radiation therapy | CD4 antigen | Cytometry | Tumor necrosis factor | Antigen-presenting cells | Medical prognosis | Yttrium | Chemokines | Cell migration | Tumors | 2312 | Hepatology | biomarkers | immune activation | Time-of-flight Mass Cytometry (CyTOF) | tumor microenvironment | 1506 | Y90 radioembolization | hepatocellular carcinoma | chemotaxis
Journal Article
Cellular and Molecular Immunology, ISSN 1672-7681, 01/2019, Volume 16, Issue 1, pp. 86 - 97
ILC2s are implicated in asthma pathogenesis, but little is known about the mechanisms underlying their accumulation in airways. We investigated the time course... 
Chemokine | ILC2s | IL-25 | IL-33 | Accumulation | Asthma | MIGRATION | IMMUNITY | ACTIVATION | IMMUNOLOGY | RESPONSES | INFLAMMATION | REGULATORS | DIFFERENTIATION | AIRWAYS | EXPRESSION | T-CELLS
Journal Article
Developmental and Comparative Immunology, ISSN 0145-305X, 08/2018, Volume 85, pp. 86 - 94
Chemokine CXCL16 and its receptor CXCR6 are associated with a series of physiological and pathological processes in cooperative and stand-alone fashions. To... 
Hominoids | CXCR6 | Diversification | Cercopithecoids | CXCL16 | MAXIMUM-LIKELIHOOD | PROTEIN | CANCER CELLS | AMINO-ACID SITES | IMMUNOLOGY | EXPRESSION CLONING | MEMBRANE-BOUND CHEMOKINE | FISHERIES | SCAVENGER RECEPTOR | ZOOLOGY | TNF-ALPHA | FUNCTIONAL DIVERGENCE | T-CELLS | Animal genetics | Genetic research | Monkeys | Phylogeny | Analysis
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2012, Volume 188, Issue 5, pp. 2093 - 2101
Journal Article
Atherosclerosis, ISSN 0021-9150, 2007, Volume 197, Issue 2, pp. 487 - 495
Abstract Chemokines, scavenger receptors and adhesion molecules have long been known as important players in the pathogenesis of atherosclerosis. A series of... 
Cardiovascular | Atherosclerosis | SR-PSOX | Scavenger receptors | Adhesion molecules | CXCR6 | Coronary artery disease | Chemokines | Chemokine receptors | CXCL16 | atherosclerosis | coronary artery disease | chemokine receptors | CARDIAC & CARDIOVASCULAR SYSTEMS | IFN-GAMMA | LOW-DENSITY-LIPOPROTEIN | TRANSMEMBRANE CHEMOKINES | chemokines | scavenger receptors | LIVER-INFILTRATING LYMPHOCYTES | SCAVENGER RECEPTOR | INTERFERON-GAMMA | adhesion molecules | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CXC CHEMOKINE LIGAND-16 | T-CELLS | Chemokines, CXC - immunology | Receptors, CXCR - immunology | Atherosclerosis - immunology | Atherosclerosis - physiopathology | Chemokine CXCL16 | Humans | Endothelium, Vascular - physiopathology | Chemokine CXCL6 - immunology | Receptors, Chemokine - immunology | Receptors, Scavenger - immunology | Animals | Biomarkers | Receptors, Virus - immunology | Mice | Disease Models, Animal | Endothelium, Vascular - immunology | Receptors, CXCR6 | Development and progression | Blood lipids | Medical and Health Sciences | Receptors; Scavenger/immunology | Medicin och hälsovetenskap | Biological Markers | Chemokine CXCL6/immunology | Endothelium; Vascular/immunology/physiopathology | Klinisk medicin | Receptors; Chemokine/immunology | MEDICINE | Kardiologi | Invärtesmedicin | Dermatologi och venerologi, klinisk genetik, invärtesmedicin | Disease Models; Animal | Internal medicine | Receptors; CXCR/immunology | Atherosclerosis/immunology/physiopathology | Kardiovaskulär medicin | Dermatology and venerology,clinical genetics, internal medicine | Clinical Medicine | Cardiac and Cardiovascular Systems | Chemokines; CXC/immunology | Receptors; Virus/immunology | MEDICIN | Cardiovascular medicine
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 140, Issue 1, pp. 177 - 189.e9
Background In patients with vitiligo, an increased reactive oxygen species (ROS) level has been proved to be a key player during disease initiation and... 
Allergy and Immunology | skin migration | Oxidative stress | unfolded protein response | CD8+ T cells | CXCR6 | CXCL16 | CD8 | T cells | RHEUMATOID-ARTHRITIS | IMMUNE-RESPONSE | AUTOIMMUNE VITILIGO | IMMUNOLOGY | HUMAN MELANOCYTES | GENERALIZED VITILIGO | ALLERGY | MOUSE MODEL | CD8(+) T cells | HUMAN-MELANOMA CELLS | CHEMOKINE CXCL16 | ENDOPLASMIC-RETICULUM | HYDROGEN-PEROXIDE | Cell Line | Up-Regulation | Endoribonucleases - genetics | Humans | Vitiligo - immunology | Cells, Cultured | Oxidative Stress - genetics | Oxidative Stress - immunology | Protein-Serine-Threonine Kinases - genetics | RNA, Messenger - metabolism | Unfolded Protein Response | CD8-Positive T-Lymphocytes - physiology | Keratinocytes - immunology | Vitiligo - blood | Hydrogen Peroxide - immunology | Eukaryotic Initiation Factor-2 - genetics | CD8-Positive T-Lymphocytes - immunology | X-Box Binding Protein 1 - genetics | Cell Movement | Cytokines - immunology | Skin - immunology | eIF-2 Kinase - genetics | Hydrogen peroxide | Leukocyte migration | CD8 antigen | Trafficking | Melanocytes | Lymphocytes T | Activation | Western blotting | Proteins | Homing | Vitiligo | Lymphocytes | Protein folding | Rodents | CXCL16 protein | Activating transcription factor 1 | Lesions | Enzyme-linked immunosorbent assay | Inositol | Immunoglobulins | Keratinocytes | Light therapy | Real time | Ribonucleic acid--RNA | Chemotaxis | Patients | Studies | Polymerase chain reaction | Rheumatoid arthritis | Skin | Infiltration | Aberration | Immunofluorescence | Endoplasmic reticulum | Cell migration | Chemokines
Journal Article
Journal Article
Journal Article
Inflammatory Bowel Diseases, ISSN 1078-0998, 11/2010, Volume 16, Issue 11, pp. 1871 - 1881
Background: CXCL16 mediates adhesion and phagocytosis of both Gram‐negative and Gram‐positive bacteria and is a strong chemoattractant for CXCR6+ T cells. In... 
cytokine | intestinal epithelial cells | chemokine | signaling | mouse model | Crohn's disease | ulcerative colitis | CXCR6 | intestinal inflammation | inflammatory bowel disease | CXCL16 | Crohns disease | FRACTALKINE | MIGRATION | ACTIVATION | CANCER CELLS | PROTEIN-KINASE | PROLIFERATION | EPITHELIAL-CELLS | RECEPTOR CXCR6 | TNF-ALPHA | LIGAND | GASTROENTEROLOGY & HEPATOLOGY | Up-Regulation | Chemokines, CXC - immunology | Receptors, CXCR - immunology | Receptors, Virus - metabolism | Chemokines, CXC - blood | Colitis - virology | Humans | Chemokine CXCL6 - blood | Crohn Disease - virology | Crohn Disease - metabolism | Colitis - pathology | Colon - immunology | Chemokine CXCL6 - metabolism | Ileitis - pathology | Herpesviridae Infections - pathology | Mitogen-Activated Protein Kinases - analysis | T-Lymphocytes - metabolism | Receptors, CXCR - metabolism | Herpesviridae Infections - metabolism | Ileitis - metabolism | Colitis - immunology | Proto-Oncogene Proteins c-akt - metabolism | Receptors, Scavenger - metabolism | Cell Line | Chemokine CXCL16 | Receptors, Chemokine - metabolism | Chemokine CXCL6 - immunology | Receptors, Scavenger - blood | Receptors, Chemokine - immunology | Crohn Disease - immunology | Colon - metabolism | Muromegalovirus - metabolism | Muromegalovirus - immunology | Receptors, Scavenger - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Crohn Disease - pathology | Ileitis - immunology | Chemokines, CXC - metabolism | Colitis - metabolism | Receptors, Virus - immunology | T-Lymphocytes - immunology | Mice | Herpesviridae Infections - immunology | Mitogen-Activated Protein Kinases - metabolism | Receptors, CXCR6 | Chemotactic factors | Animal models | Inflammatory bowel diseases | Epithelial cells | Colorectal cancer | Antibodies | Gram-positive bacteria | AKT protein | Lymphocytes T | Small intestine | Signal transduction | Intestine | CXCL16 protein | Enzyme-linked immunosorbent assay | Interleukin 8 | Ileitis | MAP kinase | Inflammation | Gene expression | Polymerase chain reaction | Serum levels | Biopsy | Colitis | Chemokines | Phagocytosis
Journal Article
Kidney International, ISSN 0085-2538, 06/2012, Volume 81, Issue 11, pp. 1098 - 1107
TWEAK (tumor necrosis factor–like weak inducer of apoptosis) is a TNF superfamily cytokine that activates the fibroblast growth factor–inducible 14 (Fn14)... 
chemokine | acute kidney injury | glomerulonephritis | inflammation | LOW-DENSITY-LIPOPROTEIN | SR-PSOX | INJURY | PROLIFERATION | VEIN ENDOTHELIAL-CELLS | TUBULAR EPITHELIUM | SCAVENGER RECEPTOR | MESANGIAL CELLS | UROLOGY & NEPHROLOGY | CHEMOKINE CXCL16 | NF-KAPPA-B | Up-Regulation | Humans | Middle Aged | Male | NF-kappa B - metabolism | Gene Expression Profiling | TWEAK Receptor | RNA, Messenger - metabolism | Chemokine CXCL6 - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | T-Lymphocytes - metabolism | Adult | Female | Kidney Tubules - pathology | Kidney Tubules - metabolism | Chemokine CXCL6 - genetics | Disease Models, Animal | Receptors, Scavenger - metabolism | Folic Acid | Kidney Tubules - immunology | Nephritis, Interstitial - chemically induced | Receptors, Tumor Necrosis Factor - metabolism | Recombinant Proteins - metabolism | Cell Line | NF-kappa B - antagonists & inhibitors | Kidney Tubules - drug effects | Chemokine CXCL16 | Mice, Inbred C57BL | Tumor Necrosis Factors - metabolism | Antibodies, Neutralizing - pharmacology | Tumor Necrosis Factors - immunology | Chemotaxis | Cytokine TWEAK | Animals | Nephritis, Interstitial - genetics | Biopsy | Chemokines, CXC - metabolism | T-Lymphocytes - immunology | Mice | Sesquiterpenes - pharmacology | Nephritis, Interstitial - immunology | Tumor Necrosis Factors - antagonists & inhibitors | Index Medicus
Journal Article