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Contact Dermatitis, ISSN 0105-1873, 10/2010, Volume 63, Issue 4, pp. 215 - 222
Objectives: To investigate epidermal mRNA expression of mediators implicated in the regulation of inflammation induced by tape stripping according to our... 
tumour necrosis factor-α | tape stripping | inflammation | strip patch test | interleukin-33 | interleukin-8 | epidermal barrier | heat shock proteins 70 and 90 | ALLERGIC CONTACT-DERMATITIS | HUMAN SKIN | STRATUM-CORNEUM | TIME QUANTITATIVE PCR | INDUCTION | DERMATOLOGY | HEAT-SHOCK PROTEINS | ELICITATION | ALLERGY | IMMUNE-RESPONSES | GENE-EXPRESSION | tumour necrosis factor-alpha | HYPERSENSITIVITY | Interleukin-8 - genetics | Up-Regulation | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Patch Tests - methods | Reverse Transcription | HSP27 Heat-Shock Proteins - genetics | Young Adult | Interleukins - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | HSP90 Heat-Shock Proteins - genetics | Interleukins - biosynthesis | Cytokines - genetics | HSP70 Heat-Shock Proteins - biosynthesis | Interleukin-33 | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Keratinocytes - metabolism | Intercellular Adhesion Molecule-1 - genetics | Adolescent | Chemokine CCL5 - genetics | Chemokine CCL2 - biosynthesis | RNA, Messenger | Tumor Necrosis Factor-alpha - biosynthesis | HSP90 Heat-Shock Proteins - biosynthesis | Ribosomal Proteins - biosynthesis | Surgical Tape | Intercellular Adhesion Molecule-1 - biosynthesis | HSP27 Heat-Shock Proteins - biosynthesis | Adult | Protein Biosynthesis - genetics | Interferon-gamma - genetics | Cytoskeletal Proteins - biosynthesis | Epidermis - metabolism | Interleukin-8 - biosynthesis | Ribosomal Proteins - genetics | Carrier Proteins - biosynthesis | Chemokine CCL2 - genetics | HSP70 Heat-Shock Proteins - genetics | Carrier Proteins - genetics | Chemokine CCL5 - biosynthesis | Epidermis - immunology | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Cytokines - biosynthesis | Interferon-gamma - biosynthesis | Caspase 1 - biosynthesis | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 09/2004, Volume 44, Issue 3, pp. 264 - 270
Angiotensin II (Ang II) upregulates vascular endothelial growth factor (VEGF) and activates vascular inflammation. However, the decisive role of VEGF in Ang... 
Growth substances | Endothelial growth factors | Angiotensin II | Remodeling | Arteriosclerosis | MACROPHAGE INFILTRATION | FACTOR VEGF | remodeling | NITRIC-OXIDE SYNTHESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | endothelial growth factors | FACTOR-KAPPA-B | TUMOR ANGIOGENESIS | CHRONIC BLOCKADE | TYPE-1 RECEPTOR | arteriosclerosis | growth substances | angiotensin II | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | TISSUE ANGIOTENSIN | Genetic Therapy | Tetrazoles - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Male | Gene Expression Profiling | Vasculitis - prevention & control | Vascular Endothelial Growth Factor A - genetics | Angiotensin II Type 1 Receptor Blockers - pharmacology | Transforming Growth Factor beta - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit | Interleukin-1 - biosynthesis | Vascular Endothelial Growth Factor Receptor-2 - genetics | Imidazoles - therapeutic use | Receptors, Chemokine - genetics | Vasculitis - chemically induced | Coronary Vessels - pathology | Natriuretic Peptide, Brain - genetics | Transforming Growth Factor beta1 | Hypoxia-Inducible Factor 1 | Interleukin-6 - genetics | Extracellular Matrix Proteins - genetics | Imidazoles - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Aorta - pathology | Macrophages - metabolism | Intercellular Adhesion Molecule-1 - genetics | Chemokine CCL2 - biosynthesis | Nonmuscle Myosin Type IIB | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Vascular Endothelial Growth Factor A - physiology | Hypertrophy | Interleukin-1 - genetics | Tunica Media - pathology | Vascular Endothelial Growth Factor A - biosynthesis | Extracellular Matrix Proteins - biosynthesis | Tunica Media - drug effects | Vasculitis - physiopathology | Recombinant Fusion Proteins - physiology | Ventricular Remodeling | Nuclear Proteins - biosynthesis | Cell Division | Intercellular Adhesion Molecule-1 - biosynthesis | Receptors, Chemokine - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Vascular Endothelial Growth Factor Receptor-2 - biosynthesis | Angiotensin II - toxicity | Nuclear Proteins - genetics | Olmesartan Medoxomil | Hypertrophy, Left Ventricular - etiology | Mice, Inbred C57BL | Extracellular Matrix Proteins - physiology | Natriuretic Peptide, Brain - biosynthesis | Chemokine CCL2 - genetics | Renin-Angiotensin System - physiology | Transcription Factors - biosynthesis | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Animals | Transforming Growth Factor beta - genetics | Interleukin-6 - biosynthesis | Myosin Heavy Chains | Tetrazoles - therapeutic use | DNA-Binding Proteins - biosynthesis | Receptors, CCR2 | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2011, Volume 300, Issue 4, pp. 1418 - 1426
Post-myocardial infarction (MI), chemokine homing of inflammatory cells into the injured left ventricle (LV) regulates ventricular remodeling, in part by... 
CC chemokines | Inflammation | Matrix metalloproteinases | RECRUITMENT | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ANGIOGENESIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INCREASES | ISLET XENOGRAFTS | HEART | inflammation | PERIPHERAL VASCULAR DISEASE | MICE | matrix metalloproteinases | EXPRESSION | LEFT-VENTRICULAR FUNCTION | Myocardial Infarction - genetics | Macrophage Activation - genetics | Antigens, CD - biosynthesis | Mannose-Binding Lectins - biosynthesis | Receptors, CCR5 - genetics | Arginase - biosynthesis | Male | Lectins, C-Type - biosynthesis | Gene Deletion | Myocardial Infarction - pathology | Ventricular Remodeling - genetics | Female | Interleukin-1beta - biosynthesis | Receptors, Cell Surface - biosynthesis | Receptors, CCR5 - physiology | Procollagen - biosynthesis | Transforming Growth Factor beta1 - biosynthesis | HSP47 Heat-Shock Proteins - biosynthesis | Antigens, Differentiation, Myelomonocytic - biosynthesis | Macrophages - pathology | Ventricular Remodeling - physiology | Collagen Type I - biosynthesis | Macrophages - metabolism | Animals | Interleukin-6 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Gene mutations | Physiological aspects | Genetic aspects | Research | Macrophages | Health aspects | Chemokine receptors | Heart attack | Risk factors | Proteins | Heart attacks | Collagen | Rodents | Metabolism | Cells | Tumors | Index Medicus | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 03/2011, Volume 2, Issue 1, pp. 240 - 240
The immune system can both promote and suppress cancer. Chronic inflammation and proinflammatory cytokines such as interleukin (IL)-1 and IL-6 are considered... 
LUNG-CANCER | INTERFERON-INDUCIBLE PROTEIN-10 | NECROSIS IN-VIVO | PROGNOSTIC-FACTORS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CD4(+) T-CELLS | ADJUVANT THERAPY | CARCINOMA | EXPRESSION | Angiostatic Proteins - immunology | Immunohistochemistry | Neoplasms - metabolism | Interleukin-1alpha - immunology | Chemokine CXCL9 - biosynthesis | Tumor Microenvironment | Multiple Myeloma - immunology | Th2 Cells - immunology | Th1 Cells - immunology | CD4-Positive T-Lymphocytes - immunology | Chemokine CXCL10 - biosynthesis | Inflammation - metabolism | Interleukin-2 - immunology | Chemokine CXCL10 - immunology | Lymphoma, B-Cell - immunology | Interleukin-1beta - biosynthesis | Macrophages - immunology | Interleukin-12 - biosynthesis | Lymphoma, B-Cell - metabolism | Signal Transduction | Angiostatic Proteins - biosynthesis | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Interleukin-1beta - immunology | Chemokine CXCL9 - immunology | Inflammation - immunology | Macrophages - cytology | Mice, SCID | Multiple Myeloma - metabolism | Macrophages - metabolism | Multiple Myeloma - pathology | Animals | Neoplasms - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Interleukin-6 - immunology | Lymphoma, B-Cell - pathology | Cell Line, Tumor | Interleukin-6 - biosynthesis | Interleukin-1alpha - biosynthesis | Mice | Interleukin-2 - biosynthesis | Neoplasms - pathology | Interferon-gamma - biosynthesis | Index Medicus
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2007, Volume 194, Issue 1, pp. 54 - 61
Abstract Interleukin (IL)-17-producing helper T cells may play a pivotal role in the pathogenesis of multiple sclerosis. Here, we examined the effects of IL-17... 
Neurology | Allergy and Immunology | EAE | Cytokine | IL-17 | Microglia | cytokine | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MULTIPLE-SCLEROSIS LESIONS | MESSENGER-RNA EXPRESSION | NEUROTROPHIC FACTOR | IMMUNOLOGY | NEUROSCIENCES | NERVE GROWTH-FACTOR | INTERLEUKIN-17 | NEURONAL DEATH | NITRIC-OXIDE | TNF-ALPHA | microglia | T-CELLS | Brain-Derived Neurotrophic Factor - genetics | Microglia - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-17 - physiology | Receptors, Interleukin-17 - genetics | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | Interleukin-23 - genetics | RNA, Messenger - biosynthesis | Chemokine CXCL2 - genetics | Brain-Derived Neurotrophic Factor - biosynthesis | Interleukin-1beta - biosynthesis | Multiple Sclerosis - metabolism | Autocrine Communication | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Interleukin-6 - genetics | Microglia - drug effects | Receptors, Interleukin-17 - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - genetics | Interleukin-17 - biosynthesis | Recombinant Proteins - pharmacology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Interleukin-23 - biosynthesis | Interleukin-6 - biosynthesis | Cells, Cultured - metabolism | Chemokine CXCL2 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Multiple sclerosis | Interleukins | T cells | Nitric oxide | Index Medicus
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 06/2006, Volume 79, Issue 6, pp. 1279 - 1285
Dendritic cells (DCs) play an important role in innate and adaptive immune responses. In addition to their phagocytic activity, DCs present foreign antigens to... 
human | cytokines | cell activation | Human | Cytokines | Cell activation | SIGNAL-TRANSDUCTION PATHWAYS | ACTIVATION | RECEPTOR | ANTIGEN PRESENTATION | IMMUNOLOGY | CUTTING EDGE | CELL BIOLOGY | B-CELLS | HUMAN NK | COMMON GAMMA-CHAIN | HEMATOLOGY | T-CELLS | IFN-ALPHA | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Receptors, Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Suppressor of Cytokine Signaling 1 Protein | Gene Expression Profiling | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - biosynthesis | Interleukins - physiology | T-Lymphocytes - metabolism | Chemokines, CC - biosynthesis | Cells, Cultured - immunology | Chemokines, CXC - biosynthesis | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Interleukin-12 - biosynthesis | Chemokine CCL5 | Cells, Cultured - drug effects | Receptors, Interleukin-21 | Lymphocyte Activation | Repressor Proteins - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Toll-Like Receptor 4 - genetics | Receptors, Interleukin-1 - biosynthesis | Receptors, Interleukin - genetics | Dendritic Cells - secretion | Feedback, Physiological | Macrophages - metabolism | Repressor Proteins - biosynthesis | T-Lymphocytes - immunology | Cells, Cultured - metabolism | HLA-DR Antigens - biosynthesis | Suppressor of Cytokine Signaling Proteins - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | Receptors, Interleukin - biosynthesis | Toll-Like Receptor 4 - biosynthesis | Interleukin-12 - genetics | Dendritic Cells - drug effects | Interleukins - pharmacology | Receptors, Interferon - genetics | Dendritic Cells - metabolism | RNA, Messenger - genetics | Cell Communication | Recombinant Proteins - pharmacology | Chemokines, CXC - genetics | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Suppressor of Cytokine Signaling 3 Protein | Chemokines, CC - genetics | Macrophages - drug effects | Interleukin-21 Receptor alpha Subunit | Receptors, Interferon - biosynthesis | Cytokines - biosynthesis | Interferon-gamma - pharmacology | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 10/2005, Volume 106, Issue 7, pp. 2366 - 2374
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2013, Volume 191, Issue 3, pp. 1200 - 1209
Journal Article
Cellular Immunology, ISSN 0008-8749, 01/2013, Volume 281, Issue 1, pp. 51 - 61
Journal Article
The Journal of Immunology, ISSN 0022-1767, 08/2008, Volume 181, Issue 4, pp. 2790 - 2798
Asthma and chronic obstructive pulmonary disease (COPD) are associated with Th2 and Th1 differentiated T cells. The cytokine thymic stromal lymphopoietin... 
AIRWAY EPITHELIAL-CELLS | ACTIVATION | BRONCHIAL BIOPSIES | INFLAMMATION | SMOKERS | KAPPA-B | IMMUNOLOGY | RECEPTORS CCR4 | T-CELLS | PERIPHERAL AIRWAYS | CUTTING EDGE | T-Lymphocyte Subsets - immunology | Th1 Cells - pathology | Asthma - metabolism | Stromal Cells - pathology | Humans | Middle Aged | Male | Th2 Cells - immunology | Pulmonary Disease, Chronic Obstructive - pathology | Th1 Cells - immunology | Cytokines - physiology | Respiratory Mucosa - pathology | Th1 Cells - metabolism | ADAM Proteins - biosynthesis | Chemokine CXCL10 - biosynthesis | Chemokine CCL17 - biosynthesis | Respiratory Mucosa - immunology | Asthma - immunology | Thymus Gland - pathology | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Thymus Gland - metabolism | Th2 Cells - pathology | Severity of Illness Index | Chemokines - biosynthesis | Chemokine CXCL11 - biosynthesis | Stromal Cells - metabolism | Chemokine CCL1 - biosynthesis | Stromal Cells - immunology | Chemokines - genetics | Th2 Cells - metabolism | T-Lymphocyte Subsets - pathology | T-Lymphocyte Subsets - metabolism | Thymus Gland - immunology | Aged | Respiratory Mucosa - metabolism | Chemotaxis, Leukocyte - immunology | Pulmonary Disease, Chronic Obstructive - immunology | Tumor Suppressor Proteins - biosynthesis | Asthma - pathology | Cytokines - biosynthesis | Index Medicus | Abridged Index Medicus
Journal Article
American journal of respiratory cell and molecular biology, ISSN 1044-1549, 11/2015, Volume 53, Issue 5, pp. 676 - 688
Macrophages are dynamic cells that mature under the influence of signals from the local microenvironment into either classically (M1) or alternatively (M2)... 
classically activated macrophages (CAM/M1) | Phagocytosis/ endocytosis | phagocytosis/endocytosis | phenotypic stability | alternatively activated macrophages (AAM/M2) | reprogramming of polarization | PHAGOCYTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONOCYTES | DIFFERENTIAL REGULATION | MURINE | CELL BIOLOGY | GM-CSF | RESPIRATORY SYSTEM | THP-1 CELLS | MANNOSE | RECEPTOR-ACTIVITY | POLARIZATION | EXPRESSION | Chemokines, CC - secretion | Gene Expression - drug effects | Monocytes - cytology | Humans | Monocytes - immunology | Chemokine CCL5 - secretion | Chemokine CCL17 - secretion | Chemokine CXCL10 - biosynthesis | Interleukin-4 - pharmacology | Chemokine CCL17 - biosynthesis | Gene Expression - immunology | Chemokines, CC - biosynthesis | Interleukin-8 - secretion | Cell Differentiation | Interleukin-1beta - biosynthesis | Macrophages - immunology | Endocytosis - immunology | Interleukin-1beta - secretion | Interleukin-8 - biosynthesis | Endocytosis - drug effects | Tumor Necrosis Factor-alpha - secretion | Immunophenotyping | Macrophages - cytology | Interleukin-13 - pharmacology | Monocytes - drug effects | Chemokine CCL5 - biosynthesis | Chemokine CXCL10 - secretion | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Primary Cell Culture | Macrophage Activation - drug effects | Tumor Necrosis Factor-alpha - biosynthesis | Interferon-gamma - pharmacology | Studies | Biology | Immunology | Blood & organ donations | Cytokines | Gene expression | Index Medicus
Journal Article
The Journal of Immunology, ISSN 0022-1767, 06/2005, Volume 174, Issue 11, pp. 6592 - 6597
We demonstrate that functional and phenotypic equivalents of mouse splenic CD8(+) and CD8(-) conventional dendritic cell (cDC) subsets can be generated in... 
SPLEEN | COLONY-STIMULATING FACTOR | SUBTYPES | SOLUBLE-ANTIGEN | MOUSE | SUBSETS | ANTIGEN PRESENTATION | IMMUNOLOGY | FLT3 LIGAND | DIFFERENTIAL PRODUCTION | IFN-ALPHA | Interferon Regulatory Factors | Spleen - immunology | Dendritic Cells - immunology | Membrane Glycoproteins - biosynthesis | fms-Like Tyrosine Kinase 3 | Repressor Proteins - physiology | Cell Differentiation - genetics | Toll-Like Receptors | Bone Marrow Cells - immunology | Cystatin C | Receptors, Chemokine - biosynthesis | Cross-Priming - genetics | Membrane Proteins - metabolism | Receptors, Cell Surface - biosynthesis | Dendritic Cells - metabolism | CD4 Antigens - genetics | Proto-Oncogene Proteins - metabolism | Chemokines - biosynthesis | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Cells, Cultured | Repressor Proteins - genetics | Antigen Presentation - genetics | CD8 Antigens - biosynthesis | Immunophenotyping | Mice, Transgenic | Spleen - cytology | Antigen Presentation - immunology | Receptor Protein-Tyrosine Kinases - metabolism | Mice, Knockout | Cell Differentiation - immunology | Animals | Repressor Proteins - biosynthesis | Cross-Priming - immunology | Spleen - metabolism | CD8 Antigens - genetics | Cystatins - biosynthesis | Ligands | Dendritic Cells - cytology | Mice | CD4 Antigens - biosynthesis | Bone Marrow Cells - metabolism | Cytokines - biosynthesis | Index Medicus | Abridged Index Medicus
Journal Article