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PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e50385
ATP released in the early inflammatory processes acts as a danger signal by binding to purinergic receptors expressed on immune cells. A major contribution of... 
Chemokines, CXC - immunology | Receptors, Purinergic P2Y2 - immunology | Interleukin-12 - genetics | Th2 Cells - virology | Dendritic Cells - immunology | Bronchoalveolar Lavage Fluid - immunology | Th2 Cells - immunology | Th1 Cells - immunology | Murine pneumonia virus - immunology | Pneumovirus Infections - metabolism | Pneumonia, Viral - metabolism | Th1 Cells - virology | Chemokine CCL20 - immunology | Inflammation - metabolism | Lung - virology | Adenosine Triphosphate - metabolism | Chemokine CXCL10 - immunology | Chemokine CCL20 - genetics | Female | Lung - metabolism | Dendritic Cells - virology | Receptors, Purinergic P2Y2 - genetics | Gene Expression | Interleukin-6 - genetics | Inflammation - virology | Neutrophils - immunology | Survival Rate | Inflammation - immunology | Chemokines, CXC - genetics | Pneumovirus Infections - virology | Neutrophils - virology | Pneumonia, Viral - immunology | Receptors, Purinergic P2Y2 - deficiency | Mice, Knockout | Pneumovirus Infections - immunology | Animals | CD8-Positive T-Lymphocytes - virology | Chemokine CXCL10 - genetics | Interleukin-12 - immunology | Interleukin-6 - immunology | Mice | Bronchoalveolar Lavage Fluid - chemistry | CD8-Positive T-Lymphocytes - immunology | Pneumonia, Viral - virology | Lung - immunology | Pneumonia | Purine receptors | CD8 antigen | Immune clearance | Viruses | Cytotoxicity | Lymphocytes T | Infections | Recruitment | Interleukin 6 | Respiratory tract | Airway management | Receptors | Lymphocytes | Rodents | IP-10 protein | Respiratory tract diseases | Alveoli | Immune system | Dendritic cells | Mortality | Cloning | Bronchus | Cystic fibrosis | Interleukin 12 | Inflammation | Hazards | Interdisciplinary aspects | Morbidity | Asthma | CD4 antigen | Respiratory syncytial virus | Lungs | Fibrosis | Signal processing | Infiltration | ATP | Chemokines | Viral infections
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2009, Volume 183, Issue 10, pp. 6469 - 6477
Alternatively activated macrophages (AAM) play a crucial role in type 2 immunity. Mice deficient in ST2, a receptor for the latest member of the IL-1 family,... 
IL-1-LIKE CYTOKINE IL-33 | IMMUNE-RESPONSE | ST2 RECEPTOR | EFFECTOR FUNCTION | MAST-CELLS | IN-VIVO | INDUCED ARTHRITIS | ALVEOLAR MACROPHAGES | INTERLEUKIN-1 RECEPTOR | IMMUNOLOGY | T-CELLS | Interleukin-1 Receptor-Like 1 Protein | Asthma - metabolism | Epithelial Cells - metabolism | Ovalbumin - immunology | Humans | Middle Aged | Chemokine CCL24 - metabolism | Interleukin-13 - immunology | Male | Interleukins - metabolism | Receptors, Interleukin - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Interleukins - immunology | Asthma - immunology | Chemokine CCL17 - immunology | Adult | Female | Interleukin-4 - genetics | Macrophages, Alveolar - immunology | Recombinant Proteins - metabolism | Cell Polarity - immunology | Lung - pathology | Receptors, Interleukin - immunology | Interleukin-33 | Macrophage Activation - immunology | Receptors, Interleukin-1 - immunology | Interleukin-4 - metabolism | Receptors, Interleukin-4 - metabolism | Inflammation - immunology | Receptors, Interleukin-4 - immunology | Interleukin-13 - metabolism | Chemokine CCL24 - immunology | Receptors, Interleukin - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Interleukin-4 - immunology | Animals | Eosinophilia - immunology | Recombinant Proteins - immunology | Epithelial Cells - immunology | Receptors, Interleukin-4 - genetics | Mice | Mice, Inbred BALB C | Macrophages, Alveolar - metabolism | Chemokine CCL17 - metabolism | Lung - immunology
Journal Article
Annual review of immunology, ISSN 1545-3278, 2007, Volume 25, Issue 1, pp. 297 - 336
Journal Article
Journal of neurovirology, ISSN 1538-2443, 2015, Volume 22, Issue 2, pp. 191 - 200
Cell-free mitochondiral DNA (mtDNA) is an immunogenic molecule associated with many inflammatory conditions. We evaluated the relationship between cell-free... 
Neurology | Neurosciences | Biomedicine | Immunology | Infectious Diseases | Pleocytosis | Mitochondrial DNA | Inflammation | Droplet digital PCR | HAND | Virology | BRAIN-INJURY | CEREBROSPINAL-FLUID | NEUROSCIENCES | IMPAIRMENT | HEART | PLASMA | VIROLOGY | NEOPTERIN | DISEASE | DNA, Mitochondrial - cerebrospinal fluid | Chemokine CCL2 - immunology | Lipopolysaccharide Receptors - cerebrospinal fluid | Humans | Memory | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Male | Antigens, CD - genetics | Learning | Neurofilament Proteins - cerebrospinal fluid | Chemokine CCL2 - cerebrospinal fluid | HIV Infections - immunology | Neurofilament Proteins - genetics | Cognitive Dysfunction - immunology | Tumor Necrosis Factor-alpha - immunology | Interleukin-6 - cerebrospinal fluid | Antigens, Differentiation, Myelomonocytic - cerebrospinal fluid | HIV Infections - cerebrospinal fluid | Gene Expression | Interleukin-6 - genetics | Tumor Necrosis Factor-alpha - cerebrospinal fluid | Lipopolysaccharide Receptors - immunology | Cognitive Dysfunction - cerebrospinal fluid | Cognitive Dysfunction - pathology | Receptors, Cell Surface - immunology | Neuropsychological Tests | Antigens, CD - cerebrospinal fluid | HIV Infections - complications | Interleukin-6 - immunology | Receptors, Cell Surface - genetics | Antigens, CD - immunology | HIV-1 - physiology | HIV Infections - pathology | Chemokine CXCL10 - immunology | Adult | Female | Neopterin - immunology | Cognitive Dysfunction - complications | Neurofilament Proteins - immunology | Executive Function | Antigens, Differentiation, Myelomonocytic - immunology | Severity of Illness Index | Chemokine CXCL10 - cerebrospinal fluid | Neopterin - cerebrospinal fluid | Cross-Sectional Studies | Chemokine CCL2 - genetics | Antigens, Differentiation, Myelomonocytic - genetics | Chemokine CXCL10 - genetics | Lipopolysaccharide Receptors - genetics
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2018, Volume 49, Issue 6, pp. 1175 - 1190.e7
The number of leukocytes present in circulation varies throughout the day, reflecting bone marrow output and emigration from blood into tissues. Using an... 
migration | circadian | immunology | leukocyte | EMIGRATION | ADHESION | RECRUITMENT | CODE | LYMPHOCYTE RECIRCULATION | TRANSENDOTHELIAL MIGRATION | LIVER | TRAFFICKING | IMMUNOLOGY | CLOCKS | CD8(+) T-CELLS | Cell Adhesion Molecules - genetics | Humans | Middle Aged | Male | Gene Expression Profiling | Cell Adhesion Molecules - immunology | Cell Movement - genetics | Organ Specificity - immunology | Cell Movement - immunology | Leukocytes - immunology | Organ Specificity - genetics | Adult | Female | Circadian Rhythm - immunology | Transcription Factors - immunology | Leukocytes - cytology | Endothelial Cells - metabolism | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Homeostasis - immunology | Mice, Transgenic | Transcription Factors - genetics | Cell Adhesion Molecules - metabolism | Mice, Knockout | Endothelial Cells - immunology | Transcription Factors - metabolism | Animals | Leukocytes - metabolism | Homeostasis - genetics | Emigration and immigration | Endothelium | Leukocyte migration | Oscillations | Homeostasis | Leukocytes | Tissues | Immunity | Experiments | Blood | Cell adhesion & migration | Homing | Time dependence | Bone marrow | Circadian rhythms | Medical research | Antigens | Organs | Rhythms | Circadian rhythm | Ablation | Endothelial cells | White blood cells | BMAL1 protein | Ligands | Chemokines | Cell migration | Cancer | Life Sciences
Journal Article
Trends in immunology, ISSN 1471-4906, 2011, Volume 32, Issue 9, pp. 412 - 419
Journal Article
Nature immunology, ISSN 1529-2916, 2012, Volume 14, Issue 2, pp. 162 - 171
Signaling through the G protein-coupled receptors for the complement fragments C3a and C5a (C3aR and C5aR, respectively) by dendritic cells and CD4(+) cells... 
Forkhead Transcription Factors - immunology | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | TOR Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - metabolism | Complement C5a - metabolism | Receptors, Complement - metabolism | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Transforming Growth Factor beta1 - immunology | Forkhead Transcription Factors - metabolism | Class Ib Phosphatidylinositol 3-Kinase - metabolism | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Receptor, Anaphylatoxin C5a - immunology | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Complement C3a - metabolism | Interleukin-6 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Receptors, Chemokine - metabolism | Gene Expression Regulation | Mice, Transgenic | Receptors, Chemokine - immunology | Receptor Cross-Talk - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Receptors, Complement - immunology | Cyclic AMP-Dependent Protein Kinases - immunology | Interleukin-6 - immunology | Mice | Complement C3a - immunology | Complement C5a - immunology | Class Ib Phosphatidylinositol 3-Kinase - immunology | Interleukin-10 - immunology | Receptor, Anaphylatoxin C5a - metabolism
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 2014, Volume 40, Issue 6, pp. 896 - 909
Animal host defense against infection requires the expression of defense genes at the right place and the right time. Understanding such tight control of host... 
LIFE-SPAN | CAENORHABDITIS-ELEGANS | RECOGNITION | AUTOPHAGY GENES | REGULATES AUTOPHAGY | STAPHYLOCOCCUS-AUREUS | INFECTION | IMMUNOLOGY | PROTEINS | C. ELEGANS | LYSOSOMAL BIOGENESIS | Pseudomonas aeruginosa - immunology | Caenorhabditis elegans - microbiology | Transcriptional Activation - genetics | Caenorhabditis elegans Proteins - immunology | Salmonella Infections - immunology | Transcriptional Activation - immunology | Autophagy - immunology | Signal Transduction - immunology | RNA Interference | Autophagy - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - immunology | Macrophages - immunology | Enterococcus faecalis - immunology | Basic Helix-Loop-Helix Transcription Factors - genetics | Staphylococcal Infections - immunology | Immunity, Innate | Caenorhabditis elegans - immunology | Pseudomonas Infections - immunology | Animals | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Salmonella enterica - immunology | Basic Helix-Loop-Helix Transcription Factors - immunology | Staphylococcus aureus - immunology | Mice | RNA, Small Interfering | Caenorhabditis elegans Proteins - genetics | Genetic aspects | Genetic transcription | Analysis | Gastrointestinal diseases | Genes | Genomics | Signal transduction | Transcription factors | Nematodes | Staphylococcus infections | Kinases | Mammals | Gene expression | Experiments | Autophagy | Chemokines
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article