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Journal Article
Clinical Science, ISSN 0143-5221, 04/2014, Volume 126, Issue 8, pp. 567 - 580
Journal Article
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2013, Volume 14, Issue 2, pp. 162 - 171
Signaling through the G protein-coupled receptors for the complement fragments C3a and C5a (C3aR and C5aR, respectively) by dendritic cells and CD4(+) cells... 
Forkhead Transcription Factors - immunology | Cell Communication - immunology | T-Lymphocytes, Regulatory - metabolism | TOR Serine-Threonine Kinases - metabolism | Transforming Growth Factor beta1 - metabolism | Complement C5a - metabolism | Receptors, Complement - metabolism | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Transforming Growth Factor beta1 - immunology | Forkhead Transcription Factors - metabolism | Class Ib Phosphatidylinositol 3-Kinase - metabolism | T-Lymphocytes, Regulatory - cytology | Interleukin-10 - metabolism | Receptor, Anaphylatoxin C5a - immunology | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Complement C3a - metabolism | Interleukin-6 - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Receptors, Chemokine - metabolism | Gene Expression Regulation | Mice, Transgenic | Receptors, Chemokine - immunology | Receptor Cross-Talk - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Proto-Oncogene Proteins c-akt - immunology | Receptors, Complement - immunology | Cyclic AMP-Dependent Protein Kinases - immunology | Interleukin-6 - immunology | Mice | Complement C3a - immunology | Complement C5a - immunology | Class Ib Phosphatidylinositol 3-Kinase - immunology | Interleukin-10 - immunology | Receptor, Anaphylatoxin C5a - metabolism | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 686 - 695.e1
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2014, Volume 192, Issue 4, pp. 1982 - 1989
Journal Article
Vaccine, ISSN 0264-410X, 10/2017, Volume 35, Issue 45, pp. 6143 - 6153
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2007, Volume 47, Issue 5, pp. 632 - 641
Background/Aims CXCR3 and CCR5 play a major role in recruiting cytotoxic T cells (Tc) and secreting secondary type 1 cytokines (Tc1) in the liver. HCV could... 
Gastroenterology and Hepatology | CXCL10 | CXCR3 | CCL3 | Liver damage | Hepatitis C virus | CCR5 | Chemotaxis | Chemokine receptors | Chemokines | Viral escape mechanism | chemokine receptors | PLUS RIBAVIRIN | viral escape mechanism | chemokines | PATHOGENESIS | THERAPY | INFLAMMATION | DISEASE | hepatitis C virus | FLOW-CYTOMETRY | GASTROENTEROLOGY & HEPATOLOGY | liver damage | T-LYMPHOCYTES | chemotaxis | Liver - virology | Interferon-alpha - pharmacology | Chemokine CCL3 - immunology | Hepatitis C - drug therapy | Hepacivirus - immunology | Humans | Middle Aged | Cell Death - immunology | Hepacivirus - genetics | Male | Liver - physiopathology | Receptors, CCR5 - immunology | Hepatitis C - immunology | Liver - immunology | Lymphocyte Activation - immunology | Chemokine CCL3 - blood | Chemokine CXCL10 - immunology | Adult | Female | Cell Death - drug effects | Cytokines - immunology | Hepacivirus - drug effects | T-Lymphocytes, Cytotoxic - immunology | Chemokine CXCL10 - blood | Receptors, CXCR3 - blood | Cytokines - secretion | Interferon-alpha - therapeutic use | Receptors, CCR5 - blood | Genotype | Up-Regulation - drug effects | Up-Regulation - immunology | Lymphocyte Activation - drug effects | Chronic Disease - therapy | Hepatitis C - virology | Receptors, CXCR3 - immunology | CD8-Positive T-Lymphocytes - immunology | Control | Cytokines | Biological response modifiers | Hepatitis C | Health aspects | Liver | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 11/2012, Volume 13, Issue 11, pp. 1072 - 1082
Hematopoietic stem and progenitor cells (HSPCs) are regulated by various bone marrow stromal cell types. Here we identified rare activated bone marrow... 
PROGENITOR CELLS | OXIDATIVE STRESS | MICROENVIRONMENT | MYELOPOIESIS | RECEPTOR LIGANDS | NICHE | STEM-CELL | IMMUNOLOGY | STROMAL CELLS | RECONSTITUTION | PROSTAGLANDIN-E2 | Cell Communication - immunology | Gamma Rays | Bone Marrow - immunology | Mesenchymal Stromal Cells - radiation effects | Reactive Oxygen Species - metabolism | Dinoprostone - biosynthesis | Monocytes - cytology | Monocytes - radiation effects | Cell Survival - genetics | Bone Marrow - radiation effects | Monocytes - immunology | Actins - immunology | Dinoprostone - immunology | Cell Movement - genetics | Proto-Oncogene Proteins c-akt - genetics | Actins - genetics | Mesenchymal Stromal Cells - immunology | Cell Movement - immunology | Signal Transduction - immunology | Hematopoietic Stem Cells - immunology | Mesenchymal Stromal Cells - cytology | Chemokine CXCL12 - genetics | Cyclooxygenase 2 - genetics | Reactive Oxygen Species - immunology | Bone Marrow - metabolism | Macrophages - radiation effects | Signal Transduction - radiation effects | Cyclooxygenase 2 - immunology | Hematopoietic Stem Cells - radiation effects | Chemokine CXCL12 - immunology | Macrophages - immunology | Gene Expression Regulation - immunology | Signal Transduction - genetics | Cell Communication - genetics | Macrophages - cytology | Cell Survival - immunology | Cell Survival - radiation effects | Animals | Proto-Oncogene Proteins c-akt - immunology | Hematopoietic Stem Cells - cytology | Gene Expression Regulation - radiation effects | Mice | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 9, pp. 1 - 14
Background: Hepatitis C virus (HCV) genomes and proteins are present in human brain tissues although the impact of HIV/ HCV co-infection on neuropathogenesis... 
HIV-1 | NERVOUS-SYSTEM | IN-VITRO | LENTIVIRUS INFECTION | ALZHEIMERS-DISEASE | BIOLOGY | NECROSIS-FACTOR-ALPHA | ANTIRETROVIRAL THERAPY | CELL-CULTURE | EXPRESSION | NEURONAL AUTOPHAGY | Hepacivirus - immunology | Humans | Neurons - cytology | Autophagy | HIV Infections - immunology | Hepatitis C - immunology | HIV-1 - physiology | Microglia - immunology | Astrocytes - immunology | Female | Hepacivirus - physiology | Hepatitis C - complications | Cytokines - immunology | Astrocytes - cytology | Microglia - cytology | Cell Line | HIV Infections - virology | Neurons - immunology | Mice, Transgenic | HIV-1 - immunology | Animals | HIV Infections - complications | Hepatitis C - virology | Hepatitis C - physiopathology | Viral Core Proteins - immunology | Mice | HIV Infections - physiopathology | Medical research | Brain | RNA | Neurons | Genes | Genomics | Genomes | HIV (Viruses) | Gene expression | Infection | Interleukins | Viral proteins | Medicine, Experimental | Genetic engineering | Hepatitis C virus | Health aspects | HIV infection | Cell culture | Proteins | Hepatitis | Vpr protein | Tubulin | Neurodegeneration | Animal tissues | Infectivity | Human immunodeficiency virus--HIV | Membrane currents | Cytokines | Astrocytes | Cultures | Exposure | Tumor necrosis factor-α | Membrane proteins | Microglia | Studies | Neurology | Gliosis | Core protein | Human performance | CXCL10 protein | TNF inhibitors | Viability | Phagocytosis | Neuropathogenesis | Interleukin | Viruses | Infections | Activation | Kinases | Molecular weight | Interleukin 6 | Neurotoxicity | Immunology | Implants | Tumor necrosis factor-TNF | Interleukin 8 | Encephalitis | Cell survival | Dendritic cells | Neutrophils | Transgenic mice | Implantation | Inflammation | Ribonucleic acid--RNA | Brain research | West Nile virus | Immunoreactivity | Hepatitis C | Chemokines | Apoptosis | Index Medicus | Human immunodeficiency virus | Ribonucleic acid | HIV
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e54675 - e54675
Type 1 diabetes (T1D) results from the autoimmune destruction of insulin-producing beta-cells in the pancreas. Recruitment of inflammatory cells is... 
PATHOGENESIS | CELLS | IN-VITRO | TRANSENDOTHELIAL MIGRATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | LEUKOCYTE ADHESION | TRANSGENIC MODEL | ISLET-SPECIFIC EXPRESSION | ACUTE-PANCREATITIS | JAM-C | Lymphocytic choriomeningitis virus - immunology | Cell Line | Gene Expression | Diabetes Mellitus, Type 1 - virology | Islets of Langerhans - pathology | Junctional Adhesion Molecule C - antagonists & inhibitors | Endothelial Cells - metabolism | Junctional Adhesion Molecule C - genetics | Diabetes Mellitus, Type 1 - genetics | Islets of Langerhans - immunology | Male | Mice, Transgenic | Autoantigens - genetics | Antibodies, Neutralizing - immunology | Animals | Lymphocytic choriomeningitis virus - pathogenicity | Autoantigens - immunology | Islets of Langerhans - virology | Female | T-Lymphocytes - immunology | Junctional Adhesion Molecule C - immunology | Mice | Diabetes Mellitus, Type 1 - immunology | Antibodies, Monoclonal - immunology | Proteins | Autoimmunity | Rheumatoid factor | Ischemia | Type 1 diabetes | Pancreatitis | Genetic engineering | T cells | Health aspects | Endothelium | Leukocyte migration | Pathogenesis | C protein | Trafficking | Viruses | Lymphocytes T | Arthritis | Infections | Jamming | Cell adhesion & migration | Recruitment | Reperfusion | Immunology | Lymphocytes | Transgenic animals | Rodents | Gangrene | Pancreas | Antigens | Immunoglobulins | Diabetes mellitus | Cloning | Neutrophils | Transgenic mice | Organs | Inflammation | Insulin | Adhesion | Endothelial cells | White blood cells | Beta cells | Rheumatoid arthritis | Infiltration | Islets of Langerhans | Diabetes | Cell migration | Chemokines | Index Medicus
Journal Article