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The Journal of Infectious Diseases, ISSN 0022-1899, 2015, Volume 211, Issue 1, pp. 156 - 165
Background. A predominantly T-helper type 2 (Th2) immune response is critical in the prognosis of pulmonary Pseudomonas aeruginosa infection. But the mucosal... 
human-milk | cytokine production | butyrate | in-vitro | chain fatty-acids | galacto-oligosaccharides | t-cells | lactobacillus | virus-infection | cystic-fibrosis patients | intestinal microbiota | prebiotics | Th1 and M1 polarization | P. aeruginosa lung infection | INFECTIOUS DISEASES | VIRUS-INFECTION | GALACTO-OLIGOSACCHARIDES | MICROBIOLOGY | IMMUNOLOGY | CYTOKINE PRODUCTION | HUMAN-MILK | METABOLISM | CYSTIC-FIBROSIS PATIENTS | CHAIN FATTY-ACIDS | BUTYRATE | T-CELLS | LACTOBACILLUS | Intestines - drug effects | Lung Diseases - microbiology | GATA3 Transcription Factor - immunology | Pseudomonas aeruginosa - growth & development | Microbiota - drug effects | Male | T-Box Domain Proteins - immunology | Lung Diseases - drug therapy | Th1 Cells - immunology | Intestines - immunology | Leukocytes - immunology | Oligosaccharides - pharmacology | Leukocytes - microbiology | Tumor Necrosis Factor-alpha - immunology | Keratinocytes - microbiology | Macrophages - immunology | Macrophages - microbiology | Chemotactic Factors - immunology | Microbiota - immunology | Pseudomonas Infections - drug therapy | Pseudomonas aeruginosa - drug effects | Pseudomonas Infections - microbiology | Keratinocytes - immunology | Pectins - chemistry | Pseudomonas Infections - immunology | Lung Diseases - immunology | Interleukin-4 - immunology | Th1 Cells - microbiology | Animals | Intestines - microbiology | Interferon-gamma - immunology | Mice | Mice, Inbred BALB C | Interleukin-10 - immunology | Pseudomonas Infections/microbiology | Macrophages/microbiology | Oligosaccharides/pharmacology | Macrophages/immunology | Lung Diseases/immunology | Lung Diseases/microbiology | Interferon-gamma/immunology | Keratinocytes/microbiology | Microbiota/drug effects | Life Sciences | Leukocytes/microbiology | Pseudomonas Infections/immunology | Interleukin-10/immunology | Lung Diseases/drug therapy | T-Box Domain Proteins/immunology | Leukocytes/immunology | Pseudomonas aeruginosa/drug effects | Th1 Cells/microbiology | GATA3 Transcription Factor/immunology | Interleukin-4/immunology | Intestines/drug effects | Intestines/microbiology | Keratinocytes/immunology | Chemotactic Factors/immunology | Tumor Necrosis Factor-alpha/immunology | Microbiota/immunology | Pseudomonas aeruginosa/growth & development | Pectins/chemistry | Pseudomonas Infections/drug therapy | Th1 Cells/immunology | Intestines/immunology
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6489 - 6499
Chemerin is the ligand of the ChemR23 receptor and a chemoattractant factor for human immature dendritic cells (DCs), macrophages, and NK cells. In this study,... 
ADAPTIVE IMMUNITY | NATURAL-KILLER-CELLS | RESOLVIN E1 | COLONY-STIMULATING FACTOR | IN-VIVO | BONE-MARROW | HUMAN BLOOD | IMMUNOLOGY | CHEMOKINE RECEPTORS | INFLAMED LYMPH-NODES | PULMONARY ALVEOLAR MACROPHAGES | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Dendritic Cells - immunology | Bronchoalveolar Lavage Fluid - immunology | Calcium - immunology | Aequorin - immunology | Aequorin - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Chemotactic Factors - pharmacology | Peptides - metabolism | Chemotactic Factors - metabolism | Pneumonia - chemically induced | Killer Cells, Natural - immunology | Pneumonia - immunology | Dendritic Cells - drug effects | Bronchoalveolar Lavage Fluid - cytology | Chemotaxis - immunology | Neutrophils - metabolism | Apoproteins - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Disease Models, Animal | Recombinant Proteins - metabolism | Chemotactic Factors - immunology | Acute Disease | Lung - pathology | Mice, Inbred C57BL | Neutrophils - drug effects | Peptides - immunology | Neutrophils - immunology | Chemotaxis - drug effects | Mice, Knockout | Macrophages - metabolism | Animals | Recombinant Proteins - immunology | Intercellular Signaling Peptides and Proteins - pharmacology | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Killer Cells, Natural - drug effects | Mice | Receptors, G-Protein-Coupled - genetics | Apoproteins - immunology | Killer Cells, Natural - metabolism | Pneumonia - metabolism | Chemokines | Intercellular Signaling Peptides and Proteins - immunology | Lung - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Neuro-Oncology, ISSN 1522-8517, 11/2010, Volume 12, Issue 11, pp. 1113 - 1125
Macrophages (MΦs)/microglia that constitute the dominant tumor-infiltrating immune cells in glioblastoma are recruited by tumor-secreted factors and are... 
Immune suppression | Innate immunity | Cancer stem cells | Macrophages | Signal transducer and activator of transcription 3 | Microglia | ACTIVATION | TOLL-LIKE RECEPTOR | STAT3 | TGF-BETA SUPERFAMILY | signal transducer and activator of transcription 3 | CLINICAL NEUROLOGY | macrophages | GLIOBLASTOMA | innate immunity | ONCOLOGY | TUMOR-ASSOCIATED MACROPHAGES | cancer stem cells | SUPPRESSOR-CELLS | immune suppression | microglia | T-CELLS | PROGRESSION | Microglia - metabolism | Phosphorylation | Humans | Brain Neoplasms - pathology | Neoplastic Stem Cells - immunology | Brain Neoplasms - metabolism | Glioma - metabolism | Cell Movement - immunology | Signal Transduction - immunology | Brain Neoplasms - immunology | Flow Cytometry | Microglia - immunology | Transfection | Chemotactic Factors - metabolism | Neoplastic Stem Cells - metabolism | Glioma - pathology | Neoplastic Stem Cells - pathology | Macrophages - immunology | STAT3 Transcription Factor - metabolism | Cytokines - immunology | Chemotactic Factors - immunology | Enzyme-Linked Immunosorbent Assay | Cell Separation | Cells, Cultured | Glioma - immunology | Blotting, Western | Immunity, Innate - immunology | Macrophages - metabolism | STAT3 Transcription Factor - immunology | Chemotaxis, Leukocyte - immunology | Cytokines - biosynthesis | Basic and Translational Investigations
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 07/2012, Volume 209, Issue 8, pp. 1427 - 1435
Infiltration of specialized immune cells regulates the growth and survival of neoplasia. Here, in a survey of public whole genome expression datasets we found... 
TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | DENDRITIC CELLS | PROGNOSIS | TIG2 | REGULATORY T | SKIN | IMMUNOLOGY | GENE-EXPRESSION CHANGES | CANCER | TUMORIGENESIS | CARCINOMA | Cell Growth Processes - immunology | RNA, Messenger - immunology | Receptors, G-Protein-Coupled - metabolism | Dendritic Cells - immunology | Humans | Melanoma, Experimental - metabolism | RNA, Messenger - metabolism | Receptors, G-Protein-Coupled - immunology | Intercellular Signaling Peptides and Proteins - metabolism | Melanoma, Experimental - immunology | Chemotactic Factors - metabolism | Myeloid Cells - immunology | Chemotactic Factors - genetics | HEK293 Cells | Killer Cells, Natural - immunology | Receptors, Chemokine - genetics | Dendritic Cells - metabolism | Chemokines - immunology | Chemotactic Factors - immunology | Down-Regulation | Mice, Inbred C57BL | RNA, Messenger - genetics | Receptors, Chemokine - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Melanoma, Experimental - pathology | Receptors, Chemokine - immunology | Chemokines - genetics | Mice, Knockout | Animals | Melanoma, Experimental - genetics | Cell Line, Tumor | Myeloid Cells - metabolism | Chemokines - metabolism | Mice | Receptors, G-Protein-Coupled - genetics | Killer Cells, Natural - metabolism | Intercellular Signaling Peptides and Proteins - immunology | 307 | Brief Definitive Report
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2008, Volume 283, Issue 36, pp. 24748 - 24759
The generation of reactive oxygen species is a central feature of inflammation that results in the oxidation of host phospholipids. Oxidized phospholipids,... 
ACTIVATION | COMPLEX | ESCHERICHIA-COLI LIPOPOLYSACCHARIDE | INDUCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIPID RAFTS | LOW-DENSITY-LIPOPROTEIN | MD-2 | HEME OXYGENASE-1 | MONOCYTE CHEMOTACTIC PROTEIN-1 | OXIDATION-PRODUCTS | Phosphatidylcholines - immunology | Reactive Oxygen Species - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lymphocyte Antigen 96 - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Macrophages, Peritoneal - cytology | Flagellin - pharmacology | Lymphocyte Antigen 96 - agonists | Oxidation-Reduction - drug effects | Tumor Necrosis Factor-alpha - immunology | Toll-Like Receptor 4 - agonists | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Carrier Proteins - immunology | Guanosine - analogs & derivatives | Poly I-C - pharmacology | Macrophages, Peritoneal - immunology | Toll-Like Receptor 4 - genetics | Lipopolysaccharide Receptors - immunology | Toll-Like Receptor 4 - immunology | p38 Mitogen-Activated Protein Kinases - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | I-kappa B Kinase - immunology | Mice | Mice, Inbred BALB C | Macrophages, Peritoneal - metabolism | Oligodeoxyribonucleotides - pharmacology | Immunologic Factors - pharmacology | Lymphocyte Antigen 96 - immunology | Tumor Necrosis Factor-alpha - metabolism | Toll-Like Receptor 2 - agonists | Acute-Phase Proteins - genetics | Lipopolysaccharide Receptors - metabolism | Lymphocyte Antigen 96 - genetics | Carrier Proteins - agonists | I-kappa B Kinase - metabolism | Reactive Oxygen Species - immunology | Female | Membrane Glycoproteins - immunology | Cell Line | Acute-Phase Proteins - metabolism | Phosphatidylcholines - pharmacology | Membrane Glycoproteins - agonists | p38 Mitogen-Activated Protein Kinases - genetics | I-kappa B Kinase - genetics | Membrane Glycoproteins - genetics | Carrier Proteins - genetics | Phosphatidylcholines - genetics | Animals | Carrier Proteins - metabolism | RNA - pharmacology | Guanosine - pharmacology | Lipopolysaccharide Receptors - genetics | Signal Transduction - physiology | Acute-Phase Proteins - agonists | Acute-Phase Proteins - immunology | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 10/2018, Volume 22, Issue 10, pp. 4617 - 4629
Histones are positively charged nuclear proteins that facilitate packaging of DNA into nucleosomes common to all eukaryotic cells. Upon cell injury or cell... 
innate immunity | inflammation | immunothrombosis | extracellular histones | Innate immunity | Immunothrombosis | Inflammation | Extracellular histones | MAMMALIAN TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | DEEP-VEIN THROMBOSIS | CELL BIOLOGY | NEUTROPHIL EXTRACELLULAR TRAPS | IN-VITRO | SECONDARY NECROSIS | MAJOR MEDIATORS | PROMOTE THROMBIN GENERATION | ANTIMICROBIAL PEPTIDE | VII-ACTIVATING PROTEASE | ACUTE-PANCREATITIS | Communicable Diseases - pathology | Communicable Diseases - therapy | Receptors, Pattern Recognition - immunology | Humans | Communicable Diseases - genetics | Necrosis - pathology | Alarmins - blood | Necrosis - immunology | Extracellular Traps - immunology | Extracellular Traps - chemistry | Chemotactic Factors - genetics | Anti-Inflammatory Agents - therapeutic use | Chemotaxis - immunology | Thrombosis - therapy | Blood Coagulation Factors - immunology | Chemotactic Factors - immunology | Alarmins - genetics | Histones - immunology | Signal Transduction | Gene Expression Regulation | Neutrophils | Thrombosis - immunology | Blood Coagulation Factors - genetics | Immunity, Innate | Extracellular Space - immunology | Histones - blood | Chemotactic Factors - blood | Thrombosis - pathology | Histones - genetics | Necrosis - therapy | Communicable Diseases - immunology | Extracellular Space - chemistry | Receptors, Pattern Recognition - genetics | Thrombosis - genetics | Necrosis - genetics | Alarmins - immunology | Histones | Health aspects | DNA binding proteins | Toxicity | Tissue engineering | Immune clearance | Infections | Inflammatory diseases | Necrosis | Proteins | Regeneration | Injury prevention | Cell injury | Microbicides | Packaging | Nucleosomes |