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Endocrinology, ISSN 0013-7227, 10/2007, Volume 148, Issue 10, pp. 4687 - 4694
Soluble protein hormones are key regulators of a number of metabolic processes, including food intake and insulin sensitivity. We have used a signal sequence... 
3T3-L1 ADIPOCYTES | CHINESE MAURITIANS | DENDRITIC CELLS | GENE | ADIPONECTIN | PATHWAY | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | IMPAIRED GLUCOSE-TOLERANCE | EXPRESSION | ADIPOSE-TISSUE | Blood Pressure | Chemokines - blood | Receptors, G-Protein-Coupled - metabolism | Humans | Middle Aged | Adipocytes - cytology | Metabolic Syndrome - metabolism | Diabetes Mellitus, Type 2 - metabolism | Intercellular Signaling Peptides and Proteins - blood | Adipose Tissue - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Chemotactic Factors - metabolism | Chemotactic Factors - genetics | Viscera | Metabolic Syndrome - physiopathology | Adult | Receptors, Chemokine - genetics | Cell Differentiation - physiology | Subcutaneous Tissue - metabolism | Body Mass Index | Gene Expression | Down-Regulation | Receptors, Chemokine - metabolism | Adipose Tissue - pathology | Intercellular Signaling Peptides and Proteins - genetics | Mesentery | 3T3-L1 Cells | Adipocytes - pathology | Chemokines - genetics | Obesity - metabolism | Chemotactic Factors - blood | Diabetes Mellitus, Type 2 - blood | Phenotype | Animals | Gerbillinae | Adipocytes - metabolism | Aged | Chemokines - metabolism | Mice | Receptors, G-Protein-Coupled - genetics | Diabetes Mellitus, Type 2 - pathology | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
Nature, ISSN 0028-0836, 09/2009, Volume 461, Issue 7261, pp. 282 - 286
Phagocytic removal of apoptotic cells occurs efficiently in vivo such that even in tissues with significant apoptosis, very few apoptotic cells are detectable.... 
UTP | THYMUS | RECEPTOR SUBTYPES | EPITHELIAL-CELLS | CHEMOTAXIS | INFLAMMATION | ENGULFMENT | ELEGANS | MULTIDISCIPLINARY SCIENCES | DEATH | EXTRACELLULAR NUCLEOTIDES | Uridine Triphosphate - metabolism | Monocytes - cytology | Humans | Culture Media, Conditioned - chemistry | Culture Media, Conditioned - pharmacology | Monocytes - metabolism | Receptors, Purinergic P2Y2 | Adenosine Triphosphate - pharmacology | Chemotactic Factors - pharmacology | Chemotactic Factors - metabolism | Adenosine Triphosphate - metabolism | Receptors, Purinergic P2 - genetics | Phagocytes - cytology | Cell Line | Phagocytosis - drug effects | Jurkat Cells | Phagocytes - drug effects | Mice, Inbred C57BL | Uridine Triphosphate - pharmacology | Cells, Cultured | Phagocytosis - physiology | Thymus Gland - cytology | Purinergic P2 Receptor Antagonists | Chemotaxis - drug effects | Macrophages - cytology | Phagocytes - metabolism | Monocytes - drug effects | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Receptors, Purinergic P2 - metabolism | Macrophages - drug effects | Mice | Apoptosis - physiology | Macrophage Activation - drug effects | Receptors, Purinergic P2 - deficiency | Culture Media, Conditioned - metabolism | Phagocytes | Nucleotides | Research | Properties | Apoptosis | Rodents | Cells | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 498, Issue 7454, pp. 371 - 375
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6489 - 6499
Chemerin is the ligand of the ChemR23 receptor and a chemoattractant factor for human immature dendritic cells (DCs), macrophages, and NK cells. In this study,... 
ADAPTIVE IMMUNITY | NATURAL-KILLER-CELLS | RESOLVIN E1 | COLONY-STIMULATING FACTOR | IN-VIVO | BONE-MARROW | HUMAN BLOOD | IMMUNOLOGY | CHEMOKINE RECEPTORS | INFLAMED LYMPH-NODES | PULMONARY ALVEOLAR MACROPHAGES | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Dendritic Cells - immunology | Bronchoalveolar Lavage Fluid - immunology | Calcium - immunology | Aequorin - immunology | Aequorin - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Chemotactic Factors - pharmacology | Peptides - metabolism | Chemotactic Factors - metabolism | Pneumonia - chemically induced | Killer Cells, Natural - immunology | Pneumonia - immunology | Dendritic Cells - drug effects | Bronchoalveolar Lavage Fluid - cytology | Chemotaxis - immunology | Neutrophils - metabolism | Apoproteins - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Disease Models, Animal | Recombinant Proteins - metabolism | Chemotactic Factors - immunology | Acute Disease | Lung - pathology | Mice, Inbred C57BL | Neutrophils - drug effects | Peptides - immunology | Neutrophils - immunology | Chemotaxis - drug effects | Mice, Knockout | Macrophages - metabolism | Animals | Recombinant Proteins - immunology | Intercellular Signaling Peptides and Proteins - pharmacology | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Killer Cells, Natural - drug effects | Mice | Receptors, G-Protein-Coupled - genetics | Apoproteins - immunology | Killer Cells, Natural - metabolism | Pneumonia - metabolism | Chemokines | Intercellular Signaling Peptides and Proteins - immunology | Lung - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 05/2017, Volume 94, Issue 4, pp. 790 - 799.e3
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 31 - 44
Polarization of tumor-associated macrophages (TAMs) to a proangiogenic/immune-suppressive (M2-like) phenotype and abnormal, hypoperfused vessels are hallmarks... 
Clodronic Acid - pharmacology | Gene Expression - genetics | Dendritic Cells - immunology | Humans | Pregnancy Proteins - genetics | Microvessels - pathology | Pregnancy Proteins - metabolism | Chemotactic Factors - metabolism | Neoplasm Metastasis - immunology | Antibodies - immunology | Neovascularization, Pathologic - immunology | Macrophages, Peritoneal - drug effects | Macrophages - pathology | Neoplasms - blood supply | Down-Regulation - genetics | Mice, Knockout | Macrophages - metabolism | Neoplasms - immunology | Cell Line, Tumor | Placenta Growth Factor | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Macrophages, Peritoneal - metabolism | Neoplasms - metabolism | CD8-Positive T-Lymphocytes - pathology | Gene Expression - drug effects | Regional Blood Flow - genetics | Dendritic Cells - pathology | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | Regional Blood Flow - drug effects | Transfection | Lung Neoplasms - secondary | Neoplasms - genetics | Dendritic Cells - metabolism | Macrophages - immunology | Lung Neoplasms - genetics | Microvessels - ultrastructure | Cytokines - metabolism | Mice, Inbred C57BL | Microvessels - drug effects | Pregnancy Proteins - immunology | Antibodies - pharmacology | Proteins - genetics | Neoplasm Metastasis - genetics | Hypoxia - genetics | Lung Neoplasms - immunology | Animals | Proteins - metabolism | Endothelial Cells - cytology | Neoplasm Metastasis - pathology | Macrophages - drug effects | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Proteins - pharmacology | Neoplasms - pathology | Endothelial Cells - drug effects | Index Medicus
Journal Article
Neuro-Oncology, ISSN 1522-8517, 11/2010, Volume 12, Issue 11, pp. 1113 - 1125
Macrophages (MΦs)/microglia that constitute the dominant tumor-infiltrating immune cells in glioblastoma are recruited by tumor-secreted factors and are... 
Immune suppression | Innate immunity | Cancer stem cells | Macrophages | Signal transducer and activator of transcription 3 | Microglia | ACTIVATION | TOLL-LIKE RECEPTOR | TGF-BETA SUPERFAMILY | signal transducer and activator of transcription 3 | FACTOR-KAPPA-B | CLINICAL NEUROLOGY | macrophages | GLIOBLASTOMA | innate immunity | ONCOLOGY | TUMOR-ASSOCIATED MACROPHAGES | cancer stem cells | ANTITUMOR IMMUNITY | SUPPRESSOR-CELLS | immune suppression | microglia | T-CELLS | Microglia - metabolism | Phosphorylation | Humans | Brain Neoplasms - pathology | Neoplastic Stem Cells - immunology | Brain Neoplasms - metabolism | Glioma - metabolism | Cell Movement - immunology | Signal Transduction - immunology | Brain Neoplasms - immunology | Flow Cytometry | Microglia - immunology | Transfection | Chemotactic Factors - metabolism | Neoplastic Stem Cells - metabolism | Glioma - pathology | Neoplastic Stem Cells - pathology | Macrophages - immunology | STAT3 Transcription Factor - metabolism | Cytokines - immunology | Chemotactic Factors - immunology | Enzyme-Linked Immunosorbent Assay | Cell Separation | Cells, Cultured | Glioma - immunology | Blotting, Western | Immunity, Innate - immunology | Macrophages - metabolism | STAT3 Transcription Factor - immunology | Chemotaxis, Leukocyte - immunology | Cytokines - biosynthesis | Index Medicus | Basic and Translational Investigations
Journal Article
Nature, ISSN 0028-0836, 10/2009, Volume 461, Issue 7268, pp. 1282 - 1286
Journal Article