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Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 13, pp. 2549 - 2566
DOCK8 mutations result in an inherited combined immunodeficiency characterized by increased susceptibility to skin and other infections. We show that when... 
MEDICINE, RESEARCH & EXPERIMENTAL | HERPES-SIMPLEX-VIRUS | IN-VITRO | DENDRITIC CELLS | LEUKOCYTE MIGRATION | VARICELLA-ZOSTER-VIRUS | RM CELLS | TISSUE | IMMUNOLOGY | ALDRICH-SYNDROME PROTEIN | MEMORY T-CELLS | IMMUNOLOGICAL SYNAPSES
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 5, pp. 1607 - 1620
Chronic inflammation, a hallmark of obliterative bronchiolitis, is known to induce lymphangiogenesis. We therefore studied the role of lymphangiogenic vascular... 
Pathology | CELLS | LUNG TRANSPLANTATION | ACTIVATION | TGF-BETA | RAT TRACHEAL ALLOGRAFTS | FACTOR-C | INFLAMMATION | LYMPHANGIOGENESIS | PATHOLOGY | UP-REGULATION | ENDOTHELIAL GROWTH-FACTOR | Inflammation - pathology | Epithelial Cells - metabolism | Cyclosporine - pharmacology | Dendritic Cells - immunology | Epithelial Cells - drug effects | Trachea - drug effects | Bronchiolitis Obliterans - metabolism | Transplantation, Homologous | Dose-Response Relationship, Drug | Trachea - transplantation | Graft Rejection - complications | Inflammation - complications | Signal Transduction - immunology | Th17 Cells - drug effects | Dendritic Cells - drug effects | Adaptive Immunity - immunology | Bronchiolitis Obliterans - pathology | Graft Rejection - pathology | Macrophages - immunology | Epithelium - drug effects | Immunoglobulins - pharmacology | Epithelium - pathology | Epithelium - metabolism | Immunity, Innate - drug effects | Graft Rejection - physiopathology | Neutrophils - drug effects | Bronchiolitis Obliterans - immunology | Rats | Epithelial Cells - pathology | Inflammation - immunology | Chemotaxis - drug effects | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Down-Regulation - drug effects | Lymphangiogenesis - drug effects | Trachea - pathology | Up-Regulation - drug effects | Immunity, Innate - immunology | Bronchiolitis Obliterans - physiopathology | Adaptive Immunity - drug effects | Animals | Vascular Endothelial Growth Factor C - antagonists & inhibitors | Signal Transduction - drug effects | Vascular Endothelial Growth Factor C - metabolism | Th17 Cells - immunology | Macrophages - drug effects | Graft Rejection - immunology
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 10, p. e0206530
... and can decontaminate and sterilize surfaces, but its exact mode of action is still under debate. This study demonstrates the effect of CAP on the whole proteome of Pseudomonas... 
INACTIVATION | CELLS | MULTIDISCIPLINARY SCIENCES | BIOFILMS | RESISTANCE | LIBRARY | FERRITIN | ATMOSPHERIC-PRESSURE PLASMA | Biofilms - drug effects | Transcription, Genetic - drug effects | Microbial Viability - drug effects | Cystic Fibrosis - microbiology | Proteome - drug effects | Cytochrome b Group - drug effects | Pseudomonas aeruginosa - drug effects | Atmospheric Pressure | Bacterial Proteins - drug effects | Plasma - metabolism | Protein Biosynthesis - drug effects | Anti-Bacterial Agents - pharmacology | Cell Death - drug effects | Cystic Fibrosis - drug therapy | Ferritins - drug effects | High temperature plasmas | Usage | Proteomics | Physiological aspects | Pseudomonas aeruginosa | Influence | Genetic aspects | Liquid chromatography | Research | Microbial mats | Mass spectrometry | Plasma (Ionized gases) | Oxidative stress | Plasma | Cold plasmas | Transcription | Genomics | Radiation | Genomes | Infections | Drug resistance | Inactivation | Proteins | Microorganisms | Decontamination | Medical electronics | Bacteria | Antibacterial activity | Manufacturing | Bacterioferritin | Stress response | Isocitrate dehydrogenase | Pathogens | Medical devices | Cold pressing | Bacterial infections | Deactivation | U.V. radiation | Cystic fibrosis | Mass spectroscopy | Chemotaxis | Disease control | Electric currents | Chromatography | Mutants | Mode of action | Gases | Ultraviolet radiation | Biofilms | Antibiotics | Cell death | Fibrosis | Trigger factor | Proteomes | Apoptosis
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 4, p. e35685
... stem cells to reverse the deleterious effects of myocardial infarction, and the results have all shown an improvement in cardiac function. However, the mechanism... 
MIGRATION | SURVIVAL | BAD | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | H9C2 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INHIBIT APOPTOSIS | CHEMOKINE RECEPTORS | STROMAL CELLS | Neovascularization, Physiologic - drug effects | Gene Expression - drug effects | Apoptosis - drug effects | Caspase 3 - metabolism | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Mesenchymal Stromal Cells - cytology | Caspase 3 - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - drug effects | Endothelial Cells - metabolism | Cytokines - secretion | Mesenchymal Stromal Cells - metabolism | Chemotaxis - drug effects | Mesenchymal Stromal Cells - secretion | Phosphatidylinositol 3-Kinases - genetics | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Endothelial Cells - cytology | Dogs | Mice | Primary Cell Culture | Cytokines - biosynthesis | Cytokines - pharmacology | Endothelial Cells - drug effects | Cytokines | Comparative analysis | Vascular endothelial growth factor | Stem cells | Apoptosis | Heart | Phosphorylation | Leukocyte migration | Mesenchyme | Intracellular signalling | AKT protein | Kinases | Macrophages | Caspase-3 | Proteins | Angiogenesis | Signal transduction | Mitochondria | Cell growth | Ischemia | Rodents | Cell cycle | Tumor necrosis factor-TNF | Conditioning | Localization | Cardiology | Growth factors | Extracellular signal-regulated kinase | Caspase | Cardiomyocytes | Inflammation | Ribonucleic acid--RNA | Chemotaxis | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Inhibitors | γ-Interferon | Myocardium | Hypoxia | Interferon | Laboratory animals | Cell migration | Monocyte chemoattractant protein 1 | RNA | Ribonucleic acid
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 10/2013, Volume 210, Issue 11, pp. 2371 - 2385
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 2, p. e0117356
.... However, the inhibitory effects of R1R2 during vascular remodeling have not been explored. We periadventitially... 
INTERCELLULAR-ADHESION MOLECULE-1 | ARTERIAL STIFFNESS | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | DOMAIN RECEPTOR 2 | GROWTH-FACTOR-BINDING | EXTRACELLULAR-MATRIX | I COLLAGEN | NEOINTIMA FORMATION | OSTEOCLAST MATURATION | KAPPA-B LIGAND | Epithelial Cells - metabolism | Adhesins, Bacterial - chemistry | Epithelial Cells - drug effects | Humans | Hyperplasia | Extracellular Matrix - metabolism | Monocytes - metabolism | Peptide Fragments - pharmacology | Transendothelial and Transepithelial Migration - drug effects | Inflammation - metabolism | Chemotaxis, Leukocyte - drug effects | Inflammation - drug therapy | Protein Binding - drug effects | Monocytes - pathology | Vascular Remodeling - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Cell Line | Collagen - antagonists & inhibitors | Inflammation - immunology | Cell Adhesion - drug effects | Cell Adhesion Molecules - metabolism | Fibronectins - metabolism | Monocytes - drug effects | Cell Movement - drug effects | Collagen - metabolism | Phenotype | Animals | Cell Differentiation - drug effects | Muscle Contraction - drug effects | Neointima - pathology | Cell Proliferation - drug effects | Mice | Fibronectins | Smooth muscle | Inflammation | Peptides | Analysis | Collagen | Vascular cell adhesion molecule 1 | Collagen (type I) | Leukocyte migration | Collagens | Homology | Remodeling | Kinases | Carotid arteries | Fibronectin | Arteries | Cell adhesion & migration | Cell activation | Cell growth | Vascular diseases | Fibroblasts | Extracellular matrix | Carotid artery | Growth factors | Deposition | Thickening | Phenotypes | Intercellular adhesion molecule 1 | Muscles | Muscle contraction | Endothelial cells | Blood flow | Studies | Monocytes | Infiltration | Cell migration | Cellular structure | Veins & arteries
Journal Article
Journal Article
Biomaterials, ISSN 0142-9612, 2018, Volume 178, pp. 504 - 516
... in the U.S since its introduction by the Food and Drug Administration in 1998. However, the advantages and possible mechanisms of action of using folic acid for peripheral nerve engineering and neurological diseases still remain largely elusive... 
Medical prosthetics | Regenerative medicine | Neural engineering | Folic acid | NERVOUS-SYSTEM | HOMOCYSTEINE | MATERIALS SCIENCE, BIOMATERIALS | DNA METHYLATION | ENGINEERING, BIOMEDICAL | REGENERATION | INJURY | VITAMIN-B12 | DISEASE | SCHWANN-CELLS | HEALTH | FOLATE | Folic Acid - pharmacology | Rats, Wistar | Microtubule-Associated Proteins - metabolism | Humans | Male | Nerve Growth Factors - metabolism | PC12 Cells | Schwann Cells - cytology | Tissue Scaffolds - chemistry | Neurites - drug effects | Schwann Cells - drug effects | Astrocytes - cytology | Astrocytes - drug effects | Cell Survival - drug effects | Tissue Engineering - methods | Folic Acid - chemistry | Rats | Neurites - metabolism | Schwann Cells - metabolism | Chemotaxis - drug effects | Polyesters - chemistry | Animals | Nervous System - drug effects | Cell Differentiation - drug effects | Cell Proliferation - drug effects | DNA Methylation - drug effects | Astrocytes - metabolism | Drugs | Medical colleges | Nervous system diseases | Drug delivery systems | Prosthesis | Neurons | Mechanical engineering | Vehicles | Implants, Artificial | Research institutes | Methylation | Health aspects | Biomedical engineering | neural engineering | folic acid | medical prosthetics | regenerative medicine
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 04/2016, Volume 310, Issue 8, pp. R697 - R706
We tested the hypothesis that an increase in the anti-inflammatory cholinergic pathway, when induced by pyridostigmine (PY), may modulate subtypes of... 
Myocardial infarction | T lymphocytes | Anticholinesterase drugs | Macrophage activation | Cholinergic anti-inflammatory reflex | IN-VITRO | PHYSIOLOGY | STIMULATION | BAROREFLEX SENSITIVITY | PATHWAY | HEART-RATE-VARIABILITY | AUTONOMIC FUNCTION | REGULATORY T-CELLS | NICOTINIC ACETYLCHOLINE-RECEPTOR | INFLAMMATORY RESPONSE | FAILURE | T-Lymphocytes, Regulatory - metabolism | Myocardium - immunology | Spleen - immunology | Rats, Inbred WKY | Male | Spleen - drug effects | Myocardial Infarction - immunology | T-Lymphocytes, Regulatory - immunology | Heart Rate - drug effects | Chemotaxis, Leukocyte - drug effects | Time Factors | Forkhead Transcription Factors - metabolism | Myocardial Infarction - pathology | Myocardium - metabolism | CD8-Positive T-Lymphocytes - metabolism | Myocardial Infarction - physiopathology | Blood Pressure - drug effects | Macrophages - immunology | Disease Models, Animal | Biomarkers - metabolism | Pyridostigmine Bromide - pharmacology | Anti-Inflammatory Agents - pharmacology | Cholinergic Neurons - metabolism | Ventricular Function, Left - drug effects | Myocardium - pathology | Cholinergic Neurons - drug effects | Myocardial Infarction - metabolism | Macrophages - metabolism | Phenotype | T-Lymphocytes, Regulatory - drug effects | Animals | Myocardial Infarction - drug therapy | Cholinesterase Inhibitors - pharmacology | Interleukin-2 Receptor alpha Subunit - metabolism | Spleen - metabolism | CD8-Positive T-Lymphocytes - drug effects | Macrophages - drug effects | CD8-Positive T-Lymphocytes - immunology | Macrophage Activation - drug effects | Cholinergic Neurons - pathology | Anti-inflammatory drugs | Heart attack | Euthanasia | Physiological aspects | Dosage and administration | Lymphocytes | Cholinesterase inhibitors | Health aspects | cholinergic anti-inflammatory reflex | anticholinesterase drugs | Translational Physiology | macrophage activation | myocardial infarction
Journal Article