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Immunity, ISSN 1074-7613, 03/2017, Volume 46, Issue 3, pp. 457 - 473
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Blood, ISSN 0006-4971, 02/2013, Volume 121, Issue 6, pp. 1008 - 1015
The majority of peripheral serotonin is stored in platelets, which secrete it on activation. Serotonin releases Weibel-Palade bodies (WPBs) and we asked... 
PULMONARY-HYPERTENSION | PERMEABILITY | 5-HYDROXYTRYPTAMINE | ENDOTHELIAL-CELLS | MAST-CELLS | P-SELECTIN | RECEPTOR | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | VON-WILLEBRAND-FACTOR | SELECTIN-DEFICIENT MICE | Blood Platelets - immunology | Endothelium, Vascular - drug effects | Male | Shock, Septic - immunology | Lipopolysaccharides | Serotonin - blood | Inflammation - metabolism | L-Selectin - immunology | Leukocytosis - metabolism | Weibel-Palade Bodies - metabolism | Chemotaxis - immunology | Neutrophils - metabolism | Serotonin - immunology | Serotonin Uptake Inhibitors - immunology | Endothelium, Vascular - immunology | Neutrophil Infiltration - immunology | Fluoxetine - pharmacology | Acute Disease | Neutrophils - drug effects | Leukocytosis - immunology | Chemotaxis - drug effects | Mice, Knockout | Blood Platelets - metabolism | Endothelium, Vascular - metabolism | Mice | Serotonin Uptake Inhibitors - pharmacology | Leukocyte Rolling - drug effects | Weibel-Palade Bodies - immunology | Histamine - immunology | L-Selectin - metabolism | Flow Cytometry | Tryptophan Hydroxylase - deficiency | Blood Platelets - drug effects | Fluoxetine - immunology | Shock, Septic - genetics | Leukocyte Rolling - genetics | Histamine - pharmacology | Shock, Septic - chemically induced | Mice, Inbred C57BL | Kaplan-Meier Estimate | Neutrophils - immunology | Inflammation - immunology | Leukocyte Rolling - immunology | Tryptophan Hydroxylase - genetics | Weibel-Palade Bodies - drug effects | Animals | Serotonin - metabolism | Inflammation - genetics | Leukocytosis - genetics | Phagocytes, Granulocytes, and Myelopoiesis | Platelets and Thrombopoiesis
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1037 - 1048
Resolution of inflammation is now recognized as a biosynthetically active process involving pro-resolving mediators. Here, we show in zymosan-initiated... 
CIRCUITS | MEDICINE, RESEARCH & EXPERIMENTAL | PROTECTINS | CHEMOTAXIS | INHIBITS LEUKOCYTE MIGRATION | NETRIN-1 | ATHEROSCLEROSIS | MECHANISMS | IMMUNOLOGY | RECEPTORS | HUMAN PHAGOCYTES | PRORESOLVING LIPID MEDIATORS | Netrin-1 | Humans | Lipids - immunology | Monocytes - metabolism | Monocytes - immunology | Nerve Growth Factors - metabolism | Docosahexaenoic Acids - immunology | Inflammation - metabolism | Lipids - chemistry | Tumor Suppressor Proteins - genetics | Inflammation Mediators - metabolism | Chemotaxis - immunology | Neutrophils - metabolism | Macrophages - immunology | Inflammation Mediators - immunology | Recombinant Proteins - metabolism | Zymosan | Tumor Suppressor Proteins - metabolism | Vagus Nerve - metabolism | Mice, Inbred C57BL | Neutrophils - drug effects | Peritonitis - metabolism | Cells, Cultured | Lipoxins - immunology | Neutrophils - immunology | Vagus Nerve - surgery | Inflammation - immunology | Nerve Growth Factors - immunology | Peritonitis - chemically induced | Chemotaxis - drug effects | Peritonitis - immunology | Recombinant Proteins - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Tumor Suppressor Proteins - immunology | Vagotomy | Blotting, Western | Mice, Knockout | Macrophages - metabolism | Animals | Recombinant Proteins - immunology | Nerve Growth Factors - genetics | Vagus Nerve - immunology | Inflammation - genetics | Mice | Lipoxins - metabolism | Microscopy, Fluorescence | Docosahexaenoic Acids - metabolism | 303
Journal Article
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA
Journal Article
The Journal of Immunology, ISSN 0022-1767, 11/2009, Volume 183, Issue 10, pp. 6489 - 6499
Chemerin is the ligand of the ChemR23 receptor and a chemoattractant factor for human immature dendritic cells (DCs), macrophages, and NK cells. In this study,... 
ADAPTIVE IMMUNITY | NATURAL-KILLER-CELLS | RESOLVIN E1 | COLONY-STIMULATING FACTOR | IN-VIVO | BONE-MARROW | HUMAN BLOOD | IMMUNOLOGY | CHEMOKINE RECEPTORS | INFLAMED LYMPH-NODES | PULMONARY ALVEOLAR MACROPHAGES | Receptors, G-Protein-Coupled - metabolism | Calcium - metabolism | Dendritic Cells - immunology | Bronchoalveolar Lavage Fluid - immunology | Calcium - immunology | Aequorin - immunology | Aequorin - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Chemotactic Factors - pharmacology | Peptides - metabolism | Chemotactic Factors - metabolism | Pneumonia - chemically induced | Killer Cells, Natural - immunology | Pneumonia - immunology | Dendritic Cells - drug effects | Bronchoalveolar Lavage Fluid - cytology | Chemotaxis - immunology | Neutrophils - metabolism | Apoproteins - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Disease Models, Animal | Recombinant Proteins - metabolism | Chemotactic Factors - immunology | Acute Disease | Lung - pathology | Mice, Inbred C57BL | Neutrophils - drug effects | Peptides - immunology | Neutrophils - immunology | Chemotaxis - drug effects | Mice, Knockout | Macrophages - metabolism | Animals | Recombinant Proteins - immunology | Intercellular Signaling Peptides and Proteins - pharmacology | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Killer Cells, Natural - drug effects | Mice | Receptors, G-Protein-Coupled - genetics | Apoproteins - immunology | Killer Cells, Natural - metabolism | Pneumonia - metabolism | Chemokines | Intercellular Signaling Peptides and Proteins - immunology | Lung - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Molecular Immunology, ISSN 0161-5890, 05/2014, Volume 59, Issue 1, pp. 55 - 63
Appropriate modulation of immunity is beneficial in antimicrobial therapy and vaccine development. Host defense peptides (HDPs) constitute critically important... 
Adjuvant | Host defense peptides | Antimicrobial resistance | Immunomodulation | MRSA | Antimicrobial peptides | CELLS | BACTERIAL-INFECTIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE | MONOCYTES | RECEPTOR | IMMUNOLOGY | ANTIINFECTIVE PEPTIDE | RESPONSES | CPG OLIGONUCLEOTIDE | CHICKEN | Antimicrobial Cationic Peptides - immunology | Cathelicidins - immunology | Chemokine CCL3 - immunology | Chemokine CCL2 - immunology | Anti-Infective Agents - pharmacology | Male | Peptide Fragments - pharmacology | Disease Resistance - immunology | Interleukin-1beta - genetics | Anti-Infective Agents - immunology | Host-Pathogen Interactions - immunology | Flow Cytometry | Chemokine CCL3 - genetics | Interleukin-1beta - metabolism | Peptide Fragments - immunology | Inflammation Mediators - metabolism | Chemokine CCL3 - metabolism | Histocompatibility Antigens Class II - metabolism | Chemokine CCL2 - metabolism | Staphylococcal Infections - microbiology | Chemotaxis - immunology | B7-2 Antigen - immunology | Cathelicidins - pharmacology | Neutrophils - metabolism | Macrophages - immunology | Inflammation Mediators - immunology | Methicillin-Resistant Staphylococcus aureus - physiology | Cell Line | Immunity, Innate - drug effects | Staphylococcal Infections - immunology | Peptides - immunology | Antimicrobial Cationic Peptides - pharmacology | Neutrophils - immunology | Interleukin-1beta - immunology | Chemokine CCL2 - genetics | Methicillin-Resistant Staphylococcus aureus - immunology | Chemotaxis - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Peptides - pharmacology | B7-2 Antigen - metabolism | Immunity, Innate - immunology | Macrophages - metabolism | Animals | Histocompatibility Antigens Class II - immunology | Chickens | Macrophages - drug effects | Mice | Mice, Inbred BALB C | Peptides
Journal Article