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Nature Cell Biology, ISSN 1465-7392, 07/2017, Volume 19, Issue 7, pp. 774 - 786
Journal Article
Gastroenterology, ISSN 0016-5085, 2008, Volume 134, Issue 2, pp. 511 - 522
Background & Aims: Loss of gastric parietal cells is a critical precursor to gastric metaplasia and neoplasia. However, the origin of metaplasia remains... 
Gastroenterology and Hepatology | OXYNTIC ATROPHY | LOCALIZATION | COMPLEX | ZYMOGENIC CELLS | ADENOCARCINOMA | RISK | INFECTION | DEFICIENT MICE | TISSUES | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Metaplasia - metabolism | Humans | Parietal Cells, Gastric - metabolism | Stomach Neoplasms - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | beta-Defensins | Fetal Proteins - metabolism | Gastric Mucosa - metabolism | Stomach Neoplasms - physiopathology | Minichromosome Maintenance Complex Component 3 | Atrophy | DNA-Binding Proteins - metabolism | Peptides - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Mucins - metabolism | Muscle Proteins - metabolism | Precancerous Conditions - pathology | Trefoil Factor-2 | Gastric Fundus - metabolism | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Microtubule-Associated Proteins | Nuclear Proteins - metabolism | Gastrins - metabolism | Cell Transformation, Neoplastic - metabolism | Metaplasia - pathology | Mice, Knockout | Animals | Carrier Proteins - metabolism | Parietal Cells, Gastric - pathology | Epididymal Secretory Proteins - metabolism | Chief Cells, Gastric - pathology | Mice | Cell Transformation, Neoplastic - pathology | Gastric Fundus - pathology | Intrinsic Factor - metabolism | Vasoactive intestinal peptides | Analysis | Polypeptides
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 4, pp. 918 - 930.e13
Background & Aims Gastric cancer develops in the context of parietal cell loss, spasmolytic polypeptide-expressing metaplasia (SPEM), and intestinal metaplasia... 
Gastroenterology and Hepatology | MAP Kinase | Signal Transduction | Differentiation | Carcinogenesis | PROGENITORS | INTESTINAL METAPLASIA | MARKER | HELICOBACTER-FELIS | INDUCTION | MODEL | LUNG-CANCER | KRAS | GASTROENTEROLOGY & HEPATOLOGY | PROGRESSION | Humans | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Male | Stomach Neoplasms - pathology | Mitogen-Activated Protein Kinase Kinases - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Female | Cell Differentiation | Anticarcinogenic Agents - pharmacology | Stomach Neoplasms - genetics | Genetic Predisposition to Disease | Macrophages - pathology | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Mice, Transgenic | Cell Transformation, Neoplastic - metabolism | Disease Progression | Chief Cells, Gastric - drug effects | Cell Lineage | Metaplasia | Macrophages - metabolism | Phenotype | Animals | Stomach Neoplasms - prevention & control | Benzimidazoles - pharmacology | Chief Cells, Gastric - pathology | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Cell Transformation, Neoplastic - drug effects | Mutation | Cell Transformation, Neoplastic - pathology | Genes, ras | Medical colleges | Developmental biology | Hospitals, Military | Index Medicus | Abridged Index Medicus | MAP kinase | differentiation | carcinogenesis | signal transduction
Journal Article
Journal Article
Developmental Biology, ISSN 0012-1606, 2009, Volume 325, Issue 1, pp. 211 - 224
In the mammalian gastrointestinal tract, the cell fate decisions that specify the development of multiple, diverse lineages are governed in large part by... 
Niche | Mucous neck cell | Chief cell | Bhlhb8 | Stem cell | CD2AP | STEM-CELLS | SONIC HEDGEHOG | GASTROINTESTINAL-TRACT | MOUSE STOMACH | DEVELOPMENTAL BIOLOGY | SEROUS EXOCRINE CELLS | PARIETAL-CELLS | GROWTH-FACTOR-ALPHA | HELICOBACTER-PYLORI | CD2-ASSOCIATED PROTEIN | TRANSGENIC MICE | Cell Polarity | Cell Proliferation | Epithelial Cells - metabolism | Cadherins - metabolism | Chief Cells, Gastric - ultrastructure | Parietal Cells, Gastric - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Cytoskeletal Proteins - deficiency | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cytoskeletal Proteins - metabolism | Cell Differentiation | Epithelial Cells - cytology | Basic Helix-Loop-Helix Transcription Factors - deficiency | Chief Cells, Gastric - metabolism | Intercellular Junctions - metabolism | Epithelial Cells - ultrastructure | Stem Cell Niche - cytology | Parietal Cells, Gastric - cytology | Stem Cells - ultrastructure | Protein Transport | Cell Lineage | Chief Cells, Gastric - cytology | Animals | Models, Biological | Parietal Cells, Gastric - ultrastructure | Adaptor Proteins, Signal Transducing - deficiency | Stem Cell Niche - metabolism | Protein Binding | Stem Cell Niche - ultrastructure | Mice | Adaptor Proteins, Signal Transducing - metabolism | Intercellular Junctions - ultrastructure | Enzymes | Analysis | chief cell | niche | mucous neck cell
Journal Article
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 11/2016, Volume 312, Issue 1, pp. G67 - G76
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 11, pp. 2438 - 2448
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 6, pp. 2050 - 2060.e2
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, pp. e0184514 - e0184514
The cytoprotective protein clusterin is often dysregulated during tumorigenesis, and in the stomach, upregulation of clusterin marks emergence of the oxyntic... 
BETA-SUBUNIT-DEFICIENT | INTESTINAL METAPLASIA | PARIETAL-CELLS | EPITHELIAL-CELLS | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION |