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Nature Cell Biology, ISSN 1465-7392, 07/2017, Volume 19, Issue 7, pp. 774 - 786
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 139, Issue 6, pp. 2028 - 2037.e9
Background & Aims Gastric cancer evolves in the setting of a pathologic mucosal milieu characterized by both loss of acid-secreting parietal cells and mucous... 
Gastroenterology and Hepatology | Metaplasia | Chief Cell | SPEM | Transdifferentiation | OXYNTIC ATROPHY | AMPHIREGULIN-DEFICIENT MICE | INTESTINAL METAPLASIA | STEM-CELLS | POLYPEPTIDE-EXPRESSING METAPLASIA | EPITHELIAL-CELLS | ZYMOGENIC CELLS | HUMAN GASTRIC-MUCOSA | PANCREATIC INTRAEPITHELIAL NEOPLASIA | GASTROENTEROLOGY & HEPATOLOGY | HELICOBACTER-PYLORI | Precancerous Conditions - physiopathology | Peptides - genetics | Lac Operon - genetics | Stomach Neoplasms - pathology | Stomach Neoplasms - physiopathology | Helicobacter Infections - pathology | Peptides - metabolism | Gastritis - pathology | Gastritis - microbiology | Precancerous Conditions - pathology | Parietal Cells, Gastric - physiology | Cell Differentiation - physiology | Helicobacter Infections - physiopathology | Disease Models, Animal | Acute Disease | Mice, Inbred C57BL | Mice, Transgenic | Gastritis - physiopathology | Cell Lineage - physiology | Cell Division - physiology | Stomach Neoplasms - microbiology | Animals | Parietal Cells, Gastric - pathology | Chief Cells, Gastric - physiology | Stem Cells - pathology | Chief Cells, Gastric - pathology | Stem Cells - physiology | Helicobacter felis | Mice | Precancerous Conditions - microbiology | Chronic Disease | Universities and colleges | Research | Developmental biology | Health aspects | Oncology, Experimental | Cancer | Hospitals, Military
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 4, pp. 918 - 930.e13
Background & Aims Gastric cancer develops in the context of parietal cell loss, spasmolytic polypeptide-expressing metaplasia (SPEM), and intestinal metaplasia... 
Gastroenterology and Hepatology | MAP Kinase | Signal Transduction | Differentiation | Carcinogenesis | PROGENITORS | INTESTINAL METAPLASIA | MARKER | HELICOBACTER-FELIS | INDUCTION | MODEL | LUNG-CANCER | KRAS | GASTROENTEROLOGY & HEPATOLOGY | PROGRESSION | Humans | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Male | Stomach Neoplasms - pathology | Mitogen-Activated Protein Kinase Kinases - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Female | Cell Differentiation | Anticarcinogenic Agents - pharmacology | Stomach Neoplasms - genetics | Genetic Predisposition to Disease | Macrophages - pathology | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Mice, Transgenic | Cell Transformation, Neoplastic - metabolism | Disease Progression | Chief Cells, Gastric - drug effects | Cell Lineage | Metaplasia | Macrophages - metabolism | Phenotype | Animals | Stomach Neoplasms - prevention & control | Benzimidazoles - pharmacology | Chief Cells, Gastric - pathology | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Cell Transformation, Neoplastic - drug effects | Mutation | Cell Transformation, Neoplastic - pathology | Genes, ras | Medical colleges | Developmental biology | Hospitals, Military | Index Medicus | Abridged Index Medicus | MAP kinase | differentiation | carcinogenesis | signal transduction
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2008, Volume 134, Issue 2, pp. 511 - 522
Background & Aims: Loss of gastric parietal cells is a critical precursor to gastric metaplasia and neoplasia. However, the origin of metaplasia remains... 
Gastroenterology and Hepatology | OXYNTIC ATROPHY | LOCALIZATION | COMPLEX | ZYMOGENIC CELLS | ADENOCARCINOMA | RISK | INFECTION | DEFICIENT MICE | TISSUES | GASTROENTEROLOGY & HEPATOLOGY | CANCER | Metaplasia - metabolism | Humans | Parietal Cells, Gastric - metabolism | Stomach Neoplasms - metabolism | Gastric Mucosa - pathology | Precancerous Conditions - metabolism | Stomach Neoplasms - pathology | beta-Defensins | Fetal Proteins - metabolism | Gastric Mucosa - metabolism | Stomach Neoplasms - physiopathology | Minichromosome Maintenance Complex Component 3 | Atrophy | DNA-Binding Proteins - metabolism | Peptides - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Mucins - metabolism | Muscle Proteins - metabolism | Precancerous Conditions - pathology | Trefoil Factor-2 | Gastric Fundus - metabolism | Chief Cells, Gastric - metabolism | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Microtubule-Associated Proteins | Nuclear Proteins - metabolism | Gastrins - metabolism | Cell Transformation, Neoplastic - metabolism | Metaplasia - pathology | Mice, Knockout | Animals | Carrier Proteins - metabolism | Parietal Cells, Gastric - pathology | Epididymal Secretory Proteins - metabolism | Chief Cells, Gastric - pathology | Mice | Cell Transformation, Neoplastic - pathology | Gastric Fundus - pathology | Intrinsic Factor - metabolism | Vasoactive intestinal peptides | Analysis | Polypeptides
Journal Article
Journal Article
Histopathology, ISSN 0309-0167, 05/2016, Volume 68, Issue 6, pp. 825 - 833
Aims Rare gastric lesions composed of a combined proliferation of chief and oxyntic cells have been variably called adenocarcinoma of fundic gland type and... 
stomach neoplasm | fundic gland | chief cells | oxyntic gland | gastric polyp | Fundic gland | Oxyntic gland | Chief cells | Gastric polyp | Stomach neoplasm | Immunohistochemistry | Biomarkers, Tumor - analysis | Humans | Middle Aged | Polyps - pathology | Male | Stomach Neoplasms - pathology | Aged, 80 and over | Adult | Chief Cells, Gastric - pathology | Female | Aged | Gastric Fundus - pathology | Morphology | Tumors
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 11/2016, Volume 312, Issue 1, pp. G67 - G76
Journal Article
Journal Article