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WORLD JOURNAL OF GASTROENTEROLOGY, ISSN 1007-9327, 02/2019, Volume 25, Issue 6, pp. 659 - 671
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 4, pp. 1509 - 1519.e3
Background & Aims Chronic, progressive hepatobiliary disease is the most severe complication of erythropoietic protoporphyria (EPP) and can require liver... 
Gastroenterology and Hepatology | Heme Biosynthesis | Protoporphyrin | Liver Disease | Ferrochelatase | CRYSTALLIZATION | HOUSE MOUSE | BILIARY FIBROSIS | KNOCKOUT MICE | P-GLYCOPROTEIN | SALT | LIVER-INJURY | FERROCHELATASE-DEFICIENT MICE | ANEMIA | GASTROENTEROLOGY & HEPATOLOGY | BILE-ACIDS | Kupffer Cells - pathology | Cholangitis, Sclerosing - prevention & control | Cholangitis, Sclerosing - genetics | Lipoprotein-X - blood | Hepatocytes - pathology | Hepatocytes - metabolism | Porphyria, Erythropoietic - genetics | Liver Cirrhosis - metabolism | Cholangitis, Sclerosing - metabolism | Kupffer Cells - metabolism | Phospholipids - metabolism | Porphyria, Erythropoietic - metabolism | Disease Models, Animal | Severity of Illness Index | Liver Cirrhosis - prevention & control | Mice, Inbred C57BL | Gene Expression Regulation | Bile Acids and Salts - metabolism | Genotype | Protoporphyrins - metabolism | Porphyria, Erythropoietic - pathology | Cholesterol - metabolism | Cholangitis, Sclerosing - pathology | Point Mutation | Phenotype | Animals | Ferrochelatase - metabolism | Ferrochelatase - genetics | Porphyria, Erythropoietic - complications | Mice | Mice, Inbred BALB C | Prevention | Liver diseases | Heme | Transplantation of organs, tissues, etc | Phospholipids | Hemoglobin synthesis | Organic chemistry | Chemical Sciences
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 2, pp. 499 - 512.e20
Background & Aims Under conditions of inflammation in the absence of micro-organisms (sterile inflammation), necrotic cells release damage-associated molecular... 
Gastroenterology and Hepatology | Immune Regulation | TLR | Cytokines | Mouse Model | ACTIVATION | MACROPHAGES | INCREASES | LUNG INJURY | DYING CELLS | IN-VIVO | ACINAR-CELLS | MICE | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | SUBUNIT | Phosphorylation | Bile Ducts - metabolism | Cholangitis, Sclerosing - prevention & control | Humans | Protein Multimerization | Transcription Factors - deficiency | Cholangitis, Sclerosing - genetics | I-kappa B Proteins - metabolism | Pancreatitis - prevention & control | Pancreatitis - genetics | Ubiquitination | Taurocholic Acid | Time Factors | Proteolysis | Bone Marrow Transplantation | Cholangitis, Sclerosing - metabolism | Proto-Oncogene Proteins - metabolism | Acute Disease | NF-KappaB Inhibitor alpha | NF-kappa B p50 Subunit - metabolism | Signal Transduction | Mice, Inbred C57BL | Pancreas - pathology | Proto-Oncogene Proteins - genetics | Pancreas - metabolism | Proto-Oncogene Proteins - deficiency | Transcription Factors - genetics | Pancreatitis - chemically induced | Mice, Knockout | Cholangitis, Sclerosing - pathology | Transcription Factors - metabolism | Animals | Transcription Factor RelA - metabolism | ATP Binding Cassette Transporter, Sub-Family B - metabolism | Pancreatitis - pathology | Pancreatitis - metabolism | Proteasome Endopeptidase Complex - metabolism | Ceruletide | ATP Binding Cassette Transporter, Sub-Family B - genetics | Bile Ducts - pathology | Analysis | Inflammation
Journal Article
Journal Article
Annals of Oncology, ISSN 0923-7534, 1/1999, Volume 10, Issue S4, pp. 208 - 211
Patients with primary sclerosing cholangitis (PSC) have a substantial predisposition to develop bile duct carcinoma. The mechanism is still unclear but the... 
dysplasia | cytology | Medicine & Public Health | Child-Pugh classification | Oncology | cholangiocarcinoma | liver transplantation | primary sclerosing cholangitis
Journal Article