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Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1393 - 14
In infants intolerant of enteral feeding because of intestinal disease, parenteral nutrition may be associated with cholestasis, which can progress to... 
LIPID EMULSIONS | ACID RECEPTOR | INTRAHEPATIC CHOLESTASIS | SALT EXPORT PUMP | SHORT-BOWEL SYNDROME | MULTIDISCIPLINARY SCIENCES | PLANT STEROLS | BILIARY CHOLESTEROL SECRETION | LIVER-DISEASE | INTESTINAL FAILURE | CHILDREN | Receptors, CCR2 - genetics | Cholestasis - genetics | Liver - pathology | Caspases - immunology | Lipoproteins - genetics | Receptors, CCR2 - immunology | Humans | Receptors, Interleukin-1 - genetics | NF-kappa B - immunology | Hepatocytes - pathology | Male | Caspase 1 - immunology | Interleukin-1beta - genetics | Cholestasis - immunology | Liver - immunology | Receptors, Cytoplasmic and Nuclear - immunology | ATP Binding Cassette Transporter, Subfamily B, Member 11 - immunology | Cholestasis - etiology | Multidrug Resistance-Associated Proteins - immunology | Liver X Receptors - immunology | Multidrug Resistance-Associated Proteins - genetics | Infant, Newborn | Macrophages - immunology | Disease Models, Animal | ATP Binding Cassette Transporter, Subfamily G, Member 5 - genetics | Parenteral Nutrition - adverse effects | Macrophages - pathology | Signal Transduction | Caspases - genetics | Receptors, Interleukin-1 - immunology | Gene Expression Regulation | Interleukin-1beta - immunology | Receptors, Cytoplasmic and Nuclear - genetics | Hepatocytes - immunology | ATP Binding Cassette Transporter, Subfamily B, Member 11 - genetics | Cholestasis - pathology | Animals | NF-kappa B - genetics | Caspase 1 - genetics | ATP Binding Cassette Transporter, Subfamily G, Member 5 - immunology | Mice | Receptors, CCR2 - deficiency | Lipoproteins - immunology | Liver X Receptors - genetics | CC chemokine receptors | Transcription | Caspase-11 | Pathogenesis | Liver | Gallbladder diseases | Infants | Interleukin 1 receptors | Macrophages | Caspase-1 | Sterols | CCR2 protein | Intestine | Rodents | Interleukin 1 | Chemical bonds | NF-κB protein | Liver diseases | Nutrition | Liver X receptors | Phytosterols | Caspase | Parenteral nutrition | Pharmacology | Catheters | Enteral feeding | Signaling | Transporter | Monocyte chemoattractant protein 1 | Cholestasis | Bile
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, p. e0199863
Cholestatic patients exhibiting high bile acid serum levels were reported to be more susceptible to bacterial and viral infections. Animal studies in bile duct... 
RESPONSES | RECRUITMENT | MURINE CYTOMEGALOVIRUS-INFECTION | REPLICATION | INTERFERON-GAMMA | LIVER-INJURY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | CHOLESTASIS | INNATE IMMUNE-SYSTEM | Inflammation - pathology | Humans | Male | Killer Cells, Natural - pathology | Monocytes - immunology | Cholestasis - virology | Cell Movement - immunology | Cholestasis - immunology | Herpesviridae Infections - pathology | Ligation | Monocytes - pathology | Killer Cells, Natural - immunology | Chemokines - immunology | Bile Acids and Salts - immunology | Inflammation - virology | Virus Replication - immunology | Inflammation - immunology | Cholestasis - pathology | Animals | Muromegalovirus - physiology | Bile Ducts - virology | Mice | Bile Ducts - immunology | Bile Ducts - pathology | Herpesviridae Infections - immunology | Jaundice, Obstructive | Care and treatment | Bile acids | Physiological aspects | Cytomegalovirus infections | Development and progression | Health aspects | Cholestasis | CC chemokine receptors | Liver | Trafficking | Viruses | Infections | Recruitment | CCR2 protein | Rodents | Cytomegalovirus | Myeloid cells | Immune response | Mortality | Inflammation | CXCR3 protein | Bile duct | Serum levels | Monocytes | Acids | Hepatocytes | CXCL10 protein | Interleukin 10 | Replication | Monocyte chemoattractant protein 1 | Bile
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2011, Volume 121, Issue 11, pp. 4244 - 4256
Biliary atresia (BA) is a destructive cholangiopathy of childhood in which Th1 immunity has been mechanistically linked to the bile duct inflammation and... 
MEDICINE, RESEARCH & EXPERIMENTAL | DISEASE | CYTOKINES INHIBIT | DIFFERENTIATION | MACROPHAGE POLARIZATION | CD8(+) T-CELLS | BILE-DUCTS | ALTERNATIVE ACTIVATION | MONONUCLEAR PHAGOCYTES | HEPATIC-FIBROSIS | LYMPHOCYTES | Liver - pathology | STAT1 Transcription Factor - deficiency | Humans | Interleukin-13 - immunology | STAT6 Transcription Factor - genetics | Th2 Cells - immunology | RNA, Messenger - metabolism | Th1 Cells - immunology | Cholestasis - immunology | Epithelium - immunology | Signal Transduction - immunology | Interleukin-13 - genetics | Liver - immunology | Cholestasis - etiology | Myeloid Cells - immunology | Child | Cytokines - genetics | Disease Models, Animal | Animals, Newborn | Biliary Atresia - etiology | Epithelium - pathology | Rotavirus - pathogenicity | Cytokines - metabolism | Biliary Atresia - immunology | Interleukin-13 - deficiency | RNA, Messenger - genetics | STAT1 Transcription Factor - genetics | STAT6 Transcription Factor - immunology | Biliary Atresia - pathology | STAT1 Transcription Factor - immunology | Mice, Knockout | Phenotype | Animals | Cholestasis - prevention & control | STAT6 Transcription Factor - deficiency | Mice | Mice, Inbred BALB C | Myeloid Cells - pathology | Biliary atresia | Care and treatment | Epithelial cells | Cellular signal transduction | Genetic aspects | Diagnosis | Research | Identification and classification
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 04/2018, Volume 1864, Issue 4, pp. 1374 - 1379
Journal Article
Hepatology, ISSN 0270-9139, 07/2014, Volume 60, Issue 1, pp. 237 - 249
Toll‐like receptor 7 (TLR7) signaling predominantly regulates production of type I interferons (IFNs), which has been suggested in clinical studies to be... 
ANTAGONIST | DENDRITIC CELLS | RNA | RECOGNITION | INFLAMMATION | CHRONIC HEPATITIS-C | MOUSE | ALPHA | MICE | INDUCTION | GASTROENTEROLOGY & HEPATOLOGY | Liver Cirrhosis - immunology | Carbon Tetrachloride - pharmacology | Cholestasis - metabolism | Membrane Glycoproteins - metabolism | Dendritic Cells - immunology | Male | Cholestasis - immunology | Signal Transduction - immunology | Interleukin 1 Receptor Antagonist Protein - metabolism | Toll-Like Receptor 7 - immunology | Liver Cirrhosis - metabolism | Interferon Type I - metabolism | Female | Kupffer Cells - metabolism | Receptor, Interferon alpha-beta - genetics | Membrane Glycoproteins - immunology | Dendritic Cells - metabolism | Disease Models, Animal | Receptor, Interferon alpha-beta - metabolism | Toll-Like Receptor 7 - genetics | Chemical and Drug Induced Liver Injury - prevention & control | Interleukin 1 Receptor Antagonist Protein - immunology | Liver Cirrhosis - prevention & control | Toll-Like Receptor 7 - metabolism | Mice, Inbred C57BL | Chemical and Drug Induced Liver Injury - immunology | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Cholestasis - prevention & control | Receptor, Interferon alpha-beta - immunology | Chemical and Drug Induced Liver Injury - metabolism | Mice | Mice, Inbred BALB C | Chronic Disease | Kupffer Cells - immunology | Liver cirrhosis | Rodents | Hepatology | Immune system | Index Medicus | liver fibrosis | TLR7 | IL-1ra | Type I IFN
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 51, pp. 83951 - 83963
Accumulation of hydrophobic bile acids in the liver contributes to cholestatic liver injury. Inflammation induced by excessive bile acids is believed to play a... 
Immune response | Bile acid | Immunology and Microbiology Section | Inflammasome | Inflammation | Liver fibrosis | IL-1β | Immunity | CELLS | OXIDATIVE STRESS | bile acid | INJURY | ALPHA | IL-1 beta | liver fibrosis | RECEPTOR TGR5 | ONCOLOGY | inflammation | OBSTRUCTIVE-CHOLESTASIS | inflammasome | MOUSE MODEL | ATP RELEASE | BILE-ACIDS | EGF RECEPTOR | Potassium - metabolism | Kupffer Cells - pathology | Liver Cirrhosis - immunology | Caspase Inhibitors - pharmacology | Liver - pathology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Cholestasis - complications | Cholestasis - metabolism | Receptors, G-Protein-Coupled - metabolism | Caspase 3 - metabolism | Chenodeoxycholic Acid - toxicity | Liver Cirrhosis - chemically induced | Cholestasis - immunology | Dose-Response Relationship, Drug | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Liver - immunology | Receptor, Epidermal Growth Factor - metabolism | Ligation | Transfection | Inflammasomes - drug effects | Liver - drug effects | RNA Interference | Adenosine Triphosphate - metabolism | Liver Cirrhosis - metabolism | Kupffer Cells - metabolism | Chemical and Drug Induced Liver Injury - etiology | Disease Models, Animal | Cell Line | Chemical and Drug Induced Liver Injury - prevention & control | Liver Cirrhosis - prevention & control | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Liver - metabolism | Mice, Inbred C57BL | Interleukin-1beta | Bile Ducts - surgery | Chemical and Drug Induced Liver Injury - immunology | Kupffer Cells - drug effects | Cholestasis - pathology | Animals | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Inflammasomes - immunology | Kupffer Cells - immunology
Journal Article
Liver International, ISSN 1478-3223, 09/2012, Volume 32, Issue 8, pp. 1253 - 1261
Background Biliary strictures after liver transplantation (LT) are a major cause of morbidity and reduced graft survival. Aims The purpose of this study was to... 
CX3CR1 | ischaemic type biliary lesions | anastomotic biliary strictures | chemokine | liver transplantation | CCR5Δ32 | Chemokine | Anastomotic biliary strictures | Ischaemic type biliary lesions | Liver transplantation | FRACTALKINE | CCR5 Delta 32 | ALLOGRAFT SURVIVAL | RECIPIENTS | BILE-DUCTS | LESIONS | ANTI-HLA | DISEASE | PREFORMED ANTIBODIES | CHEMOKINE RECEPTORS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Receptors, CCR2 - genetics | Cholestasis - genetics | Predictive Value of Tests | Graft Survival - immunology | Liver Transplantation - adverse effects | Receptors, CCR2 - immunology | Humans | Middle Aged | Anastomosis, Surgical - adverse effects | Anastomosis, Surgical - statistics & numerical data | Receptors, CCR5 - genetics | Male | Ischemia - genetics | HLA Antigens - genetics | Postoperative Complications - immunology | Receptors, CCR5 - immunology | Cholestasis - immunology | Ischemia - immunology | Postoperative Complications - genetics | Adult | Female | Receptors, Chemokine - genetics | GPI-Linked Proteins - immunology | Cross-Sectional Studies | Ischemia - epidemiology | Risk Factors | Histocompatibility Antigens Class I - immunology | Intercellular Signaling Peptides and Proteins - genetics | HLA Antigens - immunology | Cholestasis - epidemiology | Histocompatibility Antigens Class I - genetics | Postoperative Complications - epidemiology | Receptors, Chemokine - immunology | Morbidity | Liver Transplantation - statistics & numerical data | Graft Survival - genetics | Aged | CX3C Chemokine Receptor 1 | Intercellular Signaling Peptides and Proteins - immunology | GPI-Linked Proteins - genetics | Genetic research | Transplantation of organs, tissues, etc | Aspartate | Biological response modifiers | CC chemokine receptors | Liver diseases | Cytokines | Antibodies | Stenosis | Data processing | Leukocytes | Multivariate analysis | Gene polymorphism | Survival | Chemokine receptors | Immunosuppressive agents | Risk factors | Serum levels | Donors | Interleukin 10 | gamma -Interferon | Fractalkine | Histocompatibility antigen HLA | Mutation | Age
Journal Article
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