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Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 11/2008, Volume 23, Issue 11, pp. 1635 - 1648
Non‐alcoholic fatty liver disease (NAFLD) is the most common cause of referral to liver clinics, and its progressive form, non‐alcoholic steatohepatitis... 
metabolic syndrome | high‐fat diet | non‐alcoholic fatty liver disease | hepatic steatosis | overnutrition | Hepatic steatosis | Metabolic syndrome | Non-alcoholic fatty liver disease | High-fat diet | Overnutrition | OXIDATIVE STRESS | non-alcoholic fatty liver disease | FATTY LIVER-DISEASE | NONALCOHOLIC STEATOHEPATITIS | METHIONINE ADENOSYLTRANSFERASE 1A | DIABETES-MELLITUS | ACTIVATED-RECEPTOR-ALPHA | MITOCHONDRIAL ABNORMALITIES | INSULIN-RESISTANCE | high-fat diet | CHOLINE-DEFICIENT DIET | GASTROENTEROLOGY & HEPATOLOGY | Genetic Predisposition to Disease | Liver - pathology | Reproducibility of Results | Fatty Liver - metabolism | Fatty Liver - pathology | Liver - metabolism | Choline Deficiency - complications | Overnutrition - metabolism | Choline Deficiency - pathology | Hyperphagia - complications | Overnutrition - complications | Disease Progression | Hyperphagia - genetics | Diet, Atherogenic | Phenotype | Animals | Choline Deficiency - metabolism | Intubation, Gastrointestinal | Overnutrition - pathology | Dietary Fats - administration & dosage | Disease Models, Animal | Fatty Liver - etiology | Methionine - deficiency | Methionine, deficiency | Hyperphagia, complications | Hyperphagia, genetics | Choline Deficiency, complications | Overnutrition, metabolism | Liver, pathology | Fatty Liver, pathology | Dietary Fats, administration and dosage | Overnutrition, pathology | Liver, metabolism | Choline Deficiency, metabolism | Fatty Liver, metabolism | Overnutrition, complications | Fatty Liver, etiology | Choline Deficiency, pathology | Insulin resistance | Models | Liver diseases | Analysis | Risk factors
Journal Article
Molecular Nutrition & Food Research, ISSN 1613-4125, 04/2013, Volume 57, Issue 4, pp. 661 - 670
Scope Genetic or nutritional disturbances in folate metabolism lead to hyperhomocysteinemia and adverse reproductive outcomes. Folate‐dependent homocysteine... 
ApoAI | Pregnancy | Homocysteine | IFN‐γ | Folate | IFN-γ | FOOD SCIENCE & TECHNOLOGY | ACTIVATED RECEPTOR-ALPHA | COMMON MUTATION | MASS-SPECTROMETRY | HIGH-DENSITY-LIPOPROTEIN | IFN | ADVERSE REPRODUCTIVE OUTCOMES | INTERFERON-GAMMA | CARDIOVASCULAR-DISEASE | IMMUNE ACTIVATION | ONE-CARBON METABOLISM | APOLIPOPROTEIN-A-I | Choline - metabolism | S-Adenosylhomocysteine - metabolism | Liver - pathology | Psychotic Disorders - complications | Betaine - metabolism | Homocystinuria - complications | Choline - analysis | Male | Methylenetetrahydrofolate Reductase (NADPH2) - metabolism | Betaine - analysis | Interferon-gamma - metabolism | Folic Acid - administration & dosage | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Psychotic Disorders - pathology | Female | Pregnancy Complications - metabolism | Methylenetetrahydrofolate Reductase (NADPH2) - deficiency | Spleen - pathology | Liver - metabolism | Homocysteine - blood | Mice, Transgenic | Apolipoprotein A-I - metabolism | Muscle Spasticity - pathology | Homocystinuria - pathology | Placenta - metabolism | S-Adenosylmethionine - analysis | Animals | Folic Acid Deficiency - complications | Diet | Folic Acid Deficiency - pathology | Spleen - metabolism | Muscle Spasticity - complications | Placenta - pathology | Pregnancy Complications - genetics | Mice | Mice, Inbred BALB C | Methylation | S-Adenosylhomocysteine - analysis | S-Adenosylmethionine - metabolism
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 05/2015, Volume 754, pp. 19 - 24
Ipragliflozin is a selective sodium glucose cotransporter 2 (SGLT2) inhibitor that increases urinary glucose excretion by inhibiting renal glucose reabsorption... 
Nonalcoholic fatty liver disease | Nonalcoholic steatohepatitis | Choline-deficient and amino acid-defined diet | SGLT2 inhibitor | SGLT2inhibitor | Choline-deficient andaminoacid-defined diet | Nonalcoholic fattyliverdisease | OXIDATIVE STRESS | FATTY LIVER-DISEASE | PROLIFERATOR-ACTIVATED RECEPTORS | INSULIN-SECRETION | STELLATE CELL ACTIVATION | IN-VITRO | OBESITY | PIOGLITAZONE | PHARMACOLOGY & PHARMACY | TYPE-2 DIABETES-MELLITUS | Inflammation - chemically induced | Thiophenes - therapeutic use | Choline Deficiency - complications | Male | Amino Acids - pharmacology | Liver Cirrhosis - chemically induced | Non-alcoholic Fatty Liver Disease - complications | Inflammation - complications | Thiazolidinediones - therapeutic use | Non-alcoholic Fatty Liver Disease - chemically induced | Glucosides - therapeutic use | Hypoglycemic Agents - therapeutic use | Glucosides - pharmacology | Liver Cirrhosis - prevention & control | Liver Cirrhosis - complications | Hydroxyproline - metabolism | Rats | Thiophenes - pharmacology | Food, Formulated - adverse effects | Non-alcoholic Fatty Liver Disease - metabolism | Choline Deficiency - pathology | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | Triglycerides - metabolism | Animals | Non-alcoholic Fatty Liver Disease - prevention & control | Choline Deficiency - drug therapy | Liver Cirrhosis - pathology | Inflammation - prevention & control | Type 2 diabetes | Liver diseases | Choline | Fibrosis | Amino acids | Exhibitions | Glucose | Dextrose
Journal Article
Journal Article
Nutrition Reviews, ISSN 0029-6643, 11/2009, Volume 67, Issue 11, pp. 615 - 623
Choline was officially recognized as an essential nutrient by the Institute of Medicine (IOM) in 1998. There is significant variation in the dietary... 
homocysteine | choline | eggs | memory | methyl group | methylation | phosphatidylcholine | pregnancy | neural tube defects | Pregnancy | Choline | Memory | Methyl group | Neural tube defects | Phosphatidylcholine | Homocysteine | Methylation | Eggs | PLASMA TOTAL HOMOCYSTEINE | GENETIC POLYMORPHISMS | RISK FACTOR | BETAINE | HUMANS | PERINATAL CHOLINE | DEFICIENCY | HIPPOCAMPAL PLASTICITY | NUTRITION & DIETETICS | DIETARY CHOLINE | SENSORY INHIBITION | Choline - metabolism | Memory - drug effects | Public Health | Choline Deficiency - epidemiology | Humans | Middle Aged | Breast Neoplasms - etiology | Child, Preschool | Choline Deficiency - complications | Heart Diseases - prevention & control | Infant | Male | Nutrition Policy | Young Adult | Lipotropic Agents - metabolism | Health Promotion | Adult | Choline Deficiency - genetics | Female | Child | Infant, Newborn | Disease Models, Animal | Nutritional Requirements | Neural Tube Defects - etiology | Neural Tube Defects - prevention & control | Inflammation - etiology | Breast Neoplasms - prevention & control | Polymorphism, Genetic | Heart Diseases - etiology | Lipotropic Agents - administration & dosage | Animals | Choline - administration & dosage | Adolescent | Aged | Inflammation - prevention & control | Choline - genetics | Physiological aspects | Public health | Atherosclerosis
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0175675
Objective Non-alcoholic fatty liver disease (NAFLD) is an important co-morbidity associated with obesity and a precursor to steatohepatitis. However, the... 
OBESITY | RAT | PROGRAMS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | CHOLINE-DEFICIENT DIET | DYSFUNCTION | LIVER-DISEASE | ALTERS | SKELETAL-MUSCLE DEVELOPMENT | PREGNANCY | Maternal Nutritional Physiological Phenomena | Epigenesis, Genetic | Diet, High-Fat - adverse effects | Non-alcoholic Fatty Liver Disease - etiology | Choline Deficiency - complications | Male | Gene Expression Profiling | Overnutrition - complications | DNA Methylation | Prenatal Exposure Delayed Effects - microbiology | Female | Disease Models, Animal | Methionine - deficiency | Non-alcoholic Fatty Liver Disease - genetics | Liver - metabolism | Mice, Inbred C57BL | Non-alcoholic Fatty Liver Disease - microbiology | Methionine - administration & dosage | Gastrointestinal Microbiome - genetics | Prenatal Exposure Delayed Effects - etiology | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Animals | Choline - administration & dosage | Mice | Obesity | Usage | Care and treatment | Fatty liver | Physiological aspects | Development and progression | Research | Risk factors | Ketogenic diet | Pediatrics | Laboratories | Weaning | Liver | DNA damage | Body weight | Genomes | Cell interactions | Kinases | Body composition | High fat diet | Rodents | Hepatology | Cell adhesion | DNA methylation | Deoxyribonucleic acid--DNA | Nutrient deficiency | Liver diseases | Immune response | Nutrition | Complications | Methionine | Regression analysis | Metabolism | Gene expression | Ribonucleic acid--RNA | Morbidity | Steatosis | Studies | Nutrition research | Progeny | Offspring | Diet | Choline | Epigenetics | Methylation | RNA | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article