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Stem Cells, ISSN 1066-5099, 03/2010, Volume 28, Issue 3, pp. 564 - 572
Human mesenchymal stem cells (hMSCs) are multipotent cells that can differentiate into many cell types. Chondrogenesis is induced in hMSCs cultured as a... 
Cell shape | Rac1 | Chondrogenesis | Smooth muscle cells | N-cadherin | Mesenchymal stem cells | MYOBLAST FUSION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | ADHESION | ONCOLOGY | MESENCHYMAL PROGENITOR CELLS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENE-EXPRESSION | CYTOSKELETAL TENSION | DIFFERENTIATION | RHO-GTPASES | PROTEINS | HEMATOLOGY | MODULATION | MAMMARY EPITHELIAL-CELLS | Chondrocytes - cytology | Chondrogenesis - drug effects | Cadherins - metabolism | Humans | Extracellular Matrix - metabolism | Antigens, CD - genetics | Cell Lineage - drug effects | Transforming Growth Factor beta3 - metabolism | Antigens, CD - metabolism | Cell Differentiation - genetics | Chondrocytes - drug effects | Mesenchymal Stromal Cells - cytology | Cadherins - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Chondrocytes - metabolism | Transforming Growth Factor beta3 - pharmacology | Mesenchymal Stromal Cells - drug effects | Cell Adhesion - genetics | Muscle Development - physiology | Cells, Cultured | Gene Expression Regulation - physiology | Mesenchymal Stromal Cells - metabolism | Up-Regulation - genetics | Antigens, CD - drug effects | Cadherins - drug effects | Cell Adhesion - drug effects | Cell Lineage - physiology | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Chondrogenesis - physiology | Muscle Development - drug effects | rac1 GTP-Binding Protein - drug effects | Cell Differentiation - drug effects | Cell Shape - physiology | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 08/2010, Volume 466, Issue 7308, pp. 829 - 834
The cellular constituents forming the haematopoietic stem cell (HSC) niche in the bone marrow are unclear, with studies implicating osteoblasts, endothelial... 
PROGENITOR CELLS | OSTEOBLAST | OSTEOPONTIN | MICROENVIRONMENT | MULTIDISCIPLINARY SCIENCES | SELF-RENEWAL | RECEPTOR | DIFFERENTIATION | NEURAL CREST | COOPERATION | EXPRESSION | Chondrocytes - cytology | Nestin | Multipotent Stem Cells - metabolism | Parathyroid Hormone - pharmacology | Cell Lineage - drug effects | Chondrocytes - drug effects | Mesenchymal Stromal Cells - cytology | Multipotent Stem Cells - drug effects | Sympathetic Nervous System - physiology | Cell Division | Stromal Cells - drug effects | Osteoblasts - cytology | Hematopoietic Stem Cells - drug effects | Mesenchymal Stromal Cells - drug effects | Gene Expression Regulation - genetics | Osteoblasts - drug effects | Stromal Cells - metabolism | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Mice, Transgenic | Hematopoietic Stem Cells - metabolism | Stem Cell Niche - cytology | Nerve Tissue Proteins - metabolism | Granulocyte Colony-Stimulating Factor - pharmacology | Animals | Chemokine CXCL12 - metabolism | Cell Differentiation - drug effects | Multipotent Stem Cells - cytology | Hematopoietic Stem Cells - cytology | Stem Cell Niche - metabolism | Mice | Stem Cell Niche - drug effects | Osteoblasts - metabolism | Intermediate Filament Proteins - metabolism | Mesenchymal Stem Cell Transplantation | Stromal Cells - cytology | Cell Movement | Physiological aspects | Genetic aspects | Research | Bone marrow cells | Gene expression | Osteoblasts | Hematopoietic stem cells | Studies | Proteins | Bone marrow | Rodents | Stem cells | Index Medicus
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 05/2005, Volume 203, Issue 2, pp. 398 - 409
Human mesenchymal stem cells (hMSCs) expanded with and without fibroblast growth factor (FGF) supplementation were compared with respect to their proliferation... 
PROGENITOR CELLS | CROSS-TALK | PROTEIN-KINASE-C | IN-VITRO | PHYSIOLOGY | ATDC5 CELLS | DEFICIENT MDX MICE | STROMAL CELLS | GENE-EXPRESSION SIGNATURES | CHONDROCYTE DIFFERENTIATION | N-CADHERIN | CELL BIOLOGY | Chondrocytes - cytology | Chondrogenesis - drug effects | Age Factors | Oligonucleotide Array Sequence Analysis | Humans | Fibroblast Growth Factor 2 - pharmacology | Bone Marrow Cells - physiology | Gene Expression Profiling | Cell Culture Techniques - methods | Proteoglycans - drug effects | Cell Differentiation - genetics | Chondrocytes - drug effects | Mesenchymal Stromal Cells - cytology | Chondrocytes - physiology | Collagen - drug effects | Up-Regulation - physiology | Bone Marrow Cells - drug effects | Mesenchymal Stromal Cells - physiology | Mesenchymal Stromal Cells - drug effects | Tissue Engineering - methods | Bone Marrow Cells - cytology | Gene Expression Regulation - genetics | Extracellular Matrix Proteins - genetics | Cells, Cultured | Proteoglycans - metabolism | Down-Regulation - drug effects | Extracellular Matrix Proteins - drug effects | Down-Regulation - physiology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Chondrogenesis - physiology | Collagen - metabolism | Mitosis - drug effects | Mitosis - physiology | Signal Transduction - drug effects | Cell Differentiation - drug effects | Signal Transduction - physiology | Cell Proliferation - drug effects | Index Medicus
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 10/2017, Volume 32, Issue 10, pp. 2128 - 2141
Autophagy is activated during nutritionally depleted or hypoxic conditions to facilitate cell survival. Because growth plate is an avascular and hypoxic... 
ENDOCHONDRAL OSSIFICATION | AUTOPHAGY | GROWTH PLATE CHONDROCYTES | ATG7 | ER STRESS | MATRIX | CHONDROCYTES | ENDOPLASMIC-RETICULUM STRESS | CELL-DEATH | UNFOLDED PROTEIN RESPONSE | PATHWAY | ENDOCRINOLOGY & METABOLISM | GROWTH-PLATE | BONE | DIFFERENTIATION | Chondrogenesis - drug effects | Apoptosis - drug effects | eIF-2 Kinase - metabolism | Autophagy-Related Protein 7 - metabolism | Growth Plate - embryology | Cartilage - drug effects | Autophagy - drug effects | Chondrocytes - drug effects | Gene Deletion | Phenylbutyrates - pharmacology | Tibia - growth & development | Growth Plate - ultrastructure | Chondrocytes - metabolism | Femur - growth & development | Endoplasmic Reticulum Stress - drug effects | Osteogenesis - drug effects | Cells, Cultured | Femur - drug effects | Cartilage - metabolism | Growth Plate - metabolism | Organ Specificity | Tibia - drug effects | Autophagy-Related Protein 7 - genetics | Mice, Knockout | Animals | Activating Transcription Factor 4 - metabolism | Cell Differentiation - drug effects | Chondrocytes - ultrastructure | Cell Proliferation - drug effects | Embryonic Development - drug effects | Transcription Factor CHOP - metabolism | Autophagy-Related Protein 7 - deficiency | Analysis | Stress (Physiology) | Cell proliferation | Cell survival | Growth rate | Homeostasis | Collagenase 3 | Autophagy | Ossification | Cartilage | Growth plate | Bone growth | Rodents | Chondrocytes | Hypoxia | Chondrogenesis | Stress response | Endoplasmic reticulum | Bone (endochondral) | Phenylbutyric acid | Phagocytosis | Apoptosis | Index Medicus
Journal Article
Journal of Biomedical Materials Research Part A, ISSN 1549-3296, 12/2011, Volume 99A, Issue 3, pp. 467 - 478
Journal Article
Biomaterials, ISSN 0142-9612, 2011, Volume 33, Issue 10, pp. 2848 - 2857
Abstract Adult bone marrow derived mesenchymal stem cells are undifferentiated, multipotential cells and have the potential to differentiate into multiple... 
Advanced Basic Science | Dentistry | Cartilage | Silk fibroin | Chitosan | Tissue engineering | Mesenchymal stem cells | PORE-SIZE | MATERIALS SCIENCE, BIOMATERIALS | BOVINE ARTICULAR CHONDROCYTES | ENGINEERING, BIOMEDICAL | REGENERATION | CHITOSAN SCAFFOLDS | MESENCHYMAL STEM-CELLS | TISSUE ENGINEERING APPLICATIONS | 3D SCAFFOLDS | BIOMATERIALS | STROMAL CELLS | Chondrogenesis - drug effects | Rats, Wistar | Male | Cartilage - drug effects | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Flow Cytometry | Mesenchymal Stromal Cells - ultrastructure | Stromal Cells - drug effects | Chitosan - pharmacology | Bone Marrow Cells - drug effects | Extracellular Matrix Proteins - metabolism | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Cell Separation | Extracellular Matrix Proteins - genetics | Stromal Cells - metabolism | Cells, Cultured | Fibroins - pharmacology | Mesenchymal Stromal Cells - metabolism | Rats | Cartilage - metabolism | Cell Adhesion - drug effects | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Cell Differentiation - drug effects | Fluorescent Antibody Technique | Cell Proliferation - drug effects | Porosity | Bone Marrow Cells - metabolism | Microscopy, Fluorescence | Stromal Cells - cytology | Silk | Glycosaminoglycans | Biological products | Collagen | Stem cells | Polyelectrolytes | Histochemistry | Index Medicus
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 51, pp. 238 - 249
Abstract Glycopolypeptides are an emerging class of bioinspired polymers that mimic naturally occurring glycopeptides or glycoproteins, and therefore are... 
Advanced Basic Science | Dentistry | Cartilage tissue engineering | Glycopolypeptide hydrogel | Biomimic scaffold | Enzymatic crosslinking | Injectable hydrogel | MATERIALS SCIENCE, BIOMATERIALS | DRUG-DELIVERY | ENGINEERING, BIOMEDICAL | MESENCHYMAL STEM-CELLS | POLYPEPTIDES | MICHAEL ADDITION | SITU | REPAIR | BIOMEDICAL APPLICATIONS | REGENERATIVE MEDICINE | BONE | CLICK GLYCOSYLATION | Chondrocytes - cytology | Amides - chemical synthesis | Extracellular Matrix - metabolism | Glycopeptides - chemistry | Cartilage - drug effects | Hydrogels - chemical synthesis | Tissue Scaffolds - chemistry | Chondrocytes - drug effects | Polyglutamic Acid - pharmacology | Proton Magnetic Resonance Spectroscopy | Cartilage - physiology | Cell Death - drug effects | Mannose - chemistry | Amides - pharmacology | Cell Line | Cell Survival - drug effects | Rabbits | Tissue Engineering - methods | Glycosaminoglycans - metabolism | Extracellular Matrix - drug effects | Cells, Cultured | Polyglutamic Acid - chemical synthesis | Rats, Sprague-Dawley | Polyglutamic Acid - chemistry | Injections | Animals | Mice, Nude | Amides - chemistry | Cell Proliferation - drug effects | Mannose - pharmacology | Hydrogels - pharmacology | Glycopeptides - pharmacology | Hydrogels - chemistry | Subcutaneous Tissue - drug effects | Proteins | Crosslinked polymers | Glycosaminoglycans | Hydrogen peroxide | Glutamate | Tissue engineering | Index Medicus | Surgical implants | Biomedical materials | Biomimetics | Hydrogels | Biocompatibility | In vitro testing | Scaffolds
Journal Article
Acta Biomaterialia, ISSN 1742-7061, 2011, Volume 7, Issue 1, pp. 234 - 243
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 2, pp. 315 - 324
Summary Objective The aetiology of OA is not fully understood although several adipokines such as leptin are known mediators of disease progression. Since... 
Rheumatology | Smad1 | Human chondrocytes | Hip osteoarthritis | β-Catenin | Dedifferentiation | Leptin | COLLAGEN | RHEUMATOLOGY | DIFFERENTIATED PHENOTYPE | HYPERTROPHY | IN-VITRO | SYNOVIAL FIBROBLASTS | HUMAN OSTEOARTHRITIC CARTILAGE | SIGNALING PATHWAY | beta-Catenin | ARTICULAR CHONDROCYTES | ORTHOPEDICS | KNEE OSTEOARTHRITIS | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Humans | Leptin - metabolism | Middle Aged | Male | Lymphotoxin-alpha - metabolism | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Lymphotoxin-alpha - genetics | Cartilage, Articular - metabolism | Smad5 Protein - metabolism | Smad2 Protein - genetics | Aged, 80 and over | Smad2 Protein - drug effects | Protein-Serine-Threonine Kinases - metabolism | Collagen Type X - drug effects | SOX9 Transcription Factor - metabolism | Matrix Metalloproteinase 13 - genetics | Smad2 Protein - metabolism | Prednisolone - pharmacology | beta Catenin - metabolism | Matrix Metalloproteinase 13 - metabolism | Glycogen Synthase Kinase 3 - genetics | Receptors, Transforming Growth Factor beta - drug effects | Activin Receptors, Type II - genetics | Protein-Serine-Threonine Kinases - drug effects | Activin Receptors, Type II - metabolism | Osteoarthritis, Hip - metabolism | Glycogen Synthase Kinase 3 - drug effects | Activin Receptors, Type II - drug effects | Chondrocytes - drug effects | Collagen Type X - metabolism | beta Catenin - drug effects | Smad1 Protein - genetics | Adult | Female | Smad5 Protein - genetics | Chondrocytes - metabolism | RNA, Messenger - drug effects | Cell Dedifferentiation - drug effects | Cartilage, Articular - cytology | Lymphotoxin-alpha - drug effects | Protein-Serine-Threonine Kinases - genetics | Collagen Type X - genetics | SOX9 Transcription Factor - drug effects | Matrix Metalloproteinase 13 - drug effects | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - genetics | Osteoarthritis, Hip - genetics | Smad5 Protein - drug effects | Core Binding Factor Alpha 1 Subunit - drug effects | Wnt Signaling Pathway - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Wnt Signaling Pathway - genetics | Glucocorticoids - pharmacology | Smad1 Protein - metabolism | Aged | Smad1 Protein - drug effects | Cartilage, Articular - drug effects | In Vitro Techniques | Cell Dedifferentiation - genetics | Core Binding Factor Alpha 1 Subunit - genetics | Index Medicus
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 11/2013, Volume 74, Issue 1, pp. 303 - 310
Journal Article