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Stem cells (Dayton, Ohio), ISSN 1549-4918, 2010, Volume 28, Issue 3, pp. 564 - N/A
Human mesenchymal stem cells (hMSCs) are multipotent cells that can differentiate into many cell types. Chondrogenesis is induced in hMSCs cultured as a... 
N‐cadherin | Cell shape | Rac1 | Chondrogenesis | Smooth muscle cells | Mesenchymal stem cells | N-cadherin | MYOBLAST FUSION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | ADHESION | ONCOLOGY | MESENCHYMAL PROGENITOR CELLS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENE-EXPRESSION | CYTOSKELETAL TENSION | DIFFERENTIATION | RHO-GTPASES | PROTEINS | HEMATOLOGY | MODULATION | MAMMARY EPITHELIAL-CELLS | Chondrocytes - cytology | Chondrogenesis - drug effects | Cadherins - metabolism | Humans | Extracellular Matrix - metabolism | Antigens, CD - genetics | Cell Lineage - drug effects | Transforming Growth Factor beta3 - metabolism | Antigens, CD - metabolism | Cell Differentiation - genetics | Chondrocytes - drug effects | Mesenchymal Stromal Cells - cytology | Cadherins - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Chondrocytes - metabolism | Transforming Growth Factor beta3 - pharmacology | Mesenchymal Stromal Cells - drug effects | Cell Adhesion - genetics | Muscle Development - physiology | Cells, Cultured | Gene Expression Regulation - physiology | Mesenchymal Stromal Cells - metabolism | Up-Regulation - genetics | Antigens, CD - drug effects | Cadherins - drug effects | Cell Adhesion - drug effects | Cell Lineage - physiology | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Chondrogenesis - physiology | Muscle Development - drug effects | rac1 GTP-Binding Protein - drug effects | Cell Differentiation - drug effects | Cell Shape - physiology | rac1 GTP-Binding Protein - metabolism | rac1 GTP-Binding Protein - genetics
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 2, pp. 315 - 324
Summary Objective The aetiology of OA is not fully understood although several adipokines such as leptin are known mediators of disease progression. Since... 
Rheumatology | Smad1 | Human chondrocytes | Hip osteoarthritis | β-Catenin | Dedifferentiation | Leptin | COLLAGEN | RHEUMATOLOGY | DIFFERENTIATED PHENOTYPE | HYPERTROPHY | IN-VITRO | SYNOVIAL FIBROBLASTS | HUMAN OSTEOARTHRITIC CARTILAGE | SIGNALING PATHWAY | beta-Catenin | ARTICULAR CHONDROCYTES | ORTHOPEDICS | KNEE OSTEOARTHRITIS | EXPRESSION | Receptors, Transforming Growth Factor beta - genetics | Humans | Leptin - metabolism | Middle Aged | Male | Lymphotoxin-alpha - metabolism | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Lymphotoxin-alpha - genetics | Cartilage, Articular - metabolism | Smad5 Protein - metabolism | Smad2 Protein - genetics | Aged, 80 and over | Smad2 Protein - drug effects | Protein-Serine-Threonine Kinases - metabolism | Collagen Type X - drug effects | SOX9 Transcription Factor - metabolism | Matrix Metalloproteinase 13 - genetics | Smad2 Protein - metabolism | Prednisolone - pharmacology | beta Catenin - metabolism | Matrix Metalloproteinase 13 - metabolism | Glycogen Synthase Kinase 3 - genetics | Receptors, Transforming Growth Factor beta - drug effects | Activin Receptors, Type II - genetics | Protein-Serine-Threonine Kinases - drug effects | Activin Receptors, Type II - metabolism | Osteoarthritis, Hip - metabolism | Glycogen Synthase Kinase 3 - drug effects | Activin Receptors, Type II - drug effects | Chondrocytes - drug effects | Collagen Type X - metabolism | beta Catenin - drug effects | Smad1 Protein - genetics | Adult | Female | Smad5 Protein - genetics | Chondrocytes - metabolism | RNA, Messenger - drug effects | Cell Dedifferentiation - drug effects | Cartilage, Articular - cytology | Lymphotoxin-alpha - drug effects | Protein-Serine-Threonine Kinases - genetics | Collagen Type X - genetics | SOX9 Transcription Factor - drug effects | Matrix Metalloproteinase 13 - drug effects | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - genetics | Osteoarthritis, Hip - genetics | Smad5 Protein - drug effects | Core Binding Factor Alpha 1 Subunit - drug effects | Wnt Signaling Pathway - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Wnt Signaling Pathway - genetics | Glucocorticoids - pharmacology | Smad1 Protein - metabolism | Aged | Smad1 Protein - drug effects | Cartilage, Articular - drug effects | In Vitro Techniques | Cell Dedifferentiation - genetics | Core Binding Factor Alpha 1 Subunit - genetics
Journal Article
Osteoarthritis and cartilage, ISSN 1063-4584, 2016, Volume 24, Issue 1, pp. 178 - 187
Summary Objective To determine whether mandibular condylar cartilage degradation induced by experimentally abnormal occlusion could be ameliorated via systemic... 
Rheumatology | Strontium | Cartilage | Subchondral bone | NBD (NEMO-binding domain) peptide | Temporomandibular joint | Osteoarthritis | Dental occlusion | TGF-BETA | CHONDROCYTES | BLOCKS OSTEOCLASTOGENESIS | RHEUMATOLOGY | TRABECULAR BONE | SUBCHONDRAL BONE LOSS | RANELATE | INHIBITION | TEMPOROMANDIBULAR-JOINT | ORTHOPEDICS | NF-KAPPA-B | KNEE OSTEOARTHRITIS | Immunohistochemistry | RANK Ligand - metabolism | Collagen Type II - drug effects | Tumor Necrosis Factor-alpha - genetics | ADAM Proteins - drug effects | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Proteoglycans - drug effects | Collagen Type II - metabolism | Cartilage, Articular - metabolism | Interleukin-1beta - metabolism | Malocclusion | Receptor Activator of Nuclear Factor-kappa B - metabolism | Aggrecans - drug effects | Real-Time Polymerase Chain Reaction | ADAMTS5 Protein | NF-KappaB Inhibitor alpha | Proteoglycans - metabolism | I-kappa B Proteins - drug effects | Aggrecans - metabolism | ADAM Proteins - metabolism | Tumor Necrosis Factor-alpha - drug effects | Strontium - pharmacology | Mice | Osteoprotegerin - metabolism | ADAM Proteins - genetics | Interleukin-1beta - drug effects | Tumor Necrosis Factor-alpha - metabolism | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Receptor Activator of Nuclear Factor-kappa B - drug effects | Transcription Factor RelA - genetics | RANK Ligand - drug effects | Mandibular Condyle - pathology | Osteoprotegerin - genetics | Female | Mandibular Condyle - drug effects | Osteoprotegerin - drug effects | RNA, Messenger - drug effects | Mice, Inbred C57BL | Aggrecans - genetics | Osteoclasts - metabolism | Collagen Type II - genetics | Peptides - pharmacology | RANK Ligand - genetics | Animals | Transcription Factor RelA - metabolism | Receptor Activator of Nuclear Factor-kappa B - genetics | Transcription Factor RelA - drug effects | Cartilage, Articular - drug effects | Mandibular Condyle - metabolism | Dental Occlusion | Osteoclasts - drug effects | Peptides
Journal Article
Biomaterials, ISSN 0142-9612, 2012, Volume 33, Issue 10, pp. 2848 - 2857
Abstract Adult bone marrow derived mesenchymal stem cells are undifferentiated, multipotential cells and have the potential to differentiate into multiple... 
Advanced Basic Science | Dentistry | Cartilage | Silk fibroin | Chitosan | Tissue engineering | Mesenchymal stem cells | PORE-SIZE | MATERIALS SCIENCE, BIOMATERIALS | BOVINE ARTICULAR CHONDROCYTES | ENGINEERING, BIOMEDICAL | REGENERATION | CHITOSAN SCAFFOLDS | MESENCHYMAL STEM-CELLS | STROMAL CELLS | Chondrogenesis - drug effects | Rats, Wistar | Male | Cartilage - drug effects | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Flow Cytometry | Mesenchymal Stromal Cells - ultrastructure | Stromal Cells - drug effects | Chitosan - pharmacology | Bone Marrow Cells - drug effects | Extracellular Matrix Proteins - metabolism | Mesenchymal Stromal Cells - drug effects | Bone Marrow Cells - cytology | Cell Separation | Extracellular Matrix Proteins - genetics | Stromal Cells - metabolism | Cells, Cultured | Fibroins - pharmacology | Mesenchymal Stromal Cells - metabolism | Rats | Cartilage - metabolism | Cell Adhesion - drug effects | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Cell Differentiation - drug effects | Fluorescent Antibody Technique | Cell Proliferation - drug effects | Porosity | Bone Marrow Cells - metabolism | Microscopy, Fluorescence | Stromal Cells - cytology | Silk | Glycosaminoglycans | Biological products | Collagen | Stem cells | Polyelectrolytes | Histochemistry
Journal Article
Biomaterials, ISSN 0142-9612, 2011, Volume 32, Issue 27, pp. 6456 - 6470
Journal Article
Journal Article
Journal of cell science, ISSN 0021-9533, 05/2008, Volume 121, Issue 9, pp. 1455 - 1465
The Wnt/beta-catenin signaling pathway is essential for normal skeletal development because conditional gain or loss of function of beta-catenin in cartilage... 
β-catenin | Endochondral bone formation | Chondrocyte | Inhibitor of β-catenin and TCF (ICAT) | Vascular endothelial growth factor (VEGF) | beta-catenin | OSTEOBLAST DIFFERENTIATION | II COLLAGEN GENE | BINDING-PROTEIN | inhibitor of beta-catenin and TCF (ICAT) | vascular endothelial growth factor (VEGF) | CELL-FATE | endochondral bone formation | CHONDROCYTE DIFFERENTIATION | NEGATIVE REGULATOR | CELL BIOLOGY | COLON-CANCER | chondrocyte | WNT/BETA-CATENIN | BONE-FORMATION | GROWTH-PLATE | Embryo, Mammalian - drug effects | Growth Plate - pathology | Neovascularization, Physiologic - drug effects | Transcription, Genetic - drug effects | Chondrogenesis - drug effects | Cartilage - pathology | Repressor Proteins | Cartilage - drug effects | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Wnt Proteins - metabolism | Promoter Regions, Genetic - genetics | Embryo, Mammalian - metabolism | Chondrocytes - drug effects | Growth Plate - drug effects | Transgenes | Bone Morphogenetic Proteins - pharmacology | Chondrocytes - metabolism | Animals, Newborn | Cartilage - growth & development | Chondrocytes - pathology | Bone Development - drug effects | Bone Morphogenetic Protein 2 | Cell Cycle Proteins - metabolism | Gene Expression Regulation, Developmental - drug effects | Mice, Transgenic | beta Catenin - metabolism | Embryo, Mammalian - abnormalities | Transcription Factors - metabolism | Cartilage - abnormalities | Transforming Growth Factor beta - pharmacology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Mice
Journal Article
Acta biomaterialia, ISSN 1742-7061, 2013, Volume 9, Issue 12, pp. 9343 - 9350
Journal Article
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 51, pp. 238 - 249
Abstract Glycopolypeptides are an emerging class of bioinspired polymers that mimic naturally occurring glycopeptides or glycoproteins, and therefore are... 
Advanced Basic Science | Dentistry | Cartilage tissue engineering | Glycopolypeptide hydrogel | Biomimic scaffold | Enzymatic crosslinking | Injectable hydrogel | MATERIALS SCIENCE, BIOMATERIALS | DRUG-DELIVERY | ENGINEERING, BIOMEDICAL | MESENCHYMAL STEM-CELLS | POLYPEPTIDES | MICHAEL ADDITION | SITU | REPAIR | BIOMEDICAL APPLICATIONS | REGENERATIVE MEDICINE | BONE | CLICK GLYCOSYLATION | Chondrocytes - cytology | Amides - chemical synthesis | Extracellular Matrix - metabolism | Glycopeptides - chemistry | Cartilage - drug effects | Hydrogels - chemical synthesis | Tissue Scaffolds - chemistry | Chondrocytes - drug effects | Polyglutamic Acid - pharmacology | Proton Magnetic Resonance Spectroscopy | Cartilage - physiology | Cell Death - drug effects | Mannose - chemistry | Amides - pharmacology | Cell Line | Cell Survival - drug effects | Rabbits | Tissue Engineering - methods | Glycosaminoglycans - metabolism | Extracellular Matrix - drug effects | Cells, Cultured | Polyglutamic Acid - chemical synthesis | Rats, Sprague-Dawley | Polyglutamic Acid - chemistry | Injections | Animals | Mice, Nude | Amides - chemistry | Cell Proliferation - drug effects | Mannose - pharmacology | Hydrogels - pharmacology | Glycopeptides - pharmacology | Hydrogels - chemistry | Subcutaneous Tissue - drug effects | Proteins | Crosslinked polymers | Glycosaminoglycans | Hydrogen peroxide | Glutamate | Tissue engineering | Surgical implants | Biomedical materials | Biomimetics | Hydrogels | Biocompatibility | In vitro testing | Scaffolds
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2013, Volume 21, Issue 7, pp. 990 - 998
.... Muscle is a tissue that lies near cartilage in situ . However, muscle's non-loading biochemical effect on cartilage has been largely unexplored... 
Rheumatology | Cartilage tissue engineering | Myokines | Pro-inflammatory | Cytokines | Osteoarthritis | Stem cells | SILK SCAFFOLDS | INTERLEUKIN-1-BETA | NECROSIS-FACTOR-ALPHA | RHEUMATOLOGY | ANABOLIC CYTOKINES | ARTHRITIS | MESENCHYMAL STEM-CELLS | CARTILAGE | SKELETAL-MUSCLE | ORTHOPEDICS | PROGRESSION | Cell Cycle Proteins - drug effects | Gene Products, gag - drug effects | Chondrocytes - cytology | Interleukin-1beta - pharmacology | Apoptosis - drug effects | Collagen Type II - drug effects | Humans | Caspase 3 - metabolism | Ki-67 Antigen - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Case-Control Studies | Collagen Type II - metabolism | Chondrocytes - drug effects | Myoblasts - drug effects | Mesenchymal Stromal Cells - cytology | Myoblasts - metabolism | Collagen Type X - metabolism | Matrix Metalloproteinases - drug effects | Myoblasts - cytology | Aggrecans - drug effects | Chondrocytes - metabolism | Collagen Type X - drug effects | Fibroblasts - metabolism | Mesenchymal Stromal Cells - drug effects | Cell Cycle Proteins - metabolism | Mesenchymal Stromal Cells - metabolism | Aggrecans - metabolism | Gene Products, gag - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Core Binding Factor Alpha 1 Subunit - drug effects | Fibroblasts - drug effects | Cell Proliferation - drug effects | Fibroblasts - cytology | Matrix Metalloproteinases - metabolism | Developmental biology
Journal Article