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Gastroenterology, ISSN 0016-5085, 2017, Volume 152, Issue 4, pp. 880 - 894.e6
Background & Aims Hepatocellular adenomas (HCAs) are benign liver tumors that can be assigned to molecular subtypes based on inactivating mutations in... 
Gastroenterology and Hepatology | HCC | SHH | Tumor Progression | Benign | HEDGEHOG PATHWAY | FOLLOW-UP | CANCER | TUMORS | SOMATIC MUTATIONS | INACTIVATION | INHIBITION | THERAPEUTIC TARGETS | GASTROENTEROLOGY & HEPATOLOGY | LIVER ADENOMATOSIS | ACTIVATING MUTATIONS | Liver Neoplasms - classification | Hepatocyte Nuclear Factor 1-alpha - genetics | Adenoma - genetics | Cytokine Receptor gp130 - genetics | Humans | Middle Aged | Transcriptome | Hedgehog Proteins - metabolism | Male | Liver Neoplasms - chemistry | GTP-Binding Protein alpha Subunits, Gs - genetics | Young Adult | Protein-Tyrosine Kinases - genetics | Telomerase - genetics | Cell Transformation, Neoplastic - genetics | Carcinoma, Hepatocellular - genetics | Hemorrhage - etiology | Aged, 80 and over | Adult | Chromogranins - genetics | Female | Carcinoma, Hepatocellular - chemistry | Inhibin-beta Subunits - genetics | Neoplasm Proteins - genetics | Child | STAT3 Transcription Factor - genetics | Liver Neoplasms - genetics | Signal Transduction | Zinc Finger Protein GLI1 - genetics | Risk Factors | Janus Kinase 2 - genetics | Gene Fusion | Adenoma - chemistry | beta Catenin - genetics | Sequence Analysis, RNA | Adenoma - classification | Adolescent | Carcinoma, Hepatocellular - pathology | Aged | Mutation | Liver cancer | Liver | Risk factors | Adenoma | Life Sciences | Human health and pathology | Santé publique et épidémiologie | Hépatology and Gastroenterology | Tissues and Organs
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2015, Volume 47, Issue 5, pp. 435 - 444
Journal Article
Gut, ISSN 0017-5749, 09/2018, Volume 67, Issue 9, pp. 1652 - 1662
ObjectiveIntraductal papillary mucinous neoplasms (IPMNs) are precursor lesions that can give rise to invasive pancreatic carcinoma. Although approximately 8%... 
molecular genetics | pancreatic cancer | cancer genetics | mutations | carcinogenesis | PAPILLARY MUCINOUS NEOPLASMS | PANCREATIC DUCTAL ADENOCARCINOMA | GENOMIC ANALYSES | GNAS MUTATION | CYSTS | LONG-TERM SURVIVAL | SEQUENCING ANALYSIS | CONCOMITANT | INTRAEPITHELIAL NEOPLASIA | GASTROENTEROLOGY & HEPATOLOGY | CARCINOMA | Oncogene Proteins - genetics | Predictive Value of Tests | Carcinoma, Papillary - genetics | Prevalence | Follow-Up Studies | Pancreatic Neoplasms - diagnosis | United States | Humans | Middle Aged | Male | Mutation, Missense - genetics | Carcinoma, Pancreatic Ductal - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Smad4 Protein - genetics | Sensitivity and Specificity | Carcinoma, Pancreatic Ductal - diagnosis | Carcinoma, Pancreatic Ductal - mortality | Chromogranins - genetics | Female | Pancreatic Neoplasms - mortality | Retrospective Studies | Severity of Illness Index | Neoplasm Invasiveness | Pancreatic Neoplasms - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | Genes, p16 | Survival Analysis | Aged | Biomarkers, Tumor - genetics | Adenocarcinoma, Mucinous - genetics | Adenocarcinoma | Carcinoma | Invasiveness | Genes | Genetic relationship | Next-generation sequencing | Pancreatic carcinoma | Cysts | Medical prognosis | Pancreatic cancer | Genetic analysis | Mutation | Deoxyribonucleic acid--DNA | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 08/2017, Volume 146, Issue 2, pp. 305 - 313
To identify molecular alterations that contribute to vulvar cancer pathogenesis with the intent of identifying molecular targets for treatment. After... 
Molecular targets | Vulvar cancer | Molecular alterations | Targeted therapy | MULTIDRUG TRANSPORTER | ONCOLOGY-GROUP | THYMIDYLATE SYNTHASE | PHASE-II | CERVICAL-CANCER | INTRAEPITHELIAL NEOPLASIA | OBSTETRICS & GYNECOLOGY | GROWTH-FACTOR RECEPTOR | LUNG-CANCER | ONCOLOGY | SQUAMOUS-CELL CARCINOMA | ADVANCED NSCLC | Immunohistochemistry | Adenocarcinoma - pathology | Proto-Oncogene Proteins p21(ras) - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | GTP-Binding Protein alpha Subunits, Gs - genetics | Vulvar Neoplasms - pathology | Adenocarcinoma - metabolism | Vulvar Neoplasms - metabolism | Chromogranins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Proto-Oncogene Proteins B-raf - metabolism | Precision Medicine | Membrane Proteins - genetics | Vulvar Neoplasms - drug therapy | Cell Cycle Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | In Situ Hybridization, Fluorescence | Adenocarcinoma - drug therapy | Sequence Analysis, DNA | Carcinoma, Squamous Cell - drug therapy | Class I Phosphatidylinositol 3-Kinases | GTP Phosphohydrolases - metabolism | GTP Phosphohydrolases - genetics | Receptor, ErbB-4 - genetics | Mutation | Ubiquitin-Protein Ligases - genetics | F-Box Proteins - genetics | GTP-Binding Protein alpha Subunits, Gs - metabolism | Receptor, ErbB-4 - metabolism | F-Box-WD Repeat-Containing Protein 7 | Ataxia Telangiectasia Mutated Proteins - metabolism | Receptor, Fibroblast Growth Factor, Type 2 - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Molecular Targeted Therapy | Proto-Oncogene Proteins c-akt - genetics | Tumor Suppressor Protein p53 - genetics | Chromogranins - metabolism | Cell Cycle Proteins - genetics | Female | Adenocarcinoma - genetics | Membrane Proteins - metabolism | Retrospective Studies | Proto-Oncogene Proteins p21(ras) - metabolism | F-Box Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Phosphatidylinositol 3-Kinases - genetics | BRCA2 Protein - metabolism | Gene Amplification | Vulvar Neoplasms - genetics | Proto-Oncogene Proteins B-raf - genetics | Aged | High-Throughput Nucleotide Sequencing | Ataxia Telangiectasia Mutated Proteins - genetics | Neoplasm Staging | BRCA2 Protein - genetics | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Squamous cell carcinoma | Tumor proteins | Analysis
Journal Article
Archives of Pathology and Laboratory Medicine, ISSN 0003-9985, 06/2016, Volume 140, Issue 6, pp. 529 - 535
Journal Article
Gastrointestinal Endoscopy, ISSN 0016-5107, 2016, Volume 83, Issue 6, pp. 1107 - 1117.e2
Background and Aims The American Gastroenterological Association (AGA) recently reported evidence-based guidelines for the management of asymptomatic... 
Gastroenterology and Hepatology | PAPILLARY MUCINOUS NEOPLASMS | DIAGNOSIS | MALIGNANCY | MANAGEMENT | IMAGING FEATURES | HIGH-RISK | PREDICTORS | GASTROENTEROLOGY & HEPATOLOGY | TUMORS | LESIONS | INTERNATIONAL CONSENSUS GUIDELINES | Adenocarcinoma - pathology | Pancreatic Neoplasms - diagnosis | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Male | Carcinoma, Pancreatic Ductal - genetics | GTP-Binding Protein alpha Subunits, Gs - genetics | Young Adult | Cystadenoma, Serous - blood | Aged, 80 and over | Chromogranins - genetics | Endoscopic Ultrasound-Guided Fine Needle Aspiration | PTEN Phosphohydrolase - genetics | Adenocarcinoma - blood | Pancreatic Neoplasms - pathology | Pancreatic Cyst - diagnosis | Magnetic Resonance Imaging | Adenocarcinoma - diagnosis | Class I Phosphatidylinositol 3-Kinases | Adolescent | Neoplasms, Cystic, Mucinous, and Serous - diagnosis | Cystadenoma, Serous - genetics | Cystadenoma, Serous - pathology | Practice Guidelines as Topic | Endosonography | Carcinoembryonic Antigen - blood | Tumor Suppressor Protein p53 - genetics | Neoplasms, Cystic, Mucinous, and Serous - genetics | Molecular Diagnostic Techniques | Pancreatic Cyst - blood | Pancreatic Cyst - pathology | Sensitivity and Specificity | Carcinoma, Pancreatic Ductal - diagnosis | Adult | Female | Adenocarcinoma - genetics | Retrospective Studies | Von Hippel-Lindau Tumor Suppressor Protein - genetics | Pancreatic Neoplasms - blood | Neoplasms, Cystic, Mucinous, and Serous - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Pancreatic Cyst - genetics | Carcinoma, Pancreatic Ductal - blood | Phosphatidylinositol 3-Kinases - genetics | Cystadenoma, Serous - diagnosis | Cyst Fluid | Neoplasms, Cystic, Mucinous, and Serous - blood | Aged | Cysts | Medicine, Experimental | Medical research | Dysplasia | Tumor proteins | CEA (Oncology)
Journal Article
Journal Article
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 08/2014, Volume 20, Issue 16, pp. 4381 - 4389
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2014, Volume 135, Issue 11, pp. 2711 - 2720
Cyclophosphamide–dacarbazine–vincristine regimen is recommended for the treatment of malignant pheochromocytoma and paraganglioma (MPP); however, dacarbazine... 
succinate dehydrogenase B (SDHB) | temozolomide | malignant pheochromocytoma | O‐methylguanine‐DNA methyltransferase (MGMT) | paraganglioma | O-methylguanine-DNA methyltransferase (MGMT) | NEUROENDOCRINE TUMORS | SOLID TUMORS | BONE METASTASES | CYCLOPHOSPHAMIDE | FOLLOW-UP | CHROMOGRANIN-A | DACARBAZINE | ONCOLOGY | NEURON-SPECIFIC ENOLASE | SOMATOSTATIN RECEPTOR SCINTIGRAPHY | Adrenal Gland Neoplasms - drug therapy | Prognosis | Follow-Up Studies | Dacarbazine - therapeutic use | Humans | Middle Aged | Brain Neoplasms - pathology | DNA Repair Enzymes - genetics | Male | Pheochromocytoma - mortality | Promoter Regions, Genetic - genetics | Paraganglioma - secondary | Adrenal Gland Neoplasms - mortality | DNA Methylation | Neoplasm Metastasis | Paraganglioma - mortality | Tumor Suppressor Proteins - genetics | Dacarbazine - analogs & derivatives | Polymerase Chain Reaction | Aged, 80 and over | Adult | Female | Retrospective Studies | Brain Neoplasms - mortality | Pheochromocytoma - secondary | Adrenal Gland Neoplasms - pathology | Paraganglioma - genetics | Brain Neoplasms - genetics | Paraganglioma - drug therapy | Survival Rate | Brain Neoplasms - drug therapy | Mutation - genetics | Antineoplastic Agents, Alkylating - therapeutic use | Pheochromocytoma - drug therapy | DNA Modification Methylases - genetics | Succinate Dehydrogenase - genetics | Pheochromocytoma - genetics | Aged | Biomarkers, Tumor - genetics | Neoplasm Staging | Adrenal Gland Neoplasms - genetics | Hypertension | Immunohistochemistry | Antimitotic agents | Care and treatment | Pheochromocytoma | Gene mutations | Transferases | Genetic aspects | Metastasis | Methylation | Antineoplastic agents | Chemotherapy | Dehydrogenases | Mutation | Drug therapy | Tumors | Cancer | Succinate dehydrogenase | Toxicity | Methylguanine | Paraganglioma | Gene expression | Patients | Vincristine | Metastases | Anticancer properties | O6-methylguanine-DNA methyltransferase | Dacarbazine | Cyclophosphamide | Lymphopenia | DNA methylation | DNA methyltransferase | Temozolomide | Deoxyribonucleic acid--DNA | Neuroendocrine tumors | Index Medicus
Journal Article