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Nature, ISSN 0028-0836, 2014, Volume 509, Issue 7501, pp. 459 - 464
Journal Article
Nature Structural and Molecular Biology, ISSN 1545-9993, 05/2009, Volume 16, Issue 5, pp. 492 - 498
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 08/2016, Volume 34, Issue 23, pp. 2698 - 2704
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23899
The Wnt/beta-catenin signalling pathway shares a key component, beta-catenin, with the cadherin-based adhesion system. The signalling function of beta-catenin... 
TYROSINE PHOSPHORYLATION | WNT PATHWAY | DEPENDENT TRANSCRIPTION | FACTOR SCATTER FACTOR | BIOLOGY | TUMOR-SUPPRESSOR PROTEIN | ADHERENS JUNCTIONS | BETA-CATENIN | CELL-CELL ADHESION | NUCLEAR TRANSLOCATION | N-CADHERIN | Embryo, Mammalian - drug effects | Transcription, Genetic - drug effects | Epithelial Cells - metabolism | Cadherins - metabolism | Epithelial Cells - drug effects | Humans | Transcriptional Activation - drug effects | Mesoderm - drug effects | Cytoplasm - metabolism | Epithelial-Mesenchymal Transition - drug effects | Protein Transport - drug effects | Mesoderm - cytology | Epithelial-Mesenchymal Transition - genetics | Hepatocyte Growth Factor - pharmacology | Embryo, Mammalian - metabolism | Protein Binding - drug effects | HEK293 Cells | Cell Membrane - metabolism | Epithelial Cells - cytology | Cell Membrane - drug effects | Endocytosis - drug effects | beta Catenin - metabolism | Animals | Wnt Signaling Pathway - drug effects | Wnt Signaling Pathway - genetics | Dogs | Mesoderm - metabolism | Mice | Cytoplasm - drug effects | Regulators | Phosphorylation | Wnt protein | Transcription | Mesenchyme | Colorectal cancer | Activation | Biology | Cell adhesion & migration | Signal transduction | β-catenin | Embryogenesis | Endocytosis | Cell growth | Tumorigenesis | Trends | Growth factors | Chromosomes | Medical research | Gastrulation | Hepatocyte growth factor | Gene expression | Cadherin | Adhesion | Embryonic growth stage | Signaling | Insects | Morphology | Stem cells | Ligands | Mutation | Endoplasmic reticulum | Cytoplasm
Journal Article
ACS Nano, ISSN 1936-0851, 02/2009, Volume 3, Issue 2, pp. 279 - 290
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2016, Volume 29, Issue 4, pp. 464 - 476
The epigenome is a key determinant of transcriptional output. Perturbations within the epigenome are thought to be a key feature of many, perhaps all cancers,... 
targeted therapy | epigenome | histone modification | chromatin | Cancer | Histone modification | Chromatin | Epigenome | Targeted therapy | SELECTIVE-INHIBITION | THERAPEUTIC STRATEGY | ONCOLOGY | LYSINE 27 METHYLATION | HISTONE H3 | DROSOPHILA-TRITHORAX | 11Q23 TRANSLOCATIONS | CHROMATIN MODIFICATIONS | EMERGING MECHANISMS | TRANSCRIPTIONAL REGULATION | SOMATIC MUTATIONS | CELL BIOLOGY | Transcription, Genetic - drug effects | Humans | Gene Expression Regulation, Neoplastic | Neoplasms - prevention & control | Molecular Targeted Therapy | Neoplasm Proteins - metabolism | Neoplasms - therapy | Histone Code - drug effects | Neoplasms - genetics | Protein Processing, Post-Translational - drug effects | Cell Transformation, Neoplastic - genetics | Antineoplastic Agents - pharmacokinetics | Epigenesis, Genetic - drug effects | Chromatin - drug effects | Epigenomics | Oncogene Proteins - metabolism | Therapies, Investigational | Clinical Trials as Topic | DNA, Neoplasm - drug effects | Drug Resistance, Neoplasm - genetics | Animals | Epigenesis, Genetic - genetics | Chromosome Aberrations | Histone Deacetylase Inhibitors - therapeutic use | Mice | Models, Genetic | DNA, Neoplasm - genetics | Histones - metabolism | Mutation | Chromatin - genetics | DNA Methylation - drug effects | Drug Resistance, Neoplasm - drug effects | Epigenetic inheritance | Development and progression | Genetic aspects
Journal Article
Angiogenesis, ISSN 0969-6970, 3/2012, Volume 15, Issue 1, pp. 33 - 45
Exosomes, microvesicles of endocytic origin released by normal and tumor cells, play an important role in cell-to-cell communication. Angiogenesis has been... 
Biomedicine | Nanotubes | Oncology | Cancer Research | Ophthalmology | Chronic myeloid leukemia | Cardiology | Exosomes | Biomedicine general | Endothelial cells | Tyrosine kinase inhibitors | Cell Biology | PROGENITOR CELLS | BONE-MARROW | KINASE INHIBITOR | CANCER | MEMBRANE NANOTUBES | CHROMOSOME | ABL GENE | IMATINIB | PERIPHERAL VASCULAR DISEASE | TUMOR-GROWTH | ENDOTHELIAL GROWTH-FACTOR | Exosomes - drug effects | Neovascularization, Physiologic - drug effects | Laminin - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Culture Media, Conditioned - pharmacology | Thiazoles - therapeutic use | Proteoglycans - drug effects | Collagen - drug effects | Time Factors | Human Umbilical Vein Endothelial Cells - cytology | src-Family Kinases - metabolism | Exosomes - secretion | Exosomes - ultrastructure | Dasatinib | Reproducibility of Results | Human Umbilical Vein Endothelial Cells - drug effects | Endocytosis - drug effects | Piperazines - therapeutic use | Pyrimidines - pharmacology | Imatinib Mesylate | Piperazines - pharmacology | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Signal Transduction - drug effects | Cell Differentiation - drug effects | Mice, Nude | Pyrimidines - therapeutic use | Cell Communication - drug effects | K562 Cells | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Thiazoles - pharmacology | Benzamides | Drug Combinations | Usage | BCR-ABL tyrosine kinase inhibitors | Genetic aspects | Research | Health aspects | Risk factors | Drugs | Animal models | Imatinib | Phosphorylation | Tumor cells | Cell interactions | Src protein | Angiogenesis | Signal transduction | exosomes | Microenvironments | Differentiation | Protein-tyrosine kinase | Elongation
Journal Article