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Journal of Bone and Mineral Research, ISSN 0884-0431, 08/2012, Volume 27, Issue 8, pp. 1757 - 1772
Chronic kidney disease–mineral bone disorder (CKD‐MBD) is defined by abnormalities in mineral and hormone metabolism, bone histomorphometric changes, and/or... 
RENAL OSTEODYSTROPHY | OSTEOCYTE | WNT/β‐CATENIN | WNT/β-CATENIN | AORTIC CALCIFICATION | VASCULAR CALCIFICATION | ss-CATENIN | MINERAL BONE DISORDER | BETA-CATENIN | NONDIALYZED PATIENTS | CORONARY-ARTERY CALCIFICATION | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | HEMODIALYSIS-PATIENTS | PARATHYROID-HORMONE | WNT | CHRONIC KIDNEY-DISEASE | Bone and Bones - pathology | Chronic Kidney Disease-Mineral and Bone Disorder - pathology | Chronic Kidney Disease-Mineral and Bone Disorder - blood | Humans | Middle Aged | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Glycoproteins - metabolism | Cardiovascular Abnormalities - blood | Male | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Gene Expression Profiling | Cardiovascular Abnormalities - pathology | Cardiovascular Abnormalities - physiopathology | Kidney Failure, Chronic - pathology | Bone and Bones - metabolism | Female | Kidney Failure, Chronic - physiopathology | Osteoclasts - pathology | Cardiovascular Abnormalities - complications | Osteocytes - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Mutation - genetics | Osteoclasts - metabolism | Disease Progression | Animals | Kidney Failure, Chronic - blood | Wnt Signaling Pathway - genetics | Biopsy | Calcification, Physiologic | Kidney Failure, Chronic - complications | Osteocytes - pathology | Mice | Bone Remodeling | NIMA-Related Kinases | Vascular Calcification | Calcification (ectopic) | Renal function | Renal osteodystrophy | Wnt protein | Polycystic kidney | catenin | Osteocytes | Bone diseases | Minerals | Gene expression | Osteoprotegerin | Gene silencing | Signal transduction | NF- Kappa B protein | Osteoclasts | Kidney diseases | TRANCE protein | Age | Osteogenesis
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 09/2018, Volume 19, Issue 9, p. 2490
The high cardiovascular mortality associated with chronic kidney disease (CKD) is caused in part by the CKD-mineral bone disorder (CKD-MBD) syndrome. The... 
Osteoblast | Vascular calcification | Activin A | Osteoclast | CKD-MBD | DNA-BINDING | REGULATES OSTEOCLAST DIFFERENTIATION | FSH-RELEASING PROTEIN | TGF-BETA | OSTEOBLAST DIFFERENTIATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | vascular calcification | BINDING-PROTEIN | osteoblast | CHEMISTRY, MULTIDISCIPLINARY | activin A | SMOOTH-MUSCLE-CELLS | osteoclast | TYPE-2 DIABETES-MELLITUS | LACKING C-FOS | Bone and Bones - pathology | Chronic Kidney Disease-Mineral and Bone Disorder - pathology | RANK Ligand - metabolism | Kidney - pathology | Humans | Activins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Activin Receptors - genetics | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Parathyroid Hormone - genetics | Kidney - metabolism | Osteoporosis - metabolism | Bone Resorption - genetics | Parathyroid Hormone - metabolism | Bone and Bones - metabolism | Bone Resorption - metabolism | Osteoporosis - genetics | Osteogenesis - genetics | Disease Models, Animal | Signal Transduction | Gene Expression Regulation | Smad Proteins - genetics | RANK Ligand - genetics | Activin Receptors - metabolism | Animals | Osteoporosis - pathology | Bone Resorption - pathology | Activins - genetics | Mice | Bone Remodeling | Smad Proteins - metabolism | Animal models | Target recognition | Medical services | Activation | Minerals | Bone diseases | Osteoblasts | Bone resorption | Osteoporosis | Receptors | Bone growth | Biomedical materials | Biocompatibility | TRANCE protein | Growth factors | Parathyroid | Public health | Osteopenia | Bone remodelling | Diabetes mellitus | Bone turnover | Metabolism | Activin receptors | Osteoclastogenesis | Signaling | Molecular modelling | Osteodystrophy | Parathyroid hormone | Ligands | Osteoclasts | Kidney diseases | Receptor mechanisms | Cardiovascular diseases | Activin | Osteogenesis | Phosphatase | Proteins | Signal transduction | Mineralization | Bone marrow | Cytokines | Mortality | Gene expression | Calcification | Phosphorylation | Homeostasis | Kinases | Tumor necrosis factor-TNF | Diabetes
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2012, Volume 122, Issue 7, pp. 2543 - 2553
Chronic kidney disease-mineral and bone disorder (CKD-MBD) is associated with secondary hyperparathyroidism (HPT) and serum elevations in the phosphaturic... 
MEDICINE, RESEARCH & EXPERIMENTAL | FAMILIAL TUMORAL CALCINOSIS | VITAMIN-D METABOLISM | CARDIOVASCULAR EVENTS | VASCULAR CALCIFICATION | HEMODIALYSIS-PATIENTS | PARATHYROID-HORMONE | PHYSIOLOGICAL-ROLE | FIBROBLAST GROWTH FACTOR-23 | LEFT-VENTRICULAR HYPERTROPHY | STAGE RENAL-DISEASE | Chronic Kidney Disease-Mineral and Bone Disorder - blood | Kidney - pathology | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Hyperparathyroidism, Secondary - physiopathology | Male | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Parathyroid Hormone - blood | Tibia - pathology | Fibroblast Growth Factors - immunology | Luciferases - genetics | Renal Insufficiency, Chronic - metabolism | Calcium - blood | Fibroblast Growth Factors - metabolism | Phosphates - blood | Tibia - metabolism | Genes, Reporter | Kidney - physiopathology | CHO Cells | Biomarkers - metabolism | Cricetinae | Hyperparathyroidism, Secondary - metabolism | Glomerular Filtration Rate | Rats | Myocardium - pathology | Vascular Calcification - pathology | Calcitriol - blood | Rats, Sprague-Dawley | Renal Insufficiency, Chronic - physiopathology | Aorta - pathology | Renal Insufficiency, Chronic - blood | Hyperparathyroidism, Secondary - blood | Animals | Mice | Mice, Inbred BALB C | Fibroblast Growth Factors - antagonists & inhibitors | Hemodynamics | Antibodies, Monoclonal, Murine-Derived - pharmacology | Luciferases - biosynthesis | Complications and side effects | United States | Mortality | Research | Kidney diseases | Hyperparathyroidism | Risk factors
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 147 - 158
We examined activin receptor type IIA (ActRIIA) activation in chronic kidney disease (CKD) by signal analysis and inhibition in mice with Alport syndrome using... 
cardiovascular disease | chronic kidney disease | bone | fibrosis | mineral metabolism | vascular calcification | TGF-BETA | CARDIOVASCULAR EVENTS | RENAL FIBROSIS | ARTERIAL CALCIFICATION | BONE MORPHOGENETIC PROTEIN-7 | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | LINKED ALPORT-SYNDROME | OSTEOCLAST DIFFERENTIATION | ATHEROSCLEROTIC LESION | Kidney - pathology | Recombinant Fusion Proteins - pharmacology | Blood Vessels - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Vascular Calcification - metabolism | Kidney - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Bone Resorption - metabolism | Disease Models, Animal | Bone Resorption - physiopathology | Glomerular Filtration Rate | Signal Transduction | Smad2 Protein - metabolism | Cardiomegaly - physiopathology | Nephritis, Hereditary - genetics | Renal Insufficiency, Chronic - physiopathology | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Cardiomegaly - prevention & control | Nephritis, Hereditary - metabolism | Collagen Type IV - deficiency | Activin Receptors, Type II - genetics | Fibrosis | Sp7 Transcription Factor - metabolism | Collagen Type IV - genetics | Cardiomegaly - metabolism | Activin Receptors, Type II - metabolism | Bone and Bones - pathology | Phosphorylation | Vascular Remodeling | Blood Vessels - pathology | Vascular Calcification - genetics | Actins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Activin Receptors, Type II - antagonists & inhibitors | Blood Vessels - physiopathology | Renal Insufficiency, Chronic - metabolism | Ventricular Remodeling | Chronic Kidney Disease-Mineral and Bone Disorder - prevention & control | Muscle Proteins - metabolism | Vascular Calcification - physiopathology | Microfilament Proteins - metabolism | Nephritis, Hereditary - physiopathology | Kidney - physiopathology | Vascular Calcification - prevention & control | Myocardium - pathology | Bone Resorption - prevention & control | Bone and Bones - physiopathology | Animals | Nephritis, Hereditary - drug therapy | Cardiomegaly - genetics | Bone Remodeling | Chronic kidney disease
Journal Article
Seminars in Nephrology, ISSN 0270-9295, 2013, Volume 33, Issue 2, pp. 169 - 179
Summary Childhood and adolescence are crucial times for the development of a healthy skeletal and cardiovascular system. Disordered mineral and bone metabolism... 
Nephrology | parathyroid hormone | Osteodystrophy | PTH | vitamin D | vascular calcification | pediatrics | growth | CKD mineral and bone disorder | Pediatrics | Vitamin D | Growth | Parathyroid hormone | Vascular calcification | CHRONIC PERITONEAL-DIALYSIS | VITAMIN-D METABOLITES | IMMUNOMETRIC PTH ASSAYS | LEFT-VENTRICULAR HYPERTROPHY | CORONARY-ARTERY CALCIFICATION | CALCIUM-SENSING RECEPTOR | BOVINE PARATHYROID CELLS | INTERMITTENT CALCITRIOL THERAPY | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | CHRONIC-RENAL-FAILURE | Hyperparathyroidism, Secondary - therapy | Hyperparathyroidism, Secondary - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - therapy | Calcium - metabolism | Bone Development - physiology | Humans | Bone Diseases, Metabolic - etiology | Calcification, Physiologic - physiology | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Renal Insufficiency, Chronic - complications | Bone Diseases, Metabolic - metabolism | Renal Insufficiency, Chronic - metabolism | Fibroblast Growth Factors - metabolism | Vascular Calcification - metabolism | Phosphates - metabolism | Parathyroid Hormone - metabolism | Vitamin D - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - etiology | Vascular Calcification - therapy | Hyperparathyroidism, Secondary - etiology | Child | parathyroid hormone (PTH | CKD Mineral and Bonde Disorder | osteodystrophy
Journal Article
Kidney International, ISSN 0085-2538, 12/2017, Volume 92, Issue 6, pp. 1343 - 1355
Mineral and bone disease is omnipresent in patients with chronic kidney disease (CKD) and leads to a diverse range of clinical manifestations, including bone... 
dialysis | bone mineral density | parathyroid hormone | calcium | vitamin D | phosphate | bone | calcimimetics | CKD-MBD | fracture | BONE-MINERAL DENSITY | RANDOMIZED CONTROLLED-TRIAL | QUANTITATIVE COMPUTED-TOMOGRAPHY | PATIENTS RECEIVING HEMODIALYSIS | INTACT PTH LEVELS | VITAMIN-D DEFICIENCY | HIGH-DOSE CHOLECALCIFEROL | HIP FRACTURE | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | CHRONIC KIDNEY-DISEASE | Chronic Kidney Disease-Mineral and Bone Disorder - blood | Chronic Kidney Disease-Mineral and Bone Disorder - complications | Humans | Bone Morphogenetic Proteins - blood | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Parathyroid Hormone - blood | Renal Insufficiency, Chronic - complications | Bone and Bones - drug effects | Renal Insufficiency, Chronic - metabolism | Renal Insufficiency, Chronic - therapy | Kidney - metabolism | Fractures, Bone - etiology | Parathyroid Hormone - metabolism | Calcification, Physiologic - drug effects | Chronic Kidney Disease-Mineral and Bone Disorder - therapy | Bone Density Conservation Agents - therapeutic use | Biomarkers - blood | Genetic Markers | Fractures, Bone - blood | Vitamin D - blood | Bone Density Conservation Agents - blood | Renal Insufficiency, Chronic - blood | Bone Density - drug effects | Bone and Bones - physiopathology | Fibroblast Growth Factors - blood | Renal Dialysis - adverse effects | Vitamin D - therapeutic use | Fractures, Bone - prevention & control
Journal Article