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Nature, ISSN 0028-0836, 04/2013, Volume 496, Issue 7444, pp. 238 - 242
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2014, Volume 111, Issue 29, pp. 10574 - 10579
Metformin, a first-line diabetes drug linked to cancer prevention in retrospective clinical analyses, inhibits cellular transformation and selectively kills... 
Lactates | Energy metabolism | Cellular metabolism | Epithelial cells | Cell lines | Stem cells | Glycolysis | Transformed cell line | Breast cancer | Cancer | Metabolic profiling | Cancer metabolism | Metabolism | COMPLEX I | RESPIRATORY-CHAIN | MULTIDISCIPLINARY SCIENCES | glycolysis | metabolic profiling | DECREASES | CHEMOTHERAPY | cancer metabolism | BREAST-CANCER | DIABETIC-PATIENTS | GROWTH | MITOCHONDRIAL DYSFUNCTION | metabolism | INFLAMMATORY RESPONSE | Biguanides - pharmacology | Ribonucleotides - metabolism | Neoplastic Stem Cells - drug effects | Humans | Metformin - pharmacology | Citric Acid Cycle - drug effects | Glycerophosphates - metabolism | Endoplasmic Reticulum - metabolism | Nucleotides - metabolism | Glycolysis - drug effects | Phenformin - pharmacology | Lactates - metabolism | Aminoimidazole Carboxamide - analogs & derivatives | Metabolome - drug effects | Neoplastic Stem Cells - metabolism | Endoplasmic Reticulum - drug effects | Tamoxifen - pharmacology | src-Family Kinases - metabolism | Cell Line, Tumor | Folic Acid - metabolism | Neoplastic Stem Cells - pathology | Aminoimidazole Carboxamide - metabolism | Cell Line, Transformed | Pharmaceutical research | Stem cell research | Analysis | Drug interactions | Cancer cells | Dosage and administration | Metformin | Research | Health aspects | Prescription drugs | Mitochondria | Acids | Metabolites | Index Medicus | Biological Sciences
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2014, Volume 289, Issue 43, pp. 30114 - 30132
Background:Yersinia pseudotuberculosis is a human pathogen and the ancestor of Y. pestis. Results: The pyruvate-tricarboxylic acid cycle node in the carbon... 
Gene Expression | Bacterial Metabolism | Yersinia | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | Pyruvate | CHROMOSOMAL GENES | Metabolic Flux Analysis | Systems Biology | GLUCOSEPHOSPHATE STRESS | LYSINE PRODUCTION | BACILLUS-SUBTILIS | Tricarboxylic Acid Cycle (TCA Cycle) (Krebs Cycle) | Bacterial Pathogenesis | SECRETION SYSTEM | PESTIS | Bacteria | EXPRESSION | CORYNEBACTERIUM-GLUTAMICUM | Virulence - drug effects | Molecular Weight | Escherichia coli - drug effects | Adaptation, Physiological - drug effects | Yersinia pseudotuberculosis - growth & development | Biomass | Gene Regulatory Networks | Virulence - genetics | Adaptation, Physiological - genetics | Escherichia coli - metabolism | Female | Stress, Physiological - drug effects | Iron - pharmacology | Pyruvates - metabolism | Stress, Physiological - genetics | Bacterial Proteins - genetics | Citric Acid Cycle - drug effects | Glucose - pharmacology | Transcriptome - drug effects | Transcriptome - genetics | Mutation - genetics | Yersinia pseudotuberculosis - genetics | Gene Expression Regulation, Bacterial - drug effects | Citric Acid Cycle - genetics | Animals | Yersinia pseudotuberculosis - metabolism | Yersinia pseudotuberculosis Infections - microbiology | Bacterial Proteins - metabolism | Mice | Mice, Inbred BALB C | Yersinia pseudotuberculosis Infections - pathology | Yersinia pseudotuberculosis - pathogenicity | Index Medicus | Microbiology
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2007, Volume 282, Issue 7, pp. 4524 - 4532
The stability and transcriptional activity of the hypoxia-inducible factors (HIFs) are regulated by two oxygen-dependent events that are catalyzed by three HIF... 
Gene Expression Regulation, Enzymologic - drug effects | Transcription, Genetic - drug effects | Vascular Endothelial Growth Factor A - biosynthesis | Fibroblasts - enzymology | Fumarate Hydratase - chemistry | Mixed Function Oxygenases - metabolism | Fumarate Hydratase - deficiency | Mixed Function Oxygenases - chemistry | Succinate Dehydrogenase - chemistry | Procollagen-Proline Dioxygenase - metabolism | Dose-Response Relationship, Drug | Fumarate Hydratase - metabolism | Neoplasms - chemistry | Neoplasms - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - chemistry | Erythropoietin - biosynthesis | Fibroblasts - chemistry | Cell Line | Enzyme Inhibitors - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Citric Acid Cycle - drug effects | Neoplasms - enzymology | Mixed Function Oxygenases - antagonists & inhibitors | Fibroblasts - pathology | Succinate Dehydrogenase - deficiency | p300-CBP Transcription Factors - metabolism | Citric Acid Cycle - genetics | Procollagen-Proline Dioxygenase - genetics | Gene Expression Regulation, Enzymologic - genetics | Succinate Dehydrogenase - metabolism | Mixed Function Oxygenases - genetics | Neoplasms - pathology | Procollagen-Proline Dioxygenase - chemistry | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2018, Volume 115, Issue 7, pp. E1578 - E1587
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0135083 - e0135083
The chemotherapeutic use of cisplatin is limited by its severe side effects. In this study, by conducting different omics data analyses, we demonstrated that... 
APOPTOSIS | OXIDATIVE STRESS | P53 ACTIVATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | TUBULAR EPITHELIAL-CELLS | DOWN-REGULATION | AUDITORY CELLS | DEATH | CANCER | NEPHROTOXICITY | L-Lactate Dehydrogenase - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Male | Membrane Potential, Mitochondrial - drug effects | Glycolysis - drug effects | Reactive Oxygen Species - agonists | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - toxicity | Dose-Response Relationship, Drug | Cisplatin - toxicity | Tumor Suppressor Protein p53 - agonists | Epithelial Cells - cytology | Kidney Tubules - metabolism | Cell Line | Kidney Tubules - drug effects | Citric Acid Cycle - drug effects | Gene Expression Regulation | Injections, Intraperitoneal | Tumor Suppressor Protein p53 - metabolism | Rats | Rats, Sprague-Dawley | Kidney Tubules - cytology | Animals | L-Lactate Dehydrogenase - genetics | Acetylcysteine - pharmacology | Cell Proliferation - drug effects | Physiological aspects | Reactive oxygen species | Genetic aspects | Research | Cisplatin | Bioengineering | Tricarboxylic acid cycle | Oxidative stress | Correlation | Toxicity | p53 Protein | Genes | Science | Cytotoxicity | Kinases | Ovarian cancer | Mitochondria | Cell growth | Pathways | Metabolites | Rodents | Cell cycle | Inhibition | Adenosine triphosphate | Urine | Abnormalities | Mortality | Data processing | Interdisciplinary aspects | Metabolism | Gene expression | Side effects | Cell death | Glycolysis | Apoptosis | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 130, Issue 5, pp. 797 - 810
Antibiotic mode-of-action classification is based upon drug-target interaction and whether the resultant inhibition of cellular function is lethal to bacteria.... 
SYSBIO | MICROBIO | OXYGEN | SUPEROXIDE | OXIDATIVE DAMAGE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | DNA-DAMAGE | HYDROGEN-PEROXIDE | SOS RESPONSE | TRANSCRIPTIONAL REGULATION | GENE ONTOLOGY | IRON-SULFUR CLUSTERS | CELL BIOLOGY | Free Radical Scavengers - pharmacology | Hydroxyurea - pharmacology | Escherichia coli - drug effects | Gene Expression Profiling | Colony Count, Microbial | DNA, Bacterial - drug effects | Norfloxacin - pharmacology | Time Factors | Carbon-Sulfur Lyases - genetics | Escherichia coli - metabolism | Ampicillin - pharmacology | Membrane Proteins - metabolism | Cell Death - drug effects | Escherichia coli - growth & development | Rec A Recombinases - genetics | NAD - metabolism | Staphylococcus aureus - metabolism | 2,2'-Dipyridyl - pharmacology | Staphylococcus aureus - genetics | Rec A Recombinases - metabolism | Microbial Viability - drug effects | Membrane Proteins - genetics | Citric Acid Cycle - drug effects | Escherichia coli Proteins - metabolism | Hydroxyl Radical - metabolism | Carbon-Sulfur Lyases - metabolism | Kanamycin - pharmacology | Hydrogen Peroxide - metabolism | Gene Expression Regulation, Bacterial - drug effects | Citric Acid Cycle - genetics | Anti-Bacterial Agents - classification | Iron Chelating Agents - pharmacology | Escherichia coli - genetics | Ferrous Compounds - metabolism | Escherichia coli Proteins - genetics | Anti-Bacterial Agents - pharmacology | DNA Damage | Mutation | Oxidative Stress - drug effects | Staphylococcus aureus - drug effects | Staphylococcus aureus - growth & development | Metabolites | Antibiotics | Index Medicus
Journal Article
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