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1. Full Text Modulation of BMP signaling by Noggin is required for the maintenance of palatal epithelial integrity during palatogenesis
by He, Fenglei and Xiong, Wei and Wang, Ying and Matsui, Maiko and Yu, Xueyan and Chai, Yang and Klingensmith, John and Chen, YiPing
Developmental biology, ISSN 0012-1606, 11/2010, Volume 347, Issue 1, pp. 109 - 121
Noggin | Palatal epithelium | Cleft palate | Palate development | Bmp signaling | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Proliferation | Mandible - pathology | Palate - pathology | Embryo, Mammalian - metabolism | Bone Morphogenetic Proteins - metabolism | Epithelium - embryology | Gene Expression Regulation, Developmental | Bone Morphogenetic Proteins - genetics | Mandible - abnormalities | Palate - metabolism | Epithelium - pathology | Embryo, Mammalian - pathology | Epithelium - metabolism | Signal Transduction | Cleft Palate - embryology | Mutation - genetics | Palate - embryology | Organogenesis | Phenotype | Animals | Carrier Proteins - metabolism | Palate - abnormalities | Cleft Palate - pathology | Cleft Palate - metabolism | Mice | Smad Proteins - metabolism | Apoptosis | Gene expression | Bone morphogenetic proteins | Analysis | Index Medicus | cleft palate | palatal epithelium | palate development
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2. Full Text Palatogenesis: Morphogenetic and molecular mechanisms of secondary palate development
by Bush, Jeffrey O and Jiang, Rulang
Development (Cambridge), ISSN 0950-1991, 01/2012, Volume 139, Issue 2, pp. 231 - 243
Morphogenesis | Palatogenesis | Cleft palate | Secondary palate | Palate development | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Animals | Morphogenesis - genetics | Models, Biological | Cleft Palate - genetics | Cleft Palate - physiopathology | Mammals | Transforming Growth Factor beta - metabolism | Morphogenesis - physiology | Palate - embryology | Index Medicus | Reviews
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3. Full Text Molecular control of secondary palate development
by Gritli-Linde, Amel
Developmental biology, ISSN 0012-1606, 2007, Volume 301, Issue 2, pp. 309 - 326
Signaling pathways | Cleft palate | Epithelial–mesenchymal interactions | Mouse mutations | Palate patterning | Palate development | Epithelial-mesenchymal interactions | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Animals | Epithelial Cells - metabolism | Signal Transduction | Humans | Cleft Palate - embryology | Cleft Palate - metabolism | Mutation - genetics | Stem Cells - metabolism | Index Medicus
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4. Full Text Imputation of orofacial clefting data identifies novel risk loci and sheds light on the genetic background of cleft lip ± cleft palate and cleft palate only
by Ludwig, Kerstin U and Böhmer, Anne C and Bowes, John and Nikolić, Miloš and Ishorst, Nina and Wyatt, Niki and Hammond, Nigel L and Gölz, Lina and Thieme, Frederic and Barth, Sandra and Schuenke, Hannah and Klamt, Johanna and Spielmann, Malte and Aldhorae, Khalid and Rojas-Martinez, Augusto and Nöthen, Markus M and Rada-Iglesias, Alvaro and Dixon, Michael J and Knapp, Michael and Mangold, Elisabeth
Human molecular genetics, ISSN 0964-6906, 02/2017, Volume 26, Issue 4, pp. 829 - 842
Genome-Wide Association Study | Chromosomes, Human - genetics | Humans | Databases, Genetic | Male | Cleft Palate - genetics | Genetic Loci | Cleft Lip - metabolism | Cleft Lip - genetics | Animals | Cleft Lip - pathology | Cleft Palate - pathology | Female | Cleft Palate - metabolism | Mice | Polymorphism, Single Nucleotide | Index Medicus | Association Studies
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5. Full Text Wnt9b-dependent FGF signaling is crucial for outgrowth of the nasal and maxillary processes during upper jaw and lip development
by Jin, Yong-Ri and Han, Xiang Hua and Taketo, Makoto M and Yoon, Jeong Kyo
Development (Cambridge), ISSN 0950-1991, 05/2012, Volume 139, Issue 10, pp. 1821 - 1830
β-catenin | Cleft palate | Cleft lip | Mouse | FGF signaling | Orofacial morphogenesis | WNT | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Nose - embryology | Jaw - embryology | Cell Proliferation | Lip - embryology | Fibroblast Growth Factors - genetics | Apoptosis - genetics | Cleft Palate - genetics | Wnt Proteins - metabolism | Cell Movement - genetics | Cell Movement - physiology | Lip - metabolism | Fibroblast Growth Factors - metabolism | In Situ Hybridization | Wnt Proteins - genetics | Real-Time Polymerase Chain Reaction | Organ Culture Techniques | Nose - metabolism | Palate - metabolism | Microscopy, Electron, Scanning | Maxilla - embryology | Cells, Cultured | Cleft Lip - metabolism | beta Catenin - metabolism | Blotting, Western | Palate - embryology | Mice, Knockout | Cleft Lip - genetics | Animals | Maxilla - metabolism | Jaw - metabolism | Cleft Palate - metabolism | Mice | Apoptosis - physiology | Index Medicus | Cell proliferation | Fibroblast growth factor | Jaw | Mesenchyme | Wnt protein | catenin | Gene expression | Maxilla | Cleft lip/palate | Lip | Development | Palate | Mutation | β-Catenin
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6. Full Text Cleft palate defect of Dlx1/2-/- mutant mice is caused by lack of vertical outgrowth in the posterior palate
by Jeong, J and Cesario, J and Zhao, Y and Burns, L and Westphal, H and Rubenstein, J.L
Developmental dynamics, ISSN 1058-8388, 2012, Volume 241, Issue 11, pp. 1757 - 1769
palatogenesis | Dlx | cleft palate | craniofacial development | mice | Craniofacial development | Palatogenesis | Cleft palate | Mice | Palate - metabolism | Cyclin D1 - metabolism | Cell Proliferation | Homeodomain Proteins - metabolism | Gene Expression Regulation, Developmental - genetics | Transcription Factors - genetics | Homeodomain Proteins - genetics | Palate - embryology | Mice, Knockout | Transcription Factors - metabolism | Animals | Cyclin D1 - genetics | Cleft Palate - metabolism | Gene Expression Regulation, Developmental - physiology | Bone morphogenetic proteins | Fibroblast growth factors | Genetic transcription | DNA binding proteins | Analysis | Index Medicus
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7. Full Text Golgb1 regulates protein glycosylation and is crucial for mammalian palate development
by Lan, Yu and Zhang, Nian and Liu, Han and Xu, Jingyue and Jiang, Rulang
Development (Cambridge), ISSN 0950-1991, 07/2016, Volume 143, Issue 13, pp. 2344 - 2355
CRISPR | Cleft palate | Craniofacial | Genome editing | Glycosylation | Golgi | Mutation | ENU | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | RNA Editing - genetics | Cell Count | Cleft Palate - genetics | Mesoderm - cytology | RNA, Messenger - metabolism | Embryo, Mammalian - metabolism | Lectins - metabolism | Mice, Mutant Strains | Base Sequence | RNA Splicing - genetics | Mammals - embryology | Ethylnitrosourea | Female | Membrane Proteins - metabolism | Golgi Apparatus - ultrastructure | Palate - metabolism | Mesoderm - embryology | RNA Splice Sites - genetics | Membrane Proteins - genetics | RNA, Messenger - genetics | Mutation - genetics | CRISPR-Cas Systems - genetics | Palate - embryology | Animals | Hyaluronic Acid - metabolism | Fluorescent Antibody Technique | Golgi Apparatus - metabolism | Genome | Index Medicus
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8. Full Text Wnt5a regulates directional cell migration and cell proliferation via Ror2-mediated noncanonical pathway in mammalian palate development
by Fenglei He and Wei Xiong and Xueyan Yu and Ramon Espinoza-Lewis and Chao Liu and Shuping Gu and Michiru Nishita and Kentaro Suzuki and Gen Yamada and Yasuhiro Minami and YiPing Chen
Development (Cambridge), ISSN 0950-1991, 12/2008, Volume 135, Issue 23, pp. 3871 - 3879
Palatogenesis | Cleft palate | Non-canonical Wnt pathway | Ror2 | Wnt5a | Cell migration | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Proliferation | Wnt Proteins - deficiency | Receptor Tyrosine Kinase-like Orphan Receptors | Wnt-5a Protein | Wnt Proteins - metabolism | Cleft Palate - enzymology | Wnt Proteins - genetics | Gene Expression Regulation, Developmental | Mice, Mutant Strains | Mesoderm - enzymology | Body Patterning | Mammals - embryology | Bromodeoxyuridine - metabolism | Palate - cytology | Palate - metabolism | Green Fluorescent Proteins - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Chemotaxis | Palate - embryology | Phenotype | Animals | Receptor Protein-Tyrosine Kinases - genetics | Epistasis, Genetic | Cleft Palate - pathology | Palate - enzymology | Mice | Mesoderm - pathology | Cell Movement | Index Medicus
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9. Full Text Glycoprotein A repetitions predominant (GARP) positively regulates transforming growth factor (TGF) β3 and is essential for mouse palatogenesis
by Wu, Bill X and Li, Anqi and Lei, Liming and Kaneko, Satoshi and Wallace, Caroline and Li, Xue and Li, Zihai and O'Neill, Luke
The Journal of biological chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 44, pp. 18091 - 18097
Phosphorylation | Transforming Growth Factor beta3 - genetics | Humans | Protein Multimerization | Palate - pathology | Recombinant Fusion Proteins - metabolism | Transforming Growth Factor beta3 - metabolism | Embryo, Mammalian - metabolism | Gene Expression Regulation, Developmental | Transforming Growth Factor beta3 - chemistry | HEK293 Cells | Female | Membrane Proteins - metabolism | Animals, Newborn | Palate - metabolism | Embryo, Mammalian - pathology | Signal Transduction | Membrane Proteins - genetics | Smad2 Protein - metabolism | Cleft Palate - embryology | Recombinant Fusion Proteins - chemistry | Embryo, Mammalian - abnormalities | Palate - embryology | Gene Knock-In Techniques | Mice, Knockout | Organogenesis | Protein Transport | Pregnancy | Animals | Membrane Proteins - chemistry | Palate - abnormalities | Cleft Palate - pathology | Heterozygote | Cleft Palate - metabolism | Protein Processing, Post-Translational | Transforming Growth Factor beta3 - agonists | Apoptosis | Index Medicus | development | GARP, palatogenesis | cell signaling | TGFβ | apoptosis | embryo | Developmental Biology
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