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Biochimica et Biophysica Acta - Molecular Basis of Disease, ISSN 0925-4439, 04/2016, Volume 1862, Issue 4, pp. 662 - 669
Background and aims: Pretreatment with clofibrate, a peroxisome proliferator-activated receptor alpha (PPARa) agonist, protects mice from acetaminophen (APAP)... 
Cysteamine | Pantetheine hydrolase | Peroxisome proliferators | Clofibrate | Liver
Journal Article
Bioorganic & Medicinal Chemistry Letters, ISSN 0960-894X, 10/2018, Volume 28, Issue 18, pp. 3119 - 3122
[Display omitted] •Design of CF-HT with a significantly hypolipidemic activity.•CF-HT displayed potent antioxidant and hepatoprotective effects.•Study... 
Hypolipidemic | Hepatoprotective | Hydroxytyrosol-clofibrate | Antioxidant | OXIDATIVE STRESS | CHEMISTRY, MEDICINAL | ACIDS | PHENOLIC-COMPOUNDS | RATS | CHEMISTRY, ORGANIC | VIRGIN OLIVE OIL | BIOLOGICAL-ACTIVITIES | PLASMA | PHARMACOKINETICS | LIVER | HEPATOTOXICITY | Antioxidants | Lactates | Phosphatases | Dimethylformamide | Clofibrate | Liver | Superoxide | Bilirubin | Index Medicus
Journal Article
Cochrane Database of Systematic Reviews, ISSN 1469-493X, 2015, Volume 2018, Issue 4, p. CD009580
Background Fibrates are a class of drugs characterised by mainly lowering high triglyceride, raising high‐density lipoprotein (HDL) cholesterol, and lowering... 
Drugs | Cardiovascular Diseases | Costs | A. Cardiovascular Disease: Primary Prevention | Secondary Prevention | Insurance medicine | Cause of Death | Prevention | Coronary Disease | Cholesterol, HDL | Fibric Acids | Heart & circulation | A.1 Drugs | Anticholesteremic Agents | Stroke | Heart disease prevention | Clofibrate | A.1.2 Lipid Lowering | Randomized Controlled Trials as Topic | Fenofibrate | Myocardial Infarction | Bezafibrate | Hypertriglyceridemia | Gemfibrozil | Medicine General & Introductory Medical Sciences | EXTREMITY ARTERIAL-DISEASE | MEDICINE, GENERAL & INTERNAL | COMBINED HYPERLIPIDEMIA | MYOCARDIAL-INFARCTION | INTERVENTION TRIAL | COMBINATION THERAPY | DOUBLE-BLIND | CORONARY-HEART-DISEASE | TYPE-2 DIABETES-MELLITUS | FENOFIBRATE INTERVENTION | HIGH-DENSITY-LIPOPROTEIN | Hypertriglyceridemia - drug therapy | Fibric Acids - therapeutic use | Myocardial Infarction - mortality | Cardiovascular Diseases - prevention & control | Humans | Middle Aged | Gemfibrozil - therapeutic use | Male | Secondary Prevention - methods | Fenofibrate - therapeutic use | Coronary Disease - mortality | Fibric Acids - adverse effects | Clofibrate - therapeutic use | Aged, 80 and over | Cardiovascular Diseases - mortality | Adult | Female | Stroke - mortality | Bezafibrate - therapeutic use | Stroke - prevention & control | Anticholesteremic Agents - therapeutic use | Aged | Myocardial Infarction - prevention & control
Journal Article
Pharmacological Research, ISSN 1043-6618, 07/2008, Volume 58, Issue 1, p. 58
Activation of PPAR[alpha] by clofibrate has recently been shown to cause upregulation of the high-affinity carnitine transporter novel organic cation... 
Clofibrate | Levocarnitine
Journal Article
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 04/2016, Volume 1862, Issue 4, pp. 662 - 669
Journal Article
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 02/2018, Volume 93, Issue 2, pp. 375 - 389
Cardiovascular events are the leading cause of death in patients with chronic kidney disease (CKD), although the pathological mechanisms are poorly understood.... 
cardiovascular disease | chronic kidney disease | inflammation | renal pathology | PPAR-ALPHA | HEART-FAILURE | 5/6 NEPHRECTOMIZED RATS | CLOFIBRATE | GROWTH-FACTOR 23 | INTERSTITIAL FIBROSIS | UROLOGY & NEPHROLOGY | CHRONIC KIDNEY-DISEASE | EXPRESSION | WORSENING RENAL-FUNCTION | Cardio-Renal Syndrome - metabolism | Male | MicroRNAs - metabolism | Cardio-Renal Syndrome - pathology | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Hypertrophy, Left Ventricular - pathology | Ventricular Dysfunction, Left - pathology | Fatty Acids - metabolism | Clofibrate - pharmacology | Heart Ventricles - pathology | Disease Models, Animal | Signal Transduction | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Ventricular Function, Left - drug effects | Gene Expression Regulation | PPAR alpha - genetics | Cardio-Renal Syndrome - prevention & control | Rats, Sprague-Dawley | Ventricular Dysfunction, Left - prevention & control | Ventricular Dysfunction, Left - metabolism | Animals | Heart Ventricles - physiopathology | Fibrosis | PPAR alpha - agonists | Heart Ventricles - metabolism | MicroRNAs - genetics | PPAR alpha - metabolism | Cardio-Renal Syndrome - genetics | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Animal models | Nephrectomy | Therapeutic applications | miRNA | Chromosome 5 | Inflammation | Lipid metabolism | Ventricle | Peroxisome proliferator-activated receptors | Gene expression | Heart diseases | Hypertrophy
Journal Article
Toxicologic Pathology, ISSN 0192-6233, 6/2017, Volume 45, Issue 4, pp. 506 - 525
Journal Article