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PLoS Genetics, ISSN 1553-7390, 02/2011, Volume 7, Issue 2, pp. e1001308 - e1001308
Journal Article
Cancer Research, ISSN 0008-5472, 08/2013, Volume 73, Issue 16, pp. 5206 - 5217
Although androgen receptor (AR) pathway inhibitors prolong survival in castrate-resistant prostate cancer (CRPC), resistance rapidly develops and is often... 
BREAST-CANCER | SURVIVAL | TARGET | TUMOR-MODEL | CELLS | THERAPY | INDEPENDENCE | GENE | ANTIANDROGEN | ONCOLOGY | EXPRESSION | Transcription, Genetic - drug effects | Gene Silencing - drug effects | Molecular Chaperones - metabolism | Apoptosis - drug effects | Humans | Receptors, Androgen - metabolism | Apoptosis - genetics | Male | Tacrolimus Binding Proteins - metabolism | Prostatic Neoplasms, Castration-Resistant - genetics | Prostatic Neoplasms, Castration-Resistant - pathology | Proto-Oncogene Proteins c-akt - genetics | Clusterin - metabolism | Castration - methods | Prostate-Specific Antigen - metabolism | Stress, Physiological - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Gene Expression Regulation, Neoplastic - genetics | Phenylthiohydantoin - pharmacology | Prostate-Specific Antigen - genetics | Clusterin - genetics | Stress, Physiological - genetics | Molecular Chaperones - genetics | Signal Transduction - genetics | Prostatic Neoplasms, Castration-Resistant - drug therapy | Thionucleotides - pharmacology | Prostatic Neoplasms, Castration-Resistant - metabolism | Disease Progression | Phenylthiohydantoin - analogs & derivatives | Tacrolimus Binding Proteins - genetics | Proteasome Endopeptidase Complex - genetics | Animals | Receptors, Androgen - genetics | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Mitogen-Activated Protein Kinases - genetics | Mice | Proteasome Endopeptidase Complex - metabolism | Transcription, Genetic - genetics | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
American Journal of Medical Genetics Part B: Neuropsychiatric Genetics, ISSN 1552-4841, 04/2017, Volume 174, Issue 3, pp. 269 - 282
Journal Article
by Kauwe, John S. K and Cruchaga, Carlos and Karch, Celeste M and Sadler, Brooke and Lee, Mo and Mayo, Kevin and Latu, Wayne and Su'a, Manti and Fagan, Anne M and Holtzman, David M and Morris, John C and Goate, Alison M and Saradha, A and Abdi, Hervé and Abeliovich, Asa and Abellan van Kan, Gabor and Abner, Erin and Acharya, Deepa and Adams, Nicholas and Adler, Daniel and Agrusti, Antonella and Agyemang, Alex and Ahdidan, Jamila and Ahn, Jae Eun and Aisen, Paul and Aksu, Yaman and Al-Akhras, Mousa and Alarcon, Marcelo and Alberca, Roman and Alexander, Gene and Alexander, Daniel and Almeida, Fabio and Amlien, Inge and Anand, Shyam and Anew, Marilee and Angersbach, Steve and Anjum, Ayesha and Aoyama, Eiji and Arfanakis, Konstantinos and Armor, Tom and Arnold, Steven and Asatryan, Albert and Ashe-McNalley, Cody and Ashiga, Hirokazu and Assareh, Arezoo and Le Page, Aurelie and Avants, Brian and Avinash, Gopal and Awasthi, Sukrati and Ayan-Oshodi, Mosun and Babic, Tomislav and Baek, Young and Bagci, Ulas and Bai, Shuyang and Baird, Geoffrey and Baker, John and Banks, Sarah and Bard, Jonathan and Barnes, Josephine and Barret, Olivier and Bartlett, Jonathan and Bartzokis, George and Barua, Neil and Bauer, Corinna and Becker, James and Becker, J. Alex and Beckett, Laurel and Bednar, Martin and Beg, Mirza Faisal and Bek, Stephan and Belmokhtar, Nabil and Bernard, Charlotte and Bertram, Lars and Bhaskar, Uday and Biffi, Alessano and Bigler, Erin and Bilgic, Basar and Bishop, Courtney and Bittner, Daniel and Black, Ronald and Blennow, Kaj and Bogorodzki, Piotr and Bokde, Arun and Bonner-Jackson, Aaron and Boppana, Madhu and Bourgeat, Pierrick and Bowes, Mike and Bowman, Gene and Braskie, Meredith and Braunewell, Karl and Breitner, Joihn and Brewer, James and Brickman, Adam and Britschgi, Markus and Broadbent, Steve and Brogren, Jacob and Brooks, David and Browndyke, Jeffrey and Brunton, Simon and Buchert, Ralph and ... and Alzheimer's Dis Neuroimaging Initi and Alzheimer's Disease Neuroimaging Initiative and Sahlgrenska akademin and Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi and Göteborgs universitet and Gothenburg University and Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry and Sahlgrenska Academy
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, pp. e15918 - e15918
Recent genome-wide association studies of Alzheimer's disease (AD) have identified variants in BIN1, CLU, CR1 and PICALM that show replicable association with... 
IDENTIFIES VARIANTS | AMYLOID-BETA | DEPOSITION | IN-VIVO | SUSCEPTIBILITY | BIOLOGY | LOCI | APOE | GENOME-WIDE ASSOCIATION | REVEALS | Genetic Predisposition to Disease - genetics | Phosphorylation | Clusterin - genetics | Peptide Fragments - cerebrospinal fluid | Humans | Middle Aged | tau Proteins - metabolism | Male | tau Proteins - cerebrospinal fluid | Alzheimer Disease - cerebrospinal fluid | Serine - metabolism | tau Proteins - chemistry | Monomeric Clathrin Assembly Proteins - genetics | Tumor Suppressor Proteins - genetics | Adaptor Proteins, Signal Transducing - genetics | Aged, 80 and over | Amyloid beta-Peptides - cerebrospinal fluid | Female | Aged | Polymorphism, Single Nucleotide | Biomarkers - cerebrospinal fluid | Receptors, Complement 3b - genetics | Alzheimer Disease - genetics | Nuclear Proteins - genetics | Advertising executives | Medical research | Genomics | Medicine, Experimental | Development and progression | B cells | Single nucleotide polymorphisms | Biological markers | Alzheimer's disease | Physicians | Genes | Amino acids | Genomes | Biology | Single-nucleotide polymorphism | Cerebrospinal fluid | Proteins | Bioindicators | Neurological disorders | Eigen values | Threonine | Neurodegenerative diseases | Health risks | Metabolism | Apolipoproteins | Medicine | Studies | Genetic variance | Neurology | Hypotheses | Genotyping | Tau protein | Biomarkers | β-Amyloid | Gene mapping | Alzheimers disease | Psychiatry | Index Medicus | Peptide Fragments | Biological Markers | Complement 3b | Serine | tau Proteins | Single Nucleotide | Clusterin | 80 and over | Receptors | genetics | Amyloid beta-Peptides | Psykiatri | chemistry | Genetic Predisposition to Disease | Monomeric Clathrin Assembly Proteins | Alzheimer Disease | Adaptor Proteins | Nuclear Proteins | Signal Transducing | cerebrospinal fluid | metabolism | Tumor Suppressor Proteins | Polymorphism
Journal Article
Journal of Proteome Research, ISSN 1535-3893, 05/2015, Volume 14, Issue 5, pp. 2109 - 2120
Investigation of the retina proteome during hypomia-induced retinal neovascularization is valuable for understanding pathogenesis of retinopathy of prematurity... 
oxygen-induced retinopathy | peptide ion current areas | retinopathy of prematurity | label-free quantification | neovascularization | QUANTITATIVE PROTEOMICS | BRAIN-INJURY | BIOCHEMICAL RESEARCH METHODS | MASS-SPECTROMETRY | LABEL-FREE | NEWBORN RAT | FIBRILLARY ACIDIC PROTEIN | MOUSE MODEL | EXPRESSION | PREMATURITY | ENDOTHELIAL GROWTH-FACTOR | Glial Fibrillary Acidic Protein - genetics | Proteome - genetics | Humans | Eye Proteins - isolation & purification | rab GTP-Binding Proteins - isolation & purification | rhoA GTP-Binding Protein - metabolism | Glial Fibrillary Acidic Protein - metabolism | STAT1 Transcription Factor - metabolism | rho-Associated Kinases - metabolism | rhoA GTP-Binding Protein - isolation & purification | Retinal Degeneration - chemically induced | Disease Models, Animal | Animals, Newborn | Clusterin - genetics | Membrane Proteins - genetics | Retinal Degeneration - genetics | Rats | Annexin A3 - genetics | STAT1 Transcription Factor - genetics | Rats, Sprague-Dawley | Retina - chemistry | Annexin A3 - metabolism | Eye Proteins - metabolism | Glial Fibrillary Acidic Protein - isolation & purification | Retinopathy of Prematurity - metabolism | Retinal Degeneration - pathology | rho-Associated Kinases - isolation & purification | Proteome - metabolism | Retina - pathology | STAT1 Transcription Factor - isolation & purification | Retina - metabolism | rab GTP-Binding Proteins - genetics | rhoA GTP-Binding Protein - genetics | Retinopathy of Prematurity - genetics | Retinal Degeneration - metabolism | Clusterin - metabolism | Annexin A3 - isolation & purification | Membrane Proteins - metabolism | Microfilament Proteins - metabolism | Eye Proteins - genetics | Microfilament Proteins - genetics | Proteome - isolation & purification | rab GTP-Binding Proteins - metabolism | Membrane Proteins - isolation & purification | Oxygen | Microfilament Proteins - isolation & purification | Gene Expression Regulation | Clusterin - isolation & purification | rho-Associated Kinases - genetics | Animals | Neovascularization, Pathologic - genetics | Mass Spectrometry - methods | Retinopathy of Prematurity - pathology | Index Medicus
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2015, Volume 36, Issue 2, pp. 1221.e15 - 1221.e28
Abstract In this study, we have assessed the expression and splicing status of genes involved in the pathogenesis or affecting the risk of Alzheimer's disease... 
Neurology | Internal Medicine | Pathway analysis | Neurofibrillary pathology | Risk genes | Transcriptomics | Alzheimer's disease | COMMON VARIANTS | INCREASED EXPRESSION | BIOPSY | CLUSTERIN | NEUROSCIENCES | APOLIPOPROTEIN-E | GERIATRICS & GERONTOLOGY | TYPE-4 ALLELE | IDENTIFIES VARIANTS | AMYLOID-BETA | CD33 | GENOME-WIDE ASSOCIATION | Phosphorylation | Membrane Glycoproteins - metabolism | Humans | tau Proteins - metabolism | Male | Risk | Gene Expression Profiling | Alzheimer Disease - pathology | Brain - metabolism | tau Proteins - genetics | Amyloid beta-Peptides - genetics | Tumor Suppressor Proteins - genetics | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Female | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Gene Expression | Genetic Predisposition to Disease - genetics | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Nuclear Proteins - metabolism | Transcriptome - genetics | Neurofibrils - pathology | Membrane Glycoproteins - genetics | Amyloid Precursor Protein Secretases - metabolism | Adaptor Proteins, Signal Transducing - genetics | Alzheimer Disease - metabolism | Brain - pathology | Aged | Adaptor Proteins, Signal Transducing - metabolism | Alzheimer Disease - genetics | In Vitro Techniques | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Quantitative genetics | Brain | Gene expression | Genes | Protein-protein interactions | Index Medicus | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Journal of Neural Transmission, ISSN 0300-9564, 05/2013, Volume 120, Issue 5, pp. 807 - 812
Journal Article