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Journal Article
Learning & Memory, ISSN 1072-0502, 05/2017, Volume 24, Issue 5, pp. 199 - 209
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e115276
Reorganization of cytoskeleton via actin remodeling is a basic step of cell locomotion. Although cell migration of normal and cancer cells can be stimulated by... 
BREAST-CANCER | METASTASIS SUPPRESSOR | INVASION | MEDIATED ACTIN REORGANIZATION | ALPHABETA INTEGRIN | PROTEIN EZH2 | REPRESSION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | ADF/COFILIN | Caco-2 Cells | Colonic Neoplasms - therapy | Colonic Neoplasms - genetics | Polycomb Repressive Complex 2 - genetics | Actin Depolymerizing Factors - genetics | Humans | Integrin alpha2 - metabolism | Neoplasm Proteins - metabolism | Colonic Neoplasms - metabolism | Enhancer of Zeste Homolog 2 Protein | Actin Depolymerizing Factors - metabolism | Integrin alpha2 - genetics | Colonic Neoplasms - pathology | Neoplasm Proteins - genetics | Polycomb Repressive Complex 2 - metabolism | Cell Movement | Biotechnology | Chromatin | Phosphorylation | Laboratories | Serine | Colorectal carcinoma | Colorectal cancer | Biology | Metastasis | Kinases | Remodeling | Phosphatase | Cell adhesion & migration | Metastases | Proteins | Angiogenesis | Signal transduction | Colon cancer | Actin | Cofilin | Histone methyltransferase | Inhibition | Colon | Pharmaceutical sciences | Medical research | Immunoglobulins | Stability | Polymerization | Pharmacology | Breast cancer | Gene expression | Locomotion | Signaling | Molecular modelling | Cell lines | Epigenetics | Cytoskeleton | Prostate cancer | Cell migration | Tumors | Cancer
Journal Article
Oncogene, ISSN 0950-9232, 07/2014, Volume 33, Issue 27, pp. 3571 - 3582
Neurofibromatosis type 2 (NF2) is caused by mutations in the NF2 gene that encodes a tumor-suppressor protein called merlin. NF2 is characterized by formation... 
LIMK | cell proliferation | cell cycle | schwannomas | cytoskeleton dynamics | Neurofibromatosis | MERLIN | PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACTIN DYNAMICS | PROLIFERATION | CELL BIOLOGY | NF2 TUMOR-SUPPRESSOR | GENE | ONCOLOGY | RAC | GENETICS & HEREDITY | SCHWANN-CELLS | AURORA-A | COFILIN PHOSPHORYLATION | Neurofibromatosis 2 - drug therapy | Lim Kinases - deficiency | Neurofibromin 2 - genetics | Apoptosis - drug effects | Humans | Molecular Targeted Therapy | Phosphoproteins - metabolism | Aurora Kinase A - metabolism | Schwann Cells - cytology | Lim Kinases - metabolism | Actin Depolymerizing Factors - metabolism | G2 Phase Cell Cycle Checkpoints - drug effects | Neurofibromatosis 2 - metabolism | Phosphorylation - drug effects | Neurofibromin 2 - metabolism | Schwann Cells - drug effects | Cell Survival - drug effects | M Phase Cell Cycle Checkpoints - drug effects | Gene Silencing | Schwann Cells - pathology | Schwann Cells - metabolism | Neurofibromatosis 2 - enzymology | Neurofibromatosis 2 - pathology | Animals | Mitosis - drug effects | Lim Kinases - antagonists & inhibitors | Protein Kinase Inhibitors - therapeutic use | Lim Kinases - genetics | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Cytoskeleton | Genetic aspects | Research | Muscle proteins | Phosphotransferases | Health aspects | Cell growth | Genetic disorders | Mutation | Kinases | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0124355
Cucurbitacins, the natural triterpenoids possessing many biological activities, have been reported to suppress the mTORC1/p70S6K pathway and to induce... 
ACTIN AGGREGATION | CELLS | APOPTOSIS | ACTIVATED PROTEIN-KINASE | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | GROWTH | P62/SQSTM1 | CANCER | COFILIN | RNA, Small Interfering - genetics | AMP-Activated Protein Kinases - metabolism | Triterpenes - pharmacology | Microtubule-Associated Proteins - genetics | TOR Serine-Threonine Kinases - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Multiprotein Complexes - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Autophagy - drug effects | Autophagy-Related Protein-1 Homolog | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Lysosomes - metabolism | TOR Serine-Threonine Kinases - genetics | MCF-7 Cells | Phosphorylation - drug effects | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Lysosomes - drug effects | Cell Survival - drug effects | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Signal Transduction | Ribosomal Protein S6 Kinases, 70-kDa - antagonists & inhibitors | Lysosomal-Associated Membrane Protein 2 - genetics | Protein-Serine-Threonine Kinases - genetics | Ribosomal Protein S6 Kinases, 70-kDa - genetics | Microtubule-Associated Proteins - antagonists & inhibitors | Autophagy-Related Protein 5 | Cell Line, Tumor | HeLa Cells | Antineoplastic Agents, Phytogenic - pharmacology | AMP-Activated Protein Kinases - genetics | Lysosomal-Associated Membrane Protein 2 - metabolism | RNA, Small Interfering - metabolism | Molecular mechanics | Cytoskeleton | Research | Protein kinases | Analysis | Cucurbitaceae | TOR protein | Phosphorylation | Triterpenoids | Homeostasis | Activation | Kinases | Autophagy | Substrates | Recruitment | Signaling | Penicillin | Life sciences | Human behavior | Adapter proteins | Phagocytosis | Cancer
Journal Article
Journal of Neurology, Neurosurgery & Psychiatry, ISSN 0022-3050, 09/2014, Volume 85, Issue 9, pp. 1058 - 1060
To identify the molecular cause of the disease, either in one of the eight known nemaline myopathy genes not yet sequenced in the proband, or in a novel gene... 
SURGERY | DISRUPTION | GENE | PSYCHIATRY | CLINICAL NEUROLOGY | MUTATION | Base Sequence - genetics | Genetic Predisposition to Disease - genetics | Myopathies, Nemaline - genetics | Pedigree | Humans | Adult | Female | Infant | Male | Cofilin 2 - genetics | Sequence Deletion - genetics | Proteins | Disease | Biopsy | Genes | Ventilation | Mutation | Deoxyribonucleic acid--DNA
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2010, Volume 285, Issue 41, pp. 31829 - 31839
Stretch of the vascular wall stimulates smooth muscle hypertrophy by activating the MAPK and Rho/Rho kinase (ROK) pathways. We investigated the role of calcium... 
CARDIOVASCULAR DEVELOPMENT | MYOCARDIN | OPERATED CA2+ ENTRY | ACTIVATION | ACTIN POLYMERIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | GENE-EXPRESSION | CELL-PROLIFERATION | PROTEINS | TRANSCRIPTION FACTOR | Phosphorylation - physiology | Muscle, Smooth, Vascular - metabolism | Calcium - metabolism | Cell Membrane - genetics | Gene Knockdown Techniques | Lim Kinases - metabolism | RNA, Messenger - biosynthesis | Nuclear Proteins - biosynthesis | rho-Associated Kinases - metabolism | Trans-Activators - genetics | Female | Cell Membrane - metabolism | Trans-Activators - biosynthesis | Cofilin 2 - genetics | Nuclear Proteins - genetics | Cell Differentiation - physiology | Portal Vein - cytology | MEF2 Transcription Factors | Portal Vein - metabolism | Histone Deacetylases - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Myogenic Regulatory Factors - genetics | Proto-Oncogene Proteins c-jun - genetics | rho GTP-Binding Proteins - genetics | RNA, Messenger - genetics | Proto-Oncogene Proteins c-fos - metabolism | Gene Expression Regulation - physiology | Myogenic Regulatory Factors - metabolism | rho-Associated Kinases - genetics | Histone Deacetylases - metabolism | Muscle, Smooth, Vascular - cytology | Cofilin 2 - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Proto-Oncogene Proteins c-jun - metabolism | rho GTP-Binding Proteins - metabolism | Lim Kinases - genetics | Proto-Oncogene Proteins c-fos - genetics | Signal Transduction - physiology | Mice | Gene Regulation | Transcription Coactivators | MEF2 | Protein Synthesis | Signal Transduction | Calcium Channels | Actin | Rho | Cytoskeleton | siRNA | Myocardin | Smooth Muscle | Basic Medicine | Physiology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Fysiologi
Journal Article
Gene, ISSN 0378-1119, 09/2014, Volume 547, Issue 2, pp. 280 - 287
Journal Article