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1. Full Text Cerebrospinal fluid HIV escape associated with progressive neurologic dysfunction in patients on antiretroviral therapy with well controlled plasma viral load
by Peluso, Michael J and Ferretti, Francesca and Peterson, Julia and Lee, Evelyn and Fuchs, Dietmar and Boschini, Antonio and Gisslén, Magnus and Angoff, Nancy and Price, Richard W and Cinque, Paola and Spudich, Serena and Institute of Biomedicine, Department of Infectious Medicine and Sahlgrenska akademin and Institutionen för biomedicin, avdelningen för infektionssjukdomar and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
AIDS (London), ISSN 0269-9370, 09/2012, Volume 26, Issue 14, pp. 1765 - 1774
HIV pathogenesis | HIV | antiretroviral therapy | cerebrospinal fluid | neurologic manifestations | HIV drug resistance | central nervous system | Infectious Diseases | Immunology | Life Sciences & Biomedicine | Science & Technology | Virology | Antiviral agents | Immunopathology | Infectious diseases | Pharmacology. Drug treatments | Viral diseases | Viral diseases of the lymphoid tissue and the blood. Aids | Biological and medical sciences | Medical sciences | Human viral diseases | Immunodeficiencies. Immunoglobulinopathies | Immunodeficiencies | Antibiotics. Antiinfectious agents. Antiparasitic agents | AIDS Dementia Complex - etiology | HIV Seropositivity - complications | United States | Humans | Middle Aged | Blood-Brain Barrier - physiopathology | Male | Urban Population | Central Nervous System - immunology | Anti-HIV Agents - administration & dosage | Adult | Female | Blood-Brain Barrier - virology | Retrospective Studies | AIDS Dementia Complex - virology | Central Nervous System - virology | AIDS Dementia Complex - physiopathology | HIV-1 - metabolism | Neopterin - cerebrospinal fluid | Europe | HIV Seropositivity - physiopathology | Disease Progression | Blood-Brain Barrier - metabolism | Central Nervous System - physiopathology | RNA, Viral - drug effects | Viral Load - drug effects | HIV Seropositivity - virology | Index Medicus | AIDS/HIV | CSF | replication | infection | neurocognitive disorders | Immunologi inom det medicinska området | reverse-transcriptase sequence | neopterin | resistance | type-1 | encephalitis | human-immunodeficiency-virus | dementia | Immunology in the medical area
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2. Full Text Dopamine neuronal loss contributes to memory and reward dysfunction in a model of Alzheimer's disease
by Nobili, Annalisa and Latagliata, Emanuele Claudio and Viscomi, Maria Teresa and Cavallucci, Virve and Cutuli, Debora and Giacovazzo, Giacomo and Krashia, Paraskevi and Rizzo, Francesca Romana and Marino, Ramona and Federici, Mauro and De Bartolo, Paola and Aversa, Daniela and Dell'Acqua, Maria Concetta and Cordella, Alberto and Sancandi, Marco and Keller, Flavio and Petrosini, Laura and Puglisi-Allegra, Stefano and Mercuri, Nicola Biagio and Coccurello, Roberto and Berretta, Nicola and D'Amelio, Marcello
Nature communications, ISSN 2041-1723, 04/2017, Volume 8, Issue 1, pp. 14727 - 14727
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - pathology | Alzheimer Disease - complications | Dihydroxyphenylalanine - therapeutic use | Apoptosis - drug effects | Dopaminergic Neurons - pathology | Memory | Selegiline - pharmacology | Neuronal Plasticity - drug effects | Dendritic Spines - metabolism | Hippocampus - drug effects | Alzheimer Disease - pathology | Inflammation - complications | Dopaminergic Neurons - metabolism | Dihydroxyphenylalanine - pharmacology | Dopaminergic Neurons - drug effects | Cell Death - drug effects | Selegiline - therapeutic use | Disease Models, Animal | Food | Nucleus Accumbens - pathology | Nucleus Accumbens - physiopathology | Alzheimer Disease - physiopathology | Ventral Tegmental Area - drug effects | Plaque, Amyloid - pathology | Ventral Tegmental Area - physiopathology | Ventral Tegmental Area - pathology | Alzheimer Disease - drug therapy | Mice, Transgenic | Hippocampus - pathology | Nerve Degeneration - complications | Nerve Degeneration - pathology | Plaque, Amyloid - complications | Animals | Plaque, Amyloid - physiopathology | Reward | Hippocampus - physiopathology | Nerve Degeneration - drug therapy | Index Medicus
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3. Full Text Targeting Synaptic Dysfunction in Alzheimer’s Disease Therapy
by Nisticò, Robert and Pignatelli, Marco and Piccinin, Sonia and Mercuri, Nicola B and Collingridge, Graham
Molecular neurobiology, ISSN 0893-7648, 12/2012, Volume 46, Issue 3, pp. 572 - 587
Neurosciences | Alzheimer’s disease | Neurodegenerative | Long-term potentiation (LTP) | Neurobiology | Pharmacology | Cell Biology | Neurology | Biomedicine | Synaptic dysfunction | Synaptic plasticity | Amyloid | Peptide | Hippocampus | Alzheimer's disease | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neuroprotective Agents - therapeutic use | Synapses - drug effects | Alzheimer Disease - physiopathology | Animals | Neuroprotective Agents - pharmacology | Signal Transduction - drug effects | Humans | Immunotherapy | Alzheimer Disease - therapy | Alzheimer Disease - drug therapy | Synapses - pathology | Disease Models, Animal | Peptides | Alzheimers disease | Neurodegeneration | Index Medicus | Animal models | Translation | Neurodegenerative diseases | Long-term potentiation | Memory | Cognitive ability | Transgenic mice | Clinical trials | Nervous system | beta -Amyloid | Plasticity (hippocampal) | Electrophysiological recording | Plasticity (synaptic) | Reviews | Synaptic transmission | Evolution | Synapses
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4. Full Text Pharmacological and non-pharmacological interventions to improve cognitive dysfunction and functional ability in clinical depression – A systematic review
by Baune, Bernhard T and Renger, Lisa
Psychiatry research, ISSN 0165-1781, 2014, Volume 219, Issue 1, pp. 25 - 50
Psychiatry | Mood disorder | Cognitive therapy | Depression | Antidepressant treatment | Remediation therapy | Cognitive dysfunction | Cognitive impairment | Life Sciences & Biomedicine | Science & Technology | Miscellaneous | Psychology. Psychoanalysis. Psychiatry | Adult and adolescent clinical studies | Biological and medical sciences | Medical sciences | Neuropharmacology | Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer..., (alzheimer disease) | Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer | Psychopharmacology | Pharmacology. Drug treatments | Psychopathology. Psychiatry | Mood disorders | Behavior therapy. Cognitive therapy | Treatments | Cognitive Therapy - methods | Humans | Memory | Middle Aged | Serotonin Uptake Inhibitors - therapeutic use | Cognition | Activities of Daily Living | Antidepressive Agents - therapeutic use | Depression - drug therapy | Female | Cognition Disorders - therapy | Executive Function | Cognition Disorders - psychology | Serotonin uptake inhibitors | Cognition disorders | Depression, Mental | Antidepressants, Tricyclic | Index Medicus
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5. Full Text Androgen‐deprivation therapy, dementia, and cognitive dysfunction in men with prostate cancer: How much smoke and how much fire?
by McHugh, Deaglan J and Root, James C and Nelson, Christian J and Morris, Michael J
Cancer, ISSN 0008-543X, 04/2018, Volume 124, Issue 7, pp. 1326 - 1334
dementia | prostate cancer | cognitive dysfunction | Alzheimer's dementia | androgen deprivation | Life Sciences & Biomedicine | Oncology | Science & Technology | Dementia - pathology | Prognosis | Dementia - chemically induced | Humans | Risk Factors | Male | Cognitive Dysfunction - chemically induced | Cognitive Dysfunction - pathology | Androgen Antagonists - adverse effects | Prostatic Neoplasms - drug therapy | Therapy | Deprivation | Androgens | Toxicity | Cognitive ability | Dementia disorders | Smoke | Prostate cancer | Prostate | Cancer | Dementia | Metastases | Cognitive Dysfunction | Alzheimer’s Dementia | Prostate Cancer | Androgen Deprivation
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6. Full Text Astrocyte-derived CCL2 participates in surgery-induced cognitive dysfunction and neuroinflammation via evoking microglia activation
by Xu, Jiawen and Dong, Hongquan and Qian, Qingqing and Zhang, Xiang and Wang, Yiwei and Jin, Wenjie and Qian, Yanning
Behavioural brain research, ISSN 0166-4328, 08/2017, Volume 332, pp. 145 - 153
Neuroinflammation | CCL2 | Postoperative cognitive dysfunction | Astrocytes | CCR2 | Microglia | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Behavioral Sciences | Science & Technology | Inflammation - pathology | Microglia - metabolism | Neurons - pathology | Astrocytes - pathology | Postoperative Complications - metabolism | Male | Hippocampus - drug effects | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Receptors, CCR2 - antagonists & inhibitors | Tibial Fractures - metabolism | Postoperative Complications - psychology | Tibial Fractures - surgery | Inflammation - metabolism | Inflammation - drug therapy | Microglia - pathology | Chemokine CCL2 - metabolism | Neurons - metabolism | Postoperative Complications - drug therapy | Neurons - drug effects | Disease Models, Animal | Postoperative Complications - pathology | Cognitive Dysfunction - drug therapy | Astrocytes - drug effects | Microglia - drug effects | Cognitive Dysfunction - metabolism | Hippocampus - pathology | Cognitive Dysfunction - pathology | Random Allocation | Cognitive Dysfunction - etiology | Inflammation - etiology | Rats, Sprague-Dawley | Hippocampus - metabolism | Receptors, CCR2 - metabolism | Animals | Signal Transduction - drug effects | Benzoxazines - pharmacology | Tibial Fractures - complications | Tibial Fractures - pathology | Nootropic Agents - pharmacology | Spiro Compounds - pharmacology | Astrocytes - metabolism | Phosphates | Brain | Medical research | Surgery | Medicine, Experimental | Cognition disorders | Injuries | Index Medicus
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7. Full Text Dietary salt promotes neurovascular and cognitive dysfunction through a gut-initiated TH17 response
by Faraco, Giuseppe and Brea, David and Garcia-Bonilla, Lidia and Wang, Gang and Racchumi, Gianfranco and Chang, Haejoo and Buendia, Izaskun and Santisteban, Monica M and Segarra, Steven G and Koizumi, Kenzo and Sugiyama, Yukio and Murphy, Michelle and Voss, Henning and Anrather, Joseph and Iadecola, Costantino
Nature neuroscience, ISSN 1097-6256, 02/2018, Volume 21, Issue 2, pp. 240 - 249
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Antihypertensive Agents - pharmacology | Cerebrovascular Disorders - drug therapy | Intestine, Small - pathology | Cognition Disorders - chemically induced | Homeodomain Proteins - metabolism | Male | Th17 Cells - drug effects | Cell Polarity - drug effects | Phosphorylation - drug effects | Neurovascular Coupling - drug effects | Disease Models, Animal | Amides - pharmacology | Interleukin-17 - blood | Interleukin-17 - administration & dosage | Cerebrovascular Disorders - chemically induced | Acetylcholine - pharmacology | Cerebrovascular Circulation - drug effects | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Interleukin-17 - genetics | Mice, Transgenic | Homeodomain Proteins - genetics | Gene Expression Regulation - drug effects | Interleukin-17 - metabolism | Animals | Cell Differentiation - drug effects | Sodium Chloride, Dietary - toxicity | Mice | Pyridines - pharmacology | Salt | Gastrointestinal system | Neurovascular diseases | Cognition disorders | T cells | Health aspects | Risk factors | Brain | Phosphorylation | Salts | Interleukin | Cognitive ability | Helper cells | Impairment | Lymphocytes T | Small intestine | Vascular diseases | Cerebral blood flow | Rodents | Dementia disorders | Digestive tract | Cerebrovascular diseases | Immune system | Immune response | Digestive system | Health risks | Interleukin 17 | Nitric-oxide synthase | Endothelial cells | Rho-associated kinase | Blood flow | Diet | Nitric oxide | Habits | Index Medicus
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