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1. Full Text Effects of micropitted/nanotubular titania topographies on bone mesenchymal stem cell osteogenic differentiation
by Zhao, Lingzhou|Liu, Li|Wu, Zhifen|Zhang, Yumei|Chu, Paul K
Biomaterials, ISSN 0142-9612, 2011, Volume 33, Issue 9, pp. 2629 - 2641
Abstract Micro/nanotopographical modification of biomaterials constitutes a promising approach to direct stem cell osteogenic differentiation to promote... 
Advanced Basic Science | Dentistry | Osteogenic differentiation | Hierarchical topography | Cell spread | Mesenchymal stem cells | Titania nanotubes | MATERIALS SCIENCE, BIOMATERIALS | TIO2 NANOTUBES | ENGINEERING, BIOMEDICAL | PROLIFERATION | FATE | ADHESION | IN-VIVO | GROWTH | GENE-EXPRESSION | ALTERED NANOTUBE DIMENSION | LINEAGE | SURFACES | Mesenchymal Stromal Cells - enzymology | Titanium - pharmacology | Bone Marrow Cells - enzymology | Cell Count | Alkaline Phosphatase - metabolism | Titanium - chemistry | Extracellular Matrix - metabolism | Sincalide - metabolism | Nanotubes - chemistry | Cell Differentiation - genetics | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Bone Marrow Cells - drug effects | Osteogenesis - genetics | Nanotubes - ultrastructure | Mesenchymal Stromal Cells - drug effects | Calcification, Physiologic - drug effects | Bone Marrow Cells - cytology | Extracellular Matrix - drug effects | Osteogenesis - drug effects | Rats | Cell Adhesion - drug effects | Collagen - secretion | Rats, Sprague-Dawley | Bone Marrow Cells - ultrastructure | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Animals | Cell Differentiation - drug effects | Staining and Labeling | Cell Proliferation - drug effects | Cell Cycle - drug effects | Biological products | Phosphatases | Analysis | Collagen | Genes | Stem cells | Fluorescence | Gene expression | Orthodontics | Index Medicus
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2. Full Text FGF‐2 enhances the mitotic and chondrogenic potentials of human adult bone marrow‐derived mesenchymal stem cells
by Solchaga, Luis A and Penick, Kitsie and Porter, John D and Goldberg, Victor M and Caplan, Arnold I and Welter, Jean F
Journal of Cellular Physiology, ISSN 0021-9541, 05/2005, Volume 203, Issue 2, pp. 398 - 409
Human mesenchymal stem cells (hMSCs) expanded with and without fibroblast growth factor (FGF) supplementation were compared with respect to their proliferation... 
PROGENITOR CELLS | CROSS-TALK | PROTEIN-KINASE-C | IN-VITRO | PHYSIOLOGY | ATDC5 CELLS | DEFICIENT MDX MICE | STROMAL CELLS | GENE-EXPRESSION SIGNATURES | CHONDROCYTE DIFFERENTIATION | N-CADHERIN | CELL BIOLOGY | Chondrocytes - cytology | Chondrogenesis - drug effects | Age Factors | Oligonucleotide Array Sequence Analysis | Humans | Fibroblast Growth Factor 2 - pharmacology | Bone Marrow Cells - physiology | Gene Expression Profiling | Cell Culture Techniques - methods | Proteoglycans - drug effects | Cell Differentiation - genetics | Chondrocytes - drug effects | Mesenchymal Stromal Cells - cytology | Chondrocytes - physiology | Collagen - drug effects | Up-Regulation - physiology | Bone Marrow Cells - drug effects | Mesenchymal Stromal Cells - physiology | Mesenchymal Stromal Cells - drug effects | Tissue Engineering - methods | Bone Marrow Cells - cytology | Gene Expression Regulation - genetics | Extracellular Matrix Proteins - genetics | Cells, Cultured | Proteoglycans - metabolism | Down-Regulation - drug effects | Extracellular Matrix Proteins - drug effects | Down-Regulation - physiology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Chondrogenesis - physiology | Collagen - metabolism | Mitosis - drug effects | Mitosis - physiology | Signal Transduction - drug effects | Cell Differentiation - drug effects | Signal Transduction - physiology | Cell Proliferation - drug effects | Index Medicus
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3. Full Text A comprehensive panel of three-dimensional models for studies of prostate cancer growth, invasion and drug responses
by Härmä, Ville and Virtanen, Johannes and Mäkelä, Rami and Happonen, Antti and Mpindi, John-Patrick and Knuuttila, Matias and Kohonen, Pekka and Lötjönen, Jyrki and Kallioniemi, Olli and Nees, Matthias
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 5, pp. e10431 - e10431
Prostate epithelial cells from both normal and cancer tissues, grown in three-dimensional (3D) culture as spheroids, represent promising in vitro models for... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | GENE-EXPRESSION SIGNATURE | STEM-CELLS | MAMMARY EPITHELIA | METASTASIS | BIOLOGY | TUMOR-CELL INVASION | DIFFERENTIATION | CULTURE MODEL | LINES | Laminin - pharmacology | Epithelial Cells - drug effects | Humans | Mesoderm - drug effects | Spheroids, Cellular - pathology | Male | Antineoplastic Agents - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Spheroids, Cellular - enzymology | Neoplasm Proteins - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | RNA, Messenger - metabolism | Prostate - pathology | Prostatic Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Prostate - drug effects | Antineoplastic Agents - pharmacology | Collagen - pharmacology | Spheroids, Cellular - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Principal Component Analysis | Prostatic Neoplasms - drug therapy | Epithelium - drug effects | Prostatic Neoplasms - pathology | Epithelium - pathology | Neoplasm Invasiveness | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | RNA, Messenger - genetics | Epithelial Cells - pathology | Cell Shape - drug effects | Phenotype | Proteoglycans - pharmacology | Signal Transduction - drug effects | Models, Biological | Prostatic Neoplasms - enzymology | Cell Proliferation - drug effects | TOR Serine-Threonine Kinases | Cell Transformation, Neoplastic - pathology | Mesoderm - pathology | Drug Combinations | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Epigenetic inheritance | Growth | Oncology, Experimental | Genes | Research | Gene expression | Ionizing radiation | Stem cells | Physiological aspects | Models | Drug discovery | Drug therapy | Prostate cancer | Integrins | Cancer | Cell culture | Biotechnology | Transformation | Motility | Leukocyte migration | Mesenchyme | Epithelial cells | Homeostasis | AKT protein | Metastasis | Drug resistance | Tissues | Ovarian cancer | Cell adhesion & migration | Metastases | Rodents | Fibroblasts | Extracellular matrix | Basal lamina | Lipid metabolism | Medical research | Invasiveness | Phenotypic plasticity | Cultures | Tumor cell lines | Metabolism | Spheroids | 1-Phosphatidylinositol 3-kinase | Signaling | Interferon | Three dimensional models | Prostate | Cell migration | Index Medicus
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4. Full Text Nilotinib reduces muscle fibrosis in chronic muscle injury by promoting TNF-mediated apoptosis of fibro/adipogenic progenitors
by Lemos, Dario R and Babaeijandaghi, Farshad and Low, Marcela and Chang, Chih-Kai and Lee, Sunny T and Fiore, Daniela and Zhang, Regan-Heng and Natarajan, Anuradha and Nedospasov, Sergei A and Rossi, Fabio M. V
Nature Medicine, ISSN 1078-8956, 07/2015, Volume 21, Issue 7, pp. 786 - 794
Depending on the inflammatory milieu, injury can result either in a tissue's complete regeneration or in its degeneration and fibrosis, the latter of which... 
MDX | MEDICINE, RESEARCH & EXPERIMENTAL | IFN-GAMMA | MACROPHAGES | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-ALPHA | CELL BIOLOGY | DYSTROPHIC MUSCLE | SKELETAL-MUSCLE | TUMOR-NECROSIS-FACTOR | INFLAMMATION | EXPRESSION | Apoptosis - drug effects | Monocytes - cytology | Adipogenesis - drug effects | Cell Count | Transforming Growth Factor beta1 - metabolism | Male | Muscle, Skeletal - injuries | Stem Cells - cytology | RNA, Messenger - metabolism | Flow Cytometry | Muscle, Skeletal - drug effects | Muscular Diseases - drug therapy | Mice, Inbred mdx | Female | Cell Survival - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | Muscular Diseases - pathology | Macrophages - cytology | Monocytes - drug effects | Collagen - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Receptors, CCR2 - metabolism | Regeneration - drug effects | Animals | Signal Transduction - drug effects | Pyrimidines - therapeutic use | Fibrosis | Elapid Venoms | Macrophages - drug effects | Muscle, Skeletal - pathology | Receptors, CCR2 - deficiency | Chronic Disease | Tissue | Muscular system | Apoptosis | Index Medicus
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5. Full Text DOCK8 regulates lymphocyte shape integrity for skin antiviral immunity
by Zhang, Qian and Dove, Christopher G and Hor, Jyh Liang and Murdock, Heardley M and Strauss-Albee, Dara M and Garcia, Jordan A and Mandl, Judith N and Grodick, Rachael A and Jing, Huie and Chandler-Brown, Devon B and Lenardo, Timothy E and Crawford, Greg and Matthews, Helen F and Freeman, Alexandra F and Cornall, Richard J and Germain, Ronald N and Mueller, Scott N and Su, Helen C
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 13, pp. 2549 - 2566
DOCK8 mutations result in an inherited combined immunodeficiency characterized by increased susceptibility to skin and other infections. We show that when... 
MEDICINE, RESEARCH & EXPERIMENTAL | HERPES-SIMPLEX-VIRUS | IN-VITRO | DENDRITIC CELLS | LEUKOCYTE MIGRATION | VARICELLA-ZOSTER-VIRUS | RM CELLS | TISSUE | IMMUNOLOGY | ALDRICH-SYNDROME PROTEIN | MEMORY T-CELLS | IMMUNOLOGICAL SYNAPSES | Apoptosis - drug effects | Humans | Cell Shape - immunology | Male | cdc42 GTP-Binding Protein - metabolism | Killer Cells, Natural - pathology | Immunologic Memory - drug effects | Immunity - drug effects | Cattle | Cell Nucleus - pathology | Guanine Nucleotide Exchange Factors - metabolism | T-Lymphocytes - drug effects | Killer Cells, Natural - immunology | Female | Collagen - pharmacology | T-Lymphocytes - pathology | Skin - virology | Guanine Nucleotide Exchange Factors - deficiency | Skin - pathology | Skin - immunology | Mice, Inbred C57BL | Chemotaxis - drug effects | Cell Adhesion - drug effects | p21-Activated Kinases - metabolism | Cell Shape - drug effects | Animals | Signal Transduction - drug effects | Chemokine CXCL12 - pharmacology | Cytoskeleton - metabolism | T-Lymphocytes - immunology | Killer Cells, Natural - drug effects | Mice | Cell Nucleus - drug effects | Cytoskeleton - drug effects | Skin - drug effects | Index Medicus
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6. Full Text Myogenic Progenitor Cells Control Extracellular Matrix Production by Fibroblasts during Skeletal Muscle Hypertrophy
by Fry, Christopher S and Kirby, Tyler J and Kosmac, Kate and McCarthy, John J and Peterson, Charlotte A
Cell Stem Cell, ISSN 1934-5909, 01/2017, Volume 20, Issue 1, pp. 56 - 69
Satellite cells, the predominant stem cell population in adult skeletal muscle, are activated in response to hypertrophic stimuli and give rise to myogenic... 
hypertrophy | Pax7 | exosomes | muscle | collagen | microRNA | fibrosis | muscle progenitor cells | extracellular matrix | satellite cells | FIBROSIS | REGENERATION | GENE-EXPRESSION | EXOSOMES | SELF-RENEWAL | DUCHENNE MUSCULAR-DYSTROPHY | MIRNAS | SATELLITE CELL | DIFFERENTIATION | CONNECTIVE-TISSUE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Exosomes - drug effects | Exosomes - metabolism | NIH 3T3 Cells | Cell Survival - genetics | Extracellular Matrix - metabolism | MicroRNAs - metabolism | PAX7 Transcription Factor - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Stem Cells - metabolism | Gene Knockdown Techniques | Cell Differentiation - genetics | Extracellular Matrix - genetics | Cell Nucleus - metabolism | Gene Deletion | Collagen - genetics | Fibroblasts - metabolism | Cell Survival - drug effects | Ribonuclease III - metabolism | Extracellular Matrix - drug effects | Satellite Cells, Skeletal Muscle - metabolism | Down-Regulation - drug effects | Fibroblasts - pathology | Down-Regulation - genetics | Collagen - metabolism | Satellite Cells, Skeletal Muscle - drug effects | Animals | Carrier Proteins - metabolism | Muscle Development - drug effects | Cell Differentiation - drug effects | Models, Biological | Fibroblasts - drug effects | Muscle Fibers, Skeletal - pathology | Tamoxifen - pharmacology | Muscle Development - genetics | Stem Cells - drug effects | Mice | MicroRNAs - genetics | Muscle, Skeletal - pathology | Cell Nucleus - drug effects | Hypertrophy | Index Medicus | miRNA | Dicer | Muscle extracellular matrix
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7. Full Text Antimicrobial Efficacy of Riboflavin/UVA Combination (365 nm) In Vitro for Bacterial and Fungal Isolates: A Potential New Treatment for Infectious Keratitis
by Martins, Suy Anne R and Combs, Juan Castro and Noguera, Guillermo and Camacho, Walter and Wittmann, Priscila and Walther, Rhonda and Cano, Marisol and Dick, James and Behrens, Ashley
Investigative Ophthalmology & Visual Science, ISSN 0146-0404, 08/2008, Volume 49, Issue 8, pp. 3402 - 3408
PURPOSE. To demonstrate the antimicrobial properties of riboflavin/ UVA (365 nm) against common pathogens. METHODS. One group of bacteria ( Pseudomonas... 
INACTIVATION | CORNEAL COLLAGEN | VANCOMYCIN | COLLAGEN CROSS-LINKING | SUSCEPTIBILITY | OPHTHALMOLOGY | MICROBIAL KERATITIS | RESISTANT STAPHYLOCOCCUS-AUREUS | ULTRAVIOLET-RADIATION | CIPROFLOXACIN | KERATOCONUS | Staphylococcus aureus - radiation effects | Staphylococcus epidermidis - drug effects | Drug Resistance, Multiple | Bacteria - drug effects | Drug Resistance, Bacterial | Colony Count, Microbial | Flavin Mononucleotide - pharmacology | Photosensitizing Agents - pharmacology | Candida albicans - drug effects | Ultraviolet Rays | Corneal Ulcer - radiotherapy | Corneal Ulcer - microbiology | Fungi - radiation effects | Staphylococcus epidermidis - radiation effects | Streptococcus pneumoniae - drug effects | Fungi - drug effects | Ultraviolet Therapy | Pseudomonas aeruginosa - radiation effects | Combined Modality Therapy | Pseudomonas aeruginosa - drug effects | Candida albicans - radiation effects | Corneal Ulcer - drug therapy | Bacteria - radiation effects | Staphylococcus aureus - drug effects | Streptococcus pneumoniae - radiation effects | Index Medicus
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8. Full Text The promotion of bone regeneration by nanofibrous hydroxyapatite/chitosan scaffolds by effects on integrin-BMP/Smad signaling pathway in BMSCs
by Liu, Huanhuan|Peng, Hongju|Wu, Yan|Zhang, Can|Cai, Youzhi|Xu, Guowei|Li, Qin|Chen, Xiao|Ji, Junfeng|Zhang, Yanzhong|OuYang, Hong Wei
Biomaterials, ISSN 0142-9612, 2013, Volume 34, Issue 18, pp. 4404 - 4417
Abstract In bone tissue engineering, a combination of biomimetic nanofibrous scaffolds with renewable stem cells has recently emerged as a new strategy for... 
Advanced Basic Science | Dentistry | Cell proliferation | BMP signaling pathway | Integrin | Nanofibrous hydroxyapatite/chitosan | Cell adhesion | Mesenchymal stem cells | MATRIX | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | NETWORKS | INDUCTION | MESENCHYMAL STEM-CELLS | NANOCOMPOSITES | REPAIR | OSTEOBLASTS | DIFFERENTIATION | PROTEINS | CALVARIAL DEFECTS | Bone Regeneration - genetics | Alkaline Phosphatase - metabolism | Integrins - genetics | Integrins - metabolism | Tissue Scaffolds - chemistry | Extracellular Matrix - genetics | Bone Morphogenetic Proteins - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Skull - pathology | Durapatite - pharmacology | Chitosan - pharmacology | Bone Marrow Cells - drug effects | Female | Bone Morphogenetic Proteins - genetics | Osteogenesis - genetics | Nanofibers - chemistry | Bone Regeneration - drug effects | Mesenchymal Stromal Cells - drug effects | Nanofibers - ultrastructure | Implants, Experimental | Bone Marrow Cells - cytology | Cell Separation | Osteocalcin - metabolism | Extracellular Matrix - drug effects | Osteogenesis - drug effects | Membranes, Artificial | Mesenchymal Stromal Cells - metabolism | Rats | Smad Proteins - genetics | Signal Transduction - genetics | Rats, Sprague-Dawley | Bone Marrow Cells - ultrastructure | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Radiography | Animals | Signal Transduction - drug effects | Skull - diagnostic imaging | Cell Proliferation - drug effects | Smad Proteins - metabolism | Tissue engineering | Collagen | Stem cells | Integrins | Index Medicus
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9. Full Text Activation of ATM Chk1 by curcumin causes cell cycle arrest and apoptosis in human pancreatic cancer cells
by Sahu, R.P and Batra, S and Srivastava, S.K
British Journal of Cancer, ISSN 0007-0920, 05/2009, Volume 100, Issue 9, pp. 1425 - 1433
Curcumin has been shown to inhibit the growth of various types of cancer cells; however, at concentrations much above the clinically achievable levels in... 
Curcumin | Chk1 | G2/M arrest | DNA damage | Pancreatic cancer | CARCINOMA-CELLS | DNA-DAMAGE | DOWN-REGULATION | REGULATED GENE-PRODUCTS | FACTOR-KAPPA-B | BENZYL ISOTHIOCYANATE | I CLINICAL-TRIAL | pancreatic cancer | ONCOLOGY | PROSTATE-CANCER | CHEMOPREVENTIVE AGENT | curcumin | Cell Cycle Proteins - drug effects | Protein Kinases - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Protein Kinases - genetics | Gene Silencing - drug effects | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Collagen Type XI - metabolism | DNA-Binding Proteins - metabolism | Pancreatic Neoplasms - drug therapy | Transfection | Tumor Suppressor Proteins - genetics | Caspase 3 - drug effects | Cell Cycle Proteins - genetics | Gene Expression Regulation, Neoplastic - drug effects | Protein-Serine-Threonine Kinases - metabolism | DNA Damage - drug effects | Protein Kinases - drug effects | Tumor Suppressor Proteins - metabolism | Collagen Type XI - drug effects | Pancreatic Neoplasms - pathology | Cell Cycle Proteins - metabolism | Curcumin - pharmacology | DNA-Binding Proteins - drug effects | Pancreatic Neoplasms - enzymology | Protein-Serine-Threonine Kinases - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Cell Division - drug effects | Protein-Serine-Threonine Kinases - drug effects | Cell Line, Tumor | Checkpoint Kinase 1 | Tumor Suppressor Proteins - drug effects | Cell Cycle - drug effects | Index Medicus | Translational Therapeutics | M arrest
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10. Full Text Enhanced osteointegration on tantalum-implanted polyetheretherketone surface with bone-like elastic modulus
by Lu, Tao|Wen, Jin|Qian, Shi|Cao, Huiliang|Ning, Congqin|Pan, Xiaoxia|Jiang, Xinquan|Liu, Xuanyong|Chu, Paul K
Biomaterials, ISSN 0142-9612, 2015, Volume 51, pp. 173 - 183
Abstract Polyetheretherketone (PEEK) possesses a similar elastic modulus as bones but yet suffers from bio-inertness and poor osteogenesis. In this work,... 
Advanced Basic Science | Dentistry | Polyetheretherketone | Elastic modulus | Tantalum | Osteointegration | Plasma immersion ion implantation | STEM-CELLS | MATERIALS SCIENCE, BIOMATERIALS | ETHER-ETHER-KETONE | HUMAN OSTEOBLASTS | ENGINEERING, BIOMEDICAL | BIOACTIVE MATERIALS | MECHANICAL-PROPERTIES | METALLIC BIOMATERIALS | IN-VITRO | POROUS TANTALUM | IMMERSION ION-IMPLANTATION | NANOCOMPOSITE COATINGS | Mesenchymal Stromal Cells - enzymology | Bone Marrow Cells - enzymology | Alkaline Phosphatase - metabolism | Extracellular Matrix - metabolism | Fluorescent Dyes - metabolism | Elastic Modulus - drug effects | X-Ray Microtomography | Bone and Bones - drug effects | Mesenchymal Stromal Cells - cytology | Bone and Bones - diagnostic imaging | Surface Properties | Tantalum - pharmacology | Bone Marrow Cells - drug effects | Real-Time Polymerase Chain Reaction | Osteogenesis - genetics | Osseointegration - drug effects | Polyethylene Glycols - pharmacology | Ketones - pharmacology | Mesenchymal Stromal Cells - drug effects | Photoelectron Spectroscopy | Bone and Bones - physiology | Calcification, Physiologic - drug effects | Bone Marrow Cells - cytology | Extracellular Matrix - drug effects | Osteogenesis - drug effects | Cells, Cultured | Prostheses and Implants | Rats | Cell Adhesion - drug effects | Collagen - secretion | Gene Expression Regulation - drug effects | Animals | Cell Proliferation - drug effects | Collagen | Analysis | Stem cells | Medical colleges | Phosphatases | Ceramics | Implant dentures | Ceramic materials | Index Medicus | Biomedical materials | Dental materials | Biocompatibility | Bones | Modulus of elasticity | Polyetheretherketones
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