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Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1589 - 1597
Uncontrolled extracellular matrix production by fibroblasts in response to tissue injury contributes to fibrotic diseases, such as idiopathic pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | RESPONSES | PHOSPHATASE | INJURY | TENSIN HOMOLOG | MECHANISMS | GROWTH-FACTOR | IMMUNOLOGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | CANCER | MICRORNA | Oligonucleotides - genetics | Pulmonary Fibrosis - therapy | Idiopathic Pulmonary Fibrosis - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Actins - metabolism | MicroRNAs - metabolism | Pulmonary Fibrosis - genetics | Idiopathic Pulmonary Fibrosis - metabolism | Actins - genetics | Antisense Elements (Genetics) - therapeutic use | Collagen - genetics | Lung - metabolism | Phosphorylation - drug effects | Smad7 Protein - genetics | Extracellular Matrix Proteins - metabolism | Transforming Growth Factor beta1 - pharmacology | Fibroblasts - metabolism | Smad7 Protein - metabolism | Cell Line | Lung - pathology | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Smad2 Protein - metabolism | Mice, Transgenic | Pulmonary Fibrosis - pathology | Transforming Growth Factor beta1 - genetics | Fibroblasts - pathology | Fibronectins - metabolism | Collagen - metabolism | Animals | Fibroblasts - drug effects | Antisense Elements (Genetics) - genetics | Idiopathic Pulmonary Fibrosis - pathology | Pulmonary Fibrosis - chemically induced | Bleomycin - pharmacology | Fibronectins - genetics | Mice | MicroRNAs - genetics | Index Medicus | Brief Definitive Report
Journal Article
PL o S Genetics (Online), ISSN 1553-7390, 09/2012, Volume 8, Issue 9, pp. e1002932 - e1002932
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 01/2013, Volume 114, Issue 1, pp. 131 - 147
We have previously reported chronic low-intensity interval exercise training attenuates fibrosis, impaired cardiac mitochondrial function, and coronary... 
Diastolic heart failure | Exercise | Coronary flow | Fibrosis | MVO | Speckle tracking | Hypertrophic signaling | Protein Kinases - genetics | Connectin | Heart - physiology | Natriuretic Peptide, Brain - metabolism | Ventricular Function, Left - genetics | Heart Failure - physiopathology | Extracellular Matrix - metabolism | Male | Oxygen Consumption - physiology | Tissue Inhibitor of Metalloproteinases - genetics | Hypertrophy, Left Ventricular - genetics | Swine | Myocardium - metabolism | Ventricular Remodeling - genetics | Sarcomeres - physiology | Proto-Oncogene Proteins c-akt - metabolism | Natriuretic Peptide, Brain - genetics | Fibrosis - physiopathology | Matrix Metalloproteinase 2 - metabolism | Sarcomeres - metabolism | Diastole - genetics | Heart Failure - genetics | Heart Failure - metabolism | Arterial Pressure - physiology | Diastole - physiology | Muscle Proteins - genetics | Matrix Metalloproteinase 2 - genetics | MAP Kinase Kinase 4 - genetics | Hypertrophy, Left Ventricular - physiopathology | Regional Blood Flow - physiology | Protein Kinases - metabolism | Regional Blood Flow - genetics | Myocardial Contraction - physiology | Collagen Type III - metabolism | Fibrosis - metabolism | Oxygen - metabolism | Proto-Oncogene Proteins c-akt - genetics | Extracellular Matrix - genetics | Myocardial Contraction - genetics | Arterial Pressure - genetics | MAP Kinase Kinase 4 - metabolism | Sarcomeres - genetics | Muscle Proteins - metabolism | Ventricular Function, Left - physiology | Citrate (si)-Synthase - metabolism | Heart Failure - rehabilitation | Fibrosis - genetics | Hypertrophy, Left Ventricular - metabolism | RNA, Messenger - genetics | Collagen Type III - genetics | Ventricular Remodeling - physiology | Physical Conditioning, Animal - physiology | Tissue Inhibitor of Metalloproteinases - metabolism | Animals | Oxygen Consumption - genetics | Citrate (si)-Synthase - genetics | Index Medicus | speckle tracking | coronary flow | hypertrophic signaling | exercise | fibrosis | diastolic heart failure | MV̇o2
Journal Article
Journal Article
Journal of Dental Research, ISSN 0022-0345, 7/2015, Volume 94, Issue 7, pp. 913 - 920
This study evaluated associations between craniofacial candidate genes and skeletal variation in patients with malocclusion. Lateral cephalometric radiographs... 
angle class III | cephalometry | angle class II | genetics | polymorphisms | association study | CLEFT-LIP | IDENTIFICATION | CRANIOSYNOSTOSIS | TWIST | SNAIL FAMILY GENES | DENTISTRY, ORAL SURGERY & MEDICINE | MANDIBULAR PROGNATHISM | MICE | MUTATIONS | LINKAGE | GENOME-WIDE ASSOCIATION | Cephalometry - methods | Humans | Mandible - pathology | Middle Aged | PAX7 Transcription Factor - genetics | Malocclusion, Angle Class III - genetics | Young Adult | Anatomic Landmarks - pathology | Collagen Type I - genetics | Malocclusion, Angle Class II - pathology | Adult | Malocclusion, Angle Class I - pathology | Nuclear Proteins - genetics | Snail Family Transcription Factors | Malocclusion, Angle Class I - genetics | Genetic Association Studies | PAX5 Transcription Factor - genetics | Open Bite - genetics | Genotype | Image Processing, Computer-Assisted - methods | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Overbite - genetics | T-Box Domain Proteins - genetics | Malocclusion, Angle Class III - pathology | Phenotype | Malocclusion, Angle Class II - genetics | Adolescent | Twist-Related Protein 1 - genetics | Myosin Type I | Polymorphism, Single Nucleotide - genetics | Zinc Fingers - genetics | Aged | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Index Medicus | Dentistry | Research Reports
Journal Article
International Journal of Dermatology, ISSN 0011-9059, 12/2017, Volume 56, Issue 12, pp. 1406 - 1413
BackgroundGenodermatoses represent genetic anomalies of skin tissues including hair follicles, sebaceous glands, eccrine glands, nails, and teeth. Ten... 
KINDLER-SYNDROME | KINDLIN-1 | PROTEIN | MUTATION | VII COLLAGEN | SPECTRUM | DYSTROPHIC EPIDERMOLYSIS-BULLOSA | IDENTIFICATION | DERMATOLOGY | Rare Diseases - genetics | 1-Acylglycerol-3-Phosphate O-Acyltransferase - genetics | Endonucleases - genetics | Lipid Metabolism, Inborn Errors - genetics | Sjogren-Larsson Syndrome - genetics | Xeroderma Pigmentosum - genetics | Humans | Male | Mitochondrial Proteins - genetics | Ichthyosis Vulgaris - genetics | INDEL Mutation | Skin Diseases, Genetic - genetics | Exome | Epidermolysis Bullosa - genetics | DNA Mutational Analysis | Intermediate Filament Proteins - genetics | Female | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Periodontal Diseases - genetics | Protoporphyrinogen Oxidase - genetics | Porphyria, Variegate - genetics | Photosensitivity Disorders - genetics | Epidermolysis Bullosa Simplex - genetics | Flavoproteins - genetics | Membrane Proteins - genetics | Collagen Type VII - genetics | Aldehyde Oxidoreductases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Blister - genetics | Keratin-14 - genetics | Homozygote | Phenotype | Ichthyosis, Lamellar - genetics | Pedigree | Lipoxygenase - genetics | Consanguinity | Epidermolysis Bullosa Dystrophica - genetics | Muscular Diseases - genetics | Ichthyosiform Erythroderma, Congenital - genetics | Genetic research | Skin | Nucleotide sequencing | Genes | DNA sequencing | Sebaceous glands | Nails | Glands | Teeth | Disorders | Homozygosity | Tissues | Follicles | Gene sequencing | Genodermatosis | Gene mapping | Index Medicus
Journal Article
by Sandholm, Niina and Salem, Rany M and McKnight, Amy Jayne and Brennan, Eoin P and Forsblom, Carol and Isakova, Tamara and McKay, Gareth J and Williams, Winfred W and Sadlier, Denise M and Mäkinen, Ville-Petteri and Swan, Elizabeth J and Palmer, Cameron and Boright, Andrew P and Ahlqvist, Emma and Deshmukh, Harshal A and Keller, Benjamin J and Huang, Huateng and Ahola, Aila J and Fagerholm, Emma and Gordin, Daniel and Harjutsalo, Valma and He, Bing and Heikkilä, Outi and Hietala, Kustaa and Kytö, Janne and Lahermo, Päivi and Lehto, Markku and Lithovius, Raija and Österholm, Anne-May and Parkkonen, Maija and Pitkäniemi, Janne and Rosengård-Bärlund, Milla and Saraheimo, Markku and Sarti, Cinzia and Söderlund, Jenny and Soro-Paavonen, Aino and Syreeni, Anna and Thorn, Lena M and Tikkanen, Heikki and Tolonen, Nina and Tryggvason, Karl and Tuomilehto, Jaakko and Wadén, Johan and Gill, Geoffrey V and Prior, Sarah and Guiducci, Candace and Mirel, Daniel B and Taylor, Andrew and Hosseini, S. Mohsen and Parving, Hans-Henrik and Rossing, Peter and Tarnow, Lise and Ladenvall, Claes and Alhenc-Gelas, François and Lefebvre, Pierre and Rigalleau, Vincent and Roussel, Ronan and Tregouet, David-Alexandre and Maestroni, Anna and Maestroni, Silvia and Falhammar, Henrik and Gu, Tianwei and Möllsten, Anna and Cimponeriu, Danut and Ioana, Mihai and Mota, Maria and Mota, Eugen and Serafinceanu, Cristian and Stavarachi, Monica and Hanson, Robert L and Nelson, Robert G and Kretzler, Matthias and Colhoun, Helen M and Panduru, Nicolae Mircea and Gu, Harvest F and Brismar, Kerstin and Zerbini, Gianpaolo and Hadjadj, Samy and Marre, Michel and Groop, Leif and Lajer, Maria and Bull, Shelley B and Waggott, Daryl and Paterson, Andrew D and Savage, David A and Bain, Stephen C and Martin, Finian and Hirschhorn, Joel N and Godson, Catherine and Florez, Jose C and Groop, Per-Henrik and Maxwell, Alexander P and Böger, Carsten A and DCCT-EDIC Res Grp and DCCT/EDIC Research Group and Medicinska fakulteten and Institutionen för klinisk vetenskap and Pediatrik and Umeå universitet
PLoS Genetics, ISSN 1553-7390, 09/2012, Volume 8, Issue 9, pp. e1002921 - e1002921
Diabetic kidney disease, or diabetic nephropathy (DN), is a major complication of diabetes and the leading cause of end-stage renal disease (ESRD) that... 
AF4/FMR2 FAMILY | GENE | GENETICS & HEREDITY | RENAL-FUNCTION | COLLAGEN CHAINS | RISK | GROWTH-FACTOR | NATURAL-HISTORY | NEPHROPATHY | EXPRESSION | GENOME-WIDE ASSOCIATION | Receptor, Epidermal Growth Factor - genetics | Diabetic Nephropathies - pathology | Fibrosis - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Diabetic Nephropathies - etiology | Humans | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Diabetes Mellitus, Type 1 - complications | Fibrosis - metabolism | Kidney Failure, Chronic - genetics | Kidney Failure, Chronic - pathology | Polymorphism, Single Nucleotide | Kidney Tubules - pathology | Kidney Tubules - metabolism | Nuclear Proteins - genetics | Receptor, ErbB-4 | Kidney Failure, Chronic - etiology | Quantitative Trait Loci - genetics | Type 1 diabetes | Physiological aspects | Development and progression | Disease susceptibility | Genetic aspects | Research | Kidney diseases | Medical research | Diabetes | Genes | Metabolic disorders | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap | Public Health, Global Health, Social Medicine and Epidemiology | Hälsovetenskaper | Folkhälsovetenskap, global hälsa, socialmedicin och epidemiologi | genetik | Genetics | Health Sciences
Journal Article
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 3/2003, Volume 160, Issue 5, pp. 753 - 767
Cell migration and invasion are fundamental components of tumor cell metastasis. Increased focal adhesion kinase (FAK) expression and tyrosine phosphorylation... 
Cell motility | Phosphorylation | Phenotypes | Cell growth | Cell cycle | Biological invasions | Antibodies | Pseudopodia | Focal adhesions | Cells | FAK | Motility | Src | JNK | Invasion | CARCINOMA-CELLS | TYROSINE PHOSPHORYLATION | JUN NH2-TERMINAL KINASE | NULL CELLS | CELL BIOLOGY | SIGNAL-TRANSDUCTION | MATRIX METALLOPROTEINASES | invasion | IN-VIVO | motility | FOCAL ADHESION KINASE | GROWTH-FACTOR | V-SRC | Crk-Associated Substrate Protein | Laminin - pharmacology | Oncogene Protein pp60(v-src) - metabolism | Pseudopodia - ultrastructure | rac GTP-Binding Proteins - metabolism | Phosphoproteins - metabolism | Protein-Tyrosine Kinases - deficiency | Focal Adhesion Kinase 1 | Recombinant Fusion Proteins - metabolism | Cell Movement - genetics | Proteins | Matrix Metalloproteinase 9 - metabolism | Protein-Tyrosine Kinases - genetics | Cell Size - genetics | Matrix Metalloproteinase 9 - genetics | rac GTP-Binding Proteins - genetics | JNK Mitogen-Activated Protein Kinases | Collagen - pharmacology | Eukaryotic Cells - enzymology | Fibronectins - pharmacology | Cells, Cultured | Focal Adhesion Protein-Tyrosine Kinases | Genetic Vectors - metabolism | Proteoglycans - metabolism | Oncogene Protein pp60(v-src) - genetics | Signal Transduction - genetics | Phosphoproteins - genetics | Fibronectins - metabolism | Genetic Vectors - genetics | Collagen - metabolism | Neoplasm Metastasis - genetics | Animals | Proteoglycans - pharmacology | Pseudopodia - metabolism | Retinoblastoma-Like Protein p130 | Pseudopodia - genetics | Recombinant Fusion Proteins - genetics | Mitogen-Activated Protein Kinases - genetics | Eukaryotic Cells - cytology | Mice | Laminin - metabolism | Neoplasm Invasiveness - genetics | Drug Combinations | Mitogen-Activated Protein Kinases - metabolism | Tumor antigens | Cytology | Cell adhesion | Physiological aspects | Genetic aspects | Research | Gene expression | Genetic regulation | Cell migration | Cellular biology | Index Medicus | motility; invasion; FAK; Src; JNK
Journal Article
Genome Research, ISSN 1088-9051, 11/2015, Volume 25, Issue 11, pp. 1646 - 1655
Journal Article