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European Journal of Endocrinology, ISSN 0804-4643, 06/2017, Volume 176, Issue 6, pp. 685 - 693
Objective, design and methods: Roux-en-Y gastric bypass (RYGB) has proved successful in attaining sustained weight loss but may lead to metabolic bone disease.... 
MINERAL DENSITY | METABOLISM | DISTAL RADIUS | MICROARCHITECTURE | ENDOCRINOLOGY & METABOLISM | WEIGHT-LOSS | BARIATRIC SURGERY | MORBID-OBESITY | QUANTITATIVE COMPUTED-TOMOGRAPHY | POSTMENOPAUSAL WOMEN | FRACTURE RISK | Postoperative Complications - etiology | Obesity, Morbid - surgery | Humans | Leptin - metabolism | Middle Aged | Postoperative Complications - metabolism | Male | Tomography, X-Ray Computed | Luteinizing Hormone - metabolism | Osteoporosis - etiology | Peptides - metabolism | Osteoporosis - metabolism | Parathyroid Hormone - metabolism | Osteoporosis - diagnostic imaging | Adiponectin - metabolism | Adult | Female | Follicle Stimulating Hormone - metabolism | Peptide Fragments - metabolism | Bone Density | Bone Diseases, Metabolic - diagnostic imaging | Collagen Type I - metabolism | Lumbar Vertebrae - diagnostic imaging | Bone Diseases, Metabolic - etiology | Tibia - diagnostic imaging | Gastric Bypass - adverse effects | Procollagen - metabolism | Absorptiometry, Photon | Bone Diseases, Metabolic - metabolism | Hip Joint - diagnostic imaging | Insulin - metabolism | Weight Loss | Vitamin D - analogs & derivatives | Vitamin D - metabolism | Bone Remodeling | Longitudinal Studies | Postoperative Complications - diagnostic imaging | Radius - diagnostic imaging | Dual energy X-ray absorptiometry | Radius | Spine | Body weight | Gastric bypass | Body weight loss | Bone turnover | Metabolism | Bone surgery | Insulin | Hip | Osteoporosis | Bone mass | Fractures | Computed tomography | Tibia | Leptin | Bone mineral density | Gastrointestinal surgery | Bone loss | Bone strength | Index Medicus
Journal Article
Laboratory Investigation, ISSN 0023-6837, 05/2012, Volume 92, Issue 5, pp. 713 - 723
Non-alcoholic steatohepatitis (NASH) is typically associated with pro-apoptotic caspase activation. A potential role for pro-inflammatory caspases remains... 
Inflammasome | Inflammation | Caspases | Non-alcoholic fatty liver disease | Non-alcoholic steatohepatitis | Fibrosis | MEDICINE, RESEARCH & EXPERIMENTAL | POPULATION | APOPTOSIS | non-alcoholic fatty liver disease | MICE DEFICIENT | FATTY LIVER-DISEASE | INJURY | NONALCOHOLIC STEATOHEPATITIS | MODEL | PATHOLOGY | inflammation | inflammasome | fibrosis | caspases | HEPATIC STEATOSIS | non-alcoholic steatohepatitis | Tumor Necrosis Factor-alpha - metabolism | Liver - pathology | Clodronic Acid - pharmacology | Fatty Liver - pathology | Actins - metabolism | Caspase 3 - metabolism | Hepatic Stellate Cells - metabolism | Caspase 1 - metabolism | Choline Deficiency - complications | Hepatocytes - pathology | Hepatocytes - metabolism | Fatty Liver - chemically induced | LIM Domain Proteins - metabolism | Liver Cirrhosis - chemically induced | Inflammation - metabolism | Caspase 3 - blood | Interleukin-1beta - metabolism | Choline Deficiency - metabolism | Liver Cirrhosis - metabolism | Muscle Proteins - metabolism | Antigens, Differentiation - metabolism | Kupffer Cells - metabolism | Hepatic Stellate Cells - pathology | Methionine - deficiency | Collagen Type I - metabolism | Fatty Liver - metabolism | Liver - metabolism | Mice, Inbred C57BL | Nuclear Proteins - metabolism | Choline Deficiency - pathology | Kupffer Cells - drug effects | Mice, Knockout | Animals | Caspase 1 - genetics | Caspase 1 - deficiency | Liver Cirrhosis - pathology | Mice | Transforming Growth Factor beta - metabolism | Index Medicus | Nonalcoholic fatty liver disease (NAFLD) | nonalcoholic steatohepatitis (NASH)
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2015, Volume 35, Issue 4, pp. 960 - 972
Objective-Marfan's syndrome is characterized by the formation of ascending aortic aneurysms resulting from altered assembly of extracellular matrix... 
focal adhesion | actin | aortic stiffness | myocardin | TGF-β | aortic aneurysms | extracellular matrix | RhoA | TRANSCRIPTION FACTORS | SMAD2 PATHWAY | EPIGENETIC CONTROL | THORACIC AORTIC-ANEURYSMS | SIGNAL-TRANSDUCTION | TGF-beta | GROWTH-FACTOR-BETA | MOUSE MODEL | DISEASE | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | DIFFERENTIATION | HEMATOLOGY | Vascular Remodeling | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Actins - metabolism | Myocytes, Smooth Muscle - pathology | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | Aortic Aneurysm - etiology | Case-Control Studies | Muscle Proteins - metabolism | Cytoskeletal Proteins - metabolism | Cell Differentiation | Marfan Syndrome - complications | Microfilament Proteins - metabolism | Aortic Aneurysm - pathology | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Collagen Type I - metabolism | Signal Transduction | Nuclear Proteins - metabolism | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Stress Fibers - metabolism | Focal Adhesions - metabolism | Cell Line, Tumor | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Trans-Activators - metabolism | Transforming Growth Factor beta - metabolism | Index Medicus | Biological Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Naturvetenskap | Biologiska vetenskaper | Natural Sciences
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 10/2013, Volume 15, Issue 5, pp. R151 - R151
Introduction: T helper (Th)-17 cells are increased in systemic sclerosis (SSc). We therefore assessed whether Th17 cells could modulate the inflammatory and... 
RHEUMATOID-ARTHRITIS | MATRIX METALLOPROTEINASES | SYNOVIAL FIBROBLASTS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | GENE-EXPRESSION | SKIN FIBROSIS | NECROSIS-FACTOR-ALPHA | RHEUMATOLOGY | NF-KAPPA-B | T-CELLS | PULMONARY-FIBROSIS | Interleukin-8 - genetics | Gene Expression - drug effects | Skin - metabolism | Humans | Scleroderma, Systemic - pathology | Culture Media, Conditioned - pharmacology | Male | Interleukin-17 - pharmacology | Dose-Response Relationship, Drug | Collagen Type I - genetics | Th17 Cells - metabolism | Radioimmunoassay | Inflammation Mediators - metabolism | Female | Chemokine CCL2 - metabolism | Interleukin-8 - metabolism | Matrix Metalloproteinase 1 - genetics | Skin - pathology | Fibroblasts - metabolism | Collagen Type I - metabolism | Enzyme-Linked Immunosorbent Assay | Scleroderma, Systemic - metabolism | Cells, Cultured | Scleroderma, Systemic - genetics | Chemokine CCL2 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Tumor Necrosis Factor-alpha - pharmacology | Fibroblasts - drug effects | Matrix Metalloproteinase 1 - metabolism | Interferon-gamma - pharmacology | Culture Media, Conditioned - metabolism | Proteins | Medical equipment and supplies industry | Interleukins | Systemic scleroderma | Medical test kit industry | Scleroderma (Disease) | High-definition television | Skin | Biological response modifiers | Enzyme-linked immunosorbent assay | Index Medicus
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 02/2003, Volume 23, Issue 4, pp. 1428 - 1440
Journal Article
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 55, Issue 5, pp. 1086 - 1094
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2009, Volume 284, Issue 41, pp. 27780 - 27789
Depletion of mtDNA in myocytes causes insulin resistance and alters nuclear gene expression that may be involved in rescuing processes against cellular stress.... 
SKELETAL-MUSCLE | CELLS | CONTRACTION | GENE | FATTY-ACID OXIDATION | GLUT4 TRANSLOCATION | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUCOSE-TRANSPORT | ACETYL-COA CARBOXYLASE | ACRP30/ADIPONECTIN | RNA, Small Interfering - genetics | AMP-Activated Protein Kinases - metabolism | Glucose Transporter Type 4 - metabolism | Humans | Receptors, Adiponectin - metabolism | DNA, Mitochondrial | Male | Intracellular Signaling Peptides and Proteins - metabolism | Diabetes Mellitus, Type 2 - metabolism | Insulin Receptor Substrate Proteins - metabolism | Recombinant Fusion Proteins - metabolism | Acetyl-CoA Carboxylase - genetics | Proto-Oncogene Proteins c-akt - genetics | Ribonucleotides - genetics | Mitochondria - genetics | Collagen - genetics | Membrane Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Aminoimidazole Carboxamide - metabolism | Insulin Receptor Substrate Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Fatty Acids - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Disease Models, Animal | Cell Line | Ribonucleotides - metabolism | Glucose Transporter Type 4 - genetics | Membrane Proteins - genetics | Mice, Inbred C57BL | Muscle Cells - metabolism | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Mitochondria - metabolism | Muscle Cells - cytology | Collagen - metabolism | Rats, Inbred OLETF | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Glucose - metabolism | Recombinant Fusion Proteins - genetics | Mice, Obese | Mice | Enzyme Activation | AMP-Activated Protein Kinases - genetics | Acetyl-CoA Carboxylase - metabolism | Receptors, Adiponectin - genetics | RNA, Small Interfering - metabolism | Index Medicus | Metabolism and Bioenergetics
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2017, Volume 12, Issue 10, pp. e0186615 - e0186615
Background Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive and fatal disease. Histone deacetylase 6 (HDAC6) alters function and fate of various... 
EPITHELIAL-MESENCHYMAL TRANSITION | LUNG FIBROSIS | IN-VITRO | TGF-BETA | HISTONE DEACETYLASE INHIBITION | MULTIDISCIPLINARY SCIENCES | TUBULIN ACETYLATION | DISEASE | AUTOPHAGY | MYOFIBROBLAST DIFFERENTIATION | EXPRESSION | Idiopathic Pulmonary Fibrosis - genetics | Ribosomal Protein S6 Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Middle Aged | Phosphoprotein Phosphatases - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Mechanistic Target of Rapamycin Complex 1 | Autophagosomes - drug effects | Multiprotein Complexes - metabolism | Tubulin - metabolism | Bleomycin | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Aged, 80 and over | Female | Indoles - pharmacology | Lung - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Hydroxamic Acids - pharmacology | Fibroblasts - metabolism | Lung - pathology | Collagen Type I - metabolism | Histone Deacetylases - genetics | Histone Deacetylase 6 | RNA, Messenger - genetics | Histone Deacetylases - metabolism | Nuclear Proteins - metabolism | Autophagosomes - metabolism | Mice, Knockout | Transforming Growth Factor beta - pharmacology | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Hydroxamic Acids - therapeutic use | Indoles - therapeutic use | Aged | Transforming Growth Factor beta - metabolism | Idiopathic Pulmonary Fibrosis - drug therapy | Histone deacetylase | Deregulation | Collagen (type I) | Phosphorylation | Disease | Mesenchyme | Pathogenesis | Lung | Critical care | AKT protein | Leucine | Kinases | Autophagy | Proteins | Fibroblasts | Vascular endothelial growth factor | Internal medicine | Lung diseases | Environmental health | 1-Phosphatidylinositol 3-kinase | Medicine | Pulmonary fibrosis | Inhibitors | Lungs | Lysine | Deacetylation | Collagen | Fibrosis | Mice | Protein phosphatase | Phagocytosis | Index Medicus
Journal Article
Journal of Biomedical Materials Research Part A, ISSN 1549-3296, 05/2006, Volume 77A, Issue 2, pp. 242 - 252
Development of biosynthetic conduits carrying extracellular matrix molecules and cell lines expressing neurotrophic growth factors represents a novel and... 
cell proliferation | fibronectin | cell culture | nerve tissue engineering | bioprosthesis | Cell proliferation | Cell culture | Bioprosthesis | Fibronectin | Nerve tissue engineering | PERIPHERAL-NERVE REGENERATION | STEM-CELLS | MATERIALS SCIENCE, BIOMATERIALS | RAT SCHWANN-CELLS | INJURED SPINAL-CORD | ADULT RATS | ENGINEERING, BIOMEDICAL | OLFACTORY ENSHEATHING GLIA | HUMAN BONE-MARROW | GROWTH-FACTORS | MARROW STROMAL CELLS | BASEMENT-MEMBRANE | Biocompatible Materials - metabolism | Stromal Cells - transplantation | Biocompatible Materials - chemistry | Nerve Regeneration - physiology | Collagen - chemistry | Ganglia, Spinal - cytology | Alginates - metabolism | Proteoglycans - chemistry | Schwann Cells - cytology | Neurites - ultrastructure | Cell Shape | Female | Hydrogels - metabolism | Olfactory Bulb - cytology | Animals, Newborn | Bone Marrow Cells - cytology | Stromal Cells - metabolism | Cells, Cultured | Proteoglycans - metabolism | Rats | Schwann Cells - transplantation | Materials Testing | Neurites - metabolism | Schwann Cells - metabolism | Rats, Sprague-Dawley | Fibronectins - metabolism | Collagen - metabolism | Animals | Alginates - chemistry | Laminin - chemistry | Laminin - metabolism | Bone Marrow Cells - metabolism | Drug Combinations | Hydrogels - chemistry | Stromal Cells - cytology | Cell Transplantation - methods | Index Medicus | Proteoglycans/chemistry/metabolism | Cell Transplantation/methods | Collagen/chemistry/metabolism | Nerve Regeneration/physiology | Cells; Cultured | Bone Marrow Cells/cytology/metabolism | Rats; Sprague-Dawley | Animals; Newborn | Hydrogels/chemistry/metabolism | Biocompatible Materials/chemistry/metabolism | Stromal Cells/cytology/metabolism/transplantation | Alginates/chemistry/metabolism | Ganglia; Spinal/cytology | Olfactory Bulb/cytology | Neurites/metabolism/ultrastructure | Fibronectins/metabolism | Schwann Cells/cytology/metabolism/transplantation | Laminin/chemistry/metabolism
Journal Article
Kidney International, ISSN 0085-2538, 01/2018, Volume 93, Issue 1, pp. 147 - 158
We examined activin receptor type IIA (ActRIIA) activation in chronic kidney disease (CKD) by signal analysis and inhibition in mice with Alport syndrome using... 
cardiovascular disease | chronic kidney disease | bone | fibrosis | mineral metabolism | vascular calcification | TGF-BETA | CARDIOVASCULAR EVENTS | RENAL FIBROSIS | ARTERIAL CALCIFICATION | BONE MORPHOGENETIC PROTEIN-7 | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | SMOOTH-MUSCLE-CELLS | LINKED ALPORT-SYNDROME | OSTEOCLAST DIFFERENTIATION | ATHEROSCLEROTIC LESION | Kidney - pathology | Recombinant Fusion Proteins - pharmacology | Blood Vessels - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - genetics | Vascular Calcification - metabolism | Kidney - metabolism | Bone Resorption - genetics | Bone and Bones - metabolism | Myocardium - metabolism | Renal Insufficiency, Chronic - genetics | Bone Resorption - metabolism | Disease Models, Animal | Bone Resorption - physiopathology | Glomerular Filtration Rate | Signal Transduction | Smad2 Protein - metabolism | Cardiomegaly - physiopathology | Nephritis, Hereditary - genetics | Renal Insufficiency, Chronic - physiopathology | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Cardiomegaly - prevention & control | Nephritis, Hereditary - metabolism | Collagen Type IV - deficiency | Activin Receptors, Type II - genetics | Fibrosis | Sp7 Transcription Factor - metabolism | Collagen Type IV - genetics | Cardiomegaly - metabolism | Activin Receptors, Type II - metabolism | Bone and Bones - pathology | Phosphorylation | Vascular Remodeling | Blood Vessels - pathology | Vascular Calcification - genetics | Actins - metabolism | Chronic Kidney Disease-Mineral and Bone Disorder - physiopathology | Activin Receptors, Type II - antagonists & inhibitors | Blood Vessels - physiopathology | Renal Insufficiency, Chronic - metabolism | Ventricular Remodeling | Chronic Kidney Disease-Mineral and Bone Disorder - prevention & control | Muscle Proteins - metabolism | Vascular Calcification - physiopathology | Microfilament Proteins - metabolism | Nephritis, Hereditary - physiopathology | Kidney - physiopathology | Vascular Calcification - prevention & control | Myocardium - pathology | Bone Resorption - prevention & control | Bone and Bones - physiopathology | Animals | Nephritis, Hereditary - drug therapy | Cardiomegaly - genetics | Bone Remodeling | Index Medicus | Chronic kidney disease
Journal Article